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subtle opacity at the left lung base is likely atelectasis, though an early pneumonia in the right clinical setting cannot be excluded.
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<num>. narrowing of the trachea at the level of the thoracic inlet is unchanged since ct <num> days prior. <num>. no evidence of pneumothorax or pneumonia.
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no free air.
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large hiatal hernia. no evidence of pneumonia.
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no acute intrathoracic process.
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moderate pulmonary edema is mildly improved and underlying parenchymal opacities are slightly more subtle.
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no evidence of pneumonia. interval development of left basilar atelectasis.
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no acute cardiopulmonary process. et tube in appropriate position.
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interval improvement but residual mild pulmonary and interstitial edema. patchy bibasilar opacities likely reflect atelectasis in the setting of low volumes. overall cardiac and mediastinal contours are stable. no pneumothorax.
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small left pleural effusion with bibasilar opacifications likely chronic in nature, though cannot exclude infectious process.
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no evidence of acute cardiopulmonary disease.
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low lung volumes and basilar atelectasis. no focal lung consolidation. stable mild cardiomegaly.
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no acute intrathoracic process.
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orogastric and endotracheal tubes appeared are positioned appropriately.
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no acute cardiopulmonary abnormality.
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further decrease in left effusion. small residual left pneumothorax.
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<num>. final radiograph in this series of <num> images demonstrates the endotracheal tube tip to be slightly low lying, terminating approximately <num> cm from the carina. enteric tube is in standard position. <num>. multifocal airspace opacities concerning for pneumonia. <num>. mild pulmonary vascular congestion. known pleural effusions are better assessed on the previous ct. <num>. re- demonstration of multiple masses associated with the rib cage and thoracic spine.
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stable mild cardiomegaly and left lower lobe atelectasis with no pneumonia or signs of congestive heart failure
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no acute cardiopulmonary process.
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normal chest.
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known malignancy better appreciated on ct, but the parenchyma is clearly abnormal in multiple areas.
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no acute cardiopulmonary process.
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<num>. no acute cardiopulmonary process. <num>. right apical nodular opacity likely represents summation of rib shadows. lordic view may be obtained for confirmation. findings were communicated via phone call by dr. <unk> to dr. <unk> on <unk> at <unk>.
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cardiomegaly without vascular congestion is seen, suggesting either cardiomyopathy or pericardial effusion. an apparent right hilar mass is seen for which a dedicated ct is recommended.
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no acute cardiopulmonary process.
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slight interval increase in left pleural effusion. small apical right pneumothorax is stable.
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findings suggesting emphysema. patchy opacity at the left lung base, probably atelectasis but not specific.
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no acute findings.
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new right upper and probable right middle lobar opacities, associated with right hilar prominence concerning for pneumonia. no pleural effusions. recommendation(s): repeat chest radiograph in <num> weeks following treatment is recommended to assess resolution.
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questioned right basilar opacity on the prior study is not well seen on the current study and was likely artifactual. subtle right basilar opacity may be due to atelectasis.
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pulmonary edema though improved since <unk>. more dense consolidation projecting over the lung base on the lateral view could represent superimposed infection.
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no acute cardiopulmonary abnormality.
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large hiatal hernia. no acute cardiopulmonary radiographic abnormality.
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no acute cardiopulmonary process.
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likely improved left pleural effusion. left lower lobe consolidation may reflect atelectasis or pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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small right pleural effusion
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no acute cardiopulmonary process.
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substantially improved but persistent left lower lobe and lingular opacities. the left pleural effusion has resolved. followup chest radiograph in one month is recommended. these findings were entered onto the critical communications dashboard by dr. <unk> at <unk> on <unk>.
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probable epicardial fat pad accounting for subtle opacity obscuring the left heart border inferiorly. no convincing signs of pneumonia.
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no acute intrathoracic process.
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moderate size right and small left pleural effusions, both of which have decreased since the previous study. bibasilar atelectasis.
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findings suggesting pneumonia in the left lower lobe. follow-up radiographs are recommended to document resolution within <unk> weeks.
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no acute cardiopulmonary process. again seen low lung volumes, elevation of the left hemidiaphragm with gaseous distension of bowel beneath.
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interval worsening.
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no radiographic evidence for pneumonia. bibasilar subsegmental atelectasis.
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moderate left-sided pleural effusion. no evidence of free intraperitoneal air below the diaphragm.
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no acute cardiopulmonary process. s-shaped scoliosis of the thoracic and lumbar spine.
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no acute cardiopulmonary abnormality.
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previously seen left apical pneumothorax, if anything is insignificant.
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bibasilar opacities likely represent combination of atelectasis and scarring given patient's history of chronic aspiration. however, underlying pneumonia in the lower lobes particularly on the right cannot be ruled out.
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stable cardiomegaly. no acute cardiopulmonary abnormality.
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loculated moderate left pleural effusion which developed between <unk> and <unk> has not changed over the past week.
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small bilateral effusions. retrocardiac opacity could be due to left lower lobe atelectasis given the relatively lower lung volumes, however, infectious process such as pneumonia is also possible.
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new multifocal pneumonia or hemorrhage in the appropriate clinical setting. new pulmonary nodules, likely metastases.
