Inactivation properties of WT Nav1.5 and Nav1.5-F1571L alone or co-expressed with beta1. (D) Displayed from left to right are the representative whole-cell recordings of recovery from inactivation of WT Nav1.5 + beta1 and Nav1.5-F1571L + beta1. The protocol used is shown underneath the traces.


Inactivation properties of WT Nav1.5 and Nav1.5-F1571L alone or co-expressed with beta1. (E) Graph representing the recovery from inactivation, sampled after 500 ms from induction of inactivation.


3D CT reconstruction image of pelvis showing bilateral calcification of the vas deferens.


Occlusion of the left main.


After single ballon dilatation of the left main.


Right coronary artery.


showed normal sinus rhythm with prolonged QTc interval of 527 msec and some non-specific T wave changes over the inferior leads.


Coronary angiogram showing normal coronary artery.


Normal heart.


Tako-Tsubo sign. With apical ballooning (end-systolic view).


Resting ECG of the patient.


Original case TEE sequence in anaesthesia #4. A. Acute onset of left ventricular dysfunction/considerable hypokinesia (reduced contractility: outer circle: diastole; inner circle: systole).


Original case TEE sequence in anaesthesia #4. B. After successful therapeutic intervention: normal left ventricular function/no considerable wall motion abnormalities (normal contractility: outer circle: diastole; inner circle: systole).


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). A: Placement of catheters during ablation at the posterior aspect of right ventricular outflow tract (RVOT).


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). B: Placement of catheters during ablation at the right coronary cusp (RCC). Note the proximity of the RCC ablation site to the RVOT ablation site shown in panel A.


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). C: Right coronary angiography before ablation at the RCC. The white star indicates the RCC ablation site from panel B, which was located anteriorly to the right coronary artery in right anterior oblique (RAO) view.


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). D: Same images as panel C but with the ostium (white arrows) and the ostial portion (white lines) of the right coronary artery marked. Note that the ostium was located much higher than the distal tip of the ablation catheter (ABL) and the ostial portion ran inferiorly.


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). E: Intracardiac electrogram recorded at the RVOT ablation site during VT. Local ventricular activation recorded at the distal (d) bipolar (Bi) electrode of the ABL showed an upright initial R wave preceding the onset of the QRS complex by 30 ms. Simultaneous recording of the unipolar (Uni) electrode of the ablation catheter displayed a QS pattern.


Fluoroscopic images of ablation sites and right coronary artery and intracardiac electrograms during ventricular tachycardia (VT). F: Intracardiac electrogram recorded at the RCC ablation site during VT. Local ventricular activation showed not an initial positive but an early activation preceding the onset of the QRS complex by 27 ms (arrow) in the distal bipolar recording and exhibited a QS pattern in unipolar recording. See text for further discussion. EPI = epicardial mapping catheter inserted via subxiphoid puncture (catheter placed only for mapping, and catheter location not related with any endocardial ablation sites); HBE = His-bundle catheter; LAO = left anterior oblique; p = proximal; RVA = right ventricular apex catheter.


Right coronary artery stenosis. A: Twelve-lead ECGs just before (left) and after (right,asterisks) ST elevation in the inferior leads.


Right coronary artery stenosis. B: Right coronary angiography (left anterior oblique view) showing acute stenosis just after ST-segment elevation was first observed (top) and after balloon angioplasty (bottom). White arrows indicate stenotic lesion.


Right coronary artery stenosis. C: Intravascular ultrasound showing stenosis after repeated nitroglycerin injection (top) and after balloon angioplasty (bottom). White arrow indicates eccentric intimal thickening that successfully resolved after angioplasty. Neither coronary artery dissection nor thrombus was observed. See text for further discussion.


Non sustained ventricular tachycardia at exercise stress test (treadmill).


Cardiac magnetic resonance shows single coronary origin from left coronary sinus with malignant course of the right coronary artery (arrow).


Catheter coronary angiography detects anomalous origin of coronary arteries from left coronary sinus, with slit like right coronary ostium (arrow).


Catheter coronary angiography detects a bridging muscle in middle segment of the left anterior descending artery (arrow).


Axial.


Coronal images of the unenhanced computed tomography (CT) scan of the brain showed an acute left middle cerebral artery (MCA) infarction, which manifested as cortical and subcortical hypodensity with loss of gray-white matter differentiation and cortical swelling involving the left frontal, parietal, and temporal regions (asterisk); hyperdense MCA signs were also noted (arrow).


Axial maximum intensity projection (MIP) images of the CT angiogram of the brain showed complete occlusion of the left supraclinoid internal carotid artery and proximal MCA (arrows).


(E) Brain CT demonstrated the presence of a malignant MCA infarction with hemorrhagic transformation, causing significant midline shifting and mass effect.


(F) Postoperative CT.


Cardiovascular magnetic resonance imaging with inversion recovery sequence using long inversion time showed area of no contrast uptake at the basal anterior and anterolateral wall.


Blue arrows) that improved on the similar sequence repeated 30 minutes later. Arrows) consistent with microvascular obstruction.


Cardiovascular magnetic resonance imaging with inversion recovery sequence using long inversion time showed area of no contrast uptake at the basal anterior and anterolateral wall (E, F) Left atrial appendage thrombus (yellow arrow) was also discovered on this sequence. Image enlarged using letsenhance. Io.


(E, F) Left atrial appendage thrombus (yellow arrow) was also discovered on this sequence. Image enlarged using letsenhance. Io.


12-lead ECG (left, posterior and right leads) performed in our emergency room showing ST-segment elevation in the inferior leads (II, III, aVF) and V5, V6.


Final image with successful PCI of the LAD artery and good coronary flow.


Balloon sizing the atrial septum through a retrograde approach.


NSR with dynamic changes in T-waves in V1 and V2 when comparing two ECGs.


Repeat ECG after second emergency department visit.


Facial dysmorphism in velocardiofacial syndrome.


Magnetic resonance imaging (MRI). (A) Magnetic resonance angiography showing a drop in signal at the 12th thoracic level.


Magnetic resonance imaging (MRI). (B) MRI showing no ischemic or demyelinating lesion.


Macroscopic view of the right side of the heart, showing downward ventricular insertion of the tricuspid valve ring ("atrialization of the right ventricle"), leaflets partially adherent to the ventricular wall and free portion dysplastic. It also shows a right ventricle with minimal chamber and a "canal" outlet chamber to the pulmonary valve, due to the mal-positioned tricuspid leaflets (source: INMLCF, I. P. , with permission).


Macroscopic view of the Quadricuspid Pulmonary Valve, with three equal-sized cusps and an extra (No. 4) smaller one (source: INMLCF, I. P. , with permission).


Case A. Transthoracic echocardiogram demonstrates an elongated and thickened anterior mitral valve leaflet. The posterior leaflet was extremely hypoplastic with just a short visible stump. A noncompaction picture of the left ventricle lateral wall is visible.


Case A. Transthoracic echocardiogram shows a marked hypoplasia of the posterior mitral leaflet and a normal sized anterior leaflet.


Case A. Transthoracic echocardiogram-4C color Doppler demonstrating an eccentric severe regurgitation jet.


Case A. Two-dimensional transesophageal echocardiography demonstrating an anterior-posterior bicuspid aortic valve (opening phase).


Case A. Two-dimensional transesophageal echocardiography demonstrating an anterior-posterior bicuspid aortic valve (closure phase).


Case A. Three-dimensional real-time transesophageal echocardiography demonstrating the lack of posterior mitral leaflet.


Case B. Transthoracic echocardiogram shows a marked hypoplasia of the posterior mitral leaflet and a normal sized anterior leaflet.


Case B. Transthoracic echocardiogram- Parasternal Long Axis (PLax) demonstrating a severe hypoplasia of mitral posterior leaflet.


Electrocardiogram (ECG) results. The pre procedural electrocardiogram revealed an ST-segment elevation in the V2-6 leads.


Electrocardiogram (ECG) results. The post-procedural electrocardiogram showed an improvement in ST-segment elevation in the V3-6 leads.


