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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12021305/s54993955/5f2a8cb3-fba96216-a17fcb7e-36cc8113-af677bbc.jpg
interval development of small right pleural effusion with right basilar opacity, likely atelectasis. severe emphysema.
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<num>. cardiac silhouette appears to be enlarged compared to the prior study. <num>. small bilateral pleural effusions. <num>. a <num>-mm pulmonary nodule noted projecting over the second anterior rib on the right.
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no acute cardiopulmonary process.
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patchy streaky right basilar opacities, suggestive of atelectasis although not entirely specific.
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right lung opacity is improved compared to <unk>, consistent with resolving pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12317110/s50802870/f5e83083-08984c79-c9caae53-5ba03487-b022cb01.jpg
<num>. left ij central venous catheter terminates at the superior cavoatrial junction. <num>. interval improvement of left basilar atelectasis as well as bilateral perihilar opacities which reflects a combination of pulmonary edema and resolving atelectasis.
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no acute intrathoracic abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13987926/s50001064/ba8dd57c-0f556403-b9de02a9-26e605db-fd582102.jpg
similar posterior elevation of the left hemidiaphragm which may reflect eventration or hernia. no evidence of acute cardiopulmonary disease including no evidence for pulmonary edema identified.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12407889/s59684253/9472be8b-24972089-ed8b507a-0a1368c3-1dbb23fc.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15740609/s53047419/ea18cda2-29b44952-51fb0b07-43bca0ac-a9640bc4.jpg
severe emphysema with chronic interstitial abnormality. more focal opacity in the periphery of the left lung base likely reflects the known lesion seen on prior chest ct.
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no evidence of acute disease.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17470752/s54876287/b8b62f53-f50cddbd-e14e2f75-f58b3725-3ec396c4.jpg
right picc terminates in the distal svc. left-sided moderate to large pleural effusion is stable to minimally increased in size from earlier today.
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<num>. no evidence of acute cardiopulmonary process. <num>. slight improvement in bibasilar airspace opacities. <num>. reticular opacities compatible with chronic interstitial lung disease.
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decreased right middle lobe opacification but mild interstitial process which is of uncertain chronicity.
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no acute cardiopulmonary abnormality.
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persisting bilateral pleural effusions with overlying atelectasis.
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consolidation at the right base may represent a developing pneumonia or atelectasis. there is likely an associated small right pleural effusion.
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<num>. low lung volumes with slight interval improvement a left lower lobe atelectasis. <num>. minimal edema and pulmonary vascular congestion, improved.
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no evidence of pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14276803/s55111403/de36817b-ddad9d90-93b0dde7-7d2a7084-959bfaf1.jpg
no evidence of acute cardiopulmonary disease. possibility of anterior nodule which can be assessed with chest ct if needed clinically.
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mild vascular congestion.
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no acute cardiopulmonary process. limited exam demonstrating possible irregularity at the lateral left seventh rib. dedicated rib series is suggested.
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<num>. large left pleural effusion is stable since <unk>. <num>. multiple air-fluid levels in the partially evaluated small bowel are concerning for small bowel obstruction. if clinical concern, consider upright abdominal radiograph for further evaluation. <num>. stable multiple anterior wedge compression fractures of ...
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19613723/s50689139/6755a893-c3c84739-2fa81fee-84015a15-06a27fb6.jpg
no evidence of pneumonia or other acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18084077/s50839169/bef7b866-f7cc1436-2b200d0f-08fb4093-8d71457d.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11419565/s59063454/a8d43515-747d5d43-e18da561-0e33f3d3-ad6b8be5.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19990563/s50827730/3721f3e6-961cd46e-043b1950-34674cd6-2765f0be.jpg
short interval stability of bibasilar opacities, which likely represent atelectasis, however aspiration or pneumonia cannot be excluded.
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mild interval increase in pulmonary edema. no pneumothorax.
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no radiographic evidence of pneumonia.
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stable appearance of the chest with no pulmonary edema.
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left lower lobe opacity, suspicious for pneumonia in the appropriate clinical setting. no pneumothorax.
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low lung volumes with retrocardiac opacity likely atelectasis but to be correlated clinically.
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no definite pneumonia seen.
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no radiographic evidence of acute, displaced rib fracture.
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persistent normal chest findings. no evidence of chf or acute pulmonary infiltrates in this <unk>-year-old male patient with fever, cough and nasal congestion.
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no acute intrathoracic process.
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<num>. unchanged top normal heart size. <num>. no acute cardiopulmonary process.
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interval resolution of pulmonary edema with persistent small left pleural effusion.
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no evidence of pneumonia.
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<num>. no evidence of recent or non-recent tb. <num>. dense rounded opacity in the left lower lobe. recommend shallow oblique views for further evaluation. these findings were entered into the critical results dashboard by dr. <unk> at <time>pm.
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mild to large bilateral, right greater than left pleural effusions. degree of pulmonary edema may have slightly improved since prior exam although detailed evaluation is limited.
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no acute cardiopulmonary process.
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new small left pleural effusion with probable subpulmonic component. no other relevant change since recent study.
