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no acute intrathoracic abnormality.
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no acute findings in the chest.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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small bilateral effusions on the right. cardiomegaly without definite acute cardiopulmonary process.
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no acute cardiopulmonary process. no free air below the diaphragm.
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no acute findings in the chest. no signs of pneumoperitoneum.
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no convincing signs of aspiration.
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persistent small left pleural effusion with slight worsening of adjacent left basilar opacity likely reflective of compressive atelectasis.
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<num>. endotracheal tube in appropriate position after manipulation. <num>. right lung base opacity is compatible with right lower lobe pneumonia versus aspiration.
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no acute cardiopulmonary process. large hiatal hernia is again seen.
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enteric tube ends in the lumen of the upper stomach, below the diaphragm. no acute intrathoracic abnormality.
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mild pulmonary edema, similar compared to the prior exam.
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no acute cardiopulmonary process. no evidence of pneumothorax.
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left lower lobe pneumonia.
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<num>. no evidence of pneumonia. <num>. lateral left costophrenic angle blunting, which likely represents pleural thickening and less likely a small pleural effusion.
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no definite acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no change from prior.
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<num>. increasing size of multiple pulmonary metastases.
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as above.
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low lung volumes and moderate cardiomegaly. no focal consolidation.
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<num>. left basilar opacity is likely atelectasis. <num>. small left pleural effusion. <num>. displaced fractures of the left fourth through eighth ribs.
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probable early right lower lobe consolidation that may represent an infectious process. findings were communicated by dr. <unk> to dr. <unk> <unk> telephone at <time> p.m. on <unk>, five minutes after discovery.
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<num>. chronic interstitial bibasilar changes and flattening of the hemidiaphragms may represent chronic pulmonary disease. for further evaluation of interstitial lung disease, chest ct would be recommended. <num>. small hiatal hernia. recommendation(s): for further evaluation of interstitial lung disease, chest ct wou...
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no acute cardiopulmonary process.
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no acute cardiopulmonary process. copd
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no acute intrathoracic process. specifically, no evidence of intrathoracic malignancy.
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no acute intrathoracic process.
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stable moderate bilateral pleural effusions.
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no acute cardiopulmonary process.
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significantly reduced right pleural effusion status post drain placement. no pneumothorax.
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interval decrease in the right pleural effusion. persisting patchy bilateral opacities, reflective of multifocal pneumonia.
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no acute cardiothoracic process including no evidence of pneumothorax.
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mild cardiomegaly. no evidence of pneumonia. these findings were reported to dr. <unk> office at <time>am on <unk> by telephone.
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<num>. appropriately positioned lines and tubes. <num>. unchanged subsegmental left retrocardiac atelectasis and mild cardiomegaly.
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no radiographic evidence for acute cardiopulmonary process. left hilar calcification.
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no definite acute cardiopulmonary process.
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no acute cardiopulmonary process.
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hilar congestion without frank edema.
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normal chest radiograph.
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cardiomediastinal silhouette is enlarged, but grossly unchanged. chf with interstitial and possible scattered pulmonary edema, overall similar to the prior study. small right effusion may be slightly smaller. there is persistent underlying collapse and/or consolidation and probable small right effusion. increased retro...
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known left upper lobe lung mass and interval placement of a right port-a-cath with interval decrease in right paratracheal opacification, likely corresponding to known lymphadenopathy, without radiographic evidence for acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no radiographic evidence for acute cardiopulmonary process. these findings were discussed with dr. <unk> by dr. <unk> by telephone at <time> p.m. on <unk>.
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linear opacities in left lower lobe most likely represent atelectasis.
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no evidence of pneumonia.
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no acute intrathoracic process. port-a-cath positioned appropriately.
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<num>. no acute cardiopulmonary process. <num>. stable scarring in the right lower lobe.
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no new focal consolidation. probable small bilateral pleural effusions. severe pulmonary fibrosis. moderate cardiomegaly.
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right large bore internal jugular catheter unchanged in position. left-sided single lead pacer also unchanged in position, although the distal portion of the lead is difficult to see due to technique. lung volumes remain low. focal opacity seen at the left base is seen and in the correct clinical setting would be conce...
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hyperinflation without superimposed consolidation.
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little interval change from prior with continued mild congestive heart failure and bibasilar atelectasis.
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resolution of left lower lobe pneumonia with residual left lower lobe bronchial wall thickening.
