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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13976907/s50891685/3cb3e45e-17937df0-2c24d9a9-0f3fc7c7-74babb27.jpg
no definite acute cardiopulmonary process. bilateral lower lobe hazy opacities/ increased interstitial markings are unchanged compared to <unk>, and again suggestive of chronic interstitial lung disease.
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left picc line terminates in the distal svc.
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no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15245864/s56365551/7809bee4-f93c1f9a-dcf55d07-48c4715d-30b6262e.jpg
no acute cardiopulmonary process. tortuous aorta with unchanged appearance of the cardiomediastinal silhouette.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10881703/s52833401/f72b1e7a-78202f15-e172692f-65922bc3-60aa8e02.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16701759/s53266144/7bbbc3ed-f3ce230f-ecbe0d85-0f9149aa-6b1d6966.jpg
no evidence of acute cardiopulmonary process. scattered areas of atelectasis.
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limited exam with low lung volumes causing crowding of the bronchovascular structures. retrocardiac opacity could reflect aspiration or pneumonia.
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dh tube in situ with the tip in the stomach.
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enteric tube tip terminates at the anastomosis and has been intentionally placed at this site in this post esophagectomy patient. postsurgical changes in the right hemi thorax in the form of widening of upper mediastinum, right upper and lower lobe linear atelectasis and a moderate right pleural effusion persist. there...
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18040392/s57063348/f1075cfe-223305bf-90f39b95-05b447e2-f0b2faca.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14614404/s54938678/8b9edc39-1807a7ad-462b7fb2-07ff31bd-74aaed19.jpg
worsened left lower lobe atelectasis, delayed contusion, or early pneumonia. close follow up advised. small left apical pneumothorax appears newly apparent. unchanged left rib fractures, again identified.
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no evidence of injury.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13880916/s59469360/6734422f-c3e84ec8-1af0cb63-f360c146-1fca2c1e.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10055694/s54669448/a58cb9a4-1b0f64b3-1a719c69-4d2e7fce-3f2b4b38.jpg
increased cardiomegaly with signs of volume overload. opacities in the bilateral mid lungs could represent atelectasis or edema; however, superimposed infection is possible. telephone notification to dr. <unk> by dr <unk> at <time> on <unk>, <num> min after review
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no acute cardiopulmonary process.
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<num>. wide mediastinum with deviation of the trachea is suggestive of a possible mediastinal mass. if clinically indicated, further evaluation with a chest ct could further characterize the mediastinal abnormalities. <num>. mild pulmonary edema with probable small bilateral pleural effusions. <num>. probable severe ca...
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no focal consolidation.
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<num>. new right ij central venous catheter with distal tip overlying the low svc. no pneumothorax. <num>. stable appearance of the heart and lungs, including moderate cardiomegaly. no focal consolidation.
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vague opacity in the left mid lung could represent pneumonia. followup to resolution. hyperinflated lungs again noted.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14036905/s53928153/03e5fe62-122ba088-6f1d9c24-c9c1fc19-95472e74.jpg
no focal infiltrate
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no acute process.
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large right pleural effusion with overlying atelectasis, underlying consolidation not excluded.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19930086/s54040122/5b040c65-890727ca-264825ad-f8dd433a-5ef47fd5.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14412499/s51003733/f9006596-ac19bace-53f32938-fee3088c-8fcff906.jpg
no acute intrathoracic process.
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unchanged left upper lobe cavitary lesion corresponding to the known malignancy. other previously demonstrated nodules within the lungs on the pet-ct are not as well visualized on the current radiograph.
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small right and moderate left pleural effusions.
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findings compatible with pulmonary edema with bilateral effusions and compressive lower lobe atelectasis.
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increased now moderate pulmonary edema with new small bilateral pleural effusions and unchanged retrocardiac atelectasis.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13670707/s54970496/92ac16e7-4b212467-5abf3bf8-8ad110d5-5732dcd0.jpg
normal chest.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11300822/s53881153/a162c028-97d792fa-cad2c315-f9acbded-de8cc738.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15230574/s50498444/49311d73-32873c43-1266f87e-daaa3e54-d84bf638.jpg
stable chest in a patient with acute trauma including several rib fractures in right apical area and shoulder area.
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bibasilar atelectasis.
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no acute cardiopulmonary process. no focal consolidation.
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no evidence of acute disease or free air.
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no acute cardiopulmonary abnormality. normal, right mastectomy, chronic mild cardiomeagly
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no acute pulmonary process identified. no rib fracture identified on these lung technique films.
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findings suggestive of right mid lung scarring. increased interstitial markings particularly at the bases, potentially due to chronic lung disease; however, component of active inflammation is also possible.
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no acute cardiopulmonary abnormalities
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<num>. tip of et tube terminating <num> cm above the carina. <num>. satisfactory position of the enteric tube within the gastric body. <num>. bibasilar opacities which may represent atelectasis, however developing consolidation/aspiration cannot be excluded.
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no acute intrathoracic process
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left upper lobe pneumonia. recommend repeat after treatment to document resolution.