This figure (Video 1) shows the steps of the procedure; 1. Inserting the filter device (Filtrap ) and unfolding it beyond the occlusion; 2. Advancing the guide extension catheter (GuideLiner ) and capturing the huge thrombus between the tip of the GuideLiner. catheter and the filter body of the Filtrap ; 3. Pulling them back together out of the guide catheter.


Thrombus retrieved from the left anterior descending artery.


Left coronary arteriogram of a young patient with antiphospholipid syndrome and myocardial infraction on admission showing thrombotic stenosis of left descending coronary artery at the level of the proximal section.


Arteriogram of a young patient with antiphospholipid syndrome and myocardial infraction after PTCA showing evidence of successful recanalization.


Four-chamber view echocardiography of a young patient with antiphospholipid syndrome and myocardial infraction showing an apical large thrombus measuring 38x18 mm.


Presenting chest radiograph showing cardiomediastinal silhouette enlargement with mild congestion and pleural opacity on the right chest wall. Annular calcific pericardial ring later seen on echocardiogram, coronary angiography, and computed tomography scan is not well visualized.


Presenting echocardiogram showing pericardial calcification. (a) Apical four chamber and parasternal short axis.


Presenting echocardiogram showing pericardial calcification. (b) Views showing pericardial thickening (shown by arrows).


Reconstructed computed tomography scan image showing the calcific ring.


The distal first diagonal artery stent (arrow) crossing underneath the calcific band.


Autopsy gross specimens of lungs and heart. (a) Lung gross specimen showing pulmonary thromboemboli with massive right pulmonary hemorrhagic infarct and pleural hemorrhage which was thought to be the cause of death.


Autopsy gross specimens of lungs and heart. (b) Dissected specimen of left anterior groove with patent and intact recently placed first diagonal artery stent (arrow).


Autopsy gross specimens of lungs and heart. (c) Gross specimen of heart with hypertrophied left ventricle and fibrocalcific constrictive annular band in atrioventricular groove (arrow) with dense fibrosis and calcification.


CECT scan of chest showing bilateral diffuse ground glass opacities.


At onset of cerebral infarction, diffusion weighted magnetic resonance imaging (DW-MRI) revealed fresh infarctions in the left cerebral hemisphere (1-A, arrows).


MRA revealed a stenotic lesion in the cervical segment of the left ICA (1-B, arrow).


DSA (1-. Arrow).


MRA (1-. Arrows) on the second day after the onset revealed no evidence of stenosis.


Chest X - Ray Posterior - Anterior and Lateral Views Showing Dilated Main Pulmonary Artery and the Catheter.


Echocardiogram Showing the Catheter Traversing the Interatrial Septum. Abbreviations: RA, Right Atrium; LA, Left Atrium.


Intraoperative Photograph of the Entrapped Catheter in the Interatrial Septum. Abbreviations: RA, Right Atrium; SVC, Superior Venacave; IVC, Inferior Venacave (Inset-extracted catheter tip).


Intraoperative Photograph of Domed, Stenosed Bicuspid Pulmonary Valve.


Risk factors for aHUS recurrence after uDCD. Modified from Zuber et al. . uDCD = uncontrolled donation after circulatory death; cDCD = controlled donation after circulatory death; IS = immunosuppressive; IRI = ischemia-reperfusion injury.


Erect abdominal radiograph demonstrating fullness of bilateral renal fossae with obscuration of the psoas outline suggestive of renal masses. Associated mass effect evidenced by inferior displacement of adjacent bowel loops.


Sagittal ultrasound image of the right kidney depicting a large perirenal hypoechoic fluid collection with hyperechoic internal echoes compatible with complex fluid. Associated medial displacement and compression of the right kidney. Similar findings were seen on the contralateral side (not shown).


(a, b) Contrast-enhanced axial and coronal computed tomography of the abdomen demonstrating large bilateral subcapsular collections (stars) causing compression of the renal parenchyma (arrowheads).


CT scan of the brain. (A) Left side: cortico-subcortical temporo-parieto-insular hypodensity. Right side: subcortical lenticular hypodensity.


CT scan of the brain. (B) Left side: cortico-subcortical parietal hypodensity.


CT scan of the brain. (C,D) Right side: cortico-subcortical parietal hypodensity.


CT scan showing the ARSA and related pseudoaneurysm and the dissection.


3D CT reconstruction.


CT scan showing correct positioning of the endovascular stent graft.


Summative timeline showcasing key findings of the patient's evolution and detailed treatment (case 1).


Summative timeline showcasing key findings of the patient's evolution and detailed treatment (case 2).


(a-c) Para-sternal short axis views showing the non-compacted layer with numerous trabeculations. Figure 1b illustrates the two separate layers.


Cardiac MRI: four chamber view with some degree of motion artifact. Red arrow represents the non-compacted myocardium.


Echocardiography image showing large pseudoaneurysm of the posterolateral wall.


Telemetry showed ventricular tacycardia with heart rate of 149 bpm.


Pre-operative right coronary artery
angiography.


Early post-operative electrocardiogram.


Electrocardiogram at the time of chest pain 3 days after the surgery.


Non-selective aortic root injection
angiography.


Selective right coronary artery angiography after infusion of intravenous trinitroglycerine.


Electrocardiogram after transient right coronary artery spasm.


A. The electrocardiogram showed ST-segment depression in leads V3-V6 and D1-aVL.


B. Coronary angiogram in the right caudal view shows the left coronary artery, circumflex artery and intermediate artery.


C. Coronary angiogram in the left anterior oblique cranial view shows the circumflex artery and right coronary artery originating from right sinus of Valsalva. (CX: circumflex artery, IM: Intermediate artery, LAD: left anterior descending artery, LSV: Left sinus of Valsalva, RCA: Right coronary artery, RSV: Right sinus of Valsalva).


The electrocardiogram showed no ST-segment depression or elevation.


A. Coronary angiogram in the left caudal view shows the left coronary artery tree with a circumflex artery.


B. Coronary angiogram in the left anterior oblique cranial view shows the circumflex artery and right coronary artery originating from right sinus of Valsalva.


C. The significant lesion, in the left anterior oblique cranial view, shows the circumflex originating from right sinus of Valsalva.


D. After stenting the significant lesion, in left anterior oblique view shows the circumflex artery originating from right sinus of Valsalva (CX: circumflex artery, LAD: left anterior descending artery, LSV: Left sinus of Valsalva, RCA: Right coronary artery, RSV: Right sinus of Valsalva).


A: H&E, 40x, Well circumscribed tumor with compressed peripheral adrenal parenchyma (black arrow). 2b: H&E, 100x, Nests of tumor cells with circumscribed margin and adjacent normal adrenal parenchyma (black arrow). 2c: H&E 100x Nests and trabecular pattern of tumor cells, separated by thin fibrovascular septae. 2d: H&E, 400x, Tumor cells having moderate amount of granular eosinophilic cytoplasm, centrally placed round nucleus with fine granular chromatin and inconspicuous nucleoli. Scattered sustentacular cells (black arrow) are also noted.


(A) Magnetic resonance imaging shows 1.6 cm-sized mass in anterior wall of the cervix (white arrow indicates the cervical mass).


(B) Gross finding shows the tumor confirmed as poorly differentiated squamous cell carcinoma with clear resection margin (measuring 3.3 x 3.0 x 0.5 cm) (white arrow indicates the cervical mass).


The needle attached to semen containing syringe was put into the posterior cul-de-sac along the biopsy guide.


A; Right anterior oblique (RAO) lateral view. Manipulation of Gaia second guidewire with Crusade catheter during dilation for the right ventricular (RV) branch true lumen by 1.5 mm balloon.


B; While the tip of Gaia second guidewire was facing on the balloon through the strut, immediately balloon deflation was applied. The Gaia second guidewire could pass to the RV branch true lumen in the same space of the first protection guidewire.


C; Kissing balloon inflation at the right coronary artery main vessel and RV branch with 4.0 mm and 2.0 mm balloon, respectively.


D; Final angiogram. The RV branch was restored; the main coronary vessel also showed optimal result.


Transthoracic echocardiography on admission and four days later after percutaneous coronary intervention (PCI). Echocardiography on admission showing global hypokinesia, mild mitral regurgitation (MR) and left atrium (LA) enlargement (LA volume, 54 mL) (A, B).


