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Concurrent administration of drugs possessing nephrotoxic (e.g., aminoglycosides, indomethacin), myelotoxic (e.g., cytotoxic chemotherapy), cardiotoxic (e.g., doxorubicin) or hepatotoxic (e.g., methotrexate, asparaginase) effects with PROLEUKIN may increase toxicity in these organ systems.
methotrexate
PROLEUKIN
EFFECT
Aldesleukin_ddi.xml
DDI-DrugBank.d114.s2
DDI-DrugBank.d114.s2.p19
The effectiveness of progestin-only pills is reduced by hepatic enzyme-inducing drugs such as the anticonvulsants phenytoin, carbamazepine, and barbiturates, and the antituberculosis drug rifampin.
progestin
barbiturates
EFFECT
Norethindrone_ddi.xml
DDI-DrugBank.d306.s0
DDI-DrugBank.d306.s0.p3
Drugs that have been reported to diminish oral anticoagulant response, ie, decreased prothrom-bin time response, in man significantly include: adrenocortical steroids;alcohol*;antacids;antihistamines;barbiturates;carbamazepine;chloral hydrate*;chlordiazepoxide;cholestyramine;diet high in vitamin K;diuretics*;ethchlorvynol;glutethimide;griseofulvin;haloperidol;meprobamate;oral contraceptives;paraldehyde;primidone;ranitidine*;rifampin;unreliable prothrombin time determinations;vitamin C;warfarin sodium under-dosage.
adrenocortical steroids
diuretics
NONE
Anisindione_ddi.xml
DDI-DrugBank.d64.s29
DDI-DrugBank.d64.s29.p32
In a study in which patients with active RA were treated for up to 24 weeks with concurrent Kineret and etanercept therapy, a 7% rate of serious infections was observed, which was higher than that observed with etanercept alone (0%).
Kineret
etanercept
EFFECT
Anakinra_ddi.xml
DDI-DrugBank.d275.s2
DDI-DrugBank.d275.s2.p0
Therefore, cyclosporine serum levels should be monitored and appropriate cyclosporine dosage adjustments made when these drugs are used concomitantly.
cyclosporine
cyclosporine
NONE
Norfloxacin_ddi.xml
DDI-DrugBank.d217.s4
DDI-DrugBank.d217.s4.p0
Diuretics: Patients on diuretics, especially those in whom diuretic therapy was recently instituted, may occasionally experience an excessive reduction of blood pressure after initiation of therapy with Lotensin.
diuretics
Lotensin
EFFECT
Benazepril_ddi.xml
DDI-DrugBank.d561.s0
DDI-DrugBank.d561.s0.p2
Co-medications that induce CYP 3A4 (e.g., rifampicin, phenytoin, carbamazepine, phenobarbital, or St. John s wort) may significantly decrease exposure to exemestane.
carbamazepine
exemestane
MECHANISM
Exemestane_ddi.xml
DDI-DrugBank.d435.s2
DDI-DrugBank.d435.s2.p8
Among the risk factors studied, two appear to increase the risk of ARE: the prescription of thiabendazole to treat strongyloidiasis during the melarsoprol cure and the bad general clinical conditions of patients.
thiabendazole
melarsoprol
EFFECT
7496198.xml
DDI-MedLine.d97.s8
DDI-MedLine.d97.s8.p0
Ergotamine: Concurrent use of erythromycin and ergotamine or dihydroergotamine has been associated in some patients with acute ergot toxicity characterized by severe peripheral vasospasm and dysesthesia.
erythromycin
ergotamine
EFFECT
Dirithromycin_ddi.xml
DDI-DrugBank.d522.s23
DDI-DrugBank.d522.s23.p3
Other drugs which may enhance the neuromuscular blocking action of nondepolarizing agents such as NUROMAX include certain antibiotics (e. g., aminoglycosides, tetracyclines, bacitracin, polymyxins, lincomycin, clindamycin, colistin, and sodium colistimethate), magnesium salts, lithium, local anesthetics, procainamide, and quinidine.
NUROMAX
colistin
EFFECT
Doxacurium chloride_ddi.xml
DDI-DrugBank.d267.s5
DDI-DrugBank.d267.s5.p7
Bentiromide may interact with acetaminophen (e.g., Tylenol), chloramphenicol (e.g., Chloromycetin), local anesthetics (e.g., benzocaine and lidocaine), para-aminobenzoic acid (PABA)-containing preparations (e.g., sunscreens and some multivitamins), procainamide (e.g., Pronestyl), sulfonamides (sulfa medicines), thiazide diuretics (use of these medicines during the test period will affect the test results), and pancreatic supplements (use of pancreatic supplements may give false test results).
Bentiromide
procainamide
INT
Bentiromide_ddi.xml
DDI-DrugBank.d537.s0
DDI-DrugBank.d537.s0.p10
The gradual withdrawal of guafacine or a cardioselective beta-blocker could be substituted.
guafacine
cardioselective beta-blocker
NONE
Guanfacine_ddi.xml
DDI-DrugBank.d507.s7
DDI-DrugBank.d507.s7.p0
Intestinal adsorbents (e. g., charcoal) and digestive enzyme preparations containing carbohydrate-splitting enzymes (e. g., amylase, pancreatin) may reduce the effect of Acarbose and should not be taken concomitantly.
