consolidation: RESEARCH_OVERVIEW; figures w/ multi-seed error bars + cross-objective + closed-form law; polished 3 drafts (selector numbers, refs, figure callouts)
Browse files- RESEARCH_OVERVIEW.md +56 -0
- paper/figures/fig1_layer_ablation.png +0 -0
- paper/figures/fig6_cross_objective.png +0 -0
- paper/figures/fig7_rank_law.png +0 -0
- paper/make_figures.py +55 -12
- paper/paper2_rank_objectives_draft.md +6 -5
- paper/paper3_midlayer_draft.md +15 -10
- paper/working_draft.md +4 -2
RESEARCH_OVERVIEW.md
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# covtoken β Research Overview
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Label-free rare-structure geometry in frozen self-supervised vision transformers, for medical
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imaging. One principle, three papers, four gated programs β all compute run as Hugging Face Jobs,
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all experiments reproducible, labels eval-only throughout.
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## The principle (one sentence)
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**Rare/critical structure lives in the mid-layer *concentration subspace* of self-distilled
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foundation models; depth (view-invariance/globalization) erodes it; rank/spanning objectives are
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anti-aligned with it; and it is readable, certifiable, and bounded label-free.**
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## The three papers
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1. **Method** β *Where Lesions Live: Label-Free Mid-Layer Lesion Subspaces for Token-Economical
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Medical Imaging* (`paper/working_draft.md`). The label-free lesion subspace + membership pruning
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+ conformal retention certificate + lesion-routed depth, across CT (MedDINOv3) and ultrasound
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(DINOv2). Includes the coverage-floor negative result.
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2. **Theory / negative-results** β *Rank-Based Representation Objectives Fail for Rare-Signal
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Retention: A Mechanism and a Predictive Law* (`paper/paper2_rank_objectives_draft.md`). The
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exact closed-form law (gap=(mβr)/m, r\*=m), the `A(rank,SNR)` alignment surface, the
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selection-vs-scaling sharpening. Transferable beyond medicine.
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3. **Mechanism / SPIE** β *Where Lesions Live: Mid-Layer Localization in Frozen ViTs, and Why*
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(`paper/paper3_midlayer_draft.md`). Mid-layer localization + label-free tail-gap selector +
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the invariance mechanism + the cross-objective causal-by-comparison result.
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## The four programs (gated, HF-Jobs reproducible)
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- **v1 (method)** β `gate_reports/` Gates 0β6. Subspace validity, cross-modality localization,
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membership > saliency pruning, coverage-floor NEGATIVE, conformal cert (0.978 β₯ 0.90), routed
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depth (1.6Γ FLOPs @ 98% sensitivity).
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- **v2 (S1βS5)** β `research_v2/`. S1 the closed-form law (β
), S2 mid-layer + label-free selector
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(β
), S4 detection viable (β), S3 precondition-prediction hard (β), S5 conformal validity transfers (β
).
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- **v3 (F1βF4)** β `research_v3/`. F2a alignment surface (β
synthetic), F1a selectionβ scaling (β),
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F2b law needs rank-relative-to-background (β), F3a invariance mechanism (β), **F3 cross-objective
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decisive** (β
β erosion across DINOv2/supervised/MedDINOv3; MAE not separable).
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- **v4 (G1βG3)** β `research_v4/`. **G1a training-free concentration steering = NEGATIVE** (β
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honest): globalization is an *entangling* transformation, not a removable nuisance β reinforces
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S2 and sharpens F3. G2 (cross-domain universality) and G3 (deployable tool + benchmark) specced,
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not built.
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## Headline numbers (multi-seed where applicable)
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- Mid-layer lesion AUROC peak **0.866 Β± 0.010** (block 3), final-layer 0.565; label-free tail-gap
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selector regret **0.006**.
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- Cross-objective depth-erosion Ο(invariance, AUROC): DINOv2 β0.93, supervised β0.73, MedDINOv3 β0.94.
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- The law is **exact** (gap=(mβr)/m, r\*=m, std 0.0 over 40 seeds).
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- Coverage-floor ablation: 0.22 (floor) vs 0.82 (membership) small-lesion retention.