Transthoracic echocardiography on admission and four days later after percutaneous coronary intervention (PCI). Four days later after PCI, left ventricular contraction and LA enlargement have improved (LA volume, 34 mL), and MR has resolved (C, D).


Coronary artery angiography during the primary percutaneous coronary intervention. (A) Left coronary angiography showing significant stenosis from the distal left main coronary artery to the mid left anterior descending artery with thrombolysis in myocardial infarction (TIMI) 2 flow and total occlusion at the ostium of the left circumflex artery with TIMI 0 flow.


Coronary artery angiography during the primary percutaneous coronary intervention. (B) Final coronary angiography showing successful revascularization without periprocedural complications.


Computed tomography scan revealing consolidation and ground-glass opacity in the dependent portion of the right lung, with pleural effusion limited to the right lung.


(A-C) depict the axial, coronal, and sagittal panel of the preoperative CTA, respectively. The white arrows indicated the fistula and RCAA.


(D-F) depict the axial, coronal, and sagittal panel of the 6-months follow-up CTA, respectively. The white arrows indicate the mild dilated RCA.


(G,H) show the preoperative and postoperative echocardiography images of the patient. A large fistula and RCCA to RV shunt was indicated by the arrow. RCA, right coronary artery; RCAA, right coronary artery aneurysm; LV, left ventricle; RV, right ventricle.


(A,B) depicted the reconstructed image of the heart and dilated right coronary artery.


(C-F) depicted the surgical repair process. RCA, right coronary artery; RCAA, right coronary artery aneurysm; AO, aorta.


The 5-chamber view of the heart at 25.3 weeks showing the fistula between the left coronary artery and coronary sinus (arrow, A).


Power Doppler imaging confirming the communication between the left coronary artery and coronary sinus (B).


The 4-chamber view of the heart at 29.6 weeks showing the dilated coronary sinus (arrow) with the collapsed left atrium (C). . Ao, aorta; CS, coronary sinus; LA, left atrium; LV, left ventricle; RA, right atrium; RV, right ventricle.


Postnatal computed tomography showing the dilated left circumflex artery draining into the enlarged coronary sinus on the axial image (arrow.


The 3-dimensional volume rendering image (arrow,. . Ao, aorta; CS, coronary sinus; LAA, left atrial appendage; LCX, left circumflex coronary artery; LMA, left main coronary artery; PA, pulmonary artery.


Clinical fundus photo of a 44-year-old male presenting with diminution of vision following dengue fever. (A) Right eye. (A) Right eye showing additional retinal whitening exclusively inferior to the right macula.


Clinical fundus photo of a 44-year-old male presenting with diminution of vision following dengue fever. (B) Left eye. Both eyes depicting perivascular exudates, intraretinal hemorrhages, cotton wool spots and macular edema.


Fundus fluorescein angiography of a 44-year-old male presenting with diminution of vision following dengue fever. (A) Right eye.


Fundus fluorescein angiography of a 44-year-old male presenting with diminution of vision following dengue fever. (B) Left eye. Both eye showing disc and perivascular leakage limited to posterior pole indicative of vasculitis. In addition, there is capillary nonperfusion due to occlusion of multiple small arterioles supplying the macula of right eye confirming macular ischemia.


Electrocardiogram of the patient before admission.


At discharge.


Real-time monitoring of the patient's vital signs during hospitalization.


Level of cholinesterase.


Cardiac troponin I. Before admission and at discharge.


(A) Pedigree analysis of the family of patient 1. The arrow points out the proband. Circles correspond to female. Squares correspond to male. The mutation was indicated -/- if negative and ?/? if untested.


(B) Genetic testing of patient 1 showed a guanine deletion at position 973 (c.973delG) in exon 8 of the LAMP2 gene.


The echocardiogram of patient 1: substantial left ventricular hypertrophy with a maximal thickness in the posterior wall of the left ventricle of 10 mm and a ventricular septal thickness of 15 mm, and obstruction in the left ventricular outflow tract.


Right coronary artery-right ventricle (RV) fistula. A
Arrow showed the dilated right coronary artery. B
Arrow showed the aneurismal dilatation of right coronary artery. C
Arrow showed fistula ostium. D; The spectrum of fistula was continuous. Ao aorta, LA left atrium, LV left ventricle, RA right atrium, RV right ventricle.


Left anterior descending-pulmonary artery fistula. A
Arrow showed the abnormal blood flow in the pulmonary artery. B
Arrow showed the origin of the fistula is left anterior descending. C; The spectrum of fistula showed only in diastole.


Left circumflex (LCX)-coronary sinus (CS) fistula. A LCX was dilated and connected with CS. B; CS was connected with right atrium (RA). CS coronary sinus, LCX left circumflex, LV left ventricle, RA right atrium, RV right ventricle.


EKG with ST elevations in anterior leads.


Echocardiogram with severe hypokinesis of anterior, anteroseptal walls, and apex.


Chest X-ray showing dilated cardiomyopathy.


Tilt test registration. The figure shows the recording of systolic blood pressure, diastolic blood pressure, and heart rate in clinostatic and sitting position (passive orthostatic position not performable because of severe presyncopal symptoms) during a tilt test. In sitting position, an immediate reduction in systolic blood pressure to 55 mmHg and diastolic blood pressure to 28 mmHg is evident, without any change in the heart rate, which remains stable at 76 beats/min.


Electrocardiogram ,cardiac ultrasound.


The figure shows the 12-lead electrocardiogram pointing out the first-degree atrioventricular blockage (panel A). In panel B, cardiac ultrasound (parasternal long-axis view) depicts left ventricle wall thickness, diffuse myocardial hyperechogenicity, and mild pericardial effusion.


Four-chamber view in a transthoracic echocardiogram showing enlargement of
the left ventricle (LV) (The LV apical structure was unclear in this view).


Cine cardiac magnetic resonance imaging (MRI) image in a four-chamber view
shows bulging of the interventricular septum (IVS) toward the right ventricle (white
arrow) and invagination of fatty material (black arrow).


A T1-weighted image shows bright tissue replaced in the left ventricle (LV)
apical position (white arrow), which could suggest the presence of fat replacement.


A late gadolinium enhancement (LGE) image performed 10 minutes after the
contrast media injection showed no intramyocardial hyperenhancement area but indicated
the presence of myocardial tissue in the out pouching; this confirmed the congenital
nature of the diverticulum.


Medical image.


Admission head computed tomography demonstrates a metallic foreign body projecting in the region of the right transverse sinus (arrow). The bullet previously seen in the right transverse sinus is no longer evident on the follow-up head computed tomography angiography.


Chest radiograph obtained at admission (a) demonstrates no cardiopulmonary abnormality.


A follow-up chest radiograph (b) shows a 5 mm round metallic foreign body projecting over the left hilum (open arrow) with interval development of a wedge shaped pulmonary opacity a the left lung base (arrow head).


Case 1: left CAG showed a huge aneurysm with calcification of the proximal left coronary artery, and the LAD is obstructed at the outflow of the aneurysm (red arrowhead), and the blood is supplied by the collateral circulation tract from the right coronary artery. LAD, left anterior descending artery; LCX, left circumflex artery.


Case 1: macrofindings of the LAD aneurysm during surgery. A giant coronary aneurysm prior to resection is observed. A hard and calcified mass >40 mm in diameter is present. The mass was filled with a red blood clot. LAD, left anterior descending artery.


Case 2: right CAG at 1 year after CABG. The blood flow supply from the GEA to the distal site of RCA was adequate. CAG, coronary artery angiogram; CABG, coronary artery bypass graft surgery; GEA, gastroepiploic artery; RCA, right coronary artery.


Coronary angiogram in the right anterior oblique cranial view (A), showing the left circumflex artery (LCx) (arrow) and the left anterior descending artery (LAD) originating from the left main coronary artery.


When the right coronary ostium was cannulated (B), another circumflex artery (RCx) (arrow) was noticed in addition to a right coronary artery (RCA) in the left anterior oblique cranial view.


