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<num>. pleural-based opacities projecting over the upper lungs are atypical for pneumonia and concerning for malignancy. please correlate clinically and recommend chest ct to further assess. <unk> d/w dr. <unk>. <num>. mild interstitial edema.
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hyperinflated lungs likely related to asthma.
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in comparison to <unk>, increased hazy opacification of the right base could represent a layering effusion, but an underlying consolidation cannot be excluded.
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normal chest.
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no pneumonia. recommendation(s): no pneumonia.
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old fractures of the right third, fourth, fifth and eighth ribs. no displaced left rib fractures. no pneumothorax.
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prominent interstitial opacities which appear worse from <unk> are thought to reflect a path to severe fibrosis, however, this could partially be explained by an acute reaction from recent drug use. no convincing evidence for volume overload.
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<num>. right lower lobe pneumonia. <num>. interval improvement of interstitial pulmonary edema.
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left lower lobe atelectasis. otherwise, no acute cardiopulmonary abnormality.
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<num>. mild left basal atelectasis; no pneumonia. <num>. chronic mild to moderate cardiomegaly and pulmonary venous hypertension, but no pulmonary edema. <num>. chronically enlarged atherosclerotic thoracic aorta, with no focal aneurysm.
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increasing bilateral pleural effusions, moderate in size.
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no acute cardiopulmonary process.
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moderate left pleural effusion is unchanged. right lung is grossly clear.
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low lung volumes and bibasilar atelectasis persist, along with mild cardiomegaly. no pneumothorax or new consolidation to suggest pneumonia.
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no acute intrathoracic process.
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left basilar opacity again noted potentially atelectasis noting that the infection is not entirely excluded.
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no evidence of acute disease.
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<num>. no acute cardiothoracic process. <num>. superior endplate depression fracture of a mid thoracic vertebral body, of unknown chronicity.
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no acute intrathoracic abnormality.
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no pneumonia.
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no acute intrathoracic process. mild cardiomegaly, small hiatal hernia, right diaphragmatic eventration.
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left pleural effusion and worsening left basilar consolidation, the latter concerning for pneumonia.
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mild prominence the cardiac silhouette is unchanged compared with <unk> and could be accentuated due to low inspiratory volumes and lordotic positioning. minimal atelectasis in the right cardiophrenic region is also relate to low lung volumes. allowing for this, no focal infiltrate or consolidation.
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no acute cardiopulmonary abnormality. please note that previously noted pulmonary nodules on chest ct are not well assessed on the current radiograph.
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dobhoff ends along the greater curvature of the stomach.
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no acute intrathoracic process. no rib fractures identified.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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lower lung volumes without definite superimposed consolidation.
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status post removal left chest tube without significant change in the left apical pneumothorax or effusion.
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no acute cardiopulmonary abnormality. nodule previously identified on ct is not evident on radiography.
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no evidence of injury.
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enteric tube side port likely proximal to the ge junction and should be advanced for optimal positioning. bilateral parenchymal opacities, most conspicuous at the right lung apex suspicious for multifocal pneumonia with small bilateral pleural effusions.
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no evidence of acute cardiopulmonary process.
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<num>. interval increase in opacity projecting over the left hemithorax, which may indicate worsening pneumonia, loculated effusion, or parenchymal or pleural hemorrhage. <num>. interval worsening in the right lower lung opacification, which may reflect additional area of consolidation or atelectasis. findings were com...
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low lung volumes. central pulmonary vascular engorgement. the right base opacity could relate to prominence of vasculature given low lung volumes, although underlying consolidation cannot be excluded. areas of atelectasis.
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low lung volumes with increasing bibasilar linear atelectasis and likely mild pulmonary edema.
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no evidence of sarcoid or infection. atherosclerotic disease within the coronary artery bypass graft vessels.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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nasogastric tube tip within the stomach. no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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stable rib lesions.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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et tube in appropriate position. no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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acute cardiopulmonary process.
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no acute cardiopulmonary abnormalities. specifically, no evidence of pneumothorax. these findings were discussed with dr. <unk> at <time> p.m. by dr. <unk> <unk> by telephone on the day of the exam.
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<num>. et tube <num> cm from the carina. enteric tube terminates in the stomach. <num>. large bilateral pleural effusions. lateral aspect of right hemi thorax omitted from view.