Intestinal adsorbents
digestive enzyme preparations
NONE
Acarbose_ddi.xml
DDI-DrugBank.d536.s4
DDI-DrugBank.d536.s4.p1
Thyroid Physiology: The following agents may alter thyroid hormone or TSH levels, generally by effects on thyroid hormone synthesis, secretion, distribution, metabolism, hormone action, or elimination, or altered TSH secretion: aminoglutethimide, p-aminosalicylic acid, amiodarone, androgens and related anabolic hormones, complex anions (thiocyanate, perchlorate, pertechnetate), antithyroid drugs, b-adrenergic blocking agents, carbamazepine, chloral hydrate, diazepam, dopamine and dopamine agonists, ethionamide, glucocorticoids, heparin, hepatic enzyme inducers, insulin, iodinated cholestographic agents, iodine-containing compounds, levodopa, lovastatin, lithium, 6-mercaptopurine, metoclopramide, mitotane, nitroprusside, phenobarbital, phenytoin, resorcinol, rifampin, somatostatin analogs, sulfonamides, sulfonylureas, thiazide diuretics.
carbamazepine
6-mercaptopurine
NONE
Levothyroxine_ddi.xml
DDI-DrugBank.d411.s4
DDI-DrugBank.d411.s4.p273
Marked symptomatic orthostatic hypotension has been reported when calcium channel blockers and organic nitrates were used in combination.
calcium channel blockers
nitrates
EFFECT
Nitroglycerin_ddi.xml
DDI-DrugBank.d14.s2
DDI-DrugBank.d14.s2.p0
The potential effects of increased plasma concentrations of midazolam or other benzodiazepines metabolized via CYP3A4 (alprazolam, triazolam) should be considered when coadministering these agents with Aprepitant.
alprazolam
Aprepitant
ADVISE
Aprepitant_ddi.xml
DDI-DrugBank.d382.s23
DDI-DrugBank.d382.s23.p8
Cholestyramine resin may delay or reduce the absorption of concomitant oral medication such as phenylbutazone, warfarin, thiazide diuretics (acidic) or propranolol (basic), as well as tetracycline penicillin G, phenobarbital, thyroid and thyroxine preparations, estrogens and progestins, and digitalis.
Cholestyramine
warfarin
MECHANISM
Cholestyramine_ddi.xml
DDI-DrugBank.d566.s0
DDI-DrugBank.d566.s0.p2
Coumarin Anticoagulants: In a small clinical trial in which lovastatin was administered to warfarin treated patients, no effect on prothrombin time was detected.
Coumarin Anticoagulant
lovastatin
NONE
Lovastatin_ddi.xml
DDI-DrugBank.d567.s13
DDI-DrugBank.d567.s13.p0
The extent to which SSRI-TCAinteractions may pose clinical problems will depend on the degree of inhibition and the pharmacokinetics of the SSRI involved.
SSRI
TCA
INT
Clomipramine_ddi.xml
DDI-DrugBank.d238.s18
DDI-DrugBank.d238.s18.p0
The use of MAO inhibitors or tricyclic antidepressants with hydrocodone preparations may increase the effect of either the antidepressant or hydrocodone.
hydrocodone
hydrocodone
NONE
Hydrocodone_ddi.xml
DDI-DrugBank.d396.s2
DDI-DrugBank.d396.s2.p8
Therefore, it is recommended that Fluvoxamine Tablets not be used in combination with MAOIs, or within 14 days of discontinuing treatment with a MAOI.
Fluvoxamine
MAOIs
ADVISE
Fluvoxamine_ddi.xml
DDI-DrugBank.d76.s4
DDI-DrugBank.d76.s4.p0
Before taking glimepiride, tell your doctor if you are taking any of the following medicines: - aspirin or another salicylate such as magnesium/choline salicylate (Trilisate), salsalate (Disalcid, others), choline salicylate (Arthropan), magnesium salicylate (Magan), or bismuth subsalicylate (Pepto-Bismol);
glimepiride
Magan
ADVISE
Glimepiride_ddi.xml
DDI-DrugBank.d521.s1
DDI-DrugBank.d521.s1.p9
Erythromycin has been reported to decrease the clearance of triazolam and midazolam and thus may increase the pharmacologic effect of these benzodiazepines.
midazolam
benzodiazepines
NONE
Erythromycin_ddi.xml
DDI-DrugBank.d397.s6
DDI-DrugBank.d397.s6.p5
Increased toxicity (CNS depression): CNS depressants, MAO inhibitors, tricyclic antidepressants, phenothiazines.
MAO inhibitors
phenothiazines
NONE
Chlorpheniramine_ddi.xml
DDI-DrugBank.d235.s2
DDI-DrugBank.d235.s2.p4
Agents Increasing Serum Potassium: PRINIVIL attenuates potassium loss caused by thiazide-type diuretics.
PRINIVIL
thiazide-type diuretics
EFFECT
Lisinopril_ddi.xml
DDI-DrugBank.d334.s14
DDI-DrugBank.d334.s14.p0
Consequently, concomitant administration of Aprepitant with strong CYP3A4 inhibitors (e.g., ketoconazole, itraconazole, nefazodone, troleandomycin, clarithromycin, ritonavir, nelfinavir) should be approached with caution.
Aprepitant
troleandomycin
ADVISE
Aprepitant_ddi.xml
DDI-DrugBank.d382.s31
DDI-DrugBank.d382.s31.p3
- Phenothiazines (acetophenazine [e.g., Tindal], chlorpromazine [e.g., Thorazine], fluphenazine [e.g., Prolixin], mesoridazine [e.g., Serentil], perphenazine [e.g., Trilafon], prochlorperazine [e.g., Compazine], promazine [e.g., Sparine], promethazine [e.g., Phenergan], thioridazine [e.g., Mellaril], trifluoperazine [e.g., Stelazine], triflupromazine [e.g., Vesprin], trimeprazine [e.g., Temaril]) or
Thorazine
Serentil
NONE
Sulfapyridine_ddi.xml
DDI-DrugBank.d179.s21
DDI-DrugBank.d179.s21.p93
DIAMOX modifies phenytoin metabolism with increased serum levels of phenytoin.
