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comprehensive weight loss maintenance\nprograms that provide at least monthlycontact with trained individuals and focuso no n g o i n gm o n i t o r i n go fb o d yw e i g h t(weekly or more frequently) and/or other\nself-monitoring strategies such as tracking
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self-monitoring strategies such as tracking\nintake, steps, etc.; continued focus on nu-trition and behavioral changes; and par-ticipation in high levels of physical activity\n(200–300 min/week) (63,64). Some com-\nmercial and proprietary weight loss pro-\ngrams have shown promising weight lossresults; however, results...
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programs, most lack evidence of effec-\ntiveness, many do not satisfy guidelinerecommendations, and some promoteunscienti fic and possibly dangerous prac-\ntices (65,66).\nStructured, very-low-calorie meals, typ-\nically 800 –1,000 kcal/day, utilizing high-
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ically 800 –1,000 kcal/day, utilizing high-\nprotein foods and meal replacementproducts, may increase the pace and/ormagnitude of initial weight loss and glyce-\nmic improvements compared with stan-\ndard behavioral interventions (20,21).However, such an intensive nutritional in-tervention should be provided only by
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trained practitioners in medical settings\nwith close ongoing monitoring and in-tegration with behavioral support andcounseling, and only for short term (gen-\nerally up to 3 months). Furthermore, due
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erally up to 3 months). Furthermore, due\nto the high risk of complications (electro-lyte abnormalities, severe fatigue, cardiacarrhythmias, etc.), such intensive inter-vention should be prescribed only to\ncarefully selected individuals, such as
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carefully selected individuals, such as\nthose requiring weight loss and/or gly-cemic management before a neededsurgery, if the bene fits exceed the po-\ntential risks (67 –69). As weight recur-\nrence is common, such interventionsshould include long-term, comprehen-sive weight maintenance strategies and\ncounseling to ...
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counseling to maintain weight loss and be-\nhavioral changes (70,71).\nDespite widespread marketing and ex-\norbitant claims, there is no clear evidencethat nutrition supplements (such as herbs\nand botanicals, high-dose vitamins and
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and botanicals, high-dose vitamins and\nm i n e r a l s ,a m i n oa c i d s ,e n z y m e s ,a n t i o x i -dants, etc.) are effective for obesity man-agement or weight loss (72 –75). Several\nlarge systematic reviews show that most\ntrials evaluating nutrition supplements
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trials evaluating nutrition supplements\nfor weight loss are of low quality and athigh risk for bias. High-quality publishedstudies show little or no weight loss bene-\nfits. In contrast, vitamin/mineral (e.g., iron,\nvitamin B12, vitamin D) supplementationmay be indicated in cases of documented\ndeficiency (76), and pro...
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deficiency (76), and protein supplements\nmay be indicated as adjuncts to medicallysupervised weight loss therapies (77,78).\nHealth disparities adversely affect peo-
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Health disparities adversely affect peo-\nple who have systematically experiencedgreater obstacles to health based on theirrace or ethnicity, socioeconomic status,gender, disability, or other factors. Over-whelming research shows that these dis-parities may signifi cantly affect health
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outcomes, including increasing the riskfor obesity, diabetes, and diabetes-relatedcomplications. Health care professionalsshould evaluate systemic, structural, andsocioeconomic factors that may impactfood choices, access to healthful foods,and nutrition patterns; behavioral pat-terns, such as neighborhood safety andava...
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such as neighborhood safety andavailability of safe outdoor spaces forphysical activity; environmental exposures;access to health care; social contexts; and,
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ultimately, diabetes risk and outcomes.\nFor a detailed discussion of social determi-nants of health, refer to “Social Determi-\nnants of Health: A Scienti ficR e v i e w ”(79).\nPHARMACOTHERAPY\nRecommendations\n8.14 Whenever possible, minimize\nmedications for comorbid conditions\nthat are associated with weight gain....
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that are associated with weight gain. E\n8.15 When choosing glucose-lowering\nmedications for people with type 2diabetes and overweight or obesity,\nprioritize medications with bene ficial\neffect on weight. B\n8.16 Obesity pharmacotherapy should\nbe considered for people with diabetes\nand overweight or obesity along w...
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and overweight or obesity along with\nlifestyle changes. Potential bene fits and\nrisks must be considered. A\n8.17 In people with diabetes and over-\nweight or obesity, the preferred phar-\nmacotherapy should be a glucagon-likepeptide 1 receptor agonist or dual glucose-\ndependent insulinotropic polypeptide
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dependent insulinotropic polypeptide\nand glucagon-like peptide 1 receptoragonist with greater weight loss ef fi-\ncacy (i.e., semaglutide or tirzepatide),especially considering their added\nweight-independent bene fits (e.g.,\nglycemic and cardiometabolic). A\n8.18 To prevent therapeutic inertia,\nfor those not reaching...
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for those not reaching goals, reevalu-\nate weight management therapiesand intensify treatment with addi-\ntional approaches (e.g., metabolicsurgery, additional pharmacologic\nagents, and structured lifestyle man-agement programs). A\nGlucose-Lowering Therapy\nNumerous effective glucose-lowering medi-
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Glucose-Lowering Therapy\nNumerous effective glucose-lowering medi-\ncations are currently available. However, toachieve both glycemic and weight manage-\nment goals for diabetes treatment, health\ncare professionals should prioritize the useof glucose-lowering medications with a ben-eficial effect on weight. Agents ass...