Preoperative left internal carotid angiograms showing anomalous arteries arising from the internal carotid arteries, which and run anteromedially along the frontal base and making a hairpin-pin bend, and a saccular aneurysm is seen on the hairpin-pin bend, lateral view.


Oblique view.


Preoperative angiograms revealing an aneurysm with oblique view. Postoperative angiograms showing no filling of the aneurysm.


(A) Plasma glucose trend in case 1.


(B) Plasma glucose trend in case 2.


MRCP image shows intra and extrahepatic biliary dilation secondary to tumor lesion of the head of the pancreas.


Fluoroscopic image observing LAMS between duodenal bulb and bile duct.


Endoscopic ultrasound vision shows the distal flange of LAMS (HOT AXIOS) in to the CBD.


CT scan coronal view shows LAMS in proper position.


Duplex ultrasound demonstrating retrograde flow through the right external carotid artery that supplies antegrade flow to the right internal carotid artery.


Intraoperative photo of the opened common carotid artery revealing a hard calcific plaque with soft thrombus.


Excised ruptured hydatid cyst wall.


(A) MDCT image of the LAD in an enhancement scan.


(B) MDCT image of the LAD in a plain scan. LAD, left anterior descending coronary artery; MDCT, multidetector computed tomography.


Visualization of the coronary angiography. (A) Intraoperative open-heart coronary angiography shows no significant stenosis in the left coronary artery.


Visualization of the coronary angiography. (B) Intraoperative open-heart coronary angiography shows no significant stenosis in the right coronary artery. LAD, left anterior descending coronary artery; LCX, left circumflex coronary artery.


Treatment course of maggot therapy. (a) Before maggot therapy. Necrotic tissue is seen on the surface of the wound.


Treatment course of maggot therapy. (b) After one session (48 h) of treatment, the reduction of necrotic tissues is seen.


Treatment course of maggot therapy. (c) Maggots growing from second to third instar larvae.


Treatment course of maggot therapy. (d) After six sessions of treatment, debridement was conducted to the deep portion from the ulcerated base, and favourable granulation can be seen on the amputated surface.


Intra-/post-operative view. (a) Intra-operative. Additional debridement is done.


Intra-/post-operative view. (b) Three post-operative weeks. Approximately 70% of the skin graft was taken. After conducting partial simple reefing, split thickness skin graft of 20/1000 inches was used.


Short LAD arises from the left sinus of Valsalva and gives rise to one septal branch, little intermedius branch, and one diagonal branch.


Circumflex artery (LCX) is aplastic, as shown in the left caudal angiographic projection.


Right coronary artery (RCA) is normal in its origin and course.


Long LAD arises from the right sinus of Valsalva with a separate ostium, different from RCA. This LAD is longer than the first, runs on the right ventricle, and gives rise to septal and diagonal branches, supplying the mid and distal territories of septal, anterior, lateral wall, and apex.


Overlapping Figures 1 and 5, we can see the entire course of LAD along interventricular sulcus to the apex.


A 55-year-old male with confirmed IgG4 vasculopathy. Axial maximum intensity projection (MIP) image of CT coronary angiography shows partially thrombosed aneurysm of the left anterior descending artery (arrows).


A 55-year-old male with confirmed IgG4 vasculopathy. Volume rendered image shows the aneurysm of the left anterior descending artery (in pink).


A 55-year-old male with confirmed IgG4 vasculopathy. The right anterior oblique caudal view of conventional angiography shows aneurysmal dilatation of the left anterior descending artery with contrast blush (*).


A 55-year-old male with confirmed IgG4 vasculopathy. Coronal maximum intensity projection images of CT abdominal angiography in arterial.


A 55-year-old male with confirmed IgG4 vasculopathy. Venous. Phases show partially thrombosed aneurysm in the proximal part of 8th right intercostal artery (*). Enhancing nodular paravertebral soft-tissue thickening is noted adjacent to the proximal right intercostal arteries above and below this level.


A 55-year-old male with confirmed IgG4 vasculopathy. Axial image of CT abdominal angiography in the arterial phase shows a partially thrombosed aneurysm in relation to the common hepatic artery. Artifacts are seen in relation to the aneurysm (post-injection in the gastroduodenal artery). The aortic stent graft is also seen in situ.


A 55-year-old male with confirmed IgG4 vasculopathy. Coronal maximum intensity projection image of CT abdominal angiography shows multiple visceral artery aneurysms - in relation to the common hepatic artery (black arrow), inferior pancreaticoduodenal artery (white curved arrow), and the jejunal branch of superior mesenteric artery (white arrow). Artifacts are noted in relation to the gastroduodenal artery, aorta, and the superior mesenteric artery - representing stent grafts and post-procedural changes. Cholecystectomy clips are also seen.


Clinical course and IgG index. MMT, manual muscle testing; CSF, cerebrospinal fluid; IVMP, intravenous methylprednisolone pulse therapy; M, magnetic resonance imaging; N, Nerve conduction study.


Initial electrocardiography revealing ST segment elevation in leads V1-V3.


Transesophageal echocardiography image revealing spongy appearance of the right ventricle with trabeculations forming deep fissures and grooves, located in the right ventricle apical wall.


Transesophageal echocardiography contrast echo demonstrating flow from right ventricular cavity into the trabecular recesses.


Fluorescein angiography showed profound macular and peripheral ischemia in the early phase.


In the middle phase.


In the late phase. Composite view).


Follow-up fluorescein angiography showed profound macular and peripheral ischemia in the early phase.


Four-chamber transthoracic echocardiography.


Cardiac magnetic resonance (CMR) images. Reveal concentric hypertrophy with moderately impaired left ventricular function and multiple cystic structures in the septal, mediolateral and anterior parts of the left ventricle.


Medical image.


Late gadolinium enhancement (LGE) demonstrated high signal intensity of the cystic lesions indicating fibrotic tissue , Short axes of the corresponding sequences: transthoracic echocardiography.


Late gadolinium enhancement (LGE) demonstrated high signal intensity of the cystic lesions indicating fibrotic tissue , Cine-CMR.


Late gadolinium enhancement (LGE) demonstrated high signal intensity of the cystic lesions indicating fibrotic tissue. And LGE.


Cardiovascular magnetic resonance mapping of T1 ,. T1 Mapping (1425 +- 144).


T2 . T2 Mapping (60.5 +- 8.2).


Extracellular volume. At 32 months post-transplant FU revealed increased values. Extracellular volume (32.7 +- 9.6).


Timeline of postoperative course after heart transplantation. ATG, Antithymocyte globulin; CMR, Cardiac magnetic resonance tomography; HLA-AB, Human leukocyte antigen antibodies; ICU, Intensive care unit; IgG, Immunoglobulin G; IMC, Intermediate care unit; LV, Left Ventricle; LVEF, Left ventricular ejection fraction; LVAD, Left ventricular assist device; MELAS, Mitochondrial encephalomyopathy, lactic acidosis and stroke-like episodes; MMF, Mycophenolate Mofetil; nsVT, Non sustained ventricular tachycardia: pAMR, Pathologic Antibody mediated rejection; RV, Right ventricle; RVEF, Right ventricular ejection fraction; TDI-Sm, Tissue Doppler Imaging Systolic peak velocities (radial/longitudinal); TTE, Transthoracic echocardiography.


Electrocardiogram and Chest X-ray. (A) ECG showed sinus rhythm, 60 bpm, normal axis, fragmented QRS in the inferior leads (III, aVF) and flat T waves in III, aVF and V6. When compared to previous ECGs, no new abnormalities were observed.


Electrocardiogram and Chest X-ray. (B) Chest X-ray with a single lead ICD, normal heart size and no signs of pulmonary congestion.


Echocardiography. Parasternal long axis view showed mild left ventricular enlargement, both left ventricular end-diastolic.


Echocardiography. End-systolic. Diameters. Right ventricle, ascending aorta and left atrium were of normal size. Interventricular septum and posterior left ventricular wall had normal thickness. No pericardial effusion was observed.


Dynamic of high sensitivity cardiac troponin I levels. Hs-cTnI levels peaked at admission. Three days later, with ongoing recurrent episodes of chest pain, a further increase in hs-cTnI value was observed. After starting aspirin and colchicine, chest pain resolved and hs-cTnI levels progressively decreased. At discharge, hs-cTnI value was almost normal (normal range 0-58.05 ng/l).