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no acute cardiopulmonary process.
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worsening bibasilar opacities, possibly due to recurrent aspiration or developing aspiration pneumonia co-existing with linear atelectasis.
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no acute cardiopulmonary process. specifically, no evidence for pneumonia.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary abnormality.
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<num>. no evidence of acute cardiopulmonary process. <num>. stable right apical parenchymal changes related to known history of tb.
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stable cardiomegaly. pulmonary vascular congestion and mild pulmonary edema. small bilateral pleural effusions.
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<num>. cardiomediastinal silhouette is grossly unchanged. <num>. continued chf with interstitial edema, though there is evidence of slight improvement. <num>. interval improvement in left lower lobe collapse and/or consolidation.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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decreased small left pleural effusion. rounded opacity in the left mid lung may be due to infection or known pleural metastases.
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no acute cardiopulmonary process or evidence of pneumonia.
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increased interstitial markings with some areas that appear nodular. it is uncertain if this is due to vascular crowding and overlying osseous metastases, or given patient's history, the possibility of tiny pulmonary nodules from metastases. consider ct scan to further evaluate if desired. diffuse osseous mets re-demon...
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increased interstitial markings may be due to a interstitial edema or atypical infection. no focal consolidation.
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no evidence of acute injury. bilateral hazy interstitial opacities are likely representative of fibrotic changes.
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no acute cardiopulmonary abnormality.
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mild pulmonary edema and right middle lobe pneumonia.
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no active disease.
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minor left basilar atelectasis without definite focal consolidation.
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no acute intrathoracic process.
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streaky opacities the left lung base are likely atelectasis, however, infection is not excluded.
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right basilar opacity, possibly atelectasis, with small right pleural effusion, though pneumonia cannot be excluded.
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no acute cardiopulmonary abnormality.
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<num>. slightly increased prominence of opacities in the right lower lung base can be concerning for pneumonia in the right clinical setting. right moderate bibasilar pleural effusion and adjacent atelectasis is again noted. <num>. left basilar opacity is not well seen on this exam, likely atelectasis.
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changes compatible with chronic interstitial lung disease without superimposed pneumonia or chf.
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anterior lung base opacities likely represent prominent fat pad though difficult to exclude pneumonia in the correct clinical setting. <unk> consider a repeat pa radiograph with more optimized inspiratory effort to further assess.
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no acute intrathoracic process.
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right-sided pleural scarring and blunting of right costophrenic sulcus, but no pleural effusion.
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improved pulmonary edema. stable pulmonary vascular congestion and pleural effusions.
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question small early infiltrate in the right lower lobe.
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right basilar calcified pleural plaques are again identified. known anterolateral right <unk> through <unk> fractures and posterior right <num>th rib fractures are not clearly delineated on this study.
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mild chf.
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no acute intrathoracic process.
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no evidence of acute disease.
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no acute intrathoracic process.
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no evidence of pneumonia.
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<num>. edema. <num>. right lower lobe opacity concerning for infection.
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no acute cardiopulmonary process.
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the intra-aortic balloon pump remains in place with its tip <num> cm below the top of the aortic knob. overall cardiac and mediastinal contours are unchanged. lungs appear well inflated without evidence of focal airspace consolidation, pulmonary edema, pleural effusions or pneumothorax.
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<num>. mild interval increase in pulmonary vascular congestion and new small left pleural effusion. <num>. long standing l<num> vertebral wedge compression.
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no acute cardiopulmonary process.
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no radiographic evidence of pneumonia or other acute cardiopulmonary abnormalities. mild left atrial enlargement.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16926132/s58416683/7a0f90c8-d424d7f8-a6d3958b-cc51eb83-d9d3d086.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10366982/s51249749/f3ddb1c0-d8710d8d-84d69451-842b01ee-44ef1d50.jpg
no acute process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14096942/s52413637/ba903cce-20990878-da35ae59-7138fbce-ba9669bb.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16129499/s53067300/37908544-79704080-34b8906d-b05d93ca-b7db247d.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18063854/s59798157/605b5d10-b65fda21-7b125be6-d6bf7f67-57d03e6d.jpg
no acute intrathoracic abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19678952/s50113430/bc8efd03-b8a22a39-86ef395c-04a412ca-77f58a78.jpg
vascular plethora likely due to fluid overload
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10438089/s53873715/c5e0a2e8-7ff8d06f-171144c4-8f546603-ce3a7583.jpg
marked worsening of the left lung. aeration due to a combination of volume loss and effusion
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17760788/s52923686/6ab1b095-a142ac61-748aa07e-ef1ec8c6-52b6cbe1.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11644052/s55810654/501dd828-a63ddbe9-8e7b7d5b-96e6fdc8-c0cbee82.jpg
normal chest.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13213620/s52978806/a0efe844-422613dc-40604cf7-e5ce977c-803b7f50.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17037764/s58119352/0458c78f-1f9c3d35-5d2fde46-e6d91043-3dafeb68.jpg
nasogastric tube tip not well seen, but presumed to be at the ge junction as side port is evident in the mid-to-distal esophagus. minimal pulmonary edema.