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with clinically meaningful weight loss in-\nclude glucagon-like peptide 1 (GLP-1) re-\nceptor agonists, dual glucose-dependentinsulinotropic polypeptide (GIP) and GLP-1receptor agonist (tirzepatide), sodium –\nglucose cotransporter 2 inhibitors, metfor-
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glucose cotransporter 2 inhibitors, metfor-\nmin, and amylin mimetics. Dipeptidyl pepti-dase 4 inhibitors, centrally acting dopamineagonist (bromocriptine), a-glucosidase in-\nhibitors, and bile acid sequestrants (colese-\nvelam) are considered weight neutral. In
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velam) are considered weight neutral. In\ncontrast, insulin secretagogues (sulfonylur-eas and meglitinides), thiazolidinediones,and insulin are often associated with weightgain (see Section 9, “Pharmacologic\nApproaches to Glycemic Treatment ”).\nConcomitant Medications\nHealth care professionals should carefully
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Concomitant Medications\nHealth care professionals should carefully\nreview the individual ’s concomitant medi-\ncations and, whenever possible, minimize\nor provide alternatives for medications\nthat promote weight gain. Examples of
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that promote weight gain. Examples of\nmedications associated with weight gaininclude antipsychotics (e.g., clozapine,olanzapine, risperidone), some antide-pressants (e.g., tricyclic antidepressants,\nsome selective serotonin reuptake inhibi-
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some selective serotonin reuptake inhibi-\ntors, and monoamine oxidase inhibitors),glucocorticoids, injectable progestins, someanticonvulsants (e.g., gabapentin and pre-gabalin), b-blockers, and possibly sedating\nantihistamines and anticholinergics (80).\nApproved Obesity Pharmacotherapy
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Approved Obesity Pharmacotherapy\nThe U.S. Food and Drug Administration(FDA) has approved several medications forweight management as adjuncts to reducedcalorie diet and increased physical activity\nin individuals with a BMI $30 kg/m\n2or\n$27 kg/m2w i t ho n eo rm o r eo b e s i t y -\nassociated comorbid conditions (...
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associated comorbid conditions (e.g., type 2\ndiabetes, hypertension, and/or dyslipide-mia). Nearly all FDA-approved obesity\nmedications have been shown to improve\nglycemia in people with type 2 diabetesS148 Obesity and Weight Management for Type 2 Diabetes Diabetes Care Volume 47, Supplement 1, January 2024\n©Americ...
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and delay progression to type 2 diabetes\nin at-risk individuals (22), and some ofthese agents (e.g., liraglutide and sema-glutide) have an indication for glucoselowering as well as weight management.\nPhentermine and other older adrenergic\nagents are approved for short-term treat-ment (#12 weeks) (81), while all othe...
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are approved for long-term treatment\n(>12 weeks) (22) ( Table 8.1 ). (Refer to\nSection 14, “Children and Adolescents, ”\nfor medications approved for adolescents\nwith obesity.) In addition, setmelanotide,\na melanocortin 4 receptor agonist, is ap-
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a melanocortin 4 receptor agonist, is ap-\nproved for use in cases of rare geneticmutations resulting in severe hyperphagiaand extreme obesity, such as leptin recep-tor de ficiency and proopiomelanocortin\ndeficiency.\nIn people with type 2 diabetes and
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deficiency.\nIn people with type 2 diabetes and\noverweight or obesity, agents with bothglucose-lowering and weight loss ef-fects are preferred (refer to Section 9,\n“Pharmacologic Approaches to Diabetes\nTreatment ”), which include agents from\nthe GLP-1 receptor agonist class and the\ndual GIP and GLP-1 receptor agoni...
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dual GIP and GLP-1 receptor agonist\nclass. Should use of these medications\nnot result in achievement of weightmanagement goals, or if they are not tol-erated or contraindicated, other obesity\ntreatment approaches should be consid-
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treatment approaches should be consid-\nered. Two phase 3 trials have demon-strated the potential for use of the dualGIP and GLP-1 receptor agonist (tirzepa-tide) for obesity (SURMOUNT-1, individuals\nwith obesity, and SURMOUNT-2, individuals
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with obesity, and SURMOUNT-2, individuals\nwith obesity and type 2 diabetes) (82,83).In the SURMOUNT-2 trial, tirzepatide re-sulted in body weight loss of 9.6% and\n11.6% more than placebo and A1C lower-\ning of 1.55% and 1.57% more than placeboafter 72 weeks of treatment with the 10mg and 15 mg doses, respectively, wi...
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adverse effects similar to those seen with\nthe GLP-1 receptor agonist class (83).\nHealth care professionals should be\nknowledgeable about the bene fits, dosing,\nand risks for each treatment option to bal-\nance the potential benefi ts of successful\nweight loss against the potential risks for
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weight loss against the potential risks for\neach individual. The high risk and preva-lence of cardiovascular disease in peoplewith diabetes has to be balanced against\nthe lack of long-term cardiovascular out-\ncomes trial data for agents like naltrexone-bupropion and phentermine-topiramate.All these medications are c...