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<num>. pulmonary edema with stable cardiomegaly. <num>. prominent pulmonary artery likely related to pulmonary hypertension <num>. stable asbestos-related pleural disease and blunting of the left costophrenic angle likely due to pleural thickening.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11224762/s51529340/59713348-2209fd4c-f78a46ad-da5940c5-4032b136.jpg
increased bibasal airspace opacity, particular in the left lower lobe. appearances are suspicious for infection or aspiration.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16358341/s51741279/a2ad14da-7230079f-18e52492-423450d6-167c3acc.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17517983/s57072116/78c8f7e3-b66a6ef4-433c0b68-0840344d-9da39c7a.jpg
increased mild pulmonary edema, most prominent lung bases, right greater than left.
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increasing left lower lobe effusion and overlying atelectasis may be part of post lobectomy changes in an asymptomatic patient. superimposed infection cannot be excluded in a symptomatic patient.
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no acute intrathoracic process.
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<num>. interval ng tube placement, the tip of which is in the proximal stomach, this could be advanced several centimeters to ensure that the sidehole is within the stomach. <num>. unchanged left pleural effusion and left pleural thickening with adjacent rib fractures and subcutaneous emphysema. there is no pneumothora...
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no acute intrathoracic process with near complete resolution of bibasilar opacities previously described.
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stable appearance of the chest.
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no acute cardiopulmonary process.
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<num>. no radiographic evidence of pneumonia or acute heart failure. <num>. unchanged mild cardiomegaly. comment: findings were telephoned to dr. <unk> by dr. <unk> at <unk> on <unk>, <num> minutes after the time of discovery.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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normal chest radiograph.
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standard views pa and lateral chest x-ray do not disclose any new pulmonary or cardiovascular abnormality. as there is no evidence of pneumothorax, new pleural effusions or parenchymal abnormalities, the identification of local rib injuries could be performed by identifying the area of distinct local discomfort and to ...
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<num>. tiny left apical pneumothorax following chest tube removal. <num>. unchanged left upper lobe opacity and lower lobe atelectasis with a small pleural effusion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19907026/s50360470/409188d3-70ea0dbd-f89fa781-8e027cb8-73c7ade2.jpg
severe cardiomegaly with mild pulmonary vascular congestion.
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trace pleural effusions. top-normal heart size.
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right pectoral transvenous pacer with stranding in the right atrium and right ventricle. no pneumothorax. mild pulmonary edema and small right pleural effusion improved.
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chronic interstitial changes with no evidence of pneumonia.
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<num>. unchanged chest radiograph. <num>. the tip of the endotracheal tube is <num> cm above the carina. these findings were communicated to the covering team at approximately <unk> on <unk>, at which time the patient had already been extubated.
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no evidence of acute cardiopulmonary process.
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stable mild pulmonary edema and slight interval increase in the small right pleural effusion. bibasilar opacities are most consistent with atelectasis or infection in the correct clinical setting.
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normal chest radiograph.
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<num>. multiple vertebral body compression fractures are of indeterminate age. <num>. no rib fractures identified. if there are focal areas of pain, dedicated views of those areas are recommended.
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no acute cardiopulmonary process.
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no radiographic evidence of consolidation or pulmonary edema.
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resolved pulmonary edema. no evidence of pneumonia.
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low lung volumes and mild pulmonary edema. cardiomegaly. there appears to be blunting of the posterior left costophrenic angle which may be due to a pleural effusion.
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no acute cardiopulmonary abnormality. no displaced fractures noted.
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large improvement of lung ventilation with markedly reduced pulmonary edema and consolidation. heart size is still slightly enlarged.
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no acute cardiopulmonary process.
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bibasilar atelectasis and small bilateral pleural effusions are stable since the prior study.
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status post right thoracentesis, with slight decrease in right hemi thorax opacification, however, the majority of the right hemi thorax remains opacified.
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interval placement of left anterior pigtail catheter. prior seen left pneumothorax persistent though decreased in size. no pleural effusion.
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no acute cardiopulmonary abnormality.
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small right apical pneumothorax new since <unk>.
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no evidence of acute cardiopulmonary disease.
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tiny residual right pleural effusion.
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faint bibasilar opacities are noted, greater on the right than the left. pneumonia must be excluded in the proper clinical setting.
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right pleural catheter in good position with near complete resolution of right-sided effusion. mild pulmonary vascular congestion.
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no evidence of acute cardiopulmonary disease.
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low lung volumes with mild cardiomegaly and mild central pulmonary vascular congestion.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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<num>. no radiographic evidence for acute cardiopulmonary process. <num>. chronic moderate cardiomegaly.
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no focal consolidation concerning for pneumonia. patient is post right upper lobectomy. unchanged findings of pulmonary arterial hypertension.
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evidence of chronic lung disease with more confluent regions of consolidation in the mid lung zones bilaterally, which have not significantly changed since <unk>.
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possible lower lobe opacity best seen on the lateral view could be due to atelectasis. if there is continued concern for pulmonary nodules or metastatic lesions, chest ct is more sensitive.
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no acute cardiopulmonary process.
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no definite acute cardiopulmonary process. no visualized displaced fracture identified. if desired dedicated rib series can be performed.
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<num>. slight interval worsening of vascular congestion and mild pulmonary edema. additionally, the heart appears slightly larger. <num>. high position of the endotracheal tube, <num> cm from the carina. advance <num>-<num> cm for more secure placement.
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no evidence of acute cardiopulmonary disease.
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multifocal pneumonia given the distribution could be viral or atypical
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new left lower lobe infiltrate
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no acute cardiopulmonary process