Electrocardiogram at admission. Atrial fibrillation, mild ST segment depression in leads V 3-6. T wave inversion.


Diagnosis and treatment of the patient.


Electrocardiogram after resuscitation. Still atrial fibrillation, ST segment depression was significantly deepened and ST segment elevation in aVR lead.


Coronary angiography. Sub occlusive thrombus in the proximal, medial and distal part of the right coronary artery (TIMI 2).


Coronary angiography. Normal flow through the right coronary artery.


RAO caudal view shows a mild lesion in the middle of the LCX artery.


LAO view demonstrates no RCA in the right sinus of Valsalva.


(C) Reconstructed three-dimensional image obtained by the volume-rendering technique shows the anomalous RCA with an interarterial course.


(D) Similarly, the anomalous RCA between the aorta and pulmonary trunk can be seen. . Abbreviations: LAO, left anterior oblique; LCX, left circumflex coronary artery; RAO, right anterior oblique; RCA, right coronary artery.


(A) Reconstructed three-dimensional image obtained using the volume-rendering technique shows the anomalous RCA with an interarterial course.


(B) The origin of the anomalous RCA is evident. . Abbreviation: RCA, right coronary artery.


(A) LAO cranial view shows an anomalous RCA arising from the left sinus of Valsalva.


(B) LAO cranial view shows a high-grade 90% stenosis in the proximal RCA.


(C) LAO cranial view shows a final good angiographic result after PCI procedure.


(D) Reconstructed three-dimensional image obtained by the volume-rendering technique shows the anomalous RCA with an interarterial course.


(E) The anomalous RCA originates from the left sinus of Valsalva. . Abbreviations: LAO, left anterior oblique; PCI, percutaneous coronary intervention; RCA, right coronary artery.


Coronary angiography: Total occlusion of right coronary artery (RCA) and left circumflex artery. Left anterior descending artery originating from RCA.


Computed tomography coronary angiography: Common ostium for right coronary artery (RCA) and left anterior descending artery; absent left main artery. Left circumflex artery short in length and significantly narrow in caliber; completely blocked RCA in proximal segment due to dense calcification.


Angiographic image of this patient. (A) (AP + CRA30 ) the left anterior descending branch.


Angiographic image of this patient. (B) (RAO30. + CAU20 ) the circumflex branch.


Angiographic image of this patient. (C) (AP + CRA20 ) the right coronary artery.


CT angiography volume rendered reformation image. The right coronary artery (RCA) is enormously dilated and tortuous with a large number of collateral vessels. Normal course of a very large right coronary artery is seen.


CT angiography multiplanar reformatted image. The dilated right coronary artery is emerging from the right aortic sinus.


CT angiography volume rendered image. Right coronary artery is dilated and tortious with a large number of collaterals feeding the left coronary system. Left coronary artery is emerging from pulmonary artery trunk. The left anterior descending artery and their diagonal branches are also dilated and tortuous.


Magnetic resonance imaging of the chest. . Notes: Increased signal is seen on T2-weighted images in subcutaneous fat in the anterior abdominal wall at the midline. Increased signal is also noted in anterior mediastinum fat posterior to the sternum. There is periosteum enhancement of the sternum manubrium and at the upper part of the body of the sternum after contrast administration. There is also increased signal in the bone marrow noted on T2-weighted images in the sternum manubrium and in the upper part of the body of the sternum, suggesting bone marrow edema. No evidence of fracture is noted. Findings are consistent with osteomyelitis involving the manubrium and the upper part of the sternum body. Arrows indicate the area of osteomyelitis.


Twelve-lead electrocardiogram showing complete left bundle branch block.


Four-chamber echocardiogram showing a dilated left ventricle.


The constantly deficient secretion of cortisol.


Contrast-enhanced computed tomography revealing atrophic bilateral adrenal glands (red arrows).


MRI showing the posterobasal left ventricular aneurysm (arrows).


A picture of PET scan showing viability of myocardium.


Aneurysm showing thrombus (arrows).


Mitral valve excised and replaced with #29 C. E. Bioprosthesis via left ventricle (arrows).


Completed linear closure of ventriculotomy buttressed with Teflon strips (arrows).


Non-contrast enhanced.


Contrast-enhanced. Black-blood thrombus images of the head in Case 1. The red arrow indicates the focal stenosis of the internal jugular vein and cerebral vein sinus.


(A-D) Magnetic resonance arthrography images of the head in Case 2. The red arrow indicates partial filling defects.


Magnetic resonance venography images of the head.


Neck. In Case 2.


Flow diagram of the study selection process on cyclosporine-A (CsA)-induced thrombosis.


The 3-dimensional (3D) volume rendered image demonstrates both of the left anterior descending arteries (LAD), one originating from the left main coronary artery and the second anomalous one from the right side and spreading to the anterior wall of the left ventricle toward the left ventricular apex.


The segmented 3D image demonstrates both the left anterior descending arteries (LAD), their origin and course.


Coronary angiogram images of the second primary angioplasty:
A and
B show the intrastent thrombus with mycotic aneurysm of the right coronary artery. C and
D show the right coronary artery angioplasty with an overlapping bare metal stent.


Parasternal short-axis view showing the classical windsock appearance of the ruptured sinus of Valsalva arising from the noncoronary sinus.


Communicating with the right atrium.


Electrocardiography on presentation shows a sinus rhythm of 65 beats/minute and ST elevation in V3, II and III.


The CAG. Shows the compression of the LAD coronary artery during the systole resulting in narrowing.


Performed after the second chest pain attack demonstrating a dissection in the distal left anterior descending coronary artery.


Parasternal short axis view at the level of the papillary muscles during the acute phase, demonstrating a small posterior pericardial effusion. The left ventricular contractile function is normal.


Parasternal short axis view at follow-up, demonstrating normal left ventricular contractility and resolution of pericardial effusion.


Parasternal short axis view at the aortic annulus demonstrating giant aneurysms in the right coronary artery, left anterior descending artery, and another giant aneurysm posteriorly in the left circumflex artery region.


Apical four chamber view with anterior angulation demonstrating all three giant aneurysms. The right coronary aneurysm lies at the atrioventricular groove (AV) and the left sided aneurysms are noted along the interventricular septum and the left AV groove.


Still frame image showing the diameter of the right coronary artery aneurysm.


Still frame image showing size of the coronary artery aneurysms of the left anterior descending artery and circumflex artery.


Aneurysm in the region of the circumflex branch of left coronary artery with an area of increased echogenecity along the aneurysmal wall, suspicious of a thrombus.


Selective left coronary artery angiogram in slight left anterior oblique view with caudal angulation. The left main coronary artery appears normal and trifurcates into the left anterior descending (LAD), ramus and circumflex branches. The giant aneurysm in the LAD appears globular in this view and is densely opacified. The ramus aneurysm is long and saccular, located inferior to LAD. There is ectasia of the circumflex branch.


AP view of the same demonstrating overlapping of ramus and LAD aneurysms.


Straight lateral view of a selective right coronary artery angiogram demonstrating a giant aneurysm just distal to the coronary ostium. Multiple small aneurysms are noted throughout the course of the right coronary artery and posterior descending.


showed QS patter with T-wave inversion in III and a VF.


CT angiogram showing bilateral pulmonary emboli.


CT scan Head without Contrast.


MRI Brain without contrast.


MRA Brain.


Troponin trend.


Echocardiographic features of myocardial crypts. Transthoracic
echocardiogram two-chamber view showing diastolic features of a myocardial crypt:
penetration perpendicular to myocardial wall, and ,contention by thin sub epicardial layer
(white arrow).


Echocardiographic features of myocardial crypts. Systolic sub-total obliteration on transthoracic
echocardiogram two-chamber view.


Echocardiographic features of myocardial crypts. Modified two-chamber view
demonstrates the presence of second, more apical crypt parallel of the previous one
(white arrow).


Optimal delineation of the two crypts in cardiac magnetic
resonance two-chamber view sequence (black arrows).