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in individuals who are pregnant or actively\ntrying to conceive and are not recommendedfor use in individuals who are nursing. Indi-\nviduals of childbearing potential shouldreceive counseling regarding the use ofreliable methods of contraception. Ofnote, while weight loss medications are\no f t e nu s e di np e o p l ...
[ -0.05530443415045738, 0.010479075834155083, -0.07231749594211578, 0.015676064416766167, 0.003191200317814946, 0.0752188041806221, 0.04740799590945244, 0.12247702479362488, 0.0009504396002739668, 0.030362624675035477, 0.012847669422626495, 0.07790737599134445, -0.0662815049290657, 0.0127292...
o f t e nu s e di np e o p l ew i t ht y p e1d i a b e -\ntes, clinical trial data in this populationare limited.\nAssessing Efficacy and Safety of\nObesity Pharmacotherapy\nUpon initiating medications for obesity,\nassess their ef ficacy and safety at least\nmonthly for the first 3 months and at\nleast quarterly thereaf...
[ 0.016218120232224464, -0.016373194754123688, -0.060594502836465836, -0.007249202113598585, 0.020390145480632782, 0.03802330419421196, -0.05650351569056511, 0.12966810166835785, -0.004823732655495405, -0.0049723065458238125, 0.030067062005400658, 0.00857760664075613, 0.01021541003137827, 0....
least quarterly thereafter. Modeling from\npublished clinical trials consistently shows\nthat early responders have improvedlong-term outcomes (84,85); however, itis notable that the response rate with thelatest generation of obesity pharmaco-\ntherapies is much higher (48,83). Unless
[ 0.038853973150253296, 0.010659638792276382, -0.011154883541166782, 0.06682717055082321, -0.009996953420341015, 0.0163191556930542, -0.13706989586353302, 0.13627491891384125, 0.023265618830919266, -0.013604946434497833, 0.016184408217668533, 0.03729430213570595, -0.011203733272850513, -0.00...
therapies is much higher (48,83). Unless\nclinical circumstances (such as poor toler-ability) or other considerations (such as fi-\nnancial expense or individual preference)suggest otherwise, those who achieve\nsufficient early weight loss upon starting\na chronic obesity medication (typically de-\nfined as >5% weight los...
[ 0.02045232243835926, -0.01505574956536293, -0.028338847681879997, 0.03932950273156166, -0.036623869091272354, 0.0024189534597098827, -0.04780084267258644, 0.16600298881530762, -0.03974202275276184, -0.026803838089108467, -0.05983411520719528, 0.07746779918670654, -0.02000129036605358, 0.01...
fined as >5% weight loss after 3 months\nof use) should continue the medication\nlong term. When early weight loss results\nare modest (typically <5% weight loss af-\nter 3 months of use), the bene fits of on-\ngoing treatment need to be balanced in\nthe context of the glycemic response, the\navailability of other potent...
[ -0.00891935545951128, -0.020350299775600433, -0.027345983311533928, 0.018292834982275963, -0.037492651492357254, -0.033105868846178055, -0.014934878796339035, 0.13652832806110382, -0.033003613352775574, -0.04503075033426285, 0.0014835462206974626, 0.03494933620095253, -0.034779343754053116, ...
availability of other potential treatment\noptions, treatment tolerance, and overalltreatment burden.\nOngoing monitoring of the achievement\nand maintenance of weight managementgoals is recommended. For those not reach-\ning or maintaining weight-related treatment
[ -0.04036113619804382, 0.0701623484492302, -0.005888395942747593, 0.04335258901119232, -0.07079549133777618, 0.037873122841119766, -0.006030655466020107, 0.060182586312294006, -0.09015683829784393, -0.04385054484009743, 0.06239856779575348, -0.00737018883228302, -0.006509694270789623, 0.027...
ing or maintaining weight-related treatment\ngoals, reevaluate weight management ther-apies and intensify treatment with addi-tional approaches (e.g., metabolic surgery,\nadditional pharmacologic agents, and struc-\ntured lifestyle management programs).\nMEDICAL DEVICES FOR WEIGHT\nLOSS\nWhile gastric banding devices h...
[ -0.01451845932751894, 0.015785936266183853, -0.007421205751597881, 0.058466214686632156, -0.09018892049789429, -0.0020822964143007994, 0.02103690803050995, 0.09307944029569626, -0.06652435660362244, -0.04454934224486351, 0.028162769973278046, 0.016217811033129692, -0.04881131276488304, 0.0...
LOSS\nWhile gastric banding devices have fallen\nout of favor due to their limited long-term ef ficacy and high rate of complica-\ntions, several minimally invasive medicaldevices have been approved by the FDAfor short-term weight loss, including im-planted gastric balloons, a vagus nervestimulator, and gastric aspirati...
[ 0.002964877290651202, 0.03647886589169502, -0.00598467793315649, 0.05236300453543663, -0.02901027537882328, 0.0012429201742634177, 0.04413516819477081, 0.12516731023788452, -0.021755335852503777, -0.03209148719906807, -0.007767159957438707, 0.009311026893556118, -0.0527978241443634, -0.008...
