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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15787595/s59885643/1ced65d0-b5b4ccfb-a6ba3498-81131bfe-703f3ffa.jpg
no radiographic evidence for acute cardiopulmonary process.
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no acute intrathoracic abnormalities identified.
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chronic interstitial lung disease, but no definite evidence of pneumonia.
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pulmonary vascular engorgement with possible trace left pleural effusion. top-normal to mildly enlarged cardiac silhouette size.
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<num>. overinflated lungs and bronchial wall thickening, findings which could be due to chronic bronchitis in the setting of a history of smoking and chronic cough. <num>. no acute pulmonary abnormality.
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no acute cardiopulmonary abnormality.
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scattered areas of mild linear atelectasis. no evidence of free air beneath the diaphragms.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12531206/s52114949/a406a6e9-5a2f01b1-9999c6ab-03e1e052-091b235e.jpg
fluid overload with mild cardiomegaly, small bilateral pleural effusions and mild pulmonary edema. atelectasis or infection in the right lower lobe is improving.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17318449/s57272372/499bb691-a870a1f6-04eb8660-8523e964-df8bb1fb.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17305008/s56130839/5e1e3f17-898a21c1-0f443ff2-45fbf286-ca10f61a.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13650860/s57072908/569b2990-20083d61-c6ae53fc-76d3ebb4-41a22be1.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19939665/s55485502/74efe8df-83fa61ea-2989773b-4ddc7730-0fb1a4db.jpg
there is no pulmonary edema.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14982245/s53539422/7adb0882-1c8455df-282ad1dc-770f59f1-c82c9feb.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18949819/s51716589/d8021617-13fabaf4-5f257817-978da39d-e59361bc.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11551927/s55844868/39478940-45a5a6b4-34dcae2c-4e326acd-326d920c.jpg
persistent collapsed right lower lobe and probably pneumonic consolidation in the left upper lobe
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16425412/s51789277/cf62d15d-967f0335-0526234e-9da4b5f4-8bfd1caf.jpg
opacities in the lower lobes consistent with pneumonia. tiny right pleural effusion.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18458646/s56670161/97e46fea-2f057126-00a4829d-33db438c-6dce78a6.jpg
no radiographic evidence of an acute cardiopulmonary process. findings were discussed with <unk> by <unk> via telephone on <unk> at <time> pm, time of discovery.
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no pneumonia. possible mild central adenopathy requires follow <unk> <unk> <unk> was paged.
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cardiomegaly and mild chf, without significant change compared <num> day earlier.
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<num>. large hiatal hernia. <num>. no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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slight interval decrease in the left-sided hydro pneumothorax post pleural drain catheter insertion.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12486000/s51194653/35e29d62-3359d816-6311d60b-b7ec39ae-4651e040.jpg
no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19004451/s54514645/163848fe-41462170-f24b3169-5db76e2e-f789b504.jpg
patchy new right lower lobe opacity with possible small pleural effusion. depending on the clinical setting, mild pneumonia or sequelae of aspiration could be considered, although evolving scarring or atelectasis with associated with an interval effusion could also be considered.
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ground-glass opacities in the lower lungs are concerning for hemorrhage in the setting of hemoptysis. please refer to subsequently performed chest cta for further details.
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no evidence of pneumonia.
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no definite acute cardiopulmonary process.
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dobbhoff tube terminates in the proximal stomach.
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no evidence of pneumonia.
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<num>. slightly more confluent right basilar opacity, consistent with persistent, possibly worsening, pneumonia. <num>. retrocardiac opacity is likely atelectasis, though a component of continued pneumonia is difficult to exclude. <num>. small left pleural effusion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13040016/s57001290/917c5670-bb4f9311-0a40d5fd-e3cf6190-f274e4f3.jpg
no acute cardiopulmonary process.
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unchanged right-sided pleural effusion, potentially loculated. left pleural drain likely within the lower posterior pleural space, not significantly changed since <unk>.
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bibasilar atelectasis, tiny pleural effusions. no picc line seen.
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<num>. vascular congestion with mild cardiomegaly. <num>. retrocardiac opacity is worrisome for pneumonia in the appropriate clinical setting. differential includes asymmetric vascular congestion.
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<num>. no acute cardiopulmonary abnormality. <num>. radiopaque foreign body projecting over the left upper quadrant better seen on concurrent kub.
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new left lower lobe infiltrate and effusion.
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no acute cardiopulmonary process.
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no acute cardiothoracic process.
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no radiographic evidence of trauma.
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<num>. stable mild cardiomegaly with mild interstitial pulmonary edema and interval increase in small bilateral pleural effusions. <num>. mild bibasilar opacities likely reflect atalectasis, less likely pneumonia.
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no acute cardiopulmonary process.
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<num>. mild pulmonary edema is new and bilateral pleural effusions are increased, moderate to large on the right and small on the left, since <unk>. <num>. new ett tip is seen <num> cm above the carina. care should be taken to not withdraw the tube any higher.
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large hiatal hernia with compressive left lower lobe atelectasis. picc line in appropriate position.
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<num>. left pleural effusion with a left retrocardiac opacity likely reflecting atelectasis, though superimposed infection is not excluded. <num>. multiple pulmonary nodules and masses are better assessed on the dedicated chest ct from <unk>.
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chest tube projecting over the left hemithorax. small quantity of subcutaneous emphysema but no residual pneumothorax identified.
