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subsegmental right basilar atelectasis.
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moderate left pleural effusion new from <unk>.
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right lower lobe consolidation, compatible with pneumonia. followup to resolution.
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no acute findings.
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<num>. bronchial wall thickening, which may be related to history of acute bronchitis. no consolidation to suggest coexisting pneumonia. <num>. bibasilar linear atelectasis or scarring.
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bilateral lower lobe atelectasis is minimally improved since <unk>.
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low lung volumes on frontal exam contributing to crowding of the bronchovascular markings. increased conspicuity of bibasilar opacities which could represent atelectasis although superimposed infection would be difficult to exclude.
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no radiographic evidence for acute cardiopulmonary process.
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no pneumothorax. no significant interval change in bilateral effusions.
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no acute cardiopulmonary process.
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right lower lobe asymmetric opacity may reflect early bronchopulmonary pneumonia in the appropriate clinical situation.
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this exam is not dedicated for imaging of fractures. irregularity of the left lower lateral rib could be fracture. correlate with focal exam findings. dedicated rib films would be obtained with bb marker in the area of focal pain if clinically indicated.
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no acute cardiopulmonary process.
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opacity in the right lower lobe corresponds to an area felt to represent bronchial mucoid impaction on ct from the same day. no other focal consolidation identified.
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<num>. the endotracheal and nasogastric tube are now in appropriate position. <num>. the left retrocardiac opacity has almost resolved.
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no substantial interval change from the previous chest ct. continued small right pleural effusion with bibasilar atelectasis. paramediastinal radiation fibrosis re- demonstrated.
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left subclavian picc line remains in place. overall cardiac and mediastinal contours are stable. stable calcified granuloma in the right mid lung. there is increasing retrocardiac opacity which could reflect an area of pneumonia or aspiration in the correct clinical setting. no large effusions. no pneumothorax. no pulm...
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<num>. no acute cardiac or pulmonary process. <num>. stable mild cardiomegaly. <num>. unchanged blunting of the right costophrenic angle is either a tiny effusion or scarring.
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pulmonary vascular congestion. no focal consolidation.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12945162/s53046293/51fee91a-b616d944-e03d8913-f17da551-9ea25dd8.jpg
no acute cardiopulmonary abnormality.
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interval extubation. stable bilateral pleural effusions, large on the left and small on the right. possible minimal pulmonary edema.
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no acute cardiopulmonary abnormality.
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<num>. minimal if any residual right lower lung opacity. <num>. resolved pulmonary edema.
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no acute cardiopulmonary abnormality.
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no pneumonia.
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no significant interval change from prior study attention on followup to the right
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no acute cardiopulmonary process.
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pulmonary edema with bilateral pleural effusions. supervening infection cannot be excluded.
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no evidence of acute cardiopulmonary process.
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clear lungs.
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chronic scarring within the right lung base without acute cardiopulmonary process.
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no free air. no acute cardiopulmonary process.
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no evidence of pneumonia.
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<num>. multiple areas of increased opacity, unchanged from prior study, concerning for multifocal pneumonia. <num>. small bilateral pleural effusions.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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there is opacity at the right lung base, which is suspicious for pneumonia.
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no evidence of acute disease.
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no definite acute cardiopulmonary process.
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no change.
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<num>. ng tube in the mid esophagus. <num>. mild elevation of the left hemidiaphragm. results were discussed with dr. <unk> at <time> p.m. on <unk> via telephone by dr. <unk> at the time the findings were discovered.
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no pneumonia or other acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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<num>. mild cardiomegaly without evidence of pulmonary edema. <num>. broken superior-most median sternotomy wire.
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no acute cardiopulmonary abnormality.
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no chest radiographic evidence of acute thoracic disease. recommendation(s): if clinical concern for infection persists, a ct thorax is of higher sensitivity for pneumocystis and may be considered for more complete assessment.
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no acute cardiopulmonary abnormality.
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<num>. doubt significant interval change in position of swan-ganz catheter, with tip overlying the right pulmonary artery. <num>. cardiomegaly, unchanged. <num>. suspect mild chf.