(86). High cost, limited insurance coverage,\nand limited data supporting the effi cacy ofthese devices in the treatment of individu-\nals with diabetes has created uncertaintyfor their current use (87).\nAn oral hydrogel (cellulose and citric\nacid) has been approved for long-termuse in those with BMI >25 kg/m\n2to
[ -0.030327480286359787, 0.02669619768857956, 0.03844612091779709, -0.0013215031940490007, -0.08182276785373688, -0.0047178673557937145, 0.06639157235622406, 0.1483766734600067, -0.04297726973891258, -0.02307754009962082, -0.009458605200052261, 0.029475471004843712, -0.026450147852301598, -0...
2to\nsimulate the space-occupying effect ofimplantable gastric balloons. Taken withwater 30 min before meals, the hydrogelexpands to fill a portion of the stomach\nvolume to help decrease food intake dur-\ning meals. The average weight loss was
[ 0.001081752241589129, 0.033514123409986496, 0.04934101551771164, 0.027409907430410385, -0.054260365664958954, -0.04144078865647316, 0.05512451380491257, 0.07113471627235413, -0.026686642318964005, -0.04923689365386963, 0.018008893355727196, -0.04051024466753006, -0.012247774749994278, 0.00...
ing meals. The average weight loss was\nrelatively small (2.1% greater than pla-cebo), and very few participants had dia-betes at baseline ( /C2410%) (88).\nMETABOLIC SURGERY\nRecommendations\n8.19 Consider metabolic surgery as a\nweight and glycemic management ap-\nproach in people with diabetes with\nBMI$30.0 kg/m2(o...
[ 0.0075654820539057255, 0.11364491283893585, -0.010455366224050522, -0.014850670471787453, -0.08887376636266708, -0.048207804560661316, 0.004065553657710552, 0.0825691819190979, 0.00524902855977416, 0.008005043491721153, -0.018850786611437798, -0.009333529509603977, -0.07441071420907974, -0...
proach in people with diabetes with\nBMI$30.0 kg/m2(or$27.5 kg/m2in\nAsian American individuals) who areotherwise good surgical candidates. A\n8.20 Metabolic surgery should be\nperformed in high-volume centerswith interprofessional teams knowl-edgeable about and experienced\nin managing obesity, diabetes, and\ngastroin...
[ -0.023532142862677574, 0.07004514336585999, -0.013414693996310234, 0.023222964257001877, -0.07553660869598389, -0.057752326130867004, 0.05911358818411827, 0.05543805658817291, -0.013607209548354149, 0.032674696296453476, -0.025133229792118073, -0.006344256456941366, -0.06981242448091507, 0...
in managing obesity, diabetes, and\ngastrointestinal surgery (www.facs\n.org/quality-programs/accreditation-\nand-veri fication/metabolic-and-bariatric-\nsurgery-accreditation-and-quality-improvement-program/). E\n8.21 People being considered for met-\nabolic surgery should be evaluated forcomorbid psychological conditi...
[ 0.016094569116830826, 0.056954652070999146, -0.044805437326431274, 0.0020324026700109243, -0.08881824463605881, -0.01676037162542343, -0.00918254442512989, 0.025496706366539, -0.08827150613069534, -0.048062488436698914, -0.01210329681634903, 0.03603474795818329, -0.028554679825901985, -0.0...
social and situational circumstances\nthat have the potential to interfere\nwith surgery outcomes. B\n8.22 People who undergo metabolic\nsurgery should receive long-term med-\nical and behavioral support and rou-\ntine micronutrient, nutritional, and\nmetabolic status monitoring. B\n8.23 If post –metabolic surgery hypo...
[ -0.008539373986423016, 0.09469354897737503, -0.02051827870309353, -0.0062736086547374725, -0.05085734277963638, 0.0022263743449002504, -0.01423034816980362, 0.010923934169113636, -0.08196061104536057, -0.04249073937535286, 0.02527325041592121, 0.05861876159906387, -0.02748986706137657, 0.0...
8.23 If post –metabolic surgery hypogly-\ncemia is suspected, clinical evaluationshould exclude other potential disor-\nders contributing to hypoglycemia, and\nmanagement should include education,\nmedical nutrition therapy with a regis-\ntered dietitian/nutritionist experienced in\npost–metabolic surgery hypoglycemia,
[ -0.022376617416739464, 0.11431385576725006, 0.01436146255582571, 0.016179630532860756, -0.0091455252841115, -0.022615376859903336, -0.012685015797615051, 0.09420624375343323, -0.10527577996253967, -0.05971860885620117, -0.0011943442514166236, -0.016586916521191597, -0.09255656599998474, 0....
post–metabolic surgery hypoglycemia,\nand medication treatment, as needed.\nAContinuous glucose monitoring\nshould be considered as an importantadjunct to improve safety by alerting\nindividuals to hypoglycemia, especiallydiabetesjournals.org/care Obesity and Weight Management for Type 2 Diabetes S149\n©AmericanDiabete...
[ -0.027251476421952248, 0.09683413058519363, -0.029924454167485237, 0.03216953203082085, -0.04051671549677849, -0.016888316720724106, 0.03459085151553154, 0.05086646229028702, -0.07670286297798157, -0.020633846521377563, -0.03922220319509506, 0.05152638629078865, -0.07840273529291153, 0.029...
Table 8.1 —Obesity pharmacotherapy\nMedication name and\ntypical adult maintenance\ndoseAverage wholesale price\n(median and range for\n30-day supply) (142)National Average Drug\nAcquisition Cost\n(30-day supply) (143) Treatment armsWeight loss\n(% loss from\nbaseline)Common side effects(144–149)Possible safety concern...