DIAMOX
phenytoin
MECHANISM
Acetazolamide_ddi.xml
DDI-DrugBank.d368.s0
DDI-DrugBank.d368.s0.p0
Drugs that have been reported to diminish oral anticoagulant response, ie, decreased prothrom-bin time response, in man significantly include: adrenocortical steroids;alcohol*;antacids;antihistamines;barbiturates;carbamazepine;chloral hydrate*;chlordiazepoxide;cholestyramine;diet high in vitamin K;diuretics*;ethchlorvynol;glutethimide;griseofulvin;haloperidol;meprobamate;oral contraceptives;paraldehyde;primidone;ranitidine*;rifampin;unreliable prothrombin time determinations;vitamin C;warfarin sodium under-dosage.
anticoagulant
primidone
EFFECT
Anisindione_ddi.xml
DDI-DrugBank.d64.s29
DDI-DrugBank.d64.s29.p18
Warfarin: Increased INR (International Normalized Ratio) when ARAVA and warfarin were co-administered has been rarely reported.
ARAVA
warfarin
EFFECT
Leflunomide_ddi.xml
DDI-DrugBank.d41.s16
DDI-DrugBank.d41.s16.p2
However, LDL-C reduction was greater when atorvastatin and colestipol were coadministered than when either drug was given alone.
atorvastatin
colestipol
EFFECT
Atorvastatin_ddi.xml
DDI-DrugBank.d140.s5
DDI-DrugBank.d140.s5.p0
Psychoactive Drugs: Hallucinations have been reported when TORADOL was used in patients taking psychoactive drugs (fluoxetine, thiothixene, alprazolam).
TORADOL
alprazolam
EFFECT
Ketorolac_ddi.xml
DDI-DrugBank.d3.s19
DDI-DrugBank.d3.s19.p8
When estrogen therapy is initiated, a reduction in corticosteroid dosage may be required, and increased amounts may be required when estrogen is terminated.
estrogen
corticosteroid
NONE
Fludrocortisone_ddi.xml
DDI-DrugBank.d526.s24
DDI-DrugBank.d526.s24.p0
The most commonly occurring drug interactions are listed below: - Drugs that may increase plasma phenytoin concentrations include: acute alcohol intake, amiodarone, chboramphenicol, chlordiazepoxide, cimetidine, diazepam, dicumarol, disulfiram, estrogens, ethosuximide, fluoxetine, H2-antagonists, halothane, isoniazid, methylphenidate, phenothiazines, phenylbutazone, salicylates, succinimides, sulfonamides, tolbutamide, trazodone
phenothiazines
succinimides
NONE
Fosphenytoin_ddi.xml
DDI-DrugBank.d40.s10
DDI-DrugBank.d40.s10.p212
Other Cardiovascular Agents: Enalapril and enalapril IV have been used concomitantly with beta adrenergic-blocking agents, methyldopa, nitrates, calcium-blocking agents, hydralazine, prazosin and digoxin without evidence of clinically significant adverse interactions.
nitrates
hydralazine
NONE
Enalapril_ddi.xml
DDI-DrugBank.d107.s10
DDI-DrugBank.d107.s10.p27
Agents that have been found, or are expected to have decreased plasma levels in the presence of EQUETROTM due to induction of CYP enzymes are the following: Acetaminophen, alprazolam, amitriptyline, bupropion, buspirone, citalopram, clobazam, clonazepam, clozapine, cyclosporin, delavirdine, desipramine, diazepam, dicumarol, doxycycline, ethosuximide, felbamate, felodipine, glucocorticoids, haloperidol, itraconazole, lamotrigine, levothyroxine, lorazepam, methadone, midazolam, mirtazapine, nortriptyline, olanzapine, oral contraceptives(3), oxcarbazepine, Phenytoin(4), praziquantel, protease inhibitors, quetiapine, risperidone, theophylline, topiramate, tiagabine, tramadol, triazolam, valproate, warfarin(5) , ziprasidone, and zonisamide.
EQUETROTM
desipramine
MECHANISM
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s11
DDI-DrugBank.d94.s11.p11
In patients receiving nonselective monoamine oxidase inhibitors (MAOIs) (e.g., selegiline hydrochloride) in combination with serotoninergic agents (e.g., fluoxetine, fluvoxamine, paroxetine, sertraline, venlafaxine), there have been reports of serious, sometimes fatal, reactions.
selegiline hydrochloride
fluvoxamine
EFFECT
Dexfenfluramine_ddi.xml
DDI-DrugBank.d423.s0
DDI-DrugBank.d423.s0.p17
The results indicate that a spinal naloxone-sensitive endorphinergic system is involved in the production of beta-endorphin but not morphine-induced tail-flick inhibition, and suggest that intraventricular beta-endorphin and morphine elicit their pharmacological actions via the activation of different descending pain inhibitory systems;
naloxone
beta-endorphin
NONE
3155550.xml
DDI-MedLine.d63.s7
DDI-MedLine.d63.s7.p2
Thyroid Physiology: The following agents may alter thyroid hormone or TSH levels, generally by effects on thyroid hormone synthesis, secretion, distribution, metabolism, hormone action, or elimination, or altered TSH secretion: aminoglutethimide, p-aminosalicylic acid, amiodarone, androgens and related anabolic hormones, complex anions (thiocyanate, perchlorate, pertechnetate), antithyroid drugs, b-adrenergic blocking agents, carbamazepine, chloral hydrate, diazepam, dopamine and dopamine agonists, ethionamide, glucocorticoids, heparin, hepatic enzyme inducers, insulin, iodinated cholestographic agents, iodine-containing compounds, levodopa, lovastatin, lithium, 6-mercaptopurine, metoclopramide, mitotane, nitroprusside, phenobarbital, phenytoin, resorcinol, rifampin, somatostatin analogs, sulfonamides, sulfonylureas, thiazide diuretics.
