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MIMIC-CXR-JPG/2.0.0/files/p11761571/s59195945/70450045-25faba92-d1dda7ee-2546b877-5a430dd1.jpg
retrocardiac opacity is similar in appearance to the studies of , but overlying infection is not excluded. small left loculated effusion is unchanged in appearance.
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lower lobe consolidation, likely on the left, best seen on the lateral view, likely represents pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p15352491/s55521700/c0f25687-2905f73f-18a5289a-1e11314a-e4036d76.jpg
no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11515672/s59474291/bb7592dc-9e3efe3a-69743826-f3dee070-d65e821f.jpg
no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p10862710/s57743327/5f343109-c9f1cf4b-e21e75e1-b771d9f5-6c4f8c86.jpg
low lung volumes. apparent superior mediastinal widening is likely related to the presence of low lung volumes, but repeat pa and lateral views with improved inspiratory effort is recommended. no acute cardiopulmonary abnormality otherwise noted.
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mild retrocardiac opacification in the setting of low lung volumes most likely represents atelectasis. in the appropriate clinical context, superimposed infection is not entirely excluded.
MIMIC-CXR-JPG/2.0.0/files/p14758794/s54285482/ffbaf075-c9742faa-01c3dd91-e4bcfdfb-f6cfcbee.jpg
no acute cardiopulmonary abnormality.
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endotracheal tube tip in good position. mildly improved left perihilar, basilar opacity. mildly worsened right infrahilar opacity. findings likely from edema, with component of atelectasis; pneumonitis cannot be excluded in the appropriate clinical setting
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14761129/s58710136/e8b659f7-6988c60a-86244324-5abf8f72-c722436d.jpg
no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p17425647/s55169341/498ec5af-fd7f0cda-1b1bba3b-9fda3f5d-353d7be7.jpg
new patchy consolidation in the right lower lobe compatible with pneumonia. please repeat after treatment to document resolution, especially in the setting of patient with prior malignancy. chronic changes as detailed above.
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improved ventilation of the left lung when compared to the last cxr taken at pm. stable post-operative pneumopericardium.
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no acute intrathoracic abnormalities identified.
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normal chest radiograph without evidence of pneumonia
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p18700677/s54702946/b9345e11-5407e1ea-bd588f1f-450f9d6e-2f077dd6.jpg
<num> mm bilobed metallic density object within the soft tissues of the left axilla.
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no acute intrathoracic process.
MIMIC-CXR-JPG/2.0.0/files/p17542726/s52936613/56e7c971-5e3d2f39-df416438-d18b1d34-52f6009b.jpg
persistent left basilar opacities, possibly due to chronic scarring or atelectasis noting the lack of change, although it is difficult to exclude the possibility of recurring infection at the same location as before.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p17614057/s53572658/f762bf98-b2141d3c-a5c0a0b1-4fb662f7-fce29b8d.jpg
no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p14630669/s58239769/eb9ba3e6-c1e2396c-1781d883-21b9659e-3cfc585a.jpg
no evidence of acute disease. hyperinflation.
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no evidence of acute cardiopulmonary process. there is mild compression fracture of a lower thoracic vertebral body (likely t<num>) new since the prior study, but otherwise of indeterminate age. recommend clinical correlation for acuity.
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increased interstitial markings are seen bilaterally, raising possibility of pulmonary vascular congestion. superimposed bibasilar opacities, potentially due to atelectasis; however, developing consolidation from infection is also possible. clinical correlation suggested.
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minimally improved ventilation at the right lung base. otherwise no relevant change. pleural thickening on the left. moderate cardiomegaly. low lung volumes and signs of mild to moderate pulmonary edema persist. no focal parenchymal opacities suggesting pneumonia
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ng tube tip isin the stomach. no other interval change from prior study.
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left basal opacity likely reflects atelectasis and a small pleural effusion. minimal right basal atelectasis. no pneumothorax.
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no acute intrathoracic abnormalities identified.