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interval decrease in size of left-sided effusion. no pneumothorax.
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no evidence of acute disease.
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no acute cardiopulmonary process.
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mild cardiomegaly and tortuous aorta with otherwise no acute cardiopulmonary process.
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ng tube probably coiled in the distal esophagus.
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marked transverse cardiomegaly with mild cephalization of pulmonary blood flow suggesting cardiac decompensation. in the differential diagnosis consider a pericardial effusion. no pneumonia.
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no acute intrathoracic process.
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mild pulmonary vascular congestion, improved since prior. no evidence of consolidation.
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mild-to-moderate cardiomegaly, moderate pulmonary edema and small bilateral pleural effusions consistent with congestive heart failure.
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hyperinflation and cardiomegaly without acute cardiopulmonary process.
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<num>. right port-a-cath in appropriate positioning. <num>. no evidence of pneumonia.
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peribronchial thickening, which is seen diffusely and could reflect bronchial inflammation. correlate clinically. no lobar consolidation.
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no acute cardiopulmonary process.
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no convincing evidence for pneumonia or aspiration on very limited study. hazy opacities and patchy opacities at the lung bases may be due to mild vascular congestion and minor atelectasis. short-term follow-up radiographs are suggested with better inspiration, if feasible, in the event that respiratory symptoms were to persist.
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no acute cardiopulmonary process.
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new tiny opacity projecting over the right lower lung is likely confluence of shadows, but a tiny infectious focus cannot be excluded. recommend follow up radiographs after treatment.
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<num>. interval improvement in lung volumes with residual minimal left basilar atelectasis. <num>. mild chronic abnormality at left lung base could be scarring or bronchiectasis and has been stable since <unk>.
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<num>. probable small left pleural effusion and atelectasis. no evidence of pneumonia. <num>. mild cardiomegaly.
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no acute intrathoracic process.
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no acute findings in the chest.
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no evidence of acute cardiopulmonary process.
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<num>. improved pulmonary edema, now mild. small right and trace left pleural effusions, similar to prior. <num>. no displaced rib fracture visualized. dedicated rib series would increase sensitivity for detection of rib fractures.
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no acute cardiopulmonary process.
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no acute findings.
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no significant interval change. focal right lower lobe opacity compatible with known underlying lesion as seen on prior pet-ct.
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<num>. new moderate left pleural effusion. <num>. bibasilar atelectasis without evidence of pneumonia. results were communicated with the patient's primary care doctor at <time> p.m. on <unk> via telephone by dr. <unk>.
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interval placement of right chest tube and resolution of mediastinal shift. small right pneumothorax and trace left pneumothorax remains.
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no definite acute interval changes to explain patient's lingering cough. since the patient's lower breasts are dense and may obscure small findings in the bases of the lungs, if there is a high clinical suspicion for pneumonia, would recommend additional oblique views for further evaluation. these findings and recommendations were discussed with dr. <unk> by dr. <unk> at <time>pm by telephone on the day of the exam.
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nasogastric tube tip within the stomach.
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no radiographic evidence of pneumonia.
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no evidence of acute disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17761938/s55108963/50292cd3-8098dbcd-df0f0a44-c25aefe0-912569e2.jpg
no pneumonia. nodules seen on prior ct is below the resolution of radiograph.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12148014/s56662747/861a9a1a-94f42c92-559ba385-3df26645-0f6bf2a6.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11363157/s52279354/1bdc83b7-e4ed48ee-9ec43e74-a8699815-11e5e741.jpg
findings doubtful for pneumonia but suggestive of mild vascular congestion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12601251/s56648885/4313b569-ca07e7bf-ee16aefd-aa4b74d2-e98523ec.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13194374/s52754095/36420037-1a4ba60b-a81bb1fe-7feedb60-84b503f7.jpg
no acute cardiothoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16837125/s53984517/adb66c73-f3f5efd7-2ca02bd3-2966ded8-d8355435.jpg
normal chest radiograph. no obvious radiopaque esophageal and tracheal opacity
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p11902760/s50495086/d380bacb-543b3fcd-56a90480-1370d8f2-e5190a43.jpg
no acute intrathoracic process. no displaced rib fracture. if there is further concern is dedicated rib series may be performed.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16893112/s52724352/b6a88ec7-beda965e-a0f5f7f4-93521a6b-4a10c6ee.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13784168/s53716976/77bae7e4-71bf862a-eddc8260-a23c65a0-62f6c5c4.jpg
no evidence of acute disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19589747/s52174205/5091e634-4b270381-a3ba41f0-40370e78-d0bb0559.jpg
no evidence of acute intrathoracic injury.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19227457/s53411674/26c08700-c5ee5fa0-7f4d4cec-507d4e5e-1970d0f9.jpg
interval development of bilateral pleural effusions, small-to-moderate and slightly larger on the left with associated lower lobe compressive atelectasis. cardiomegaly also noted with equivocal mild interstitial pulmonary edema.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13189172/s53876605/99e7647e-2e807aa9-828ed03e-80f9079a-dcc5fc9f.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19922133/s56687543/97a3c2de-b4c0081d-27f85989-70ca0cbd-2ec24665.jpg
no evidence of acute cardiopulmonary process.