pertechnetate
lithium
NONE
Levothyroxine_ddi.xml
DDI-DrugBank.d411.s4
DDI-DrugBank.d411.s4.p197
Increasing the felbamate dose to 2400 mg/day increased the steadystate valproate Cmin to 96 25 micrograms/mL.
felbamate
valproate
MECHANISM
Felbamate_ddi.xml
DDI-DrugBank.d434.s25
DDI-DrugBank.d434.s25.p0
Verapamil also significantly decreased the incidence of lymphatic invasion of adenocarcinomas, which was enhanced by bombesin.
Verapamil
bombesin
EFFECT
11125235.xml
DDI-MedLine.d133.s5
DDI-MedLine.d133.s5.p0
Other drugs which may enhance the neuromuscular blocking action of nondepolarizing agents such as NIMBEX include certain antibiotics (e. g., aminoglycosides, tetracyclines, bacitracin, polymyxins, lincomycin, clindamycin, colistin, and sodium colistemethate), magnesium salts, lithium, local anesthetics, procainamide, and quinidine.
NIMBEX
antibiotics
EFFECT
Cisatracurium Besylate_ddi.xml
DDI-DrugBank.d60.s12
DDI-DrugBank.d60.s12.p15
Levothyroxine Sodium Absorption: The following agents may bind and decrease absorption of levothyroxine sodium from the gastrointestinal tract: aluminum hydoxide, cholestyramine resin, colestipol hydrochloride, ferrous sulfate, sodium polystyrene sulfonate, soybean flour (e.g., infant formula), sucralfate.
levothyroxine sodium
ferrous sulfate
MECHANISM
Levothyroxine_ddi.xml
DDI-DrugBank.d411.s2
DDI-DrugBank.d411.s2.p10
Co-administration of bosentan decreased the plasma concentrations of cyclosporine A (a CYP3A4 substrate) by approximately 50%.
bosentan
cyclosporine A
MECHANISM
Bosentan_ddi.xml
DDI-DrugBank.d289.s13
DDI-DrugBank.d289.s13.p0
Because of the danger of a potentially fatal prolongation of the QTc interval, halofantrine must not be given simultaneously with or subsequent to Mefloquine.
halofantrine
Mefloquine
ADVISE
Mefloquine_ddi.xml
DDI-DrugBank.d220.s4
DDI-DrugBank.d220.s4.p0
Naproxen and other NSAIDs can reduce the antihypertensive effect of propranolol and other beta-blockers.
Naproxen
propranolol
EFFECT
Naproxen_ddi.xml
DDI-DrugBank.d85.s9
DDI-DrugBank.d85.s9.p1
Drugs that may alter imatinib plasma concentrations Drugs that may increase imatinib plasma concentrations: Caution is recommended when administering Gleevec with inhibitors of the CYP3A4 family (e.g., ketoconazole, itraconazole, erythromycin, clarithromycin).
Gleevec
ketoconazole
ADVISE
Imatinib_ddi.xml
DDI-DrugBank.d115.s0
DDI-DrugBank.d115.s0.p11
Concurrent administration of drugs possessing nephrotoxic (e.g., aminoglycosides, indomethacin), myelotoxic (e.g., cytotoxic chemotherapy), cardiotoxic (e.g., doxorubicin) or hepatotoxic (e.g., methotrexate, asparaginase) effects with PROLEUKIN may increase toxicity in these organ systems.
asparaginase
PROLEUKIN
EFFECT
Aldesleukin_ddi.xml
DDI-DrugBank.d114.s2
DDI-DrugBank.d114.s2.p20
Drugs that reportedly may increase oral anticoagulant response, ie, increased prothrombin response, in man include:alcohol*;allopurinol;aminosalicylic acid;amiodarone;anabolic steroids;antibiotics;bromelains;chloral hydrate*;chlorpropamide;chymotrypsin;cimetidine;cinchophen;clofibrate;dextran;dextrothyroxine;diazoxide;dietary deficiencies;diflunisal;disulfiram;drugs affecting blood elements;ethacrynic acid;fenoprofen;glucagon;hepatotoxic drugs;ibuprofen;indomethacin;influenza virus vaccine;inhalation anesthetics;mefenamic acid;methyldopa;methylphenidate;metronidazole;miconazole;monoamine oxidase inhibitors;nalidixic acid;naproxen;oxolinic acid;oxyphenbutazone;pentoxifylline;phenylbutazone;phenyramidol;phenytoin;prolonged hot weather;prolonged narcotics;pyrazolones;quinidine;quinine;ranitidine*;salicylates;sulfinpyrazone;sulfonamides, long acting;sulindac;thyroid drugs;tolbutamide;triclofos sodium;trimethoprim/sulfamethoxazole;unreliable prothrombin time determinations;warfarin sodium overdosage.
anticoagulant
aminosalicylic acid
EFFECT
Anisindione_ddi.xml
DDI-DrugBank.d64.s87
DDI-DrugBank.d64.s87.p2
In patients receiving nonselective monoamine oxidase inhibitors (MAOIs) (e.g., selegiline hydrochloride) in combination with serotoninergic agents (e.g., fluoxetine, fluvoxamine, paroxetine, sertraline, venlafaxine), there have been reports of serious, sometimes fatal, reactions.