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the expected location of a midline catheter (upper arm) is not effectively included on this examination. in order to image a midline catheter in conventional placement, images should include the upper arm/humerus and axilla. no catheter seen within the chest. no acute pulmonary process identified.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p15568077/s56577538/e4e482d1-fa5ac7d7-c7e7dfdc-163b9741-b04bbdb3.jpg
no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11648387/s54280682/de5af302-09cc16af-f2f2f88c-bf82cea8-f174c86e.jpg
bibasilar atelectasis.
MIMIC-CXR-JPG/2.0.0/files/p19606815/s59817144/2c8c1e8a-e24f3c4b-b35a4abc-534e1493-a3ff848a.jpg
no signs for acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p13958446/s51330447/e629ff87-0249e0b6-df59e9a0-dca75274-c0e4cf8b.jpg
status post-placement of left pleurx catheter with interval decrease in left-sided pleural effusion. left-sided apical and lateral areas of hypodensity may represent pneumothorax versus expanding lung. recommend repeat radiograph of the chest for additional evaluation of possible pneumothorax.
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moderate cardiomegaly and mild pulmonary edema appear unchanged. bibasilar opacities have improved in comparison to the prior study from.
MIMIC-CXR-JPG/2.0.0/files/p19346447/s57816423/bf62f619-0e7c80ca-741a13c0-49c0b2f9-c1a727b2.jpg
ng tube tip terminates in the stomach. right picc terminates in the svc. left pleural effusion with likely left lower lobe atelectasis. no pneumothorax.
MIMIC-CXR-JPG/2.0.0/files/p11827275/s57956851/cf84fd25-f449a28f-23f8ce1f-890d35ed-c66d9bc7.jpg
findings most suggestive of mild-to-moderate pulmonary edema.
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a left picc line tip is at the level of mid svc. heart size and mediastinum are unchanged in appearance. multifocal opacities are unchanged.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14325171/s56483450/af433aa9-104d09a2-53d4c870-23f77f2d-abbfdc60.jpg
normal heart, lungs, hila, mediastinum, and pleural surfaces. no evidence of intrathoracic malignancy or infection, including tuberculosis.
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no acute intrathoracic process.
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heart size and mediastinum are stable. there is interval progression of perihilar opacities concerning for pulmonary edema although infectious process is a possibility. distortion caused by the lung cancer treatment is re- demonstrated and unchanged.
MIMIC-CXR-JPG/2.0.0/files/p13694819/s59109972/8bb7e607-ad692809-b9488a0a-08c6f9f6-fcc65578.jpg
no acute cardiopulmonary process. no displaced rib fracture.
MIMIC-CXR-JPG/2.0.0/files/p10304606/s56996892/0e75244d-bd2cc3d2-e4a1e33e-c509f8fd-472cdb9b.jpg
as compared to previous study of , marked cardiomegaly is stable, accompanied by pulmonary vascular congestion and interstitial edema as well as a small right pleural effusion. bibasilar linear atelectasis is also noted.
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in comparison with the study of , with the <num> chest tubes on water seal, there is no evidence of appreciable pneumothorax. the endotracheal tube, swan-ganz catheter, and nasogastric tube have all been removed. continued enlargement of the cardiac silhouette with bibasilar opacifications, consistent with relatively m...
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in comparison with the study of , there is continued and possibly increasing bilateral layering pleural effusions with underlying compressive atelectasis. there may be mild elevation of pulmonary venous pressure. monitoring and support devices are essentially unchanged.
MIMIC-CXR-JPG/2.0.0/files/p11471975/s50428896/eeba1f9c-39135d8b-bde92642-4a13031d-9fbd6845.jpg
no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14065959/s55715929/48f2bd81-378f9655-8565778a-754c4daa-96b5b381.jpg
worsened cardiac decompensation.
MIMIC-CXR-JPG/2.0.0/files/p10344684/s59281689/6fac1123-2d2e67a9-803304ee-2468541e-32a863e9.jpg
mild pulmonary edema.