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## What's honest about this body of work
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The clean wins (the law, the mid-layer mechanism + selector, the cross-modality method, the
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conformal certificate) are real and reproducible. The deep extensions (steering, concentration-
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preserving pretraining via reconstruction) returned **negatives that sharpened the science** rather
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than producing new methods β reported as first-class results. The remaining novel lever is G2
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(cross-domain universality), a deliberate new project, not a quick eval.
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## Reproduce
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All jobs in `jobs/` (PEP-723 uv scripts) run via `hf jobs uv run --flavor <t4-medium|t4-small|cpu>
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--secrets HF_TOKEN -v hf://buckets/Chucks90/eryon-datasets:/mnt <script>`. Specs in
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`research_specs/`. Figures: `paper/figures/` via `paper/make_figures.py`.
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paper/figures/fig1_layer_ablation.png
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paper/figures/fig6_cross_objective.png
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paper/figures/fig7_rank_law.png
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paper/make_figures.py
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@@ -13,18 +13,21 @@ plt.rcParams.update({"figure.dpi": 150, "font.size": 11, "axes.grid": True,
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def fig1_layer():
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ax.axhline(0.767, ls="--", color="#d62728", label="attention saliency (0.767)")
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ax.set_xticks(
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ax.set_ylabel("token-level lesion AUROC (LIDC)"); ax.set_ylim(0.
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ax.set_title("Finding 1: lesion signal
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ax.legend(loc="lower
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fig.tight_layout(); fig.savefig(OUT / "fig1_layer_ablation.png"); plt.close(fig)
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@@ -102,6 +105,46 @@ def fig5_conformal():
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fig.tight_layout(); fig.savefig(OUT / "fig5_conformal.png"); plt.close(fig)
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f()
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print("figures written:", sorted(p.name for p in OUT.glob("*.png")))
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def fig1_layer():
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# full 12-block depth curve with multi-seed (n=3) error bars [research_v3/rigor_results.json]
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blocks = np.arange(1, 13)
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mean = [0.8612,0.8641,0.8657,0.8375,0.8127,0.7818,0.7366,0.7059,0.6816,0.6802,0.6607,0.6365]
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std = [0.0098,0.0089,0.0098,0.0169,0.0182,0.0163,0.0253,0.0268,0.0302,0.0379,0.0373,0.0264]
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fig, ax = plt.subplots(figsize=(5.4, 3.4))
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ax.errorbar(blocks, mean, yerr=std, fmt="o-", color="#1f77b4", lw=2, ms=6,
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capsize=3, label="density-A (mean Β± std, 3 seeds)")
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ax.axhline(0.767, ls="--", color="#d62728", label="attention saliency (0.767)")
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pk = int(np.argmax(mean))
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ax.annotate(f"peak block {blocks[pk]}\n{mean[pk]:.3f}Β±{std[pk]:.3f}", (blocks[pk], mean[pk]),
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textcoords="offset points", xytext=(14, 6), ha="left", fontsize=8.5, color="#1f77b4")
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ax.set_xticks(blocks); ax.set_xlabel("transformer block (MedDINOv3)")
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ax.set_ylabel("token-level lesion AUROC (LIDC)"); ax.set_ylim(0.55, 0.92)
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ax.set_title("Finding 1: lesion signal peaks mid-layer, erodes with depth")
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ax.legend(loc="lower left", fontsize=8.5)
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fig.tight_layout(); fig.savefig(OUT / "fig1_layer_ablation.png"); plt.close(fig)
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fig.tight_layout(); fig.savefig(OUT / "fig5_conformal.png"); plt.close(fig)
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def fig6_cross_objective():
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# F3 decisive: depth-erosion across objectives [research_v3/f3_cross_objective.json]
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blocks = np.arange(1, 13)
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dino = [0.862,0.880,0.868,0.807,0.729,0.682,0.701,0.676,0.682,0.653,0.621,0.617]
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sup = [0.842,0.840,0.831,0.825,0.816,0.797,0.785,0.791,0.785,0.732,0.681,0.658]
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mae = [0.611,0.600,0.596,0.600,0.589,0.590,0.587,0.582,0.577,0.570,0.577,0.568]
