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plasma levels of several closely related tricyclic antidepressants have been reported to be increased by the concomitant administration of methylphenidate or hepatic enzyme inhibitors (e.g., cimetidine, fluoxetine) and decreased by the concomitant administration of hepatic enzyme inducers (e.g., barbiturates, phenytoin), and such an effect may be anticipated with CMI as well.
methylphenidate
cimetidine
NONE
Clomipramine_ddi.xml
DDI-DrugBank.d238.s6
DDI-DrugBank.d238.s6.p6
Agents that have been found, or are expected to have decreased plasma levels in the presence of EQUETROTM due to induction of CYP enzymes are the following: Acetaminophen, alprazolam, amitriptyline, bupropion, buspirone, citalopram, clobazam, clonazepam, clozapine, cyclosporin, delavirdine, desipramine, diazepam, dicumarol, doxycycline, ethosuximide, felbamate, felodipine, glucocorticoids, haloperidol, itraconazole, lamotrigine, levothyroxine, lorazepam, methadone, midazolam, mirtazapine, nortriptyline, olanzapine, oral contraceptives(3), oxcarbazepine, Phenytoin(4), praziquantel, protease inhibitors, quetiapine, risperidone, theophylline, topiramate, tiagabine, tramadol, triazolam, valproate, warfarin(5) , ziprasidone, and zonisamide.
clonazepam
delavirdine
NONE
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s11
DDI-DrugBank.d94.s11.p334
Dopamine Antagonists: Since apomorphine is a dopamine agonist, it is possible that dopamine antagonists, such as the neuroleptics (phenothiazines, butyrophenones, thioxanthenes) or metoclopramide, may diminish the effectiveness of APOKYN.
metoclopramide
APOKYN
EFFECT
Apomorphine_ddi.xml
DDI-DrugBank.d357.s3
DDI-DrugBank.d357.s3.p44
When indinavir at an increased dose (1000 mg every 8 hours) was given with SUSTIVA (600 mg once daily), the indinavir AUC and Cmin were decreased on average by 33-46% and 39-57%, respectively, compared to when indinavir (800 mg every 8 hours) was given alone.
indinavir
SUSTIVA
MECHANISM
Efavirenz_ddi.xml
DDI-DrugBank.d531.s39
DDI-DrugBank.d531.s39.p0
The hypoglycemic action of sulfonylureas may be potentiated by certain drugs including nonsteroidal anti-inflammatory agents and other drugs that are highly protein bound, salicylates, sulfonamides, chloramphenicol, probenecid, coumarins, monoamine oxidase inhibitors, and beta adrenergic blocking agents.
sulfonylureas
monoamine oxidase inhibitors
EFFECT
Glibenclamide_ddi.xml
DDI-DrugBank.d178.s0
DDI-DrugBank.d178.s0.p6
Diuretic: Hydrochlorothiazide, given concomitantly with ketoprofen, produces a reduction in urinary potassium and chloride excretion compared to hydrochlorothiazide alone.
Diuretic
Hydrochlorothiazide
NONE
Ketoprofen_ddi.xml
DDI-DrugBank.d499.s10
DDI-DrugBank.d499.s10.p0
Therefore, CYP3A4 substrates known to have a narrow therapeutic index such as alfentanil, astemizole, terfenadine, cisapride, cyclosporine, fentanyl, pimozide, quinidine, sirolimus, tacrolimus, or ergot alkaloids (ergotamine, dihydroergotamine) should be administered with caution in patients receiving SPRYCEL.
cyclosporine
SPRYCEL
ADVISE
Dasatinib_ddi.xml
DDI-DrugBank.d48.s15
DDI-DrugBank.d48.s15.p54
and phase 3 (days 45-52): cisapride 10 mg 4 times/day (days 45-51) plus fluoxetine 20 mg/day (days 45-52).
cisapride
fluoxetine
NONE
11213850.xml
DDI-MedLine.d46.s9
DDI-MedLine.d46.s9.p0
The most commonly occurring drug interactions are listed below: - Drugs that may increase plasma phenytoin concentrations include: acute alcohol intake, amiodarone, chboramphenicol, chlordiazepoxide, cimetidine, diazepam, dicumarol, disulfiram, estrogens, ethosuximide, fluoxetine, H2-antagonists, halothane, isoniazid, methylphenidate, phenothiazines, phenylbutazone, salicylates, succinimides, sulfonamides, tolbutamide, trazodone
cimetidine
trazodone
NONE
Fosphenytoin_ddi.xml
DDI-DrugBank.d40.s10
DDI-DrugBank.d40.s10.p94
The absorption of oral gemifloxacin is significantly reduced by the concomitant administration of an antacid containing aluminum and magnesium.