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trace bilateral pleural effusions and possible minimal interstitial edema.
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<num>. large hiatal hernia, partially fluid-filled. of note patient is at increased risk for aspiration. <num>. hypoinflated lungs with bibasilar atelectasis and small stable bilateral pleural effusions. <num>. persistent retrocardiac and left lower lobe opacity is most consistent with atelectasis however differential ...
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no acute cardiopulmonary process.
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<num>. right-sided port-a-cath tip in the mid svc. no pneumothorax. <num>. moderate size hiatal hernia.
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no acute cardiopulmonary process.
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no acute radiographic intrathoracic pulmonary disease.
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no acute cardiopulmonary process.
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<num>. rounded hazy opacity in the left mid lung field, concerning for pneumonia. <num>. left lateral bulge along the descending thoracic aortic contour at the level of the aortic hiatus, of uncertain etiology. this could be further assessed with ct or mri on a nonemergent basis.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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no acute pneumonia. increasing mild pulmonary edema with small bilateral pleural effusions.
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<num>. cardiomegaly with worsening pulmonary edema. <num>. subtle left suprahilar nodular opacity may represent a focal area of edema; repeat imaging after diuresis may be considered.
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improving left lower lobe opacity. considering rapid development and rapid improvement, aspiration is a likely possible cause. however, followup radiographs with pa and lateral technique would be helpful as well as clinical correlation to exclude an infectious pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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<num>. no acute cardiac or pulmonary findings. <num>. no definite rib fracture identified. if there is continued clinical concern for a rib fracture, further evaluation could be performed with a dedicated rib series, including an appropriately positioned radiopaque skin marker.
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no acute cardiopulmonary process.
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right lung basilar opacity is likely atelectasis, although pneumonia can not be excluded.
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blunting of the right costophrenic angle, representing either pleural thickening or small pleural effusion.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process. no radiopaque stent seen.
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<num>. increased air component of moderate left hydropneumothorax. <num>. unchanged small right pleural effusion. <num>. unchanged dense left lower lung atelectasis. pertinent findings were discussed with dr. <unk> by dr. <unk> at <time> a.m. via telephone on the day of the study, five minutes after discovery.
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high in position endotracheal tube terminating approximately <num> cm above the level of the carina was discussed with dr. <unk> at <time> a.m. on <unk> via telephone approximately <num> minutes after discovery.
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no acute cardiopulmonary process.
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removal of chest tubes with no pneumothorax. otherwise, no significant interval change.
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low lung volumes without definite acute cardiopulmonary process.
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no acute cardiopulmonary process.
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normal chest radiograph.
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cardiomegaly with mild interstitial edema and small right pleural effusion.
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new bibasilar airspace opacities are likely due to aspiration or infection.
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multifocal consolidations probably pneumonia and nodular opacities that could represent solid nodules . when clinically appropriate, ct chest is recommended. new superimposed mild pulmonary edema
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<num>. new minimal disruption of third sternotomy wire. clinical correlation recommended to assess for superimposed infection. <num>. no additional acute cardiopulmonary process.
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<num>. bilateral lower lung hazy opacities possibly combination of pleural effusion and consolidation. infection cannot be excluded. <num>. et tube terminating <num> cm from the carina. <num>. ng tube tip below the diaphragm but beyond the visualized field.
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right lower lung opacity likely represents pleural effusion and atelectasis, but supervening infection cannot be excluded.
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small right pleural effusion
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no acute cardiopulmonary process.
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increase in the extent of involvement of the left upper lobe consolidation.
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findings suggesting mild vascular congestion.
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no evidence of acute fracture. if sternal symptoms persist, recommendation is made to further evaluate with ct.
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calcified pleural plaques compatible with prior asbestos exposure. no acute cardiopulmonary abnormality otherwise visualized.
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marked interval improvement in the previously described left basal opacity and in the right perihilar and lower lobe opacities.
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clear lungs.
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<num>. moderate vascular congestion, increased since <unk>. <num>. unchanged bilateral pleural effusions, right worse than left.
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no acute intrathoracic process.
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<num>. relative increased density of the left hemithorax as compared to the right likely reflects overlying soft tissue in the setting of patient rotation. <num>. mild pulmonary edema.
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no focal consolidation to suggest pneumonia.