Obvious
hyperenhancement in the inferior wall (white arrows) with adjacent myocardial crypt
(open arrow) on the cardiac magnetic resonance-late gadolinium enhancement short-axis
sequence.


ECG upon arrival (March 2018).


ECG upon review 4 months later (July 2018).


Serial electrocardiograms, lead II strips. (a): normal, 5 months prior to admission.


Serial electrocardiograms, lead II strips. (b): Complete third-degree atrioventricular (AV) block, at primary office 12 h before admission.


Serial electrocardiograms, lead II strips. (c): Type I second-degree AV block, 2 h after admission and antibiotic therapy.


Serial electrocardiograms, lead II strips. (d): first-degree AV block, 12 h after admission and antibiotic therapy.


Serial electrocardiograms, lead II strips. (e): normal, at discharge.


The electrocardiogram recorded in emergency room (ER).


The electrocardiogram prior to antiarrhythmic treatment. The electrocardiogram shows a short PR interval, a delta wave, wide QRS complex, and ST depression.


Electrocardiograms with the sacral neuromodulation device in both on and off modes. (A) An electrocardiogram performed with the sacral neuromodulation implant in "off" mode shows no changes and a sinus rhythm in a patient on antiarrhythmic medication.


Electrocardiograms with the sacral neuromodulation device in both on and off modes. (B) An electrocardiogram performed with the sacral neuromodulation implant in "on" mode shows no changes and a sinus rhythm in a patient on antiarrhythmic medication.


ECG on admission demonstrating anterior ST-segment elevation.


Diagnostic coronary angiography of left coronary artery system . Views:. PA cranial.


Diagnostic coronary angiography of left coronary artery system . PA caudal.


Diagnostic coronary angiography of left coronary artery system . LAO Caudal.


RCA. LAO.


RCA. RAO.


Views of the RCA before.


After. Drug eluting balloon treatment. Markers indicate the RV branch which was occluded and subsequently recanalized following intervention.


Schematic drawing of the anomalous arteries and steno-occlusive changes found in this patient, showing the blood flow in the right internal carotid artery angiography.


The left vertebral artery angiography. As seen on Figures 2 and 3.


Echocardiogram in apical four chamber view. Left: Severe RV and RA dilatation. Right: Mild RV and RA dilatation in recovery.


Straight left upper border of heart.


Left ventricular (LV) apical aneurysm.


Echocardiography showing coronary sinus type of interatrial communication measuring 10 mm x 16 mm with left to right shunting.


Intraoperative finding of a pure coronary sinus type of interatrial communication measuring 1.5 cm in size and a normal coronary sinus ostium.


Resting ECG reveals delayed R wave progression.


Exercise ECG reveals significant ST elevation in lead V3.


Stanford type A DeBakey type II aortic dissection showing enlarged ascending aorta with an approximate diameter of 4.5 cm and dark reddish in color along its length. Extent of the aortic dissection reaches nearly 7 cm high.


(A) Section of the dissected ascending aorta showing the intramural hematoma that was found behind the brachiocephalic trunk.


(B) Section of the dissected ascending aorta revealing calcified left coronary cusp of the aortic valve and dissection entry, measuring around 5 mm, which was situated at the intima of the anterior wall of the ascending aorta, near the sinotubular junction.


(A) Supracoronary aortic tube graft 28 mm, 7 cm in length, placed directly at the sinotubular junction.


(B) Saphenous vein graft taken from the left lower extremity placed between the grafted ascending aorta and the LCX.


Admission electrocardiogram showing sinus rhythm and complete right bundle branch block.


Admission chest X-ray showing evidence of previous sternotomy (repaired tetralogy of Fallot), cardiomegaly and implanted cardioverter defibrillator in left pectoral region.


Right anterior oblique coronary angiography view of the patient. The arrows point to the stenotic sites, including the distal stenosis of the left main coronary artery, severe ostial stenosis of the left anterior descending artery, ramus intermedius artery, and left circumflex artery.


Left anterior oblique coronary angiography view of the patient, showing multiple stenosis at mid and distal parts of the right coronary artery (arrows).


(a) Left ventriculography (right anterior oblique view) showing a left ventricular cleft (white arrow) at the basal inferior wall in diastole.


(b) Transthoracic echocardiogram (apical 2-chamber view). Left ventricular cleft (white arrow) in the basal inferior wall in diastole.


(c) Transthoracic echocardiogram (apical 2-chamber view). More prominent demonstration of left ventricular cleft (white arrow) with contrast echocardiography in diastole.


(d) Cardiac magnetic resonance imaging (left ventricular long-axis view). Myocardial cleft (white arrow) in the basal inferior segment.


Left ventricular cleft narrowed in systole in (a) left ventriculography (right anterior oblique view).


(b) Transthoracic echocardiogram (apical 2-chamber view) with contrast.


(c) Cardiac magnetic resonance imaging (left ventricular long-axis view).


Two-dimensional echo apical four-chamber view.


X-ray image postimplantable cardioverter defibrillator placement.


Prominent 'q' wave, ST segment elevation and 'T' wave inversion in lead II, III and aVF with ST segment depression seen in lead I and aVL. Right sided chest leads (V4R-V6R) showed <1 mm ST segment elevation.


Chest radiograph showing cardiomegaly and pulmonary congestion upon admission.


Chest radiograph showing resolution of cardiomegaly with marked improvement of pulmonary congestion by day nine of hospitalization.


Free thyroxine (FT4) serum concentrations during hospitalization before and after therapeutic plasma exchange (TPE).


An 12-lead electrocardiogram showing acute ST-segment changes in leads II, III, and VF (shaded in light blue).


Follow-up cardiac catheterization demonstrates complete resolution of the right coronary artery dissection (areas corresponding to acute abnormalities noted on the initial cardiac catheterization - see Figure 2 - are circled).


Electrocardiographic examination shows left ventricular hypertrophy and T wave inversion.


Echocardiographic examination shows that the left ventricle cavity is smaller than normal and there is a significant hypertrophy in the septum.


Telecardiography shows epicardial ICD implantation.


Multiple metastasis. Although the ovary carcinoma was resected, liver, and bone metastasis (arrow) was detected. Multiple lymph nodes are swelling (arrowhead), indicating metastasis.


Sagittal computed tomography image of the brain demonstrates a posterior fossa anomaly with vermian hypoplasia, expansile arachnoid cyst, and hydrocephalus.


(a) Maximum intensity projections of computed tomography (CT) angiography demonstrate stenosis of the basilar artery and aplasia of the right common carotid artery.


(b) Multiplanar reconstruction of CT angiography of the neck demonstrates aplasia of the right internal carotid artery.


(c) Skull base CT scanning demonstrates an absence of the right carotid canal.


(d) Abnormal origin and course of the left subclavian vessels are demonstrated. Bilateral common carotid arteries have arisen from the distal side of the left subclavian artery.


(a) Single-photon emission computed tomography demonstrates reduced cerebral blood flow in the border zones between the middle and posterior cerebral artery territories in the right hemisphere.


(b) After extracranial-intracranial bypass, cerebral blood flow of the right hemisphere improves to the normal level.


The dysmorphic facies with hypertelorism and low-set ears.


Cerebral angiography of the left internal carotid artery (ICA) demonstrating severe stenosis of the left ICA at the distal cavernous-infraclinoid portion. The ocular artery was not confirmed (a).


Cerebral angiography of the left internal carotid artery (ICA) demonstrating severe stenosis of the left ICA at the distal cavernous-infraclinoid portion. Cerebral angiography of the left common carotid artery before stent placement demonstrating the ocular artery (arrows) was depicted by a collateral blood flow from the external carotid system (b).


Cerebral angiography of the left internal carotid artery (ICA) demonstrating severe stenosis of the left ICA at the distal cavernous-infraclinoid portion. Cerebral angiography of the left ICA after stent placement demonstrating the ocular artery (arrowhead) was depicted by a directed blood flow from the ICA (c).


Preoperative.


Postoperative. Fluorescent fundus angiography demonstrating the visualization of the retinal artery which improved markedly after the treatment.


A; Preoperative transesophageal echocardiography. Severe calcification was observed in all leaflets and aortic annulus (arrowheads).