MAOIs
venlafaxine
EFFECT
Dexfenfluramine_ddi.xml
DDI-DrugBank.d423.s0
DDI-DrugBank.d423.s0.p14
Barbiturates may decrease the effectiveness of oral contraceptives, certain antibiotics, quinidine, theophylline, corticosteroids, anticoagulants, and beta blockers.
Barbiturates
beta blockers
EFFECT
Hexobarbital_ddi.xml
DDI-DrugBank.d457.s0
DDI-DrugBank.d457.s0.p6
Uricosuric Agents: Aspirin may decrease the effects of probenecid, sulfinpyrazone, and phenylbutazone.
Aspirin
probenecid
EFFECT
Aspirin_ddi.xml
DDI-DrugBank.d443.s0
DDI-DrugBank.d443.s0.p4
Carbamazepine: Felbatol causes a decrease in the steady-state carbamazepine plasma concentrations and an increase in the steady-state carbamazepine epoxide plasma concentration.
Felbatol
carbamazepine
MECHANISM
Felbamate_ddi.xml
DDI-DrugBank.d434.s17
DDI-DrugBank.d434.s17.p3
There is a significant increase in exposure to imatinib when Gleevec is coadministered with ketoconazole (CYP3A4 inhibitor).
Gleevec
ketoconazole
MECHANISM
Imatinib_ddi.xml
DDI-DrugBank.d115.s2
DDI-DrugBank.d115.s2.p2
SUBUTEX and SUBOXONE should be prescribed with caution to patients on benzodiazepines or other drugs that act on the central nervous system, regardless of whether these drugs are taken on the advice of a physician or are taken as drugs of abuse.
SUBUTEX
benzodiazepines
ADVISE
Buprenorphine_ddi.xml
DDI-DrugBank.d380.s6
DDI-DrugBank.d380.s6.p1
Concomitant administration of FACTIVE and calcium carbonate, cimetidine, omeprazole, or an estrogen/progesterone oral contraceptive produced minor changes in the pharmacokinetics of gemifloxacin, which were considered to be without clinical significance.
estrogen
contraceptive
NONE
Gemifloxacin_ddi.xml
DDI-DrugBank.d347.s1
DDI-DrugBank.d347.s1.p23
Therefore, MAO inhibitors should be discontinued at least two weeks prior to the cautious initiation of therapy with SINEQUAN.
MAO inhibitors
SINEQUAN
ADVISE
Doxepin_ddi.xml
DDI-DrugBank.d223.s20
DDI-DrugBank.d223.s20.p0
Intrathecal injection of naloxone at doses of 0.4 to 40 micrograms caused a dose-related blockade of the inhibition of the tail-flick response induced by intraventricular injection of beta-endorphin, and a high dose of naloxone (40 micrograms) completely blocked the tail-flick inhibition induced by intraventricular beta-endorphin (16 micrograms).
naloxone
beta-endorphin
EFFECT
3155550.xml
DDI-MedLine.d63.s4
DDI-MedLine.d63.s4.p0
Chirocaine should be used with caution in patients receiving other local anesthetics or agents structurally related to amide-type local anesthetics since the toxic effects of these drugs could be additive.
Chirocaine
anesthetics
ADVISE
Levobupivacaine_ddi.xml
DDI-DrugBank.d320.s0
DDI-DrugBank.d320.s0.p0
Etonogestrel may interact with the following medications: acetaminophen (Tylenol), antibiotics such as ampicillin and tetracycline, anticonvulsants (Dilantin, Phenobarbital, Tegretol, Trileptal, Topamax, Felbatol), antifungals (Gris-PEG, Nizoral, Sporanox), atorvastatin (Lipitor), clofibrate (Atromid-S), cyclosporine (Neoral, Sandimmune), HIV drugs classified as protease inhibitors (Agenerase, Crixivan, Fortovase, Invirase, Kaletra, Norvir, Viracept), morphine (Astramorph, Kadian, MS Contin), phenylbutazone, prednisolone (Prelone), rifadin (rifampin), St. Johns wort, temazepam, theophylline (Theo-Dur), and vitamin C.
Etonogestrel
atorvastatin
INT
Etonogestrel_ddi.xml
DDI-DrugBank.d484.s0
DDI-DrugBank.d484.s0.p16
Other drugs Drug interactions have been reported with concomitant administration of erythromycin and other medications, including cyclosporine, hexobarbital, carbamazepine, alfentanil, disopyramide, phenytoin, bromocriptine, valproate, astemizole, and lovastatin.
erythromycin
phenytoin
INT
Dirithromycin_ddi.xml
DDI-DrugBank.d522.s24
DDI-DrugBank.d522.s24.p5
Thyroid Physiology: The following agents may alter thyroid hormone or TSH levels, generally by effects on thyroid hormone synthesis, secretion, distribution, metabolism, hormone action, or elimination, or altered TSH secretion: aminoglutethimide, p-aminosalicylic acid, amiodarone, androgens and related anabolic hormones, complex anions (thiocyanate, perchlorate, pertechnetate), antithyroid drugs, b-adrenergic blocking agents, carbamazepine, chloral hydrate, diazepam, dopamine and dopamine agonists, ethionamide, glucocorticoids, heparin, hepatic enzyme inducers, insulin, iodinated cholestographic agents, iodine-containing compounds, levodopa, lovastatin, lithium, 6-mercaptopurine, metoclopramide, mitotane, nitroprusside, phenobarbital, phenytoin, resorcinol, rifampin, somatostatin analogs, sulfonamides, sulfonylureas, thiazide diuretics.
thiocyanate
dopamine agonists
NONE
Levothyroxine_ddi.xml
DDI-DrugBank.d411.s4
DDI-DrugBank.d411.s4.p134
Drugs That Inhibit Both Aldehyde Oxidase and CYP3A4 Cimetidine: Cimetidine inhibits both aldehyde oxidase (in vitro) and CYP3A4 (in vitro and in vivo), the primary and secondary enzymes, respectively, responsible for zaleplon metabolism.