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fig, ax = plt.subplots(figsize=(5.8, 3.6))
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ax.plot(blocks, dino, "o-", color="#1f77b4", lw=2, ms=5, label="DINOv2 (self-distill) Ο=β0.93")
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ax.plot(blocks, sup, "s-", color="#2ca02c", lw=2, ms=5, label="ViT (supervised) Ο=β0.73")
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ax.plot(blocks, mae, "^--", color="#d62728", lw=2, ms=5, label="MAE (reconstruction) Ο=+0.06")
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ax.axhline(0.50, ls=":", color="k", alpha=0.4, label="chance")
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ax.set_xticks(blocks); ax.set_xlabel("transformer block (natural-trained; eval on CT)")
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ax.set_ylabel("token-level lesion AUROC"); ax.set_ylim(0.45, 0.92)
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ax.set_title("Mechanism: localizers erode with depth; MAE never localizes")
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ax.legend(loc="upper right", fontsize=8)
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ax.annotate("MAE flat & low\n(not density-separable)", (9, 0.578),
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textcoords="offset points", xytext=(0, -28), ha="center", fontsize=7.5, color="#d62728")
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fig.tight_layout(); fig.savefig(OUT / "fig6_cross_objective.png"); plt.close(fig)
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def fig7_law():
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# S1 closed-form law: spanning vs concentration retention vs signal rank [research_v2/s1_crossover.json]
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m = 8; ranks = np.array([1, 2, 3, 4, 5, 6, 7, 8, 10, 12])
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spanning = np.minimum(ranks, m) / m # = min(r,m)/m
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concentration = np.ones_like(spanning, float)
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fig, ax = plt.subplots(figsize=(5.4, 3.6))
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ax.plot(ranks, concentration, "o-", color="#1f77b4", lw=2, ms=6, label="concentration (energy/membership)")
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ax.plot(ranks, spanning, "s-", color="#d62728", lw=2, ms=6, label="spanning (effective-rank/RankMe)")
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ax.fill_between(ranks, spanning, concentration, color="#d62728", alpha=0.12)
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ax.axvline(m, ls=":", color="gray"); ax.annotate("crossover r*=m", (m, 0.55),
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textcoords="offset points", xytext=(6, 0), ha="left", fontsize=8.5, color="gray")
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ax.annotate("retention gap\n(mβr)/m", (2.0, 0.62), fontsize=8.5, color="#d62728", ha="center")
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ax.set_xlabel("signal effective rank r (lesion concentration β diversity)")
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ax.set_ylabel("fraction of rare signal retained"); ax.set_ylim(0, 1.08)
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ax.set_title("The law: rank objectives lose rare/low-rank signal")
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ax.legend(loc="lower right", fontsize=8.5)
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fig.tight_layout(); fig.savefig(OUT / "fig7_rank_law.png"); plt.close(fig)
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for f in (fig1_layer, fig2_crossmodality, fig3_pruning_gain, fig4_ablation, fig5_conformal,
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fig6_cross_objective, fig7_law):
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f()
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print("figures written:", sorted(p.name for p in OUT.glob("*.png")))
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paper/paper2_rank_objectives_draft.md
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a budget we select tokens by (i) concentration = top-energy `||P_L z||^2`, or (ii) spanning =
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farthest-point / effective-rank maximization in L.
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**Result.** spanning retention `= min(r,m)/m`; concentration retention `= 1`; **gap
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crossover `r* = m`.** A spanning objective retains a signal only in proportion to its
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concentration only when the signal is fully diverse. (Multi-seed
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**Alignment surface.** Adding an SNR axis yields `A(r, SNR)`: concentration dominates iff the
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signal is concentrated (r<m) AND distinct (high SNR); at low SNR both lose the signal. This
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### Appendix β artifacts
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`research_v2/s1_crossover.json`, `research_v3/f1a_f2a_results.json`, `research_v3/f2b_f3a_results.json`,
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`gate_reports/ablation_floor.json`, `gate_reports/NEGATIVE_RESULT.md`, multi-seed
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`research_v3/
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a budget we select tokens by (i) concentration = top-energy `||P_L z||^2`, or (ii) spanning =
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farthest-point / effective-rank maximization in L.