[ -0.005224701017141342, 0.029045602306723595, 0.016738053411245346, 0.032217271625995636, 0.010058239102363586, 0.07829402387142181, 0.03946677967905998, 0.18414552509784698, -0.11457659304141998, 0.02141416259109974, -0.008107324130833149, -0.023779762908816338, 0.03259105235338211, -0.035...
Short-term treatment (12 weeks)\nSympathomimetic amine anorectic\nPhentermine (150)\n8–37.5 mg q.d.* $43 ($5 –$90),\n37.5 mg/day$2(37.5 mg dose)15 mg q.d.7.5 mg q.d.Placebo5.04.91.9Dry mouth, insomnia,\ndizziness, irritability,increased blood pressure,\nelevated heart rate/C15Contraindicated for use in\ncombination wit...
[ 0.053862065076828, -0.02795281819999218, -0.032874248921871185, 0.007930098101496696, -0.05005770921707153, 0.029687732458114624, -0.0016023783246055245, 0.12292375415563583, 0.0055608563125133514, 0.04856278374791145, -0.01208776980638504, -0.011819225735962391, -0.004222840070724487, 0.0...
combination with monoamineoxidase inhibitors\nLong-term treatment (52 or 56 weeks)\nLipase inhibitor\nOrlistat (4)60 mg t.i.d. (OTC)120 mg t.i.d. (Rx)$52 ($41 –$82)\n$843 ($781 –$904)NA$722120 mg t.i.d. †\nPlacebo9.65.6Abdominal pain, flatulence,
[ 0.04486294835805893, -0.009383061900734901, 0.022545455023646355, 0.04082246497273445, -0.05189213901758194, 0.02425384148955345, -0.025523679330945015, 0.15096792578697205, 0.042598623782396317, 0.04398181289434433, -0.03984701260924339, -0.00235163327306509, 0.015590154565870762, 0.05625...
Placebo9.65.6Abdominal pain, flatulence,\nfecal urgency/C15Potential malabsorption of fat-soluble vitamins (A, D, E, K) and ofcertain medications (e.g.,\ncyclosporine, thyroid hormone,\nanticonvulsants)\n/C15Rare cases of severe liver injury\nreported\n/C15Cholelithiasis\n/C15Nephrolithiasis\nSympathomimetic amine anore...
[ -0.021211422979831696, -0.05618807300925255, -0.03851184621453285, 0.0019863725174218416, -0.04216304421424866, -0.03796444460749626, -0.01514914445579052, 0.14442460238933563, 0.006920989137142897, -0.028768910095095634, -0.020126385614275932, -0.03868214040994644, -0.05205395072698593, 0...
Sympathomimetic amine anorectic/antiepileptic combination\nPhentermine/topiramate ER (47)\n7.5 mg/46 mg q.d. ‡ $223(7.5 mg/46 mg dose)$179(7.5 mg/46 mgdose)15 mg/92 mg q.d. §\n7.5 mg/46 mg q.d. §\nPlacebo9.87.81.2Constipation, paresthesia,\ninsomnia, nasopharyngitis,xerostomia, increased\nblood pressure/C15Contraindica...
[ 0.058204054832458496, -0.051552172750234604, -0.055183861404657364, -0.005645452532917261, -0.03705184534192085, -0.017184285447001457, 0.09505972266197205, 0.15873682498931885, 0.02786896750330925, 0.09923435002565384, -0.016018452122807503, -0.02066463604569435, -0.010959917679429054, 0....
blood pressure/C15Contraindicated for use in\ncombination with monoamineoxidase inhibitors\n/C15Birth defects\n/C15Cognitive impairment\n/C15Acute angle-closure glaucoma\nOpioid antagonist/antidepressant combination\nNaltrexone/bupropion ER (15)16 mg/180 mg b.i.d. $750 $599 16 mg/180 mg b.i.d.Placebo5.01.8Constipation,...
[ 0.005602648016065359, 0.005855152849107981, -0.05156485363841057, -0.006245572119951248, 0.012792621739208698, 0.009567185305058956, -0.025453457608819008, 0.17058435082435608, -0.01109002809971571, 0.019643409177660942, -0.025985443964600563, -0.046529509127140045, -0.06528767198324203, -...
headache, xerostomia,\ninsomnia, elevated heart\nrate and blood pressure/C15Contraindicated in people withunmanaged hypertension and/or\nseizure disorders\n/C15Contraindicated for use with\nchronic opioid therapy\n/C15Acute angle-closure glaucoma\nBlack box warning:/C15Risk of suicidal behavior/ideation in
[ -0.026647226884961128, 0.02506771869957447, -0.037221480160951614, 0.06570085883140564, 0.00939065683633089, 0.0009461486479267478, 0.04500385373830795, 0.09095992892980576, -0.04164699465036392, -0.017714859917759895, 0.017846515402197838, -0.04531421884894371, -0.0015323467087000608, 0.0...
