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increased opacity at the left lung base with volume loss. poor visualization of trace suspected pneumothorax.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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bilateral perihilar opacities could relate to pulmonary edema although multifocal infection is not excluded given clinical history provided. moderate cardiomegaly.
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<num>. linear left basilar opacity, most consistent with atelectasis. <num>. likely small right pleural effusion.
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interval development of small bilateral pleural effusion, otherwise no interval change.
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persistent small bilateral pleural effusions with bibasilar atelectasis. no new focal consolidation to suggest pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17715495/s53155952/925af41f-c677f1e1-31a91a36-670b5765-9b87992a.jpg
no evidence of acute disease.
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mild pulmonary vascular prominence. no pulmonary edema.
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no evidence of acute disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16868103/s50895519/4eea3162-3c038f2f-e98bd353-61fbade8-326e2628.jpg
no evidence of pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14454179/s59633196/a8039881-9b366655-e4084234-80e19d72-5022bbcc.jpg
left basal opacity with slight elevation of the left hemidiaphragm, stable, likely represents scarring. please refer to subsequent cta chest for further details.
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cardiomegaly with pulmonary vascular congestion.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18524648/s58710154/4e95a898-347eb7e0-9006c4a3-2949f0fc-f4ada9c5.jpg
increased pulmonary edema. stable left and slightly smaller right pleural effusions.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16124481/s51832499/7089f5d7-81af688d-662009df-de188133-34319a01.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17514642/s52160648/e4463f28-a601c963-c6f86d75-ce35c78c-41763000.jpg
no acute cardiopulmonary process.
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stable cardiomegaly without overt pulmonary edema or pleural effusion.
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<num>. previously noted mediastinal lymphadenopathy may be slightly improved. fullness of the right hilum is unchanged and reflective of known lymphadenopathy. <num>. emphysema. <num>. known nodule within the right upper lobe is better seen on the prior exams.
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no acute cardiopulmonary process.
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subtle opacity in the left lower lobe could represent an early pneumonia.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14054139/s58294009/30eaf884-ad0f8c23-50af735e-8a333eb0-9bde5126.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16840812/s52157413/6f9f2595-375885c7-f2b3239c-f06248bf-6bb13314.jpg
no acute intrathoracic process.
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interval increase in bilateral perihilar and mid to lower lung opacities worrisome for moderate to severe pulmonary edema and bilateral pleural effusions which may be increased. bibasilar opacities likely represent combination of pleural effusion and atelectasis, however, in the appropriate clinical setting underlying ...
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slightly decreased right effusion and slightly increased left pleural effusion. compressive lower lobe atelectasis, though cannot exclude pneumonia.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10685081/s58865540/7eaeb0f5-12e7bc34-f495c26b-3057aced-86ef85d5.jpg
large left pneumothorax status post left apical lung lesion fiducial placement. results were discussed over the telephone with dr. <unk> by <unk> <unk> at <time> on <unk> at time of initial review.
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top normal heart size, otherwise normal.
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resolution of focal left lower lobe pneumonia.
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mild interstitial edema.
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no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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findings suggestive of mild vascular congestion and basilar atelectasis. if developing infection is a possible consideration, then short-term followup pa and lateral radiographs could be considered, however.
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no evidence of consolidation. chronic changes in left lung consistent with scarring.
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no evidence for retained foreign body. the case was discussed with dr. <unk> at approximately <time> p.m. while the patient was still in the operating room.
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no acute intrathoracic process.
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less marked pulmonary congestion since next preceding portable chest examination. again, no evidence of acute pneumonic infiltrate can be suspected to be the culprit for patient's rising white blood count. can liver abscess explain these findings?
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known small pneumothorax identified on ct is not evident on radiograph. lower lung volumes with slightly increased retrocardiac opacification, atelectasis versus aspiration in setting of trauma.
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no acute cardiopulmonary abnormality. mild elevation of the left hemidiaphragm of unknown chronicity with mild left basilar atelectasis.
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no acute cardiopulmonary process.
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when compared to prior study dated <unk>, there has been little interval change. low lung volumes and mild cardiomegaly persist. no acute intrathoracic abnormality is detected.
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no acute cardiopulmonary process. relatively low lung volumes.
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<num>. unchanged small right pleural effusion from <unk>. <num>. no pulmonary edema or pneumonia.
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mild cardiomegaly with mild pulmonary vascular congestion.
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normal radiographs of the chest.
