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no acute cardiopulmonary abnormalities ovoid opacity in the right mid lung associated with adjacent pleural abnormalities is stable, of unclear etiology, ct again is recommended for further evaluation.
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no pneumothorax. significant decrease in right pleural effusion with similar appearance of the left pleural effusion. residual linear atelectasis of the right mid lung is noted.
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persistent bilateral pleural effusions.
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findings suggests mild vascular congestion.
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limited, negative for acute intrathoracic process.
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postoperative change with right chest tube. there is some residual lateral pleural fluid. small right pneumothorax is present. there is considerable atelectasis or consolidation in the right base. patchy density left base
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<num>. no focal consolidations concerning for pneumonia or pleural effusions. <num>. unchanged rightward tracheal deviation, likely due to enlarged thyroid. <num>. unchanged mixed sclerotic and lytic bone lesions, consistent with history of metastatic breast cancer.
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hyperinflated lungs, no signs of pneumonia.
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large pleural effusions with worsening left perihilar consolidation, likely due to worsening pneumonia. questionable small pneumothorax at the left apex. these findings were discussed with <unk> by dr. <unk> via telephone on <unk> at <time>, time of discovery.
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unchanged chest examination with no evidence of pneumonia.
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no acute cardiopulmonary process. abnormal contour in the region of the ap window which appears in continuity with the descending thoracic aorta. dedicated chest ct is suggested to further characterize.
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<num>. no evidence of pneumonia. <num>. slight interval increase in the cardiac size, which is now at the upper limits of normal.
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left costophrenic angle not fully included on the frontal image. otherwise, no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormalities
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low lung volumes with bibasilar atelectasis. no focal consolidation concerning for pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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unchanged enlargement of the pulmonary arteries bilaterally consistent with pulmonary hypertension. no acute cardiopulmonary process.
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no radiographic evidence of acute cardiopulmonary disease. a left suprahilar opacity is slightly more conspicuous when compared to the prior radiograph. recommendation(s):
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relatively low lung volumes without acute cardiopulmonary process seen.
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no significant interval change since recent exam. cardiomegaly and mild pulmonary vascular congestion with small effusions.
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<num>. an apparent right picc line ends in the right axilla. <num>. numerous rounded opacities throughout both lung fields are consistent with septic pulmonary emboli, better evaluated on ct <unk>. <num>. small, right pleural effusion and associated mild basilar atelectatic changes.
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lung volumes remain low. linear opacity at the left base likely reflects subsegmental atelectasis or scarring. no evidence of pulmonary edema. overall cardiac and mediastinal contours are likely unchanged. right picc line has its tip in the right atrium. pull-back of approximately <num>-<num> cm to position the tip in ...
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compared to the preoperative film the left lower lobe is worse
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no rib fracture is identified. if there are focal areas of pain dedicated views of those areas are recommended.
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no evidence of acute cardiopulmonary disease.
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no acute cardiopulmonary process or rib fractures.
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<num>. no evidence of acute disease. <num>. no evidence for free air. <num>. nodular focus projecting over the left lung apex, a possible lung nodule. when clinically appropriate, evaluation with chest ct is recommended. <num>. hyperinflation and suspected emphysema. <num>. suspected bony demineralization and mild comp...
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no acute cardiopulmonary process.
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no significant interval change. mild interstitial edema and cardiomegaly. no evidence of consolidation.
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no significant interval change of left-sided pleural effusion with adjacent atelectasis.
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<num>. left apical pneumothorax is no longer identified. <num>. stable small left pleural effusion.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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bibasilar opacities, improved on the right and slightly more pronounced on the left. small bilateral effusions, improved on the right. suspect mild cardiomegaly, unchanged.
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improved right pneumothorax which is now small. resolved right pleural effusion.
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<num>. small right-sided pleural effusion. <num>. no parenchymal opacities concerning for pneumonia. a well-defined bandlike sclerotic density projecting over the left anterior <num>nd rib is not clearly seen in the lateral view and is of unclear significance. this is felt to represent a chest wall or bony abnormality....
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no evidence of acute cardiopulmonary disease.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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relatively low lung volumes. increased prominence of the right lower paratracheal and hilar soft tissue could be due to lymphadenopathy versus prominent vasculature or mediastinal/ hilar fat. this can be further assessed on nonurgent chest ct.
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limited, negative.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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stable mediastinal prominence compatible with known left paramediastinal mass. emphysema. no acute injury is seen. please refer to subsequent ct chest for additional details.