Black box warning:/C15Risk of suicidal behavior/ideation in\npeople younger than 24 years oldwho have depression\nContinued on p. S151S150 Obesity and Weight Management for Type 2 Diabetes Diabetes Care Volume 47, Supplement 1, January 2024\n©AmericanDiabetesAssociation
[ -0.02434708923101425, 0.07046161592006683, -0.09809837490320206, 0.09323439002037048, 0.05141042545437813, 0.06043350324034691, 0.046151310205459595, 0.11576970666646957, -0.05140512436628342, -0.03420964255928993, -0.009487085975706577, -0.01885165460407734, -0.04501368850469589, -0.00302...
Table 8.1 —Continued\nMedication name and\ntypical adult maintenancedoseAverage wholesale price(median and range for30-day supply) (142)National Average DrugAcquisition Cost(30-day supply) (143) Treatment armsWeight loss(% loss frombaseline)Common side effects(144–149)Possible safety concerns andconsiderations (144–149...
[ 0.017782380804419518, 0.03456982597708702, 0.024169689044356346, -0.0008690130780451, -0.0001532838068669662, 0.06380578875541687, 0.030876418575644493, 0.21071502566337585, -0.12457737326622009, 0.019673913717269897, 0.01115445513278246, -0.016492241993546486, 0.049623169004917145, -0.009...
Glucagon-like peptide 1 receptor agonist\nLiraglutide (16,49) jj\n3 mg q.d. $1,619 $1,294 3.0 mg q.d.\n1.8 mg q.d.Placebo6.04.72.0Gastrointestinal side effects\n(nausea, vomiting,diarrhea, esophageal\nreflux), injection site\nreactions, elevated heart
[ 0.017121044918894768, -0.06242522597312927, -0.061813581734895706, 0.035353366285562515, -0.06173879653215408, -0.014941577799618244, 0.04372569918632507, 0.14385342597961426, 0.07295921444892883, 0.019569119438529015, -0.020296689122915268, 0.01808265782892704, -0.03409349545836449, 0.020...
reflux), injection site\nreactions, elevated heart\nrate, hypoglycemia/C15Pancreatitis has been reported inclinical trials, but causality has notbeen established. Discontinue if\npancreatitis is suspected.\n/C15Use caution in people with kidney\ndisease when initiating or increasingdose due to potential risk of acute\nk...
[ 0.006300562992691994, 0.004809112753719091, 0.01911100186407566, -0.009423754177987576, -0.034932274371385574, -0.03439321741461754, 0.004043196327984333, 0.09534909576177597, -0.012339558452367783, 0.02474578097462654, 0.007078649941831827, 0.021333105862140656, 0.01768629439175129, 0.051...
kidney injury.\n/C15May cause cholelithiasis and gallstone-\nrelated complications.\n/C15Gastrointestinal disorders (severeconstipation and small bowel\nobstruction/ileus progression)\n/C15Monitor for potential consequences of\ndelayed absorption of oral medications.\nBlack box warning:\n/C15Risk of thyroid C-cell tumo...
[ -0.028785299509763718, -0.01566126011312008, 0.022903140634298325, 0.011655713431537151, -0.029231896623969078, 0.0081727784126997, -0.017751792445778847, 0.11571613699197769, -0.03994216397404671, -0.03834705799818039, -0.05902761593461037, 0.004251593258231878, -0.05137064680457115, -0.0...
/C15Risk of thyroid C-cell tumors inrodents; human relevance not\ndetermined\nSemaglutide (48,151) jj\n2.4 mg once weekly $1,619 $1,295 2.4 mg weekly\n1.0 mg weekly\nPlacebo9.6\n7.0\n3.4Gastrointestinal side effects\n(nausea, vomiting,diarrhea, esophagealreflux), injection site\nreactions, elevated heart
[ -0.01140118483453989, 0.06465645879507065, -0.02022409997880459, -0.011116144247353077, 0.01600060425698757, 0.0422997921705246, 0.021902291104197502, 0.13304392993450165, 0.015959182754158974, 0.011224955320358276, -0.0811459869146347, -0.08097998797893524, 0.02659345045685768, -0.0911770...
reactions, elevated heart\nrate, hypoglycemia/C15Pancreatitis has been reported inclinical trials, but causality has not\nbeen established. Discontinue ifpancreatitis is suspected.\n/C15Use caution in people with kidney\ndisease when initiating or increasing\ndose due to potential risk of acutekidney injury.
[ -0.0034295159857720137, 0.01764814741909504, 0.023396672680974007, -0.0047636451199650764, 0.013164365664124489, -0.022703487426042557, 0.012997613288462162, 0.10331559181213379, -0.023528164252638817, 0.050922270864248276, 0.004152047913521528, 0.000042898427636828274, -0.019004812464118004...
dose due to potential risk of acutekidney injury.\n/C15May cause cholelithiasis and gallstone-related complications.\n/C15Gastrointestinal disorders (severeconstipation and small bowel\nobstruction/ileus progression)\n/C15Monitor for potential consequences of\ndelayed absorption of oral medications.
[ 0.027205519378185272, -0.06135199964046478, -0.005165286362171173, -0.004912863951176405, -0.03233896195888519, -0.05218496173620224, 0.008812607266008854, 0.1435946226119995, 0.013207059353590012, -0.048468217253685, 0.007495791185647249, 0.012434737756848335, -0.03686647489666939, 0.0539...
delayed absorption of oral medications.\nBlack box warning:/C15Risk of thyroid C-cell tumors in\nrodents; human relevance notdetermined\nContinued on p. S152diabetesjournals.org/care Obesity and Weight Management for Type 2 Diabetes S151\n©AmericanDiabetesAssociation
[ -0.015082476660609245, 0.039671335369348526, -0.07235673815011978, 0.020890790969133377, 0.008828007616102695, 0.01653362810611725, 0.013886037282645702, 0.07048340886831284, -0.030528409406542778, -0.027780843898653984, -0.0514841191470623, 0.03876723349094391, -0.014231076464056969, -0.0...