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<num>. resolving pneumoperitoneum. <num>. bilateral plaque dominant perihilar opacities are largely unchanged since <unk>.
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<num>. stable or improved small left pleural effusion compared to <unk>. <num>. stable postoperative appearance of the cardiomediastinal silhouette, with mild pulmonary vascular congestion.
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left basilar atelectasis.
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no significant change since <unk>.
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interval intubation with endotracheal tube having its tip approximately <num> cm above the carina. interval placement of a nasogastric tube which courses below the diaphragm with the tip not identified. the left internal jugular central line is unchanged in position with its tip in the proximal svc. status post median ...
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ap chest compared to <unk> at <time>. new dobbhoff feeding tube, with the wire stylet in place, loops of the gastroesophageal junction and ends in the upper esophagus. previous mild pulmonary edema has improved, but small bilateral pleural effusions have increased. moderate to severe left lower lobe atelectasis is unch...
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mild pulmonary edema, unchanged to slightly worse from <unk>.
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<num>. interval improvement in lung volumes with small bilateral pleural effusions. <num>. interval resolution of left apical pneumothorax.
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stable right paratracheal mass with deviation and critical compression of the trachea.
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no acute cardiopulmonary process.
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<num>. edema in the right lung and atelectasis in the left mid lung are mildly worse compared to prior. other findings are similar to prior. <num>. et tube remains several cm superior to optimal position. <num>. significant retrocardiac opacity is similar to multiple priors, likely atelectasis.
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stable large left pleural effusion. consolidation in the left mid lung zone with air bronchograms concerning for pneumonia, comparable to recent ct findings. no pneumothorax. these findings were entered into the critical results dashboard by dr. <unk> at <num>pm.
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no acute intra thoracic abnormality. if clinical concern for rib fracture persists, dedicated rib pelvis with radiopaque marker can be performed. no displaced rib fracture is identified.
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<num>. large hiatal hernia. <num>. old healed right rib fractures. <num>. no acute consolidation, pleural effusion, or pneumothorax.
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no chest injury. if there is further concern for rib fracture, recommend repeat dedicated views with a bb marker to mark the site of pain.
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chf with interstitial and alveolar edema. this appears slightly worse compared with <unk> opacity at both lung bases which likely represents combination of pleural effusions and underlying collapse and/or consolidation. ng tube not well visualized in lower esophagus and beyond due to underpenetration.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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no focal consolidation or pneumothorax.
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there is diffuse bilateral interstitial prominence which may represent mild pulmonary edema or alternatively lymphangitic spread of tumor. multiple known metastatic nodules seen on ct are not well evaluated by radiography. interstitial abnormalities limit detection of subtle consolidation however there is no large cons...
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no radiographic evidence for acute cardiopulmonary process.
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mild left basal atelectasis and top normal heart size. otherwise, normal.
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status post endotracheal tube removal and tracheostomy tube placement. no acute cardiopulmonary process.
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mild bibasilar atelectasis. no acutely displaced fractures identified.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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emphysema without superimposed pneumonia.
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<num>. no areas of consolidation to suggest the presence of pneumonia. <num>. bronchial wall thickening, which could reflect bronchitis in the appropriate clinical setting. <num>. enlarged central pulmonary artery suggesting pulmonary arterial hypertension.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no radiographic evidence for acute cardiopulmonary process.
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no acute cardiopulmonary process.
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right upper lobe opacity with apparent associated volume loss is suspicious for parenchymal lesion, though shadows from overlapping structures is possible. recommend apical-lordotic radiographs for further evaluation. recommendation(s): right upper lobe opacity with apparent associated volume loss is suspicious for par...
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no pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10173600/s57767334/3a889390-02548516-59764501-b842da68-24a8b6b2.jpg
no acute intrathoracic process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12298456/s57638644/c77c454a-20778ce8-10e9afc6-d986cf64-6d62aea9.jpg
lung hyperinflation, no evidence of pneumonia. mild increase in left basilar atelectasis.
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no radiographic evidence for pneumonia.
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limited, negative.
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minimal bibasilar atelectasis. otherwise, no acute pulmonary process identified.
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an opacity projecting over the lung bases may represent pleural disease or may be parenchymal. ct may be useful given the patient's pleural in interstitial lung disease.
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no acute cardiopulmonary process, no pulmonary edema.
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no acute cardiopulmonary process are noted.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15605848/s58163071/bd5e4711-88976a9e-9df52112-f6fdcb31-1313d22c.jpg
no acute cardiopulmonary process.