gemifloxacin
magnesium
MECHANISM
Gemifloxacin_ddi.xml
DDI-DrugBank.d347.s6
DDI-DrugBank.d347.s6.p2
CELEBREX should be introduced at the lowest recommended dose in patients receiving fluconazole.
CELEBREX
fluconazole
ADVISE
Celecoxib_ddi.xml
DDI-DrugBank.d172.s18
DDI-DrugBank.d172.s18.p0
When combined with ofloxacin, KRM-1648 exhibited strong synergistic activity while only additive effects were observed with the combination of rifampicin (or rifabutin) and ofloxacin.
ofloxacin
KRM-1648
EFFECT
11137650.xml
DDI-MedLine.d8.s6
DDI-MedLine.d8.s6.p0
In addition to bleeding associated with heparin and vitamin K antagonists, drugs that alter platelet function (such as acetylsalicylic acid, dipyridamole and Abciximab) may increase the risk of bleeding if administered prior to, during, or after Activase therapy.
heparin
vitamin K antagonists
NONE
Alteplase_ddi.xml
DDI-DrugBank.d508.s1
DDI-DrugBank.d508.s1.p0
Phospholine Iodide potentiates other cholinesterase inhibitors such as succinylcholine or organophosphate and carbamate insecticides.
Phospholine Iodide
organophosphate insecticide
EFFECT
Echothiophate Iodide_ddi.xml
DDI-DrugBank.d377.s0
DDI-DrugBank.d377.s0.p2
Skeletal muscle relaxants: amphotericin B-induced hypokalemia may enhance the curariform effect of skeletal muscle relaxants (e.g., tubocurarine).
amphotericin B
tubocurarine
EFFECT
Amphotericin B_ddi.xml
DDI-DrugBank.d318.s12
DDI-DrugBank.d318.s12.p4
Although this effect was noted even when cholestyramine was given 4 hours prior to fluvastatin, this regimen did not result in diminished efficacy.
cholestyramine
fluvastatin
EFFECT
19489169.xml
DDI-MedLine.d119.s16
DDI-MedLine.d119.s16.p0
Quinolone Antibiotics: VIDEX should be administered at least 2 hours after or 6 hours before dosing with ciprofloxacin because plasma concentrations of ciprofloxacin are decreased when administered with antacids containing magnesium, calcium, or aluminum.
ciprofloxacin
calcium
MECHANISM
Didanosine_ddi.xml
DDI-DrugBank.d43.s8
DDI-DrugBank.d43.s8.p20
Multivalent Cation-Containing Products: Concurrent administration of a quinolone, including ciprofloxacin, with multivalent cation-containing products such as magnesium or aluminum antacids, sucralfate, VIDEX chewable/buffered tablets or pediatric powder, or products containing calcium, iron, or zinc may substantially decrease the absorption of ciprofloxacin, resulting in serum and urine levels considerably lower than desired.
ciprofloxacin
zinc
MECHANISM
Ciprofloxacin_ddi.xml
DDI-DrugBank.d123.s8
DDI-DrugBank.d123.s8.p17
Administration of diltiazem hydrochloride concomitantly with propranolol in five normal volunteers resulted in increased propranolol levels in all subjects and bioavailability of propranolol was increased approximately 50%.
