IdA
string
IdB
string
labels
int64
mechanism
string
effect
string
score
float64
sentence
string
signor_id
string
P06493
P49459
1
phosphorylation
up-regulates
0.37
Hhr6a is phosphorylated in vitro by cdk-1 and -2 on ser120, a residue conserved in all hhr6a homologues, resulting in a 4-fold increase in its ubiquitin-conjugating activity.
SIGNOR-116504
O96004
Q15797
0
transcriptional regulation
up-regulates quantity
0.2
Chromatin immunoprecipitation (ChIP) revealed a subset of the BIG (BMP4 induced genes) signature, including Satb2, Smad6, Hand1, Gadd45γ and Gata3, that was bound by Smad1/5 in the developing mandible, revealing direct Smad-mediated regulation
SIGNOR-268936
P62330
Q99418
0
guanine nucleotide exchange factor
up-regulates activity
0.887
Effects of ARNO upon Axonogenesis Are Mediated by Downstream Activation of ARF6. ARNO/ARF6 signaling pathways that could modulate actin reorganization in the axonal growth cone. ARNO stimulates GTP exchange on ARF6, thereby increasing the amount of active ARF6.
SIGNOR-264910
P17252
Q96T49
1
phosphorylation
down-regulates activity
0.2
PKCα phosphorylated the full length recombinant TIMAP in in vitro kinase assay and Ser331 of TIMAP was shown to be phosphorylated by PKC. Phosphorylation of TIMAP upon PKC activation in endothelial cells results in enrichment of TIMAP in the membrane, but no such change can be observed in PKC depleted cells. Phosphorylation state of TIMAP, through affecting PP1 activity, has a remarkable effect on endothelial barrier function.
SIGNOR-273802
Q92630
O43255
1
phosphorylation
up-regulates
0.41
In the present study, we identify the serine/threonine kinase dyrk2 as siah2 interaction partner that phosphorylates siah2 at five residues (ser16, thr26, ser28, ser68, and thr119). accordingly, phosphorylated siah2 is more active than the wild-type e3 ligase and shows an increased ability to trigger the hif-1?-Mediated transcriptional response and angiogenesis.
SIGNOR-198729
Q16665
P28482
0
phosphorylation
up-regulates
0.586
We show that at least two different nuclear protein kinases, one of them identified as p42/p44 mapk, can modify hif-1_. Analysis of in vitro phosphorylated hif-1_ by mass spectroscopy revealed residues ser-641 and ser-643 as possible mapk phosphorylation sites these data suggest that phosphorylation of ser-641/643 by mapk promotes the nuclear accumulation and transcriptional activity of hif-1_
SIGNOR-178723
O43683
Q12834
1
phosphorylation
down-regulates activity
0.992
Bub1 directly phosphorylates Cdc20 in vitro and inhibits the ubiquitin ligase activity of APC/C(Cdc20) catalytically. A Cdc20 mutant with all six Bub1 phosphorylation sites removed is refractory to Bub1-mediated phosphorylation and inhibition in vitro. 
SIGNOR-250606
Q13177
P49593
0
dephosphorylation
down-regulates
0.261
Pop x2, a pp 2c serine/threonine phosphatase, is known to dephosphorylate pak and downregulate its activity.
SIGNOR-162149
Q9NYV4
P24928
1
phosphorylation
up-regulates activity
0.784
Cyck/cdk12 can activate transcription and phosphorylate ser2 in the ctd of rnapii / phosphorylation of serine at position 2 (ser2) is thought to be responsible for productive transcriptional elongation and synthesis of full-length mature mrna
SIGNOR-176805
Q13315
P48730
1
phosphorylation
up-regulates activity
0.34
These results elucidated the specificity of this in vivo degradation assay, and further implicated that CKI\u03b4-dependent phosphorylation events is the major signaling route through which DNA damage-dependent activation of ATM might control timely turnover of Mdm2 during genotoxic stress. (A) ATM-mediated phosphorylation of CK1\u03b4 promotes CK1\u03b4 nuclear localization.|We further demonstrated that DNA damage-induced activation of ATM directly phosphorylated CKI\u03b4 at two well-conserved S/TQ sites, which promotes CKI\u03b4 nuclear localization to increase CKI\u03b4-mediated phosphorylation of Mdm2, thereby facilitating subsequent Mdm2 ubiquitination by SCF\u03b2-TRCP.
SIGNOR-279504
P10071
P17612
0
phosphorylation
down-regulates quantity
0.468
Ci/gli zinc finger proteins mediate the transcriptional effects of hedgehog protein signals. In drosophila, ci action as transcriptional repressor or activator is contingent upon hedgehog-regulated, pka-dependent proteolytic processingall six pka phosphorylation sites are required for processing of gli3.