B; Preoperative transesophageal echocardiography. The left coronary ostium was intact; there was no significant stenosis (dotted circle).


Preoperative coronary angiography. Left coronary ostium did not show significant stenosis (dotted circle).


Anesthetic record. start/end of surgery. PAP pulmonary artery pressure, CVP central venous pressure, AP arterial pressure, (1) start of CPB, (2) AVR, (3) withdrawal of CPB, (4) signs of left heart failure and appearance of left ventricular wall motion abnormality, (5) second run of CPB, (6) second withdrawal of CPB.


Electrocardiogram on admission and after stent implantation. . Notes:. Showing ST segment slope-down depression, and ,T-wave inversion in leads II, III, aVF, and ,V4-V6. T-wave amplitudes in leads II, III, aVF were decreased, compared with (A). . Abbreviation: ECG, electrocardiogram.


Electrocardiogram on admission and after stent implantation. ECG after stent implantation.


Colon under colonoscopy. . Notes: (A, B) Colonoscopy showing tumor with dirty white-yellow furs in the transverse colon.


Colon under colonoscopy. (C, D) Repeat colonoscopy showing normal colon 10 months after the operation.


ECG during stent thrombosis. . Notes: (A) ECG before recurrent chest pain.


ECG during stent thrombosis. (B) Inverted T-waves in leads II, III, and aVF. Increased T-wave amplitudes in leads III, aVF, and V1-V6, compared with.


ECG during stent thrombosis. ECG after PTCA, decreased T-wave amplitudes in leads II, III, and aVF, compared with . Abbreviations: ECG, electrocardiogram; PTCA, percutaneous transluminal coronary angioplasty.


ECG during stent thrombosis.


ECG changes of acute inferior wall myocardial infarction post-operation. . Notes:. Obvious elevation of ST segment in leads II and III, aVF compared with . Abbreviation: ECG, electrocardiogram.


ECG changes of acute inferior wall myocardial infarction post-operation. ECG 1 day before surgery.


Left anterior oblique caudal view in the coronary angiogram, showing a giant left coronary aneurysm.


Left anterior oblique cranial view in the coronary angiogram, showing a giant left coronary aneurysm (arrow).


Three-dimensional volume-rendering computed tomography, showing a giant aneurysm of the left main coronary artery.


Another view of the giant aneurysm with three-dimensional volume-rendering computed tomography.


CT chest on admission showed SVC thrombus that extended to involve the brachiocephalic vein.


Self-captured photo for prior oral ulcer.


Photo of psudo-folliculitis lesion.


Multiple Sections through CMR revealing normal myocardium with no evidence of endomyocardial fibrosis or scarring.


Diffuse thyroid hypervascularity.


Multiple nodules shown by 'x'.


Upsloping ST depression at J point in the inferior lead (de Winter sign) (red arrow) with positive T wave (yellow arrow) along with minimal ST elevation in aVR (black arrow).


(a) 99% thrombotic occlusion seen in the mid right coronary artery.


(b) After placement of drug-eluting stent.


ST elevation was not noted in serial electrocardiography. Post-post angioplasty resolution of De Winter pattern in inferior leads.


(A) Ischemia-free kidney transplant procedure. The diagram shows procurement, preservation, and implantation of the donor kidney without cessation of blood supply using normothermic machine perfusion.


(B) Normothermic machine perfusion device, Liver Assist (Organ Assist, Groningen, the Netherlands).


(C) Donor kidney circuit on the organ reservoir of the NMP device.


Normothermic machine perfusion and allograft viability. (A) Arterial flow rates and pressure.


Normothermic machine perfusion and allograft viability. (B) Creatinine (Crea) and urea concentration in the perfusate.


Normothermic machine perfusion and allograft viability. (C) Volume of urine production during machine perfusion.


Normothermic machine perfusion and allograft viability. (D) pH values and specific gravity levels of the urine produced before procurement, during machine perfusion, and post-reperfusion.


Initial presenting electrocardiogram: sinus rhythm with premature atrial and ventricular contractions, ST Elevation and Q waves in II, III, and aVF and tall R wave in V2 consistent with infero-post wall MI, STEMI.


Right coronary artery (RCA) pre-intervention revealing 90% occlusion of the mid RCA with thrombus formation.


Right coronary artery (RCA) after bare metal implantation in the mid RCA.


Computed tomography of a 77-year-old man with nonocclusive mesenteric ischemia, carried out at 5 h postadmission with contrast enhancement. A, Spreading of hepatic portal venous gas in the left lobe (white arrows) compared with the initial scan.


Computed tomography of a 77-year-old man with nonocclusive mesenteric ischemia, carried out at 5 h postadmission with contrast enhancement. B, Remarkable bowel dilatation and pneumatosis intestinalis (white arrows) in the absence of contrast-induced bowel wall enhancement.


Computed tomography without contrast undertaken on admission of a 77-year-old man with nonocclusive mesenteric ischemia. A, A small amount of hepatic portal venous gas in the left lobe (white arrows).


Computed tomography without contrast undertaken on admission of a 77-year-old man with nonocclusive mesenteric ischemia. B, Computed tomography did not reveal remarkable acute mesenteric ischemia findings, such as bowel wall thickening, a hyperattenuating bowel wall, and paper-thin bowel wall (white arrows).


Emergency laparotomy carried out in a 77-year-old man with nonocclusive mesenteric ischemia. The small bowel appears necrotic, with segmental and skip lesions visible at the initial operation.


Optical coherence tomography angiography (OCTA) with the right eye represented by the right column.


The left eye by the left column The structural slabs a, b of the lesions in both eyes were obtained using an RPE-RPE fit segmentation.


Optical coherence tomography angiography (OCTA) with the right eye represented by the right column En face OCTA with customized segmentation between RPE and Bruch's membrane shows a type 1 neovascular network with evident aneurysmal dilatation, as demarcated by the crossing blue and green lines (c, d). Dense B-scan with angiographic flow overlay e, f demonstrates the corresponding cross-sectional view and the customized segmentation of the respective lesions.


The left eye by the left column The structural slabs a, b of the lesions in both eyes were obtained using an RPE-RPE fit segmentation. En face OCTA with customized segmentation between RPE and Bruch's membrane shows a type 1 neovascular network with evident aneurysmal dilatation, as demarcated by the crossing blue and green lines (c, d). Dense B-scan with angiographic flow overlay e, f demonstrates the corresponding cross-sectional view and the customized segmentation of the respective lesions.


Polymorphous rash on the infant's inferior limbs.


Cross-sectional view at the level of the great arteries: left anterior descending (LAD) artery (at 3 o'clock).


(A) Cross-sectional view at the level of the great arteries: left anterior descending (LAD) with an aneurysm (diameter > 6 mm, Z score + 16.45).


(B) The same image as 3A with color Doppler.


Chest X-ray posteroanterior view, arrow showing asymmetrical cardiomegaly.


Two-dimensional transthoracic echocardiography image (parasternal short-axis view) showing cystic mass over lateral wall of left ventricle extending into inferior side.


Computed tomography angiogram showing relation of the cyst with the major epicardial vessels and relation of cyst with left ventricle cavity.


White pale daughter cysts after thoracotomy on the lateral wall of the left ventricle.


Daughter cysts after delivery.


(A-D) Case presentation of 65-year-old male patient with COVID-19, 5 days after tocilizumab (TCZ), non-contrast abdominal CT. Axial.


(A-D) Case presentation of 65-year-old male patient with COVID-19, 5 days after tocilizumab (TCZ), non-contrast abdominal CT. Coronal.


(A-D) Case presentation of 65-year-old male patient with COVID-19, 5 days after tocilizumab (TCZ), non-contrast abdominal CT. 3D reconstruction, pneumatosis intestinalis (PI) involving ascending colon (yellow arrows), with dilated multiple right lower quadrant small bowel loops with mesenteric and portal venous gas (yellow arrowheads).


Chordoid glioma in 34-year-old man. . A. Axial unenhanced CT showing hyperattenuated mass compressing frontal horn of right lateral ventricle. Note small calcification (arrow) in periphery of mass.