propranolol
propranolol
NONE
Diltiazem_ddi.xml
DDI-DrugBank.d565.s8
DDI-DrugBank.d565.s8.p4
Ascorbic acid: Doses of ascorbic acid (vitamin C) 1 g/day have been reported to increase plasma concentration of synthetic estrogens by ~47%, possibly by inhibiting conjugation;
ascorbic acid
synthetic estrogens
MECHANISM
Ethynodiol Diacetate_ddi.xml
DDI-DrugBank.d485.s14
DDI-DrugBank.d485.s14.p4
Thyroid Physiology: The following agents may alter thyroid hormone or TSH levels, generally by effects on thyroid hormone synthesis, secretion, distribution, metabolism, hormone action, or elimination, or altered TSH secretion: aminoglutethimide, p-aminosalicylic acid, amiodarone, androgens and related anabolic hormones, complex anions (thiocyanate, perchlorate, pertechnetate), antithyroid drugs, b-adrenergic blocking agents, carbamazepine, chloral hydrate, diazepam, dopamine and dopamine agonists, ethionamide, glucocorticoids, heparin, hepatic enzyme inducers, insulin, iodinated cholestographic agents, iodine-containing compounds, levodopa, lovastatin, lithium, 6-mercaptopurine, metoclopramide, mitotane, nitroprusside, phenobarbital, phenytoin, resorcinol, rifampin, somatostatin analogs, sulfonamides, sulfonylureas, thiazide diuretics.
aminoglutethimide
ethionamide
NONE
Levothyroxine_ddi.xml
DDI-DrugBank.d411.s4
DDI-DrugBank.d411.s4.p13
Because there is a theoretical basis that these effects may be additive, use of ergotamine-containing or ergot-type medications (like dihydroergotamine or methysergide) and naratriptan within 24 hours is contraindicated.
methysergide
naratriptan
ADVISE
Naratriptan_ddi.xml
DDI-DrugBank.d478.s1
DDI-DrugBank.d478.s1.p9
Vecuronium: When used in the perioperative period, piperacillin has been implicated in the prolongation of the neuromuscular blockade of vecuronium.
piperacillin
vecuronium
EFFECT
Piperacillin_ddi.xml
DDI-DrugBank.d462.s1
DDI-DrugBank.d462.s1.p2
Clinical trials have indicated that Pulmozyme can be effectively and safely used in conjunction with standard cystic fibrosis therapies including oral, inhaled and/or parenteral antibiotics, bronchodilators, enzyme supplements, vitamins, oral or inhaled corticosteroids, and analgesics.
Pulmozyme
vitamins
NONE
Dornase Alfa_ddi.xml
DDI-DrugBank.d93.s0
DDI-DrugBank.d93.s0.p2
Clonidine hydrochloride may enhance the CNS-depressive effects of alcohol, barbiturates or other sedatives.
Clonidine hydrochloride
alcohol
EFFECT
Clonidine_ddi.xml
DDI-DrugBank.d495.s1
DDI-DrugBank.d495.s1.p0
Agents that have been found, or are expected to have decreased plasma levels in the presence of EQUETROTM due to induction of CYP enzymes are the following: Acetaminophen, alprazolam, amitriptyline, bupropion, buspirone, citalopram, clobazam, clonazepam, clozapine, cyclosporin, delavirdine, desipramine, diazepam, dicumarol, doxycycline, ethosuximide, felbamate, felodipine, glucocorticoids, haloperidol, itraconazole, lamotrigine, levothyroxine, lorazepam, methadone, midazolam, mirtazapine, nortriptyline, olanzapine, oral contraceptives(3), oxcarbazepine, Phenytoin(4), praziquantel, protease inhibitors, quetiapine, risperidone, theophylline, topiramate, tiagabine, tramadol, triazolam, valproate, warfarin(5) , ziprasidone, and zonisamide.
EQUETROTM
ziprasidone
MECHANISM
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s11
DDI-DrugBank.d94.s11.p43
Clinical trials have indicated that Pulmozyme can be effectively and safely used in conjunction with standard cystic fibrosis therapies including oral, inhaled and/or parenteral antibiotics, bronchodilators, enzyme supplements, vitamins, oral or inhaled corticosteroids, and analgesics.
vitamins
corticosteroids
NONE
Dornase Alfa_ddi.xml
DDI-DrugBank.d93.s0
DDI-DrugBank.d93.s0.p12
Beta-adrenergic Blocking Agents: Experience in over 1400 patients in a non-comparative clinical trial has shown that concomitant administration of nifedipine and beta-blocking agents is usually well tolerated, but there have been occasional literature reports suggesting that the combination may increase the likelihood of congestive heart failure, severe hypotension or exacerbation of angina.
nifedipine
beta-blocking agents
EFFECT
Nifedipine_ddi.xml
DDI-DrugBank.d373.s0
DDI-DrugBank.d373.s0.p2
Antifungals: In vitro and/or in vivo data indicate that fluconazole, itraconazole, and oral ketoconazole markedly inhibit the metabolism of cisapride, which can result in an increase in plasma cisapride levels and prolongation of the QT interval on the ECG.