SIGNOR-75359
P46934
Q00987
1
ubiquitination
up-regulates quantity by stabilization
0.466
NEDD4 promotes MDM2 ubiquitination in a dose- and time-dependent manner, whereas depletion of NEDD4 reduced the half-life of endogenous MDM2 [ xref ].
SIGNOR-278769
P07333
Q9H334
0
transcriptional regulation
down-regulates quantity by repression
0.296
Overexpression of MFH/Foxp1 markedly attenuated phorbol ester-induced expression of c-fms, which encodes the M-CSF receptor and is obligatory for macrophage differentiation.
SIGNOR-269048
P46783
O96006
0
transcriptional regulation
up-regulates quantity by expression
0.2
HDRE-like sequences act as positive regulatory elements for RP gene promoter activities in vivo. | Cotransfection of a plasmid expressing hDREF increased luciferase expression directed by each RP gene promoter more than 30% compared with the values obtained without the hDREF-expressing plasmid.
SIGNOR-266083
O96013
P17676
1
phosphorylation
up-regulates activity
0.2
Importantly, we found that PAK4 enhanced CEBPB phosphorylation on Thr 235.|In summary, we showed that PAK4 mediated CEBPB activation upregulated CLDN4 expression to promote breast cancer cell migration and invasion.
SIGNOR-280055
P27361
Q15788
1
phosphorylation
up-regulates
0.268
Mapk also directly phosphorylates src-1 at thr1179 and ser1185. Phosphorylation of src-1 by mitogen-activated protein kinase (mapk) is required for optimal progesterone receptor-dependent transcription and for functional cooperation with camp response element-binding protein-binding protein
SIGNOR-91143
Q15910
P31269
1
transcriptional regulation
down-regulates quantity by repression
0.396
These data support the proposed regulatory impact of particular PRC2-proteins in expression of HOXA9 and HOXA10 in NK/T-cells. In mammalian cells knockdown of PRC2 components EZH2 or PHF1 led to upregulated HOXA gene expression.
SIGNOR-260068
Q96JC1
Q13485
1
relocalization
down-regulates activity
0.318
The data demonstrating binding of TLP to TGF-β and activin type II receptors and selective inhibition of Smad3/Smad4 complex formation by deregulated TLP suggest that TLP is involved in localizing these receptors and Smad4 to specific intracellular compartments, where it regulates formation of Smad3/Smad4 but not Smad2/Smad4 complexes.
SIGNOR-261377
O14920
P12931
0
phosphorylation
up-regulates
0.362
These results indicate that c-src can associate with ikkbeta and phosphorylate its tyrosine residues after tnf-alfa or tpa stimulation.
SIGNOR-99318
P09651
P17252
0
phosphorylation
down-regulates
0.2
A survey of seven protein kinases showed that a1 was heavily phosphorylated by protein kinase c (pkc) and also was phosphorylated by casein kinase iiamino acid sequencing revealed that these sites were ser95, ser192, and ser199;phosphorylation at ser192 was more abundant than at ser95 and ser199. Phosphorylation by pkc inhibited the strand annealing activity of a1.
SIGNOR-32291
P19174
Q06187
0
phosphorylation
up-regulates activity
0.581
Then, BTK and ITK are activated by Src kinases and proceed to phosphorylate the lipase PLCgamma2 / PLCgamma1, which cleaves PIP2 in the plasma membrane and generates the secondary messengers IP3 and DAG.
SIGNOR-279444
P26045
P03372
1
dephosphorylation
up-regulates activity
0.2
Our recent studies further demonstrated that PTPH1 dephosphorylates estrogen receptor at Y537, increases estrogen receptor stability and nuclear accumulation, and enhances breast cancer sensitivity to anti-estrogens [ ].
SIGNOR-277127
Q13485
Q92905
0
ubiquitination
down-regulates
0.433
We report a novel mechanism of smad4 degradation. Jab1 interacts directly with smad4 and induces its ubiquitylation for degradation
SIGNOR-114697
Q13158
Q9BV47
0
dephosphorylation
down-regulates activity
0.366
This multi-functionality of fadd may depend primarily on its subcellular location. Fadd shuttles between the cytosol and the nucleus and this signal is unclear;however, fadd trafficking requires phosphorylation of the protein on ser194dusp26 suppresses cell proliferation by fadd dephosphorylation
SIGNOR-204559
O60260
O43236
1
ubiquitination
down-regulates quantity by destabilization
0.2
Thus, Parkin degrades ARTS in a proteasome dependent manner.|Together, these results suggest that Parkin specifically ubiquitinates and degrades ARTS, and that this degradation may be linked to the pro-apoptotic function of ARTS.