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duo lead pacemaker in standard position with no evidence of pneumothorax.
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no acute cardiopulmonary process.
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no evidence of recent or non-recent tb. findings consistent with known interstitial abnormality, better seen on prior ct. no evidence of progression and no acute abnormality.
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the endotracheal tube has its tip <num> cm above the carinal. a nasogastric tube is seen coursing below the diaphragm with the tip not identified. there are stable bilateral diffuse parenchymal opacities in this patient with known wegener's granulomatosis. overall cardiac and mediastinal contours are unchanged. there a...
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12749129/s57354295/d7f2482c-d8b5ba37-28ea6f67-c342aa9a-df13516e.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16612444/s52964198/6d0ca591-e8e8ab3f-517d3f8e-47aec07c-df884ef2.jpg
no acute cardiopulmonary process
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13468314/s55541508/df6e7b47-8e32eec6-fc13b9d0-d549a045-8dc2266f.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10682314/s58341551/24301aa8-7d0908c1-55759fd8-39a4fbe3-82812d79.jpg
no acute cardiopulmonary process, no free intraperitoneal air.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17574940/s52963255/81455ece-851b695c-b3b6d8cd-54498a53-27d230cc.jpg
no acute cardiopulmonary process. if high clinical concern for rib fracture, dedicated rib series could be performed.
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in addition to the clear peribronchovascular infiltration and fibrosis in the upper lungs, less severe micro nodulation and early fibrosis is present in the lower lungs laterally and has progressed slightly since <unk>. although this could be a community acquired lung infection, i suspect sarcoid or other granulomatous...
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<num>. interstitial abnormality which has been slowly progressive on chest radiographs since <unk> and ct chest in <unk>. <num>. likely superimposed heart failure. the findings were entered into the critical communications dashboard by dr. <unk> at <unk> on <unk>.
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no acute cardiopulmonary abnormality. unchanged positioning of the tracheobronchial stent.
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<num>. low lung volumes. no acute cardiopulmonary process. <num>. right port-a-cath terminating in the high right atrium versus cavoatrial junction. <num>. diffuse osseous sclerotic metastases. <num>. large hiatus hernia.
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no acute cardiopulmonary process. chronic interstitial lung disease is similar to before.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12945136/s57487634/4bd0aaca-da80a62a-70ff5dfb-bbe3451b-707fb1a7.jpg
no acute cardiopulmonary abnormality. emphysema.
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<num>. pulmonary vascular engorgement consistent with mild interstitial edema and small bilateral pleural effusions. <num>. mild cardiomegaly.
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<num>) increase in size of small right pleural effusion and new small left pleural effusion. <num>) right basilar atelectasis. <num>) no evidence of pneumonia.
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findings compatible with right middle lobe pneumonia.
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no acute cardiopulmonary disease including pneumonia. findings were relayed to <unk> <unk> minutes following review by dr. <unk> by telephone on <unk> at approximately <time>.
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normal chest radiograph.
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patchy left base opacity could be due to pneumonia, aspiration, or atelectasis. cardiomegaly. central pulmonary vascular engorgement without overt pulmonary edema.
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no acute cardiopulmonary process.
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nasogastric tube terminates in the stomach.
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no acute cardiopulmonary process.
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<num>. stable large hiatal hernia with esophageal dilatation. <num>. no radiographic evidence of pneumonia or intrathoracic metastases.
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significant interval improvement of bibasilar patchy opacities and pleural effusions compatible with resolving pneumonia. <unk> weeks should be allowed for clearance of inflammatory changes in the lungs.
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heterogeneous new right lower lobe opacity concerning for developing infection.
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no definite acute cardiopulmonary process.
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right lower lobe pneumonia. dr. <unk> was called on <unk> at <time> p.m. and the findings were discussed by phone.
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small right pleural effusion has decreased since the prior study. right middle and lower lobes atelectasis has also significantly decreased in the interval. relative ovoid lucency projecting over the lateral right lower hemi thorax is felt to most likely be artifactual versus less likely loculated pneumothorax.
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increased opacification of the left lower lobe is consistent with worsening atelectasis; however, in the appropriate clinical context, pneumonia is possible.
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mild interstitial edema, slightly worse compared to <num> hours prior. superimposed pneumonia, particularly in the right lower lung is possible.
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as above
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<num>. right internal jugular central venous catheter tip in the lower svc. no pneumothorax. <num>. right basilar atelectasis.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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<num>. small left apical pneumothorax. <num>. marked decrease in small left pleural effusion.
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no acute cardiopulmonary abnormality.
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subtle opacification at the lung bases, which may represent atypical pneumonia or dependent edema. this finding was discussed with dr. <unk> by dr. <unk> by phone at <time> a.m. on <unk>.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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in comparison to <unk> exam, there is interval progression of bilateral heterogeneous opacities, suggestive of multifocal pneumonia.
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<num>. right picc terminates in the superior cavoatrial junction. <num>. no acute cardiopulmonary process.
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persistent diffuse bilateral peribronchial thickening and areas of bronchiectasis, in keeping with history of cystic fibrosis. no new focal consolidation seen.
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<num>. decreased but persistent right apical pneumothorax. <num>. increased right mid and lower lung opacities, which correspond with pulmonary contusions seen on recent ct.
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small stable bilateral pleural effusions.