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new pulmonary vascular congestion and mild pulmonary edema, with new small bilateral pleural effusions.
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pneumonia in the anterior portion of the right upper lung. recommend treatment and followup radiographs in <unk> weeks to document resolution. these findings were communicated to <unk> by telephone <unk> min after discovery by dr. <unk>.
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mild engorgement of the pulmonary vasculature. low lung volumes.
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right lower lobe pneumonia. moderate cardiomegaly.
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bibasilar opacities favoring atelectasis, although pneumonia or aspiration cannot be entirely excluded.
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no acute cardiopulmonary process.
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no evidence of acute disease.
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no acute intrathoracic process. please refer to subsequent cta for further details.
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persistently low lung volumes with patchy opacities in lung bases, likely atelectasis, though infection is not excluded in the correct clinical setting.
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right middle lobe opacity has partially improved. an additional followup chest radiograph in four weeks is recommended to document complete resolution.
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no evidence of acute disease.
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a new <unk> pacemaker lead extends through the coronary sinus and terminates in the position of the obtuse marginal coronary vein with unchanged position of other <num> leads. no evidence of complication, particularly no pneumothorax.
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small bilateral pleural effusions, which are new since <unk>.
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bilateral opacities occupying nearly the totality of the lungs consistent with acute pulmonary edema. diffuse reactive inflammatory process such as pneumonitis or infection cannot be excluded.
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no focal consolidation to suggest pneumonia is seen.
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no acute cardiopulmonary process.
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persistently low lung volumes. no acute cardiopulmonary process. hrct is better for evaluation of interstitial lung disease, if suspected.
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no acute cardiopulmonary process. no radiopaque foreign body.
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bilateral pleural effusions, slightly increasing and suggestive of chf. left-sided retrocardiac atelectasis persists and possibility of infective course is likely. no other interval changes are seen.
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no significant interval change. there is atelectasis at the lung bases, small right-sided pleural effusion prominence of the pulmonary interstitial markings, and emphysematous changes, which are all stable.
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<num>. no pneumothorax. <num>. interval improvement in pulmonary vascular congestion and left lower lobe atelectasis. <num>. persistent small left pulmonary effusion.
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no acute findings in the chest.
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no acute cardiopulmonary process.
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no evidence of acute disease.
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no signs of pneumonia or other acute intrathoracic process.
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patchy opacity adjacent to the left heart border raises concern for lingular consolidation, pneumonia.
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findings concerning for free intraperitoneal air.
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no radiographic evidence of hilar lymph node enlargement to suggest sarcoidosis.
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low lung volumes with probable mild bibasilar atelectasis.
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no acute cardiopulmonary process.
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stable atelectasis or scarring at the lung bases. no evidence of pneumonia.
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normal chest x-ray.
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no evidence of pneumonia. calcified pleural plaque in the left upper hemithorax.
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no acute cardiopulmonary process.
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pulmonary edema with cardiomegaly.
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vague opacity in the left lower lung could represent atelectasis versus pneumonia.
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overall cardiac and mediastinal contours are stable given lordotic technique. lungs appear somewhat low in volume but no focal airspace consolidation, pulmonary edema or pneumothorax is appreciated. no large effusions.
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no acute cardiopulmonary process.
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no acute pulmonary process. mild unfolding of the aorta, which can be seen with aortic stenosis and/or hypertension.
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interval increase in size in the right upper lobe fluid collection.
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cardiomegaly unchanged with congestion and mild edema.
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moderate right pleural effusion may be smaller.
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no radiographic evidence for acute cardiopulmonary process.
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<num>. coiled dobbhoff with tip in the body of the stomach. left picc line now terminates in the proximal atria. <num>. worsening perihilar edema, right greater than left. the findings were communicated with dr. <unk> by dr.<unk> <unk> telephone at time of observation at <time> on <unk>.
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slowly progressive hilar adenopathy and interstitial lung abnormality should be evaluated with chest ct.
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no acute intrathoracic process.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary abnormality. dr. <unk> <unk> these results with dr. <unk> <unk> telephone on <unk> at <time> pm.
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no evidence of acute disease.
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no acute cardiopulmonary abnormality. no displaced rib fracture.
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no evidence of acute disease.
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diffuse bilateral, right much greater than left alveolar opacities could relate to asymmetric edema and/or infection. correlate clinically and consider repeat after diuresis. comparison with prior radiographs would be helpful.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process. please refer to subsequent cta chest for further details.