SIGNOR-278642
Q03933
P35227
0
sumoylation
down-regulates activity
0.325
MEL-18, in contrast to the polycomb protein PC2/CBX4, in which SUMO E3 activity stimulates sumoylation of certain proteins, actually functions like an anti-SUMO E3 protein, interacting with both HSF2 and the SUMO E2 UBC9 but acting to inhibit UBC9 activity and thereby decreasing sumoylation of a target protein, in this case that of HSF2. sumoylation of HSF2 is up-regulated during mitosis and is important for the interaction of this factor with a subunit of the condensin complex during the bookmarking process
SIGNOR-226245
Q9Y4A8
P15559
1
transcriptional regulation
down-regulates quantity by repression
0.338
Deletion mutation analysis revealed that Nrf3 repression of NQO1 gene expression required heterodimerization and DNA binding domains but not transcriptional activation domain of Nrf3.
SIGNOR-268976
Q9NQC7
P20749
1
deubiquitination
down-regulates
0.512
Cyld binds and deubiquitinates bcl-3in cyld+/+ keratinocytes, tpa or uv light triggers the translocation of cyld from the cytoplasm to the perinuclear region, where cyld binds and deubiquitinates bcl-3, thereby preventing nuclear accumulation of bcl-3 and p50/bcl-3- or p52/bcl-3-dependent proliferation.
SIGNOR-146774
P61586
P35241
1
phosphorylation
up-regulates activity
0.47
Rev-erbα interacted with OPHN-1, promoted RhoA activity and phosphorylation of ERM. etection of phosphorylated ezrin (Thr567)/radixin (Thr564)/moesin (Thr558)(p-ERM) in Rev-erbαfl/flCre− and Rev-erbαfl/flPF4Cre+ platelets using phospho-specific antibodies.
SIGNOR-268430
Q14012
O00429
1
phosphorylation
up-regulates activity
0.333
For example, protein kinase A (PKA) phosphorylation of Drp1S600 has been reported to decrease Drp1 GTPase activity in vitro (23, 24), whereas phosphorylation of the same conserved serine residue by Ca2+-calmodulin–dependent protein kinase Iα (CaMKIα) in Drp1 isoform 3 has been reported to cause a significant increase in mitochondrial fission
SIGNOR-262552
P09471
P19474
1
null
down-regulates
0.2
Mechanistically, GNAO1 recruited TRIM21 and facilitated TRIM21-mediated ubiquitination. 
SIGNOR-278888
O00311
O75475
1
phosphorylation
up-regulates
0.334
We now report identification of the cdc7-activator of s-phase kinase (ask) heterodimer as a novel interactor of ledgf. the kinase phosphorylated ledgf in vitro, with ser-206 being the major target, and ledgf phosphorylated at this residue could be detected during s phase of the cell cycle. Ledgf potently stimulated the enzymatic activity of cdc7-ask, increasing phosphorylation of mcm2 in vitro by more than 10-fold.
SIGNOR-25763
Q99683
Q9UNI6
0
dephosphorylation
down-regulates activity
0.25
Our study showed that DUSP12 inhibited ASK1-JNK activation and DUSP12 can directly bind to and dephosphorylate ASK1.
SIGNOR-277032
Q12778
O75874
1
transcriptional regulation
up-regulates quantity by expression
0.258
We identify FOXOs as transcriptional activators of IDH1. FOXOs promote IDH1 expression and thereby maintain the cytosolic levels of α-ketoglutarate and NADPH.
SIGNOR-260101
Q9BZI1
P28482
0
phosphorylation
up-regulates activity
0.2
We tested the transcriptional properties of Irx2 by dividing it into amino- and carboxy terminal parts and found that Mek1-mediated phosphorylation activates and derepresses the amino and carboxyl parts, respectively. When Ser46 and Ser65 were mutated to alanine (S46A and S65A), phosphorylation was reduced, whereas substitution of Ser83 and Ser103 (S83A and S103A) did not affect phosphorylation.
SIGNOR-263052
P28482
P30041
1
phosphorylation
up-regulates
0.693
These results show that the mapks can mediate phosphorylation of prdx6 at thr-177 with a consequent marked increase in its aipla(2) activity.
SIGNOR-183379
Q13627
P49418
1
phosphorylation
down-regulates
0.396
Recent studies show that phosphorylation of amphiphysin1 prd by cdk5 inhibited the association of amphiphysin1 with ap-2 in synaptic vesicle endocytosis (7, 8) similar to that by mapk (present report). Cdk5 appears to phosphorylate amphiphysin1 at serines 261, 272, 276, and 285 and threonine 310, located in the prd
SIGNOR-126843
Q5S007
P46734
1
phosphorylation
up-regulates activity
0.393
LRRK2 phosphorylates MKK3 and MKK7 in vitro but has a relatively minor effect on MKK6 phosphorylation.