MIMIC-CXR-JPG/2.0.0/files/p13687044/s54470875/aa4d168a-9c98bdb8-39ed398c-30e8ff37-51606b50.jpg
left-sided picc line tip projects over the right subclavian vein. repositioning is recommended. these findings were discussed with , rn by dr via telephone on at , at time of discovery.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p14012923/s53641710/b4b8fc51-75999dd3-1ef0e038-917b1adb-19bbb62f.jpg
no radiographic evidence of an acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11921191/s58955362/92422977-69de1343-40b6b6cd-04965170-45cad440.jpg
no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p16167870/s53804282/bfe857d9-40be2055-d1776aba-d52b7d7a-834a4094.jpg
minimal patchy right basilar opacity may reflect atelectasis, with a trace right pleural effusion. infection is not completely excluded.
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in comparison with the study of , the questioned nodular opacity overlying a mid to upper vertebral body is no longer appreciated. otherwise, little change in the appearance the heart and lungs.
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moderate right pleural effusion concerning for malignant involvement. subtle left basilar opacity of uncertain etiology. close attention on follow-up imaging is recommended.
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no evidence of acute disease. bulging right atrial contour; this appearance is not necessarily abnormal but follow-up echocardiogram should be considered in addition to correlation with clinical history.
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no acute cardiopulmonary abnormalities
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decreased right mid-to-lower lung opacities, consistent with improving atelectasis. unchanged volume loss at the left lung base. marked left hemidiaphragm elevation, not significantly changed. small bilateral pleural effusions, containing less than <num> ml of fluid.
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no acute cardiopulmonary process.
MIMIC-CXR-JPG/2.0.0/files/p11945569/s53144834/8986e3d7-61b2a83c-89ea9cf6-aa93bd6a-269cc290.jpg
left-sided pleural effusion appears similar to slightly decreased compared to previous imaging.
MIMIC-CXR-JPG/2.0.0/files/p11887646/s56149094/d57a5b81-dfef4be6-de62cb4c-80ea092e-3d1806fe.jpg
no acute cardiopulmonary abnormality.
MIMIC-CXR-JPG/2.0.0/files/p12376118/s51475211/04435412-b49ea085-eccaa514-6a3c4256-0071034f.jpg
right internal jugular central venous catheter ends in the low svc. no pneumothorax. decreased moderate interstitial pulmonary edema with unchanged moderate-to-severe cardiomegaly. dense retrocardiac atelectasis. likely small bilateral pleural effusions.
MIMIC-CXR-JPG/2.0.0/files/p13197784/s51523022/496b2ce2-c113d802-b460a5db-74471023-669f3c48.jpg
mild interstitial pulmonary edema is new. lung volumes remain quite low and there is greater atelectasis at the right base. small pleural effusions are likely but not appreciably changed. heart size is normal.
MIMIC-CXR-JPG/2.0.0/files/p17977928/s50760492/181a994e-098b46fb-f02512e4-dc3d1c80-3b6dd4ad.jpg
no acute cardiopulmonary process.
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right lung base opacity appears new from exam, with associated small right pleural effusion, suggestive of pneumonia.
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heart size and mediastinum are unremarkable. bilateral pleural effusions are small to moderate associated with bibasal consolidations. loculated effusion along the right major fissure is most likely present. no pneumothorax is demonstrated.
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comparison to. unchanged massive cardiomegaly. mild pulmonary edema is present on today's examination. no pleural effusions. no pneumonia.
MIMIC-CXR-JPG/2.0.0/files/p10670818/s51340521/09a7a2ef-90d79ed1-357532dc-63d429ac-67fb568f.jpg
minimal increase in size of right pleural effusion. slightly decreased left pleural effusion.
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no acute cardiopulmonary process and no evidence of heart failure.
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no acute cardiopulmonary process.
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increase in right pleural effusion and adjacent atelectasis. no evident pneumothorax copd
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compared to chest radiographs since , most recently. interval increase in pulmonary vascular caliber and heart size accompanied by mild interstitial edema reflects cardiac decompensation and/or volume overload. new well-circumscribed round <num> mm wide opacity projecting over the right upper lung could be a fissural p...