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enteric tube in the upper stomach. no significant interval change.
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mild patchy bibasilar atelectasis in the setting of low lung volumes.
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elevated left hemidiaphragm with adjacent left basilar scar or atelectasis.
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new left lower lung zone opacification for which etiology includes infectious process or alternatively sequela of aspiration. increased perihilar opacities suggest central vascular congestion and mild pulmonary edema.
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no acute cardiopulmonary abnormality.
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normal chest radiographs.
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satisfactorily positioned endotracheal tube. right upper lobe pneumonia.
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<num>. slight worsening mild to moderate pulmonary edema. <num>. small bilateral pleural effusions. <num>. unchanged positioning of swan-ganz catheter, with its tip within the interlobar portion of the right pulmonary artery. this catheter could be withdrawn a few centimeters for standard positioning.
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no acute intrathoracic process.
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opacity in the left lower lobe suggesting pneumonia.
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no significant interval change.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13996091/s54047068/6dde6727-ad2a607e-4ae0c709-9036e911-c3cdcfa2.jpg
no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process.
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moderately enlarged cardiac silhouette and a mild central pulmonary vascular engorgement without overt pulmonary edema. <num> cm ovoid density projecting over the lower right paratracheal region of unclear clinical significance, but may represent a calcified lymph node. this could be confirmed on a non urgent chest ct.
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multifocal opacities are concerning for aspiration or bronchopneumonia. the above findings were communicated to dr. <unk> by dr. <unk> <unk> telephone at <time> am, <unk> min after discovery.
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no pneumomediastinum or pneumothorax.
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no acute cardiopulmonary process.
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no acute findings.
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no evidence of acute disease.
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rounded retrocardiac opacity, may represent a hiatal hernia. top normal heart size.
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no acute cardiopulmonary process.
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possible minimal interstitial edema which may in part be technical. no focal consolidation seen.
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findings likely represent moderate pulmonary edema. superimposed infection cannot be excluded. recommend repeat radiograph after treatment.
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no evidence of acute cardiopulmonary process. appropriate lead positioning.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10035780/s59076224/81601813-88063656-fdacf6b6-2f97c7dd-ace5238e.jpg
no acute cardiopulmonary process. no focal consolidation to suggest pneumonia.
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progressive, now moderate cardiomegaly. diffuse interstitial abnormality, similar to prior, compatible with chronic interstitial lung disease. if symptoms persist, repeat chest radiographs can be performed.
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no acute cardiopulmonary process. endotracheal tube terminates <num> cm above the carina.
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clear lungs.
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no radiographic evidence of active or latent pulmonary tuberculosis infection.
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clear lungs. no acute or chronic lung disease.
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no significant interval change since the prior study of <unk>. persistently elevated right hemidiaphragm and bibasilar opacities.
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bibasal atelectasis and small bilateral pleural effusions, left more than right, has worsened since <unk>.
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<num>. increased bibasilar airspace opacities which are nonspecific but could reflect infection. <num>. large hiatal hernia and fat containing right posterior diaphragmatic hernia. <num>. probable small right pleural effusion. <num>. slight interval increase in extent of a compression deformity of a mid thoracic verteb...
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mild to moderate pulmonary interstitial edema with small bilateral pleural effusions.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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ill-defined focal opacity over the left mid lung. findings were discussed with dr.<unk> <unk> telephone at <num>pm on <unk> and in this patient with a smoking history, follow-up imaging with ct scan is recommended.
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<num>. no acute cardiopulmonary process. <num>. no displaced rib fracture.
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posterior basilar consolidation worrisome for pneumonia, best seen on the lateral view.
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no acute cardiopulmonary process.
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<num>. findings suggesting mild vascular congestion. <num>. patchy left mid lower lung opacities that appear unchanged and suggest minor atelectasis or scarring. <num>. similar severe elevation of the right hemidiaphragm. <num>. air-fluid levels in bowel of the upper abdomen, a non-specific pattern.
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worsened appearance to the chest with new focal infiltrate in the left midlung
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no acute cardiopulmonary abnormalities
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no evidence of acute cardiopulmonary disease.
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stable cardiomegaly.
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no acute cardiopulmonary process.
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mild interstitial pulmonary edema, slightly improved compared to the previous radiograph.
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no definite fracture identified.
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no acute cardiopulmonary process.
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et tube in appropriate location ending <num> cm above the carina. new enteric tube in appropriate location. otherwise, unchanged chest radiograph.