Cimetidine
zaleplon
MECHANISM
Zaleplon_ddi.xml
DDI-DrugBank.d324.s28
DDI-DrugBank.d324.s28.p2
Azlocillin should not be administered concomitantly with amikacin, ciprofloxacin, gentamicin, netilmicin, or tobramycin.
Azlocillin
gentamicin
ADVISE
Azlocillin_ddi.xml
DDI-DrugBank.d9.s0
DDI-DrugBank.d9.s0.p2
ACE-inhibitors:Reports suggest that NSAIDs may diminish the antihypertensive effect of ACE-inhibitors.
ACE-inhibitors
NSAIDs
NONE
Valdecoxib_ddi.xml
DDI-DrugBank.d328.s8
DDI-DrugBank.d328.s8.p0
5-HT3 antagonists: In clinical drug interaction studies, aprepitant did not have clinically important effects on the pharmacokinetics of ondansetron or granisetron.
5-HT3 antagonists
aprepitant
NONE
Aprepitant_ddi.xml
DDI-DrugBank.d382.s7
DDI-DrugBank.d382.s7.p0
The increase of phenobarbital level, however, is small (15%) when given with Trileptal.
phenobarbital
Trileptal
MECHANISM
Oxcarbazepine_ddi.xml
DDI-DrugBank.d307.s37
DDI-DrugBank.d307.s37.p0
Ibandronate should be taken at least 60 minutes before any oral medications containing multivalent cations (including antacids, supplements or vitamins).
Ibandronate
vitamins
ADVISE
Ibandronate_ddi.xml
DDI-DrugBank.d440.s2
DDI-DrugBank.d440.s2.p1
Nephrotoxic agents : Concomitant administration of VISTIDE and agents with nephrotoxic potential [e.g., intravenous aminoglycosides (e.g., tobramycin, gentamicin, and amikacin), amphotericin B, foscarnet, intravenous pentamidine, vancomycin, and non-steroidal anti-inflammatory agents] is contraindicated.
VISTIDE
amphotericin B
ADVISE
Cidofovir_ddi.xml
DDI-DrugBank.d260.s3
DDI-DrugBank.d260.s3.p4
Interactions with Other CNS Agents: Concurrent use of Levo-Dromoran with all central nervous system depressants (eg, alcohol, sedatives, hypnotics, other opioids, general anesthetics, barbiturates, tricyclic antidepressants, phenothiazines, tranquilizers, skeletal muscle relaxants and antihistamines) may result in additive central nervous system depressant effects.
Levo-Dromoran
barbiturates
EFFECT
Levorphanol_ddi.xml
DDI-DrugBank.d257.s0
DDI-DrugBank.d257.s0.p6
Agents that are CYP inducers that have been found, or are expected, to decrease plasma levels of EQUETROTM are the following: Cisplatin, doxorubicin HCL, felbamate, rifampin, phenobarbital, Phenytoin(2), primidone, methsuximide, and theophylline Thus, if a patient has been titrated to a stable dosage on EQUETROTM, and then begins a course of treatment with one of these CYP3A4 inducers, it is reasonable to expect that a dose increase for EQUETROTM may be necessary.
rifampin
primidone
NONE
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s8
DDI-DrugBank.d94.s8.p40
Because busulfan is eliminated from the body via conjugation with glutathione, use of acetaminophen prior to ( 72 hours) or concurrent with BUSULFEX may result in reduced busulfan clearance based upon the known property of acetaminophen to decrease glutathione levels in the blood and tissues.
acetaminophen
BUSULFEX
MECHANISM
Busulfan_ddi.xml
DDI-DrugBank.d72.s4
DDI-DrugBank.d72.s4.p4
Other drugs which may enhance the neuromuscular blocking action of nondepolarizing agents such as NIMBEX include certain antibiotics (e. g., aminoglycosides, tetracyclines, bacitracin, polymyxins, lincomycin, clindamycin, colistin, and sodium colistemethate), magnesium salts, lithium, local anesthetics, procainamide, and quinidine.
nondepolarizing agents
bacitracin
EFFECT
Cisatracurium Besylate_ddi.xml
DDI-DrugBank.d60.s12
DDI-DrugBank.d60.s12.p4
Interactions with Other CNS Agents: Concurrent use of Levo-Dromoran with all central nervous system depressants (eg, alcohol, sedatives, hypnotics, other opioids, general anesthetics, barbiturates, tricyclic antidepressants, phenothiazines, tranquilizers, skeletal muscle relaxants and antihistamines) may result in additive central nervous system depressant effects.
Levo-Dromoran
tricyclic antidepressants
EFFECT
Levorphanol_ddi.xml
DDI-DrugBank.d257.s0
DDI-DrugBank.d257.s0.p7
Digoxin - In subjects who had received 21 days of 40 mg/day racemic citalopram, combined administration of citalopram and digoxin (single dose of 1 mg) did not significantly affect the pharmacokinetics of either citalopram or digoxin.
citalopram
digoxin
NONE
Escitalopram_ddi.xml
DDI-DrugBank.d568.s9
DDI-DrugBank.d568.s9.p6
Dexbrompheniramine can interact with alcohol or other CNS depressants (may potentiate the CNS depressant effects of either these medications or antihistamines), anticholinergics or other medications with anticholinergic activity (anticholinergic effects may be potentiated when these medications are used concurrently with antihistamines), and monoamine oxidase (MAO) inhibitors (concurrent use with antihistamines may prolong and intensify the anticholinergic and CNS depressant effects of antihistamines).