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bilateral multifocal opacities with increasing opacities in the left lung base, small left pleural effusion, and left retrocardiac consolidation. evolution of pulmonary findings with distention of the azygos vein raises the possibility of volume overload superimposed on known infection.
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stable chest findings with poor inspirational effort and low respiratory volumes with basal plate atelectasis but no evidence of acute pneumonic infiltrate.
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no intrathoracic process. dilated loops of air and fluid-filled small bowel are not completely evaluated. further characterization with abdominal radiograph is recommended.
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new dual lead pacemaker leads terminating in the right atrium and right ventricle. no complications. possible lesion in the lingula. recommend correlation with any other chest imaging available to see if a clinically significant, lung lesion can be excluded. recommendation(s): if no other chest radiographs or ct perfor...
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in comparison with the earlier study of this date, of the monitor and support devices are unchanged. the patient has taken a better inspiration, and this may account for the slight improved aeration. there is still extensive bilateral pulmonary opacification bilaterally. the tip of the endotracheal tube measures approx...
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no acute cardiopulmonary process.
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as compared to the previous image, the patient has been intubated. the tip of the endotracheal tube projects <num> cm above the carina. the new nasogastric tube shows a normal course. the tip projects over the middle parts of the stomach. the right jugular vein catheter remains in unchanged constant position. moderate ...
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no acute cardiopulmonary process.
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persistent blunting of the left costophrenic angle may be due to underlying pleural effusion. increased left mid to lower lung opacity raises concern for overlying infection.
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large right pleural effusion with right basilar opacification likely reflective of compressive atelectasis. infection cannot be completely excluded.
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no acute cardiopulmonary process.
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dense consolidation involving the right mid and lower lung zones with more patchy opacities in the right upper lung concerning for pneumonia. possible small left pleural effusion or volume loss at the right base.
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right middle lobe pneumonia.
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the heart is stably enlarged which most likely reflects cardiomegaly, although pericardial effusion cannot be entirely excluded. lungs are without evidence of focal airspace consolidation to suggest pneumonia. no pulmonary edema. no pneumothorax. no pleural effusions. bones are osteopenic with mild degenerative changes...
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in comparison with the study of , the cardiac silhouette is within normal limits in size and there is again mild tortuosity of the aorta. no evidence of acute pneumonia, vascular congestion, or pleural effusion.
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interval improvement in opacity at the right lung base. no pneumothorax.
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compared to chest radiographs most recently. lungs are fully expanded and clear. cardiomediastinal and hilar silhouettes and pleural surfaces are normal. previous borderline cardiomegaly and vascular congestion have resolved.
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status post pacemaker with leads in appropriate position.
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in comparison with the study of , the monitoring and support devices are stable, as is the cardiomediastinal silhouette. continued low lung volumes. engorgement of pulmonary vessels is consistent with elevated pulmonary venous pressure. continued bibasilar opacifications, some of which represents pulmonary vascular con...
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multi focal areas of infection or infarction have generally improved since. mild pulmonary edema is new. pleural effusions small if any. there is no pneumothorax. cardiomediastinal silhouette is probably normal size, given severely diminished lung volumes. it would be difficult to detect a rib fracture unless severely ...
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in comparison with the study of , the monitoring and support devices are unchanged. an there is little change in the appearance of the heart and lungs.
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decrease in small to moderate left pleural effusion.
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interval placement of right chest tube with improvement in right pneumothorax which still contains apical and medial components.
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no acute intrathoracic process.
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mild pulmonary edema, improved since prior imaging.
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comparison to. status post left thoracocentesis. there is no evidence of pneumothorax. mild decrease in extent of the pre-existing left pleural effusion. stable normal appearance of the right lung.
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no evidence of acute cardiopulmonary process. possible aortic valve calcifications raise concern for valvular disease. clinical exam is recommended to assess for evidence of aortic stenosis.
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no acute cardiopulmonary process.