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**Result (Fig. 1).** spanning retention `= min(r,m)/m`; concentration retention `= 1`; **gap
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`= (m-r)/m`; crossover `r* = m`.** A spanning objective retains a signal only in proportion to its
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rank; it ties concentration only when the signal is fully diverse. (Multi-seed: Β§5.)
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**Alignment surface.** Adding an SNR axis yields `A(r, SNR)`: concentration dominates iff the
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signal is concentrated (r<m) AND distinct (high SNR); at low SNR both lose the signal. This
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### Appendix β artifacts
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`research_v2/s1_crossover.json`, `research_v3/f1a_f2a_results.json`, `research_v3/f2b_f3a_results.json`,
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`gate_reports/ablation_floor.json`, `gate_reports/NEGATIVE_RESULT.md`, multi-seed in
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`research_v3/rigor_results.json`. Figure: `paper/figures/fig7_rank_law.png` (the law). All
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experiments reproducible as HF Jobs.
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paper/paper3_midlayer_draft.md
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loss of spatial information** β a mechanism that holds across self-distillation and supervised
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objectives but is absent for masked reconstruction (which never localizes). A label-free
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density/membership probe over patch tokens localizes lesions; its AUROC on chest CT rises from
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0.565 at the final block to 0.871 at block 3
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Across three objectives β DINOv2 (self-distillation, peak 0.88), supervised ViT (peak 0.84),
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MedDINOv3 (CT self-distillation) β localizability peaks early/mid and erodes with depth, strongly
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anti-correlated with rising flip-invariance (Ο = β0.73 to β0.94). Masked-reconstruction (MAE)
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## 1. The finding (depth)
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Token-level lesion-membership AUROC by block (LIDC, MedDINOv3): final 0.565 β block 6 0.769 β
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block 4 0.865 β **block 3 0.871**.
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## 2. Label-free layer selection
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## 3. The mechanism (why mid-layer)
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Depth-erosion + invariance-coupling hold for both objectives that produce a localizer
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(self-distillation, supervised), and for CT-native MedDINOv3 (Ο=β0.94) β three objectives, same
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law. **MAE is the clarifier:** it is flat *and low* (0.59) β masked reconstruction features are not
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density-separable for lesions at any depth, so "no collapse" is trivial (nothing to lose). The
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method needs self-distillation/supervised features; reconstruction is the wrong pretext.
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### Appendix β artifacts
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`research_v2/s2_depth_localizability.json`, `research_v3/f2b_f3a_results.json`,
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`research_v3/f3_cross_objective.json`, `research_v3/
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loss of spatial information** β a mechanism that holds across self-distillation and supervised
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objectives but is absent for masked reconstruction (which never localizes). A label-free
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density/membership probe over patch tokens localizes lesions; its AUROC on chest CT rises from
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0.565 at the final block to 0.871 at block 3 (peak 0.866 Β± 0.010 over 3 seeds), and the **tail-gap**
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of the membership-score distribution selects a layer within **0.006 AUROC** of the masked oracle
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(multi-seed) β entirely label-free.
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Across three objectives β DINOv2 (self-distillation, peak 0.88), supervised ViT (peak 0.84),
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MedDINOv3 (CT self-distillation) β localizability peaks early/mid and erodes with depth, strongly
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| 22 |
anti-correlated with rising flip-invariance (Ο = β0.73 to β0.94). Masked-reconstruction (MAE)
|
|
|
|
| 27 |
## 1. The finding (depth)
|
| 28 |
|
| 29 |
Token-level lesion-membership AUROC by block (LIDC, MedDINOv3): final 0.565 β block 6 0.769 β
|
| 30 |
+
block 4 0.865 β **block 3 0.871** (Fig. 1; multi-seed peak 0.866 Β± 0.010, monotone decline to
|
| 31 |
+
0.637 Β± 0.026). Final-layer features serve the global self-distillation objective; the dense local
|
| 32 |
+
lesion signal is mid/early.
|
| 33 |
|
| 34 |
## 2. Label-free layer selection
|
| 35 |
|
| 36 |
+
We select the operating layer with NO masks from the shape of the membership-score distribution.