SIGNOR-279057
P13051
O15297
0
dephosphorylation
down-regulates activity
0.377
PPM1D dephosphorylation of UNG2 is correlated with reduced UNG2 activity on uracil-containing templates.|This result suggests that PPM1D specifically inhibits UNG2 and not other uracil DNA glycosylases.
SIGNOR-277156
P29320
Q8NBZ7
1
phosphorylation
up-regulates activity
0.2
However, it is possible that etk is a principal activator of Ugd.|The phosphorylation of the Ugd protein (a UDP-glucose dehydrogenase) by Etk increases Ugd dehydrogenase activity.
SIGNOR-280006
O43541
P51817
0
phosphorylation
up-regulates activity
0.431
In vitro phosphorylation assays demonstrated that PrKX phosphorylates Smad6 at a serine residue.
SIGNOR-279270
Q13535
Q9NZC9
1
phosphorylation
down-regulates activity
0.538
ATR phosphorylates SMARCAL1 on S652, thereby limiting its fork regression activities and preventing aberrant fork processing. Thus, phosphorylation of SMARCAL1 is one mechanism by which ATR prevents fork collapse, promotes the completion of DNA replication, and maintains genome integrity.
SIGNOR-273516
P18887
P19784
0
phosphorylation
up-regulates activity
0.46
XRCC1 is phosphorylated in vivo and in vitro by CK2, and CK2 phosphorylation of XRCC1 on S518, T519, and T523 largely determines aprataxin binding to XRCC1 though its FHA domain | In addition, we present data to show that the acute loss of aprataxin by small interfering RNA (siRNA) renders HeLa cells sensitive to MMS through a mechanism that destabilizes XRCC1.
SIGNOR-251050
P24385
Q96PU4
0
ubiquitination
down-regulates quantity by destabilization
0.294
We found that NIRF directly ubiquitinated cyclins D1 and E1, as evidenced by the appearance of the tail (Fig. 4B). In summary, the above findings suggest that NIRF tightly cooperates with the core cell cycle machinery and induces G1 arrest, which is accompanied by ubiquitination of cyclins D1 and E1.
SIGNOR-271885
O43521
P27361
0
phosphorylation
down-regulates quantity by destabilization
0.731
In vitro, bimel was phosphorylated by extracellular signal-regulated kinase on ser(69), which resides in the bimel-specific insert region. Using phosphospecific antibody against this site, we show that this residue is actually phosphorylated in cells. We also show that phosphorylation of ser(69) promotes ubiquitination of bimel. We conclude that mek inhibitors sensitize mda-mb231 and hbc4 cells to anoikis by blocking phosphorylation and hence degradation of bimel
SIGNOR-129878
P12882
Q14814
0
transcriptional regulation
up-regulates quantity by expression
0.34
Myocyte enhancer factor-2 and serum response factor binding elements regulate fast Myosin heavy chain transcription in vivo. We show that the upstream promoter region of the gene most abundantly expressed in mouse skeletal muscles, IIb MyHC, retains binding activity and transcriptional activation for three positive transcription factors, the serum response factor, Oct-1, and myocyte enhancer factor-2, whereas the other two genes (IIa and IId/x) have nucleotide substitutions in these sites that reduce binding and transcriptional activation
SIGNOR-238751
Q00535
P21860
1
phosphorylation
up-regulates activity
0.341
We demonstrated that Cdk5 phosphorylated Ser-1176 in the neuregulin receptor ErbB2 and phosphorylated Thr-871 and Ser-1120 in the ErbB3 receptor. We identified the Ser-1120 sequence RSRSPR in ErbB3 as a novel phosphorylation consensus sequence of Cdk5. Finally, we found that Cdk5 activity is involved in neuregulin-induced Akt activity and neuregulin-mediated neuronal survival. 
SIGNOR-250663
Q92985
Q9Y4K3
0
ubiquitination
up-regulates activity
0.732
We have shown that TRAF6 E3 ligase promotes IRF7 K63-linked ubiquitination that is required for EBV LMP1 activation of IRF7 [ xref ]; however, A20, a member with both E3 ligase and deubiquitinase activities in the OTU family, inhibits LMP1-stimulated IRF7 activity by acting as a deubiquitinase [ xref ].
SIGNOR-278788
P17676
P28482
0
phosphorylation
up-regulates
0.716
Phosphorylation of cebpb at thr(235) peaked at 16 hours in il-1beta-stimulated cells. The mek inhibitor u0126 inhibited this phosphorylation and reduced mmp-1 gene induction.