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endotracheal tube tip has been withdrawn and still is slightly low lying, terminating approximately <num> cm from the carina. consider withdrawing the tube by approximately <num> cm.
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interval improvement of right-sided loculated pleural effusion. otherwise, no new acute cardiopulmonary process or acute worsening of tracheal narrowing.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process. no change.
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possible left lower lobe pneumonia with a small left pleural effusion.
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no pleural effusion concerning for hemothorax or evidence of pneumonia or other significant cardiopulmonary abnormalities.
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no acute cardiopulmonary abnormalities copd
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low lung volumes. interval appearance of retrocardiac consolidation with volume loss suggestive of left lower lobe collapse. pneumonia cannot be excluded. no obvious edema. no pneumothorax. right subclavian picc line is unchanged in position. interval removal of the feeding tube. heart remains enlarged. mediastinal con...
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bibasilar opacities, potentially atelectasis noting that infection is not entired excluded.
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no acute cardiopulmonary process.
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no acute interval change in diffuse interstitial lung disease.
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no acute cardiopulmonary abnormality.
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mild improvement in bilateral vascular congestion and right upper lobe opacity. stable retrocardiac opacity. left picc tip in upper svc.
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no acute intrathoracic process.
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<num>. elevation of left hemidiaphragm with minimal associated atelectasis. <num>. no evidence of pneumonia. <num>. mildly distended, fluid-filled stomach.
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mild fluid overload.
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no evidence of acute cardiopulmonary disease.
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probable linear atelectasis at the right lung base. no convincing signs of pneumonia.
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air-fluid in the distal esophagus but no visualization or radiodense foreign body.
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mild pulmonary edema, slightly improved compared to the previous exam.
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if present, tiny left apical pneumothorax has not changed in size. if further evaluation is clinically warranted, obtain a frontal expiration view.
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no convincing signs of pneumonia on this limited portable radiograph.
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findings which may suggest developing interstitial lung disease. if further assessment is desired, then dedicated chest ct could be considered in followup.
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chronic moderate cardiomegaly. no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process. if desired, dedicated rib series can be performed.
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mild increase of left-sided pleural effusion apparently induced by tki administration (does this stand for tyrosine kinase inhibitor?).
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no acute intrathoracic abnormalities identified.
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previously noted moderate pulmonary edema is vastly improved, right moderate partially loculated pleural effusion is decreased, and the left moderate pleural effusion is unchanged since <unk>.
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normal chest radiograph.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary disease.
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no definite focal consolidation.
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<num>. interval development of moderate-to-large left pleural effusion and small right effusion and adjacent compressive atelectasis. <num>. mediastinal and hilar lymphadenopathy is better appreciated on ct.
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pericardiocentesis line in place.
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vague increased retrocardiac opacity which could represent left lower lobe pneumonia. please note this finding was similar when compared to prior and followup will be necessary to ensure resolution.
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no acute intrathoracic process identified. mild cardiomegaly.
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no acute cardiopulmonary abnormality. no evidence of pneumoperitoneum.
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no acute cardiopulmonary abnormality.
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no pneumonia or pulmonary edema. heart size top-normal. mild pulmonary hyperinflation could be due to small airway obstruction or emphysema.
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no evidence of pneumonia.
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resolving right upper lobe process; however, increased right middle lobe process with a greater area of opacification as well as central lucency as well as some new small right pleural effusion. given multiple cavitating lung lesions consider immunosupression possibly from hiv vs valvular abnormality.
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interval development of a large right pleural effusion, possibly malignant.
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moderate cardiomegaly and mild pulmonary vascular congestion without frank pulmonary edema.
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no significant interval change noting persistent left pleural effusion with pleural catheter in place and similar underlying parenchymal changes compatible with underlying lesion with associated parenchymal changes as above.
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no acute cardiopulmonary process.
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unchanged appearance of left dual-chamber pacemaker.
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no acute cardiopulmonary process.
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subtle opacity in the right mid lung may represent early or developing pneumonia in the appropriate clinical setting. no dense consolidation.