Chordoid glioma in 34-year-old man. B. Sagittal T1-weighted (upper left panel) and axial T2-weighted (upper central panel) images showing isointense lobulated mass (arrows) relative to cerebral cortex in anterior third ventricle. Axial susceptibility-weighted image (upper right panel) showing no evidence of intratumoral hemorrhage. Sagittal (lower left panel), axial (lower central panel), and coronal (lower right panel) post-contrast T1-weighted images showing strong enhancing main tumor with lobulated margin (arrows) in anterior part of third ventricle and smaller enhancing masses (arrowheads) along wall of lateral ventricles and fourth ventricle.


Chordoid glioma in 34-year-old man. C. CBV map of perfusion MRI showing elevated CBV within tumor (arrows) in third ventricle.


Chordoid glioma in 34-year-old man. D. Photomicrograph of hematoxylin and eosin stained slide showing solid cellular components composed of clusters and cords of epithelioid tumor cells (arrows) within variable mucinous stroma (original magnification x 400).


Chordoid glioma in 34-year-old man. Photomicrographs of immunostained slides for GFAP.


Chordoid glioma in 34-year-old man. CBV = cerebral blood volume E, F. CD 34. Showing diffuse and strong expression in tumor cells (dark yellow and brown colors) (original magnification x 400). GFAP = glial-fibrillary acid protein.


Coronary angiography with a catheter located in the coronary artery. The dotted circle shows the ruptured distal fragment of the catheter, before it detached.


Skull radiography, anteroposterior view. The dotted white circle shows the catheter fragment lodged in the right middle cerebral artery.


Right internal carotid artery angiography, left anterior oblique view. The dotted white circle shows a double-lumen balloon inflated within the fragment of the catheter, the latter lodged in a branch of the right middle cerebral artery.


Skull radiography, right anterior oblique view. Note inside the white circle the balloon inflated displacing the fragment distally.


(a) Skull radiography. Continued white arrow shows the inflated balloon half inside the catheter fragment and half distal to it. Dotted white arrow shows the catheter fragment.


(b) Skull radiography. Dotted white circle shows the inflated balloon pushing the catheter fragment at M1 segment of cerebral media artery.


(c) Facial bones radiography. Dotted white circle shows the catheter fragment near the tip of the guide catheter at cervical segment of carotid artery.


(d) Chest radiography centered on the clavicle. Inside the dotted white circle, note the inflated balloon and catheter fragment at the level of the right common carotid's origin. White arrow shows the tip of the guide catheter at the level of the right brachiocephalic trunk.


Postprocedural computed tomography of the brain, showing a subarachnoid hemorrhage.


Follow-up angiography with no evidence of vascular lesions or residual intravascular evidence of the retrieved catheter fragment.


Follow-up computed tomography of the brain, without secular lesions.


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-AP view fluoroscopy shows blunt left sinus (arrow) with no coronary origin.


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-AP view fluoroscopy shows blunt left sinus with no coronary origin.


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-right anterior oblique angiographic view shows both right and left coronaries originating from the right sinus (arrow).


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-lateral angiographic view shows both RCA and LCA originating from same ostium (arrow).


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-right anterior oblique angiographic view shows both right and left coronaries originating from the right sinus.


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. Coronary angiography-lateral angiographic view shows both RCA and LCA originating from same ostium.


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. CT coronary angiography-CT image with 3D reconstruction shows retroaortic course of LCA (arrow).


60-year-old male with ACS-inferior wall MI, post thrombolysis and post MI angina. CT coronary angiography-CT axial image shows all three coronary arteries.


Bedside chest X ray showing diffuse and bilateral lung infiltrates, cephalization of the pulmonary vessels, bronchial wall thickening giving the aspect or railway track in the upper right lung field and blunting of the right costophrenic angle. The cardiac silhouette is not interpretable (not standing chest x ray).


(A-D) High Resolution Computed Tomography showing scattered ground glass opacities, with predominant bilateral postero-basal and central distribution. It is associated occasionally with a septal thickening giving the aspect of crazy paving.


Bedside chest X ray showing diffuse interstitial lung disease, affecting predominantly the right upper lobe. The right scissure is well visible. We remark also the blunting of the right costophrenic angle. The mediastinum is not interpretable.


(A-D) Computed Tomography Scan showing Crazy paving with ground glass opacities and bilateral thickening of the interlobular septa, located predominantly in the right upper lobe, the middle lobe and the basal segments of the right lower lobe, the apical segment of the left upper lobe and the upper segment of the left lower lobe.


An electrocardiogram showing the ST segment elevation in DII, DIII, and aVF, as well as the ST segment depression in V1-V5.


A coronary angiographic image showing the total occlusion of the LCx (RAO 12. - Caudal 36 ).


An aortic root angiographic image revealing the left main coronary artery arising from the left Valsalva sinus and an absent right coronary ostium (LAO 36. - Caudal 7 ) (Video 1).


A post-stent angiographic image revealing that the RCA originated from the LCx (RAO 10. - Caudal 39 ) (Video 3).


A; CT angiographic image showing a single coronary artery arising from aorta.


Twelve-lead electrocardiography on admission showed atrial fibrillation with a heart rate of 78 beats/min and ST elevation at II, III, and aVF and ST depression at V2-V6.


The head computed tomography (CT) image showed no intracranial hemorrhages.


But the hyper-dense arterial sign was observed at the basilar artery. Arrowhead), which suggested the basilar artery occlusion.


The contrast-enhanced chest CT did not indicate aortic dissection nor cardiac tamponade (c).


The coronary angiography revealed distal occlusion in the posterior descending branch of the right coronary artery (d, arrow).


Mild stenotic lesions suggestive of atherosclerotic pathology were observed in some parts (e and f).


The diffusion-weighted image showed hyper-intense lesions in the region of the posterior inferior cerebellar artery (g and h).


Magnetic resonance angiography did not reveal the flow of the basilar artery, and severe stenotic lesions of the major arteries were scarcely observed (i).


After 38 h from the onset, his consciousness level was depressed as Japan coma scale I-3. The head computed tomography (CT) 38 h after the onset showed hydrocephalus.


After 38 h from the onset, his consciousness level was depressed as Japan coma scale I-3. Due to cerebellar edema and hemorrhagic infarction of the ischemic cerebellum, which compressed the aqueduct.


After 38 h from the onset, his consciousness level was depressed as Japan coma scale I-3. The head CT after cerebral ventricular drainage showed improvement in hydrocephalus (c).


After 38 h from the onset, his consciousness level was depressed as Japan coma scale I-3. The chest CT showed cardiac tamponade (d). Emergency pericardiotomy was immediately performed to allow the fluid to drain. A left mural and atrial appendage thrombi were observed.


After 38 h from the onset, his consciousness level was depressed as Japan coma scale I-3. There were no apparent bleeding sources, aortic dissections, nor myocardial ruptures intraoperatively (e).


12 lead electrocardiogram (ECG) of the patient at the time of presentation. ECG showing T wave inversion in precordial leads from V1 to V6. There is no shift of ST segments.


Coronary angiogram in the left anterior oblique caudal view showing eccentric narrowing the proximal left anterior descending coronary artery with 75% stenosis.


Coronary angiogram showing successful result of angioplasty and stenting to proximal left anterior descending coronary artery.


12-lead ECG showing normalization of the ischemic changes on follow up.


Twelve-lead electrocardiogram on admission. The electrocardiogram demonstrates hyperacute T waves in V2-4.


Left coronary angiography. A; Initial left coronary angiography of cranial view revealing embolic obstruction straddling the bifurcation in the left anterior descending artery and diagonal branch (arrow).


Left coronary angiography. B; Left coronary angiography after aspiration showing the restoration of blood flow.


Contrast-enhanced computed tomography showed that tumor invading the left renal vein (arrow).


Transesophageal echocardiography showing a patent foramen ovale and shunt flow (arrow). LA: left atrium, RA: right atrium.


Transthoracic echocardiography on readmission showing right ventricular dilatation and a mobile mass attached to the tricuspid valve (arrow). RV: right ventricle, LV: left ventricle, RA: right atrium.


Electrocardiogram 4 h post-presentation, with recurrence of chest pain showing ST-elevation in leads V1-V2.