SIGNOR-187798
P62136
Q9BYX4
1
dephosphorylation
up-regulates activity
0.2
Exogenous PP1alpha or PP1gamma substantially decreased the S88 phosphorylation of Flag-MDA5|we identified PP1alpha and PP1gamma as primary phosphatases responsible for MDA5 and RIG-I dephosphorylation, leading to their activation.
SIGNOR-264577
Q13085
Q9NP71
0
transcriptional regulation
up-regulates quantity by expression
0.435
The present study provides evidence for a direct and dominant role of ChREBP in the glucose regulation of two key liver lipogenic enzymes, acetyl-CoA carboxylase (ACC) and fatty acid synthase (FAS)
SIGNOR-267946
P00519
P46527
1
phosphorylation
down-regulates quantity
0.595
A conserved tyrosine residue (Y88) in the Cdk-binding domain of p27 can be phosphorylated by the Src-family kinase Lyn and the oncogene product BCR-ABL
SIGNOR-245293
O15117
P06241
0
phosphorylation
up-regulates activity
0.2
 two tyrosines, Tyr595 and Tyr651, of FYB are major sites of phosphorylation by FYN-T and mediate binding to SLP-76 in Jurkat T cells. We further demonstrate that the loss of SLP-76 binding by mutation of these sites markedly reduced the ability of FYN-T-FYB-SLP-76 to up-regulate IL-2 transcription.
SIGNOR-251163
Q13322
P42345
0
phosphorylation
up-regulates
0.412
The adaptor protein grb10 was identified as an mtorc1 substrate that mediates the phosphoinositide 3-kinase.
SIGNOR-174071
Q15418
P19634
1
phosphorylation
up-regulates
0.452
The results indicate that p90rsk phosphorylates serine 703 of nhe-1, and this phosphorylation is required for growth factor stimulation of na+/h+ exchange.
SIGNOR-69171
Q9H410
Q8IWV8
0
ubiquitination
down-regulates quantity
0.2
Mub1 and Ubr2 mediate Dsn1 ubiquitylation and degradation.|The levels of WT Dsn1 were also increased in the mub1Delta and ubr2Delta strains, suggesting that Mub1 and Ubr2 mediate the degradation of WT Dsn1 protein.
SIGNOR-278795
P48729
O00257
1
phosphorylation
down-regulates quantity by destabilization
0.2
The phosphorylation of CBX4 at T437 by casein kinase 1α (CK1α) facilitated its ubiquitination at both K178 and K280 and subsequent degradation by CHIP, and this phosphorylation of CBX4 could be reduced by TNFα. 
SIGNOR-277512
P05455
P68400
0
phosphorylation
up-regulates
0.337
Prior studies indicate that hla is activated by phosphorylation of serine-366 by protein kinase ck2, neutralizing a negative effect of a short basic motif (sbm)
SIGNOR-160761
O14920
P19838
1
phosphorylation
down-regulates quantity by destabilization
0.853
Ikkbeta phosphorylates p105 resulting in its degradation, which releases tpl2 resulting in activation of the pro-proliferative map kinase- pathway.
SIGNOR-70473
P52954
O95863
1
transcriptional regulation
up-regulates quantity by expression
0.32
Compared with the empty vector, LBX1 induced increased promoter activity of threefold to fourfold and fivefold to sixfold for ZEB1 and Snail1, respectively 
SIGNOR-266053
P00533
P09874
1
phosphorylation
up-regulates activity
0.394
EGFR and MET heterodimer interacts with and phosphorylates PARP1.
SIGNOR-279169
Q9HC77
O00444
0
phosphorylation
up-regulates
0.799
Plk2 phosphorylates the s589 and s595 residues of cpap in vitro and in vivo. This phosphorylation is critical for procentriole formation during the centrosome cycle. Plk4 also phosphorylates s595 of cpap
SIGNOR-166007
P21127
O96017
0
phosphorylation
up-regulates activity
0.2
CHK2 kinase promotes pre-mRNA splicing via phosphorylating CDK11p110|Unexpectedly, CHK2 kinase constitutively phosphorylated CDK11(p110) in a DNA damage-independent manner.
SIGNOR-279458
Q96GD4
O95239
1
phosphorylation
up-regulates activity
0.517
Using in vitro kinase assays, we found that active AMPK and Aurora B phosphorylated KIF4A at Ser801 and Thr799 respectively in a time-dependent manner (Figure 5D). KIF4A is phosphoregulated by AMPK and Aurora B. Although AMPK phosphorylation increased the ATPase activity of KIF4A, Aurora B phosphorylation resulted in a stronger increase (Figure 5I), which might be consistent with the more powerful kinase function of Aurora B during mitosis.