Table 8.1 —Continued\nMedication name and\ntypical adult maintenance\ndoseAverage wholesale price\n(median and range for\n30-day supply) (142)National Average Drug\nAcquisition Cost\n(30-day supply) (143) Treatment armsWeight loss\n(% loss from\nbaseline)Common side effects(144–149)Possible safety concerns andconsidera...
[ 0.0013349219225347042, 0.015993738546967506, 0.01879427395761013, 0.00318282563239336, 0.01218020636588335, 0.07809602469205856, 0.04039304703474045, 0.21143870055675507, -0.12618747353553772, 0.01843281462788582, 0.031014064326882362, -0.012831241823732853, 0.03010403737425804, -0.0097291...
Dual glucose-dependent insulinotropic polypeptide and glucagon-like peptide 1 receptor agonist\nTirzepatide (83)\n5 mg, 10 mg, or\n15 mg once weeklyNA NA 10 mg weekly15 mg weeklyPlacebo12.814.73.2Gastrointestinal side effects\n(nausea, vomiting,diarrhea, esophagealreflux), injection site
[ 0.03800778463482857, -0.03522013500332832, -0.08195765316486359, 0.039763566106557846, -0.03569626808166504, -0.0361117348074913, 0.08448928594589233, 0.08577663451433182, -0.030933838337659836, 0.004841991234570742, -0.060669489204883575, 0.02654057927429676, -0.041484534740448, -0.020022...
(nausea, vomiting,diarrhea, esophagealreflux), injection site\nreactions, elevated heartrate, hypoglycemia/C15Pancreatitis has been reported inclinical trials, but causality has notbeen established. Discontinue ifpancreatitis is suspected.
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/C15Use caution in people with kidneydisease when initiating or increasingdose due to potential risk of acutekidney injury.\n/C15May cause cholelithiasis andgallstone-related complications.\n/C15Gastrointestinal disorders (severe\nconstipation and small bowel\nobstruction/ileus progression)\n/C15Monitor effects of oral...
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/C15Monitor effects of oral medications\nwith narrow therapeutic index\n(warfarin) or whose ef ficacy is\ndependent on threshold\nconcentration.\n/C15Advise those using oral hormonal\ncontraception to use or add a non-\noral contraception method for4 weeks after initiation and doseescalations.
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Black box warning:/C15Risk of thyroid C-cell tumors inrodents; human relevance not\ndetermined.\nSelect safety and side effect information is provided; for a comprehensive discussion of safety considerations, please refer to the prescribing information for each agent. b.i.d., twice daily; ER, extended release;
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OTC, over the counter; NA, data not available; Rx, prescription; t.i.d., three times daily, p.o., by mouth; SC, subcutaneous injection; AWP, average wholesale price; NADAC, National Average Drug AcquisitionCost. *Use lowest effective dose; maximum appropriate dose is 37.5 mg. Weight loss data were extracted from the 12...
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mg. Weight loss data were extracted from the 12-week time point, as phentermine is approved for use for up to 12 weeks. †Enrolled partic-
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ipants had normal (79%) or impaired (21%) glucose tolerance. ‡Maximum dose, depending on response, is 15 mg/92 mg q.d. §Approximately 68% of enrolled participants had type 2 diabetes or impaired glu-
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cose tolerance. jjAgent has indication for reduction of cardiovascular events (49,151). AWP and NADAC prices for 30-day supply of maximum or maintenance dose as of 6 Sep tember 2023.S152 Obesity and Weight Management for Type 2 Diabetes Diabetes Care Volume 47, Supplement 1, January 2024\n©AmericanDiabetesAssociation
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for those with severe hypoglycemia\nor hypoglycemia unawareness. E\n8.24 In people who undergo metabolic\nsurgery, routinely screen for psychoso-cial and behavioral health changes and\nrefer to a quali fied behavioral health\nprofessional as needed. C\n8.25 Monitor individuals who have\nundergone metabolic surgery for i...
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8.25 Monitor individuals who have\nundergone metabolic surgery for in-\nsufficient weight loss or weight recur-\nrence at least every 6 –12 months. E\nIn those who have insuf ficient weight
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In those who have insuf ficient weight\nloss or experience weight recurrence,assess for potential predisposing fac-tors and, if appropriate, consider addi-tional weight loss interventions (e.g.,obesity pharmacotherapy). C\nSurgical procedures for obesity treat-\nment —often referred to interchangeably\nas bariatric surg...
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as bariatric surgery, weight loss surgery,\nmetabolic surgery, or metabolic/bariatric\nsurgery —can promote signi ficant and du-\nrable weight loss and improve type 2 dia-betes. Given the magnitude and rapidity\nof improvement of hyperglycemia and\nglucose homeostasis, these procedures\nhave been suggested as treatments...