Dexbrompheniramine
alcohol
EFFECT
Brompheniramine_ddi.xml
DDI-DrugBank.d192.s0
DDI-DrugBank.d192.s0.p0
Cholestyramine and Charcoal Administration of cholestyramine or activated charcoal in patients (n=13) and volunteers (n=96) resulted in a rapid and significant decrease in plasma M1 (the active metabolite of leflunomide) concentration .
cholestyramine
leflunomide
MECHANISM
Leflunomide_ddi.xml
DDI-DrugBank.d41.s0
DDI-DrugBank.d41.s0.p8
In addition, results from regression analyses of patient pharmacokinetic data suggest that co-administration of other inducers of drug clearance (efavirenz, nevirapine, phenytoin, dexamethasone, or carbamazepine) with CANCIDAS may result in clinically meaningful reductions in caspofungin concentrations.
carbamazepine
CANCIDAS
MECHANISM
Caspofungin_ddi.xml
DDI-DrugBank.d350.s12
DDI-DrugBank.d350.s12.p18
Codeine in combination with other narcotic analgesics, general anesthetics, phenothiazines, tranquilizers, sedative-hypnotics, or other CNS depressants (including alcohol) has additive depressant effects.
tranquilizers
sedative-hypnotics
NONE
Codeine_ddi.xml
DDI-DrugBank.d464.s0
DDI-DrugBank.d464.s0.p22
Spontaneous reports of serotonin syndrome associated with co-administration of ZYVOX and serotonergic agents, including antidepressants such as selective serotonin reuptake inhibitors (SSRIs), have been reported.
ZYVOX
selective serotonin reuptake inhibitors
EFFECT
Linezolid_ddi.xml
DDI-DrugBank.d441.s6
DDI-DrugBank.d441.s6.p2
When therapeutic concentrations of furosemide, propranolol, dipyridamole, warfarin, quinidine, or naproxen were added to human plasma (in vitro), the plasma protein binding of nicardipine HCl was not altered.
propranolol
naproxen
NONE
Nicardipine_ddi.xml
DDI-DrugBank.d468.s10
DDI-DrugBank.d468.s10.p9
May interact with thyroid medication (e.g., levothyroxine), iodine-containing products, antacids, H2-antagonists (e.g., famotidine, ranitidine), and proton pump inhibitors (e.g., lansoprazole, omeprazole).
ranitidine
lansoprazole
NONE
Diatrizoate_ddi.xml
DDI-DrugBank.d293.s0
DDI-DrugBank.d293.s0.p31
Since apraclonidine may reduce pulse and blood pressure, caution in using drugs such as beta-blockers (ophthalmic and systemic), antihypertensives, and cardiac glycosides is advised.
apraclonidine
antihypertensives
ADVISE
Apraclonidine_ddi.xml
DDI-DrugBank.d224.s8
DDI-DrugBank.d224.s8.p1
Intraventricular injection of naloxone at doses of 1.2 to 12 micrograms equally antagonized in a dose-dependent manner the tail-flick inhibition induced by intraventricular beta-endorphin and morphine.
naloxone
beta-endorphin
EFFECT
3155550.xml
DDI-MedLine.d63.s6
DDI-MedLine.d63.s6.p0
Agents that are CYP3A4 inhibitors that have been found, or are expected, to increase plasma levels of EQUETROTM are the following: Acetazolamide, azole antifungals, cimetidine, clarithromycin(1), dalfopristin, danazol, delavirdine, diltiazem, erythromycin(1), fluoxetine, fluvoxamine, grapefruit juice, isoniazid, itraconazole, ketoconazole, loratadine, nefazodone, niacinamide, nicotinamide, protease inhibitors, propoxyphene, quinine, quinupristin, troleandomycin, valproate(1), verapamil, zileuton.
EQUETROTM
fluoxetine
MECHANISM
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s4
DDI-DrugBank.d94.s4.p9
May interact with the following: beta-adrenergic blocking agents (these medicines may make your condition worse and prevent the adrenergic bronchodilators from working properly) and disopyramide, quinidine, phenothiazines, and procainamide (these medicines may increase the risk of heart problems).
beta-adrenergic blocking agents
procainamide
NONE
Isoetharine_ddi.xml
DDI-DrugBank.d541.s0
DDI-DrugBank.d541.s0.p4
- Phenothiazines (acetophenazine [e.g., Tindal], chlorpromazine [e.g., Thorazine], fluphenazine [e.g., Prolixin], mesoridazine [e.g., Serentil], perphenazine [e.g., Trilafon], prochlorperazine [e.g., Compazine], promazine [e.g., Sparine], promethazine [e.g., Phenergan], thioridazine [e.g., Mellaril], trifluoperazine [e.g., Stelazine], triflupromazine [e.g., Vesprin], trimeprazine [e.g., Temaril]) or
chlorpromazine
Temaril
NONE
Sulfapyridine_ddi.xml
DDI-DrugBank.d179.s21
DDI-DrugBank.d179.s21.p89
Therefore, co-administration of bupropion with drugs that are metabolized by CYP2D6 isoenzyme including certain antidepressants (e.g., nortriptyline, imipramine, desipramine, paroxetine, fluoxetine, sertraline), antipsychotics (e.g., haloperidol, risperidone, thioridazine), beta-blockers (e.g., metoprolol), and Type 1C antiarrhythmics (e.g., propafenone, flecainide), should be approached with caution and should be initiated at the lower end of the dose range of the concomitant medication.
bupropion
thioridazine
ADVISE
Bupropion_ddi.xml
DDI-DrugBank.d5.s17
DDI-DrugBank.d5.s17.p10
Carbamazepine: Isoniazid is known to slow the metabolism of carbamazepine and increase its serum levels Carbamazepine levels should be determined prior to concurrent administration with isoniazid, signs and symptoms of carbamazepine toxicity should be monitored closely, and appropriate dosage adjustment of the anticonvulsant should be made.
