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et tube in appropriate position. rapid interval development of bilateral perihilar opacities suggesting pulmonary edema. please note that blossoming infection is also possible.
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cardiomegaly, hilar congestion with pleural effusions, left greater than right. increasing right basal opacity which could represent atelectasis versus pneumonia.
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<num>. moderate pulmonary edema has worsened. <num>. moderate to severe cardiomegaly is unchanged. <num>. small bilateral pleural effusions.
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no acute cardiopulmonary abnormality.
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<num>. limited examination due to marked patient rotation and overlying motion artifact on the lateral view. cardiac enlargement which may reflect cardiomegaly or pericardial effusion. probable hiatal hernia. no definite pneumonia or pulmonary edema. followup imaging would be prudent. .
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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no acute intrathoracic abnormalities identified.
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normal chest radiograph.
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mildly increased interstitial markings compared to previous exam suggesting mild pulmonary vascular congestion. linear bibasilar opacities which have the appearance of atelectasis however clinical correlation is suggested regarding possibility of early pneumonia.
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no acute intrathoracic process.
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mild prominence and crowding of vascular structures primarily on the left suggestive of pulmonary vascular congestion or mild edema. no clear consolidative process is present.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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mild bibasilar atelectasis.
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no pneumothorax, post left-sided chest tube placement with decrease in left pleural effusion.
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bibasilar subsegmental atelectasis.
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nonspecific patchy bibasilar opacities, possibly due to aspiration or developing aspiration pneumonia given clinical concern for this entity. apparent co-existing bronchial wall thickening could be due to aspirated secretions or a potentially more chronic airways disease. followup radiograph may be helpful in this regard.
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<num>. typical findings of chronic obstructive pulmonary disease with lower lobe predominant emphysema, distribution suggestive of alpha <num> antitrypsin deficiency. <num>. no radiographic evidence of an acute cardiopulmonary process.
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mild interstitial abnormality, probably not related to the patient's acute chest pain.
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<num>. bilateral opacities as described above, consistent with multifocal pneumonia, not changed from prior. <num>. right picc line with its distal end coiled at the proximal svc and pointing proximally. recommend repositioning of the picc line. findings are discussed with dr. <unk>. recommendation(s): recommend repositioning of the right picc line.
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no significant change in the aeration of the lungs. stable bilateral pleural effusions and bibasilar atelectasis.
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no acute findings.
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no acute cardiopulmonary process.
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stable right pneumothorax. persistent postoperative widening of the mediastinum
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no acute cardiopulmonary process.
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worsening multifocal pneumonia in the right lung, possibly due to aspiration. atypical and opportunistic infections are also possible given the nodular component. diffuse interstitial opacities atypical may be related to infection or coexisting edema.
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no evidence of acute cardiopulmonary process. low lung volumes.
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no acute intrapulmonary process.
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moderate cardiomegaly, otherwise, no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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normal chest radiograph.
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slight interval decrease in the right loculated basal pneumothorax.
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no acute intrathoracic abnormality.
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no acute cardiopulmonary process.
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ng tube in the upper stomach. no short term interval change.
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calcified pleural plaques compatible with a history of prior asbestos exposure. chronically elevated left hemidiaphragm with adjacent subsegmental atelectasis. no acute cardiopulmonary abnormality otherwise demonstrated.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13630480/s59406225/e15db821-a2282ad2-62e6e680-266b1020-5c099d9b.jpg
small retrocardiac hiatal hernia with adjacent atelectasis. otherwise, no evidence of acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12034370/s54676486/2e80ef28-385708d4-223a3490-84c4dbfb-87962083.jpg
unchanged appearance of left icd since <unk>, with a single lead terminating at the right ventricle.
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mild pulmonary edema and small bilateral effusions.
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no acute cardiopulmonary process.
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no evidence of acute traumatic injury within the limitations of routine chest radiography.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality.
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status post right chest tube removal. no evidence of a pneumothorax.
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mild cardiomegaly and mild central pulmonary vascular engorgement. no focal consolidation to suggest pneumonia.
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mildly enlarged cardiac silhouette. no rib fracture seen. high-riding right humeral head can be seen in rotator cuff disease.
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small right pleural effusion with right lower lobe anterior-basal segment collapse; trace left pleural effusion.
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mild interstitial edema
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no acute cardiopulmonary process. probable eventration of the right hemidiaphragm.