SIGNOR-265992
P20810
Q8TDC3
0
phosphorylation
up-regulates activity
0.2
Here, we show that an AZ cytomatrix protein CAST and an AZ-associated protein kinase SAD-B coordinately regulate STD by controlling reloading of the AZ with release-ready synaptic vesicles. SAD-B phosphorylates the N-terminal serine (S45) of CAST, and S45 phosphorylation increases with higher firing rate.
SIGNOR-263051
Q02750
O15530
0
phosphorylation
up-regulates activity
0.27
In vitro kinase assay revealed that the direct targets of pdk1 in the mapk pathway were the upstream mapk kinases mek1 and mek2. The identified pdk1 phosphorylation sites in mek1 and mek2 are ser222 and ser226, respectively, and are known to be essential for full activation
SIGNOR-236633
P60953
Q96P48
0
gtpase-activating protein
down-regulates activity
0.531
We therefore developed a screening-compatible live-cell imaging assay, using FRET-based biosensors for the prototype GTPases RHOA, RAC1 and CDC4215,19,20 (Extended Data Fig. 2 and Supplementary Note 1)|We found catalytic activities for 45/75 RhoGEFs and 48/63 RhoGAPs| Our data thus not only reveal extensive promiscuity among regulators, but also that the inactivating RhoGAPs are less selective than the activating RhoGEFs (p-value=0.02)(Supplementary Table 2).
SIGNOR-260452
P52789
P01106
0
transcriptional regulation
up-regulates quantity
0.37
Here, using the P493-6 Burkitt's lymphoma model with an inducible MYC, we demonstrate that HIF-1 cooperates with dysregulated c-Myc to promote glycolysis by induction of hexokinase 2, which catalyzes the first step of glycolysis, and pyruvate dehydrogenase kinase 1, which inactivates pyruvate dehydrogenase and diminishes mitochondrial respiration.
SIGNOR-259986
P14373
O75385
1
ubiquitination
down-regulates quantity
0.2
STK38L ubiquitination promotes its activation and phosphorylation of ULK1 at Ser495, rendering ULK1 in a permissive state for TRIM27-mediated hyper-ubiquitination
SIGNOR-270349
P15172
Q09472
0
acetylation
up-regulates
0.681
Our results provide direct evidence that myod acetylation functionally activates the protein and show that both pcaf and cbp/p300 are candidate enzymes for myod acetylation in vivo.
SIGNOR-81053
Q15746
P28482
0
phosphorylation
up-regulates activity
0.478
Inhibition of ERK2 impaired phosphorylation of MLCK and insulin stimulated glucose uptake.|These findings suggest that ERK2 activates MLCK which then mediates the phosphorylation of RLC of MyoIIA.
SIGNOR-279226
P84077
Q9Y6D6
0
guanine nucleotide exchange factor
up-regulates activity
0.6
Brefeldin A-inhibited guanine nucleotide-exchange protein 1 (BIG1) is an approximately 200-kDa brefeldin A-inhibited guanine nucleotide-exchange protein that preferentially activates ADP-ribosylation factor 1 (ARF1) and ARF3. 
SIGNOR-272147
Q16621
Q96J02
0
ubiquitination
down-regulates activity
0.413
Itch regulates p45/NF-E2 in vivo by Lys63-linked ubiquitination| Interestingly, Itch suppressed the transactivation activity of p45/NF-E2 by adding a Lys63-linked polyubiquitin chain. Confocal microscopy revealed that ubiquitinated p45/NF-E2 became localized in the cytoplasm when Itch was over-expressed. Thus, Itch-mediated ubiquitination of p45/NF-E2 does not target the protein for proteasomal degradation, but instead retains p45/NF-E2 in the cytoplasm, where it cannot function as a transactivator.
SIGNOR-275553
P42229
P18031
0
dephosphorylation
down-regulates activity
0.676
A Cytosolic Protein-tyrosine Phosphatase PTP1B Specifically Dephosphorylates and Deactivates Prolactin-activated STAT5a and STAT5b
SIGNOR-248428
Q13153
P16949
1
phosphorylation
down-regulates
0.374
The hgf-induced wave2 transport, lamellipodia formation, stathmin/op18 phosphorylation at ser38 and binding to kinesin-wave2 complex, but not stathmin/op18 phosphorylation at ser25 and microtubule growth, were abrogated by pak1 inhibitor ipa-3
SIGNOR-183503
P20340
O43896
0
relocalization
up-regulates quantity
0.409
Here, we identify Bicaudal-D-related protein 1 (BICDR-1) as an effector of the small GTPase Rab6 and key component of the molecular machinery that controls secretory vesicle transport in developing neurons. BICDR-1 interacts with kinesin motor Kif1C, the dynein/dynactin retrograde motor complex, regulates the pericentrosomal localization of Rab6-positive secretory vesicles and is required for neural development in zebrafish. In young neurons, BICDR-1 accumulates Rab6 secretory vesicles around the centrosome, restricts anterograde secretory transport and inhibits neuritogenesis. Later during development, BICDR-1 expression is strongly reduced, which permits anterograde secretory transport required for neurite outgrowth. These results indicate an important role for BICDR-1 as temporal regulator of secretory trafficking during the early phase of neuronal differentiation.