ketoconazole
cisapride
MECHANISM
Cisapride_ddi.xml
DDI-DrugBank.d237.s7
DDI-DrugBank.d237.s7.p12
Similarly, the effects of phenytoin on phenobarbital, valproic acid and sodium plasma valproate concentrations are unpredictable
phenytoin
valproic acid
EFFECT
Fosphenytoin_ddi.xml
DDI-DrugBank.d40.s15
DDI-DrugBank.d40.s15.p1
Bentiromide may interact with acetaminophen (e.g., Tylenol), chloramphenicol (e.g., Chloromycetin), local anesthetics (e.g., benzocaine and lidocaine), para-aminobenzoic acid (PABA)-containing preparations (e.g., sunscreens and some multivitamins), procainamide (e.g., Pronestyl), sulfonamides (sulfa medicines), thiazide diuretics (use of these medicines during the test period will affect the test results), and pancreatic supplements (use of pancreatic supplements may give false test results).
anesthetics
lidocaine
NONE
Bentiromide_ddi.xml
DDI-DrugBank.d537.s0
DDI-DrugBank.d537.s0.p61
Some quinolones have also been shown to interfere with the metabolism of caffeine.
quinolones
caffeine
MECHANISM
Norfloxacin_ddi.xml
DDI-DrugBank.d217.s13
DDI-DrugBank.d217.s13.p0
Patients taking both flurbiprofen and a beta-blocker should be monitored to ensure that a satisfactory hypotensive effect is achieved.
flurbiprofen
beta-blocker
ADVISE
Flurbiprofen_ddi.xml
DDI-DrugBank.d529.s11
DDI-DrugBank.d529.s11.p0
We conclude that ADL 8-2698 prevents morphine-induced increases in gastrointestinal transit time by means of selective peripheral opioid anitagonism without affecting central opioid analgesia.
ADL 8-2698
morphine
EFFECT
11180040.xml
DDI-MedLine.d87.s8
DDI-MedLine.d87.s8.p0
High-dose cisplatin with sodium thiosulfate protection.
cisplatin
sodium thiosulfate
EFFECT
4038510.xml
DDI-MedLine.d130.s0
DDI-MedLine.d130.s0.p0
Methotrexate: Piperacillin sodium may reduce the excretion of methotrexate.
Piperacillin sodium
methotrexate
MECHANISM
Piperacillin_ddi.xml
DDI-DrugBank.d462.s7
DDI-DrugBank.d462.s7.p2
The hypoglycemic action of sulfonylurea may be potentiated by certain drugs including nonsteroidal anti-inflammatory agents and other drugs that are highly protein bound, salicylates, sulfonamides, chloramphenicol, probenecid, coumarins, monoamine oxidase inhibitors, and beta adrenergic blocking agents.
sulfonylurea
beta adrenergic blocking agents
EFFECT
Chlorpropamide_ddi.xml
DDI-DrugBank.d245.s0
DDI-DrugBank.d245.s0.p7
Rifabutin, another rifamycin, is structurally similar to rifampin and may possibly have some of the same drug interactions as rifampin.
Rifabutin
rifampin
NONE
Atovaquone_ddi.xml
DDI-DrugBank.d424.s5
DDI-DrugBank.d424.s5.p1
Interactions with Other CNS Agents: Concurrent use of Levo-Dromoran with all central nervous system depressants (eg, alcohol, sedatives, hypnotics, other opioids, general anesthetics, barbiturates, tricyclic antidepressants, phenothiazines, tranquilizers, skeletal muscle relaxants and antihistamines) may result in additive central nervous system depressant effects.
Levo-Dromoran
sedatives
EFFECT
Levorphanol_ddi.xml
DDI-DrugBank.d257.s0
DDI-DrugBank.d257.s0.p2
- a monoamine oxidase inhibitor (MAOI) such as isocarboxazid (Marplan), tranylcypromine (Parnate), or phenelzine (Nardil);
isocarboxazid
Marplan
NONE
Glimepiride_ddi.xml
DDI-DrugBank.d521.s4
DDI-DrugBank.d521.s4.p13
Human pharmacokinetics data indicate that oral ketoconazole potently inhibits the metabolism of cisapride resulting in a mean eight-fold increase in AUC of cisapride.
ketoconazole
cisapride
MECHANISM
Ketoconazole_ddi.xml
DDI-DrugBank.d458.s7
DDI-DrugBank.d458.s7.p0
Furosemide may increase the ototoxic potential of aminoglycoside antibiotics, especially in the presence of impaired renal function.