Isoniazid
carbamazepine
MECHANISM
Isoniazid_ddi.xml
DDI-DrugBank.d187.s7
DDI-DrugBank.d187.s7.p6
Magnesium: Magnesium-containing preparations (eg, antacids) may cause hypermagnesemia and should therefore not be taken during therapy with vitamin D by patients on chronic renal dialysis.
antacids
vitamin D
EFFECT
Cholecalciferol_ddi.xml
DDI-DrugBank.d404.s15
DDI-DrugBank.d404.s15.p5
Hydrochlorothiazide: In normal volunteers, concomitant administration of diflunisal and hydrochlorothiazide resulted in significantly increased plasma levels of hydrochlorothiazide.
Hydrochlorothiazide
hydrochlorothiazide
NONE
Diflunisal_ddi.xml
DDI-DrugBank.d132.s5
DDI-DrugBank.d132.s5.p1
Warfarin: Anticoagulant activity should be monitored, particularly in the first few days after initiating or changing VIOXX therapy in patients receiving warfarin or similar agents, since these patients are at an increased risk of bleeding complications.
VIOXX
warfarin
ADVISE
Rofecoxib_ddi.xml
DDI-DrugBank.d210.s31
DDI-DrugBank.d210.s31.p2
Antidiabetics: Because corticosteroids may increase blood glucose concentrations, dosage adjustments of antidiabetic agents may be required.
corticosteroids
antidiabetic agents
EFFECT
Dexamethasone_ddi.xml
DDI-DrugBank.d314.s8
DDI-DrugBank.d314.s8.p2
In vitro displacement studies with highly protein-bound drugs such as furosemide, propranolol, captopril, nicardipine, pravastatin, glyburide, warfarin, phenytoin, acetylsalicylic acid, tolbutamide, and metformin showed no influence on the extent of nateglinide protein binding.
propranolol
phenytoin
NONE
Nateglinide_ddi.xml
DDI-DrugBank.d460.s11
DDI-DrugBank.d460.s11.p16
Since PLETAL is extensively metabolized by cytochrome P-450 isoenzymes, caution should be exercised when PLETAL is coadministered with inhibitors of C.P.A. such as ketoconazole and erythromycin or inhibitors of CYP2C19 such as omeprazole.
ketoconazole
erythromycin
NONE
Cilostazol_ddi.xml
DDI-DrugBank.d358.s0
DDI-DrugBank.d358.s0.p7
Drugs that reportedly may increase oral anticoagulant response, ie, increased prothrombin response, in man include:alcohol*;allopurinol;aminosalicylic acid;amiodarone;anabolic steroids;antibiotics;bromelains;chloral hydrate*;chlorpropamide;chymotrypsin;cimetidine;cinchophen;clofibrate;dextran;dextrothyroxine;diazoxide;dietary deficiencies;diflunisal;disulfiram;drugs affecting blood elements;ethacrynic acid;fenoprofen;glucagon;hepatotoxic drugs;ibuprofen;indomethacin;influenza virus vaccine;inhalation anesthetics;mefenamic acid;methyldopa;methylphenidate;metronidazole;miconazole;monoamine oxidase inhibitors;nalidixic acid;naproxen;oxolinic acid;oxyphenbutazone;pentoxifylline;phenylbutazone;phenyramidol;phenytoin;prolonged hot weather;prolonged narcotics;pyrazolones;quinidine;quinine;ranitidine*;salicylates;sulfinpyrazone;sulfonamides, long acting;sulindac;thyroid drugs;tolbutamide;triclofos sodium;trimethoprim/sulfamethoxazole;unreliable prothrombin time determinations;warfarin sodium overdosage.
chlorpropamide
anesthetics
NONE
Anisindione_ddi.xml
DDI-DrugBank.d64.s87
DDI-DrugBank.d64.s87.p465
Consequently, the effect of iron on the retention of cobalt was lower than on absorption.
iron
cobalt
MECHANISM
7599505.xml
DDI-MedLine.d34.s9
DDI-MedLine.d34.s9.p0
The results of a study of coadministration of ethambutol (50 mg/kg) with an aluminum hydroxide containing antacid to 13 patients with tuberculosis showed a reduction of mean serum concentrations and urinary excretion of ethambutol of approximately 20% and 13%, respectively, suggesting that the oral absorption of ethambutol may be reduced by these antacid products.
ethambutol
aluminum hydroxide
MECHANISM
Ethambutol_ddi.xml
DDI-DrugBank.d160.s0
DDI-DrugBank.d160.s0.p0
Due to its nephrotoxicity, gentamicin may cause abnormal renal uptake to be seen on 99mTc-MDP bone scintigraphy.
gentamicin
99mTc-MDP
MECHANISM
7632757.xml
DDI-MedLine.d89.s2
DDI-MedLine.d89.s2.p0
Videx (Didanosine) chewable/buffered tablets or the pediatric powder for oral solution should not be administered concomitantly with, or within 2 hours of, the administration of norfloxacin, because these products may interfere with absorption resulting in lower serum and urine levels of norfloxacin.
Didanosine
norfloxacin
ADVISE
Norfloxacin_ddi.xml
DDI-DrugBank.d217.s12
DDI-DrugBank.d217.s12.p3