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left base atelectasis. no focal consolidation worrisome for infection. hilar enlargement suggests pulmonary hypertension.
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unchanged moderate cardiomegaly without overt pulmonary edema.
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no acute cardiopulmonary process. findings discussed with dr. <unk> by <unk> by telephone at <time> on <unk> at the time of initial review of the study in response to wet read request.
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doubt significant change compared with <unk>. a small left effusion would be difficult to exclude. no right-sided effusion is identified.
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<num>. decrease in size and amount of fluid within the neoesophagus (prior esophagectomy with gastric pull-through). <num>. no evidence of pneumonia.
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no acute findings.
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no change in right moderate sized pleural effusion (hemothorax on ct)
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faint opacity in the right mid lung seen on the anterior view may represent early pneumonia.
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no acute cardiopulmonary process.
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resolution of previous pneumonia.
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no acute cardiopulmonary process.
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hyperinflated lungs without superimposed acute pathology.
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ap chest at <time> compared to <unk>: the diaphragmatic region is excluded from this examination. severe cardiomegaly is unchanged during this hospitalization. mild pulmonary edema is stable, but central vascular engorgement is more pronounced, and bibasilar atelectasis which worsened after <unk> and <unk>, remains severe. pleural effusions are present, but poorly depicted and not large. et tube is in standard position. right picc line ends at the origin of the svc. no pneumothorax.
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<num>. pulmonary edema has improved. <num>. persistent bilateral effusions with small left basilar opacity, for which underline pneumonia cannot be excluded in the correct clinical setting.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13342249/s51105480/d7679c3f-e057cbde-70240015-fbb02168-2d439b66.jpg
no evidence of acute cardiopulmonary disease.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15498638/s55734033/ac618cd7-d1d356b3-0808fb4c-a1822423-70b89937.jpg
bibasilar streaky opacities, likely atelectasis and scarring. no pulmonary edema or large pleural effusion.
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mild interval improvement.
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<num>. no acute cardiopulmonary process. <num>. improved cardiomegaly.
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no focal consolidation to suggest pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p14721325/s52140957/53cfa3c7-67bb2668-32d67739-2eb1107c-ab6aee36.jpg
no acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
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mild bibasilar atelectasis. no definite signs of pneumonia.
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normal mediastinal contours.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13263226/s59669043/baa67941-0f8f29ac-7083a083-b7d55342-ed792494.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15840635/s58761299/a4307679-444720c0-53a29f7f-420894ec-3043da5f.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17351138/s55423214/9d07823a-279a4143-27ae9e0d-9ac8ee1c-242e8149.jpg
no focal consolidations concerning for pneumonia identified.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12401170/s50580709/280db3ca-95ad5eb9-b1130c6c-af2a8939-2685cfb4.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17339765/s51751674/dd9f906a-642a9d77-0b91fcf8-2336cd71-c06d4b1e.jpg
increase in bilateral pleural effusion and bibasilar atelectasis.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p16196589/s59643803/ea2b3354-572702ba-e18c986f-e0e7567f-0ed32436.jpg
decrease in left pleural effusion. no pneumothorax. remaining opacities in the left base attention in followup is recommended
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15689523/s57868783/abfdff44-3b2c06b3-c8f7456e-c95b4060-e7727cda.jpg
mild improvement in mulifocal airspace opacities since the most recent prior study, which may reflect improvement in pulmonary edema.
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no significant change given differences in technique.
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no acute cardiopulmonary abnormality.
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no evidence of acute disease. no evidence for free air.
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new diffuse hazy opacities in both lungs, likely reflective of mild pulmonary edema, although an atypical infectious process cannot be completely excluded. clinical correlation is recommended, and repeat radiographs after diuresis are recommended.
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no acute cardiopulmonary process.
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stable cardiomegaly with mild vascular congestion. no overt pulmonary edema is seen.
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no acute cardiopulmonary process.
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no evidence of acute disease.
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cardiomegaly and congestive heart failure, with less severe interstitial edema compared to <unk>.
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<num>. no acute intrathoracic process, specifically no pneumonia. <num>. chronic mid thoracic spine compression fracture.
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no pneumonia.
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no acute cardiopulmonary process. slowly progressive mild cardiomegaly.
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no acute cardiopulmonary abnormality.
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dobbhoff tube in place coursing to the region of the pylorus.
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no evidence of a pneumothorax. stable appearance of the chest otherwise.