|
| 37 |
+
The **tail-gap** `(q99βq50)/(q50βq01)` is the robust selector: multi-seed regret **0.006 AUROC**
|
| 38 |
+
(max 0.011) versus the mask-derived oracle. A bimodality statistic correlates with the depth curve
|
| 39 |
+
(Ο=0.69) but is less stable across seeds (regret 0.062), and excess kurtosis is a poor proxy (picks
|
| 40 |
+
the worst layer). So *where to read* is discoverable from the score distribution alone, without
|
| 41 |
+
annotation.
|
| 42 |
|
| 43 |
## 3. The mechanism (why mid-layer)
|
| 44 |
|
|
|
|
| 60 |
|
| 61 |
Depth-erosion + invariance-coupling hold for both objectives that produce a localizer
|
| 62 |
(self-distillation, supervised), and for CT-native MedDINOv3 (Ο=β0.94) β three objectives, same
|
| 63 |
+
law (Fig. 6). **MAE is the clarifier:** it is flat *and low* (0.59) β masked reconstruction features are not
|
| 64 |
density-separable for lesions at any depth, so "no collapse" is trivial (nothing to lose). The
|
| 65 |
method needs self-distillation/supervised features; reconstruction is the wrong pretext.
|
| 66 |
|
|
|
|
| 80 |
|
| 81 |
### Appendix β artifacts
|
| 82 |
`research_v2/s2_depth_localizability.json`, `research_v3/f2b_f3a_results.json`,
|
| 83 |
+
`research_v3/f3_cross_objective.json`, `research_v3/rigor_results.json`. Figures: `paper/figures/
|
| 84 |
+
fig1_layer_ablation.png` (depth, error bars), `fig6_cross_objective.png` (mechanism). HF-Job reproducible.
|
paper/working_draft.md
CHANGED
|
@@ -99,7 +99,7 @@ Datasets: LIDC-IDRI (lung CT), KiTS23 (kidney CT), MSD Task03 Liver, MSD Task07
|
|
| 99 |
## 4. Results: the label-free localizer
|
| 100 |
|
| 101 |
### 4.1 Lesion signal lives mid-layer (Finding 1)
|
| 102 |
-
Token-level lesion AUROC by depth (LIDC, density-A):
|
| 103 |
|
| 104 |
| layer | final (12) | block 6 | block 4 | block 3 |
|
| 105 |
|---|---|---|---|---|
|
|
@@ -107,7 +107,9 @@ Token-level lesion AUROC by depth (LIDC, density-A):
|
|
| 107 |
|
| 108 |
Final-layer features are tuned for the global self-distillation objective; the dense local lesion
|
| 109 |
signal sits mid/early. We fix block 3 (MedDINOv3) as the operating layer; for DINOv2 the optimum
|
| 110 |
-
is block 8 β backbone-dependent, but always mid/late, never final.
|
|
|
|
|
|
|
| 111 |
|
| 112 |
### 4.2 Cross-anatomy, cross-modality, cross-backbone localization (Finding 2)
|
| 113 |
density-A token-level lesion AUROC, with attention-saliency as the label-free comparator:
|
|
|
|
| 99 |
## 4. Results: the label-free localizer
|
| 100 |
|
| 101 |
### 4.1 Lesion signal lives mid-layer (Finding 1)
|
| 102 |
+
Token-level lesion AUROC by depth (LIDC, density-A; Fig. 1):
|
| 103 |
|
| 104 |
| layer | final (12) | block 6 | block 4 | block 3 |
|
| 105 |
|---|---|---|---|---|
|
|
|
|
| 107 |
|
| 108 |
Final-layer features are tuned for the global self-distillation objective; the dense local lesion
|
| 109 |
signal sits mid/early. We fix block 3 (MedDINOv3) as the operating layer; for DINOv2 the optimum
|
| 110 |
+
is block 8 β backbone-dependent, but always mid/late, never final. The curve is multi-seed stable
|
| 111 |
+
(peak 0.866 Β± 0.010, n=3), the operating layer is selectable without labels (tail-gap selector
|
| 112 |
+
regret 0.006), and the depth-erosion holds across objectives β see the companion mechanism study.
|
| 113 |
|
| 114 |
### 4.2 Cross-anatomy, cross-modality, cross-backbone localization (Finding 2)
|
| 115 |
density-A token-level lesion AUROC, with attention-saliency as the label-free comparator:
|