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have been suggested as treatments for\ntype 2 diabetes even in the absence ofsevere obesity, hence the current pre-\nferred terminology of “metabolic sur-\ngery”(89).\nA substantial body of evidence, includ-\ning data from numerous large cohort\nstudies and randomized controlled (non-\nblinded) clinical trials, demonst...
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blinded) clinical trials, demonstrates that\nmetabolic surgery achieves superior gly-\ncemic management and reduction of car-\ndiovascular risk in people with type 2diabetes and obesity compared with non-\nsurgical intervention (45). In addition to\nimproving glycemia, metabolic surgery re-\nduces the incidence of micr...
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duces the incidence of microvascular dis-\nease (90), improves quality of life (45,91,92),\ndecreases cancer risk, and improves car-\ndiovascular disease risk factors and long-\nterm cardiovascular events (93 –104).\nCohort studies that match surgical andnonsurgical subjects strongly suggest that
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metabolic surgery reduces all-cause mor-tality (105,106).\nThe overwhelming majority of proce-\ndures in the U.S. are vertical sleeve gastrec-tomy (VSG) and Roux-en-Y gastric bypass\n(RYGB). Both procedures result in an ana-\ntomically smaller stomach pouch and often\nrobust changes in enteroendocrine hor-
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robust changes in enteroendocrine hor-\nmones. In VSG, /C2480% of the stomach isremoved, leaving behind a long, thin\nsleeve-shaped pouch. RYGB creates amuch smaller stomach pouch (roughlythe size of a walnut), which is thenattached to the distal small intestine,\nthereby bypassing the duodenum and\njejunum.\nMetabolic...
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jejunum.\nMetabolic surgery has been demon-\ns t r a t e dt oh a v eb e n e ficial effects on type 2\ndiabetes irrespective of the presurgicalBMI (107). The American Society for Met-abolic and Bariatric Surgery is now recom-mending metabolic surgery for people\nwith type 2 diabetes and a BMI $30 kg/m\n2\n(or$27.5 kg/m2f...
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2\n(or$27.5 kg/m2for Asian American indi-\nviduals) in surgically eligible individuals.\nStudies have documented diabetes remis-sion after 1 –5y e a r si n3 0 –63% of individ-\nuals with RYGB (17,108).\nMost notably, the Surgical Treatment\nand Medications Potentially Eradicate Di-abetes Ef ficiently (STAMPEDE) trial, w...
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randomized 150 participants with poorlymanaged diabetes to receive either meta-bolic surgery or medical treatment, foundthat 29% of those treated with RYGB and23% treated with VSG achieved A1C of\n6.0% or lower after 5 years (45). Available\ndata suggest an erosion of diabetes re-mission over time (46); at least 35 –50...
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of individuals who initially achieve remis-\nsion of diabetes eventually experience re-\ncurrence. Still, the median disease-freeperiod among such individuals followingRYGB is 8.3 years (109,110), and the major-\nity of those who undergo surgery maintain\nsubstantial improvement of glycemiafrom baseline for at least 5 ...
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(45,91,94,95,110– 113).\nExceedingly few presurgical predictors\nof success have been identi fied, but youn-\nger age, shorter duration of diabetes (e.g.,<8 years) (84), and lesser severity of dia-\nbetes (better glycemic control, not using\ninsulin) are associated with higher rates
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insulin) are associated with higher rates\nof diabetes remission (45,94,112,114).Greater baseline visceral fat area mayalso predict improved postoperative out-\ncomes, especially among Asian American\npeople with type 2 diabetes (115).\nAlthough surgery has been shown to\nimprove the metabolic pro files and car-
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improve the metabolic pro files and car-\ndiovascular risk of people with type 1 dia-\nbetes, larger and longer-term studies are\nneeded to determine the role of meta-bolic surgery in such individuals (116).\nWhereas metabolic surgery has greater\ninitial costs than nonsurgical obesity treat-\nments, retrospective analy...
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ments, retrospective analyses and model-\ning studies suggest that surgery may becost-effective or even cost-saving for indi-viduals with type 2 diabetes. However,these results largely depend on assump-tions about the long-term effectivenessand safety of the procedures (117,118).\nThe safety of metabolic surgery has im...
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The safety of metabolic surgery has im-\nproved signi ficantly with continued re fine-\nment of minimally invasive (laparoscopic)approaches, enhanced training andcredentialing, and involvement of inter-\nprofessional teams. Perioperative mortal-\nity rates are typically 0.1 –0.5%, similar to
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ity rates are typically 0.1 –0.5%, similar to\nthose of common abdominal proceduressuch as cholecystectomy or hysterectomy\n(119–123). Major complications occur in\n2–6% of those undergoing metabolic sur-\ngery, which compares favorably with the\nrates for other commonly performed elec-\ntive operations (123). Postsurg...
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tive operations (123). Postsurgical recovery\ntimes and morbidity have also dramaticallydeclined. Minor complications and needfor operative reintervention occur in upto 15% (119– 128). Empirical data suggest\nthat the pro ficiency of the operating sur-
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that the pro ficiency of the operating sur-\ngeon and surgical team is an important fac-tor in determining mortality, complications,reoperations, and readmissions (129). Ac-\ncordingly, metabolic surgery should be\nperformed in high-volume centers withinterprofessional teams experienced inmanaging diabetes, obesity, and...
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