SIMBA / README.md
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metadata
language: en
license: cc-by-4.0
size_categories: 
pretty_name: Systematic Intracellular Motif-Binding Analysis
tags:
  - chemistry
  - biology
  - DMS
  - growth assay
  - competitive growth
dataset_summary: >-
  ' Transient protein-protein interactions play key roles in controlling dynamic
  cellular responses. Many examples involve globular protein domains that bind
  to peptide sequences known as short linear motifs (SLiMs), which are enriched
  in intrinsically disordered regions of proteins. Here we describe a novel
  functional assay for measuring SLiM binding, called systematic intracellular
  motif-binding analysis (SIMBA). In this method, binding of a foreign globular
  domain to its cognate SLiM peptide allows yeast cells to proliferate by
  blocking a growth arrest signal. A high-throughput application of the SIMBA
  method involving competitive growth and deep sequencing provides rapid
  quantification of the relative binding strength for thousands of SLiM sequence
  variants and a comprehensive interrogation of SLiM sequence features that
  control their recognition and potency. We show that multiple distinct classes
  of SLiM-binding domains can be analyzed by this method and that the relative
  binding strength of peptides in vivo correlates with their biochemical
  affinities measured in vitro. Deep mutational scanning provides
  high-resolution definitions of motif recognition determinants and reveals how
  sequence variations at noncore positions can modulate binding strength.
  Furthermore, mutational scanning of multiple parent peptides that bind human
  tankyrase ARC or YAP WW domains identifies distinct binding modes and uncovers
  context effects in which the preferred residues at one position depend on
  residues elsewhere. The findings establish SIMBA as a fast and incisive
  approach for interrogating SLiM recognition via massively parallel
  quantification of protein-peptide binding strength in vivo. '
dataset_description: Deep Mutational Scanning data for short linear motifs
repo: https://data.mendeley.com/datasets/nghf59hf4s/2
citation_bibtex: >-
  '@article{subbannaQuantitativeIntracellularPeptidebinding2025, title = {A
  Quantitative Intracellular Peptide-Binding Assay Reveals Recognition
  Determinants and Context Dependence of Short Linear Motifs}, author =
  {Subbanna, Mythili S. and Winters, Matthew J. and {\"O}rd, Mihkel and Davey,
  Norman E. and Pryciak, Peter M.}, year = 2025, month = mar, journal = {The
  Journal of Biological Chemistry}, volume = {301}, number = {3}, pages =
  {108225}, issn = {1083-351X}, doi = {10.1016/j.jbc.2025.108225}, abstract =
  {Transient protein-protein interactions play key roles in controlling dynamic
  cellular responses. Many examples involve globular protein domains that bind
  to peptide sequences known as short linear motifs (SLiMs), which are enriched
  in intrinsically disordered regions of proteins. Here we describe a novel
  functional assay for measuring SLiM binding, called systematic intracellular
  motif-binding analysis (SIMBA). In this method, binding of a foreign globular
  domain to its cognate SLiM peptide allows yeast cells to proliferate by
  blocking a growth arrest signal. A high-throughput application of the SIMBA
  method involving competitive growth and deep sequencing provides rapid
  quantification of the relative binding strength for thousands of SLiM sequence
  variants and a comprehensive interrogation of SLiM sequence features that
  control their recognition and potency. We show that multiple distinct classes
  of SLiM-binding domains can be analyzed by this method and that the relative
  binding strength of peptides in~vivo correlates with their biochemical
  affinities measured in~vitro. Deep mutational scanning provides
  high-resolution definitions of motif recognition determinants and reveals how
  sequence variations at noncore positions can modulate binding strength.
  Furthermore, mutational scanning of multiple parent peptides that bind human
  tankyrase ARC or YAP WW domains identifies distinct binding modes and uncovers
  context effects in which the preferred residues at one position depend on
  residues elsewhere. The findings establish SIMBA as a fast and incisive
  approach for interrogating SLiM recognition via massively parallel
  quantification of protein-peptide binding strength in~vivo.}, langid =
  {english}, pmcid = {PMC11879687}, pmid = {39864625}, keywords = {Amino Acid
  Motifs,cyclin-dependent kinase (CDK),high-throughput screening
  (HTS),Humans,intrinsically disordered protein,ligand-binding
  protein,mutagenesis,peptides,Peptides,phosphorylation,Protein Binding,protein
  motif,protein-protein interaction,Saccharomyces cerevisiae,Saccharomyces
  cerevisiae Proteins,yeast} }'
citation_apa: >-
  'Subbanna, M. S., Winters, M. J., Örd, M., Davey, N. E., & Pryciak, P. M.
  (2025). A quantitative intracellular peptide-binding assay reveals recognition
  determinants and context dependence of short linear motifs. The Journal of
  biological chemistry, 301(3), 108225.
  https://doi.org/10.1016/j.jbc.2025.108225'