SIGNOR-266877
P14138
P23276
0
cleavage
up-regulates activity
0.676
These data demonstrate that the Kell blood group protein is a proteolytic enzyme that processes big ET-3, generating ET-3, a potent bioactive peptide with multiple biological roles.
SIGNOR-256354
O60729
P25054
1
dephosphorylation
up-regulates
0.2
The phosphatase cdc14b translocates from the nucleolus to the nucleoplasm and induces the activation of the ubiquitin ligase apc/ccdh1
SIGNOR-179636
Q9BY78
Q13501
1
ubiquitination
up-regulates activity
0.358
SQSTM1 Is a Substrate for RNF26 and the DUB USP15. Catalytically competent RNF26 (light red) recruits SQSTM1 (blue) and mediates ubiquitin ligation (red), which serves to attract UBDs of specific vesicle-associated adaptors.
SIGNOR-269830
P46459
P43378
0
dephosphorylation
down-regulates
0.413
Our results suggest that the molecular mechanism by which ptp-meg2 promotes secretory vesicle fusion involves the local release of nsf from a tyrosine-phosphorylated, inactive state.
SIGNOR-128348
Q13535
Q8IUQ4
1
phosphorylation
down-regulates activity
0.2
We have also demonstrated that DNA damage triggers disruption of the HIPK2-Siah-1 complex, resulting in HIPK2 stabilization and activation. Disruption of the HIPK2-Siah-1 complex is mediated by the ATM/ATR pathway and involves ATM/ATR-dependent phosphorylation of Siah-1 at Ser 19.
SIGNOR-276167
P07948
Q16827
0
dephosphorylation
down-regulates activity
0.325
Both Lyn and ZAP70 were dephosphorylated by wild-type PTPROt, but not by its catalytic site mutant.|Lyn kinase and ZAP70 are substrates of PTPROt in B-cells: Lyn inactivation by PTPROt sensitizes leukemia cells to VEGF-R inhibitor pazopanib.
SIGNOR-277144
P12272
P24941
0
phosphorylation
down-regulates
0.372
Phosphorylation at the cyclin-dependent kinases site (thr85) of parathyroid hormone-related protein negatively regulates its nuclear localization
SIGNOR-68548
P15391
P00519
0
phosphorylation
up-regulates activity
0.533
The results revealed that only tyrosine (Y)490 of CD19 was phosphorylated by c-Abl.
SIGNOR-245283
Q8WV44
P05771
1
polyubiquitination
down-regulates quantity by destabilization
0.317
RINCK induces the ubiquitination of PKC both in vitro and in cells. Overexpression of RINCK reduces the levels of PKC in cells, whereas genetic knockdown of endogenous RINCK increases the levels of PKC. The RINCK-mediated ubiquitination is likely to be polyubiquitination, because the ubiquitinated PKCβII was detected as a high molecular weight smear.
SIGNOR-271667
Q9HCX3
P42772
1
transcriptional regulation
down-regulates quantity by repression
0.28
Finally, we show that ZNF304 also directs transcriptional silencing of INK4-ARF in human embryonic stem cells.
SIGNOR-266099
P09651
Q13148
0
post transcriptional regulation
up-regulates
0.406
TDP-43 regulates the alternative splicing of hnRNP A1 to yield an aggregation-prone variant in amyotrophic lateral sclerosis
SIGNOR-262824
P50750
P10412
1
phosphorylation
down-regulates activity
0.2
These data provide further evidence that CDK9 phosphorylates H1.4-S187, and that the level of pS187-H1.4 at genes is directly related to the extent of co-enrichment of CDK9.|that enrichment of pS187-H1.4 at genes is positively related to their transcription.
SIGNOR-279691
O14965
Q96R06
1
phosphorylation
up-regulates activity
0.511
We report here that Astrin acts as a mitotic phosphoprotein, and Aurora-A phosphorylates Astrin at Ser(115). The phosphorylation-deficient mutant Astrin S115A abnormally activates spindle assembly checkpoint and delays mitosis progression, decreases spindle stability, and induces chromosome misalignment. Mechanistic analyses reveal that Astrin phosphorylation mimicking mutant S115D, instead of S115A, binds and induces ubiquitination and degradation of securin, which sequentially activates Separase, an enzyme required for the separation of sister chromatids. 
SIGNOR-276648
Q07955
Q9HAZ1
0
phosphorylation
up-regulates activity
0.357
In vitro, Clk/Sty efficiently phosphorylated the SR family member ASF/SF2 on serine residues located within its serine/arginine-rich region (the RS domain). Overexpression of the active Clk/Sty kinase caused a redistribution of SR proteins within the nucleus. These results suggest that Clk/Sty kinase directly regulates the activity and compartmentalization of SR splicing factors.
SIGNOR-273860
Q8N695
P08047
0
transcriptional regulation
up-regulates quantity by expression
0.2
Luciferase reporter assays of deletion mutants of SLC5A8 promoter demonstrated that a 295-bp region is essential for the basal promoter activity of the SLC5A8 gene. Further analysis indicated that the CCAAT boxes and GC boxes were involved in positive regulation of SLC5A8 promoter. Overexpression of two transcription factors, CCAAT/enhancer binding protein beta (C/EBPbeta) and specific transcription factor 1 (Sp1), upregulated the activities of the human SLC5A8 promoter and protein expression, suggesting that both C/EBPbeta and Sp1 transcription factors might have functions in SLC5A8 transcription.
SIGNOR-254059
P06493
Q8NFH8
1
phosphorylation
down-regulates activity
0.34
Phosphorylation of POB1 and Epsin by p34cdc2 kinase. Their phosphorylation sites (Ser411 of POB1 and Ser357 of Epsin) were determined. Phosphorylated Epsin and EpsinS357D formed a complex with α-adaptin less efficiently than wild type Epsin.
SIGNOR-262724
P23467
P10912
1
dephosphorylation
down-regulates
0.295
Inally, mrna tissue distribution of these ptps by rt-pcr analysis and coexpression of the wild-type ptps to test their ability to dephosphorylate ligand-activated ghr suggest ptp-h1 and ptp1b as potential candidates involved in ghr signaling.
SIGNOR-104580
Q5TA31
Q9C029
0
ubiquitination
up-regulates quantity by stabilization
0.269
Taken together, these data suggest that Trim7 directly ubiquitinates RACO-1.
SIGNOR-278536
P31269
O00255
0
methylation
up-regulates quantity by expression
0.477
Men1 excision causes reduction of Hoxa9 expression, colony formation by hematopoietic progenitors, and the peripheral white blood cell count. Menin directly activates Hoxa9 expression, at least in part, by binding to the Hoxa9 locus, facilitating methylation of H3K4, and recruiting the methylated H3K4 binding protein chd1 to the locus. 
SIGNOR-255894
P51812
Q99683
1
phosphorylation
down-regulates activity
0.2
We provide evidence to show that RSK2 inhibits ASK1 by phosphorylating S83, T1109, and T1326 through a novel mechanism in which phospho-T1109/T1326 inhibits ATP binding to ASK1, while phospho-S83 attenuates ASK1 substrate MKK6 binding.
SIGNOR-276463
P08047
P28906
1
transcriptional regulation
up-regulates quantity by expression
0.263
Activation of the CD34 promoter by Sp1 requires the presence of a binding domain at -48 bp as well as the 5' untranslated region, which also binds Sp1
SIGNOR-241481
P30291
P53350
0
phosphorylation
down-regulates quantity by destabilization
0.642
 In the present study, we show that phosphorylation of S123 (pS123) by CDK promoted the binding of Wee1A to beta-TrCP through three independent mechanisms.  S123 phosphorylation creates a PBD-binding motif and accelerates S53 phosphorylation by Plk1.
SIGNOR-276040
P78527
P50876
0
polyubiquitination
down-regulates quantity by destabilization
0.568
We show that RNF144A induces ubiquitination of DNA-PKcs in vitro and in vivo and promotes its degradation.
SIGNOR-272213
P27707
P67775
0
dephosphorylation
down-regulates activity
0.2
Protein phosphatase 2A regulates deoxycytidine kinase activity via Ser-74 dephosphorylation|Deoxycytidine kinase (dCK) is a critical enzyme for activation of anticancer nucleoside analogs. Its activity is controlled via Ser-74 phosphorylation. Here, we investigated which Ser/Thr phosphatase dephosphorylates Ser-74. In cells, the PP1/PP2A inhibitor okadaic acid increased both dCK activity and Ser-74 phosphorylation
SIGNOR-275803