Furosemide
aminoglycoside antibiotics
EFFECT
Furosemide_ddi.xml
DDI-DrugBank.d231.s0
DDI-DrugBank.d231.s0.p0
Trecator has been found to temporarily raise serum concentrations of isoniazid.
Trecator
isoniazid
MECHANISM
Ethionamide_ddi.xml
DDI-DrugBank.d166.s0
DDI-DrugBank.d166.s0.p0
Patients receiving other narcotic analgesics, general anesthetics, phenothiazines, tranquilizers, sedative-hypnotics, tricyclic antidepressants or other CNS depressants (including alcohol) concomitantly with DILAUDID may exhibit an additive CNS depression.
narcotic analgesic
DILAUDID
EFFECT
Hydromorphone_ddi.xml
DDI-DrugBank.d26.s0
DDI-DrugBank.d26.s0.p7
Tell your doctor if you are taking any of the following drugs: blood thinners (Coumadin) other antidepressants metoprolol antihistamines carbamazepine (Tegretol) cimetidine (Tagamet) estrogens fluoxetine (Prozac) intraconazole (Sporanox) ketoconazole (Nizoral) levodopa lithium muscle relaxants birth control pills sleeping pills thyroid medications
estrogens
levodopa
NONE
Fluoxetine_ddi.xml
DDI-DrugBank.d482.s14
DDI-DrugBank.d482.s14.p122
Aminoglutethimide diminishes the effect of coumarin and warfarin.
Aminoglutethimide
warfarin
EFFECT
Aminoglutethimide_ddi.xml
DDI-DrugBank.d372.s2
DDI-DrugBank.d372.s2.p1
There was a small decrease in the clearance of cetirizine caused by a 400-mg dose of theophylline;
cetirizine
theophylline
MECHANISM
Cetirizine_ddi.xml
DDI-DrugBank.d393.s5
DDI-DrugBank.d393.s5.p0
- Cholestyramine and colestipol resins: Cholestytamine and colestipol resins have the potential of binding thiazide diuretics and reducing diuretic absorption from the gastrointestinal tract
resins
thiazide diuretics
NONE
Chlorothiazide_ddi.xml
DDI-DrugBank.d46.s7
DDI-DrugBank.d46.s7.p25
The therapeutic efficacy of tricyclic antidepressants may be compromised in these patients when cimetidine is discontinued.
tricyclic antidepressants
cimetidine
EFFECT
Nortriptyline_ddi.xml
DDI-DrugBank.d202.s4
DDI-DrugBank.d202.s4.p0
Drugs that have been reported to diminish oral anticoagulant response, ie, decreased prothrom-bin time response, in man significantly include: adrenocortical steroids;alcohol*;antacids;antihistamines;barbiturates;carbamazepine;chloral hydrate*;chlordiazepoxide;cholestyramine;diet high in vitamin K;diuretics*;ethchlorvynol;glutethimide;griseofulvin;haloperidol;meprobamate;oral contraceptives;paraldehyde;primidone;ranitidine*;rifampin;unreliable prothrombin time determinations;vitamin C;warfarin sodium under-dosage.
alcohol
cholestyramine
NONE
Anisindione_ddi.xml
DDI-DrugBank.d64.s29
DDI-DrugBank.d64.s29.p51
Thus, the interaction observed between erythromycin and terfenadine is not expected for dirithromycin.
erythromycin
terfenadine
INT
Dirithromycin_ddi.xml
DDI-DrugBank.d522.s9
DDI-DrugBank.d522.s9.p0
Additive adverse effects resulting from cholinergic blockade may occur when LEVSIN is administered concomitantly with other antimuscarinics, amantadine, haloperidol, phenothiazines, monoamine oxidase (MAO) inhibitors, tricyclic antidepressants or some antihistamines.
LEVSIN
antimuscarinics
EFFECT
Hyoscyamine_ddi.xml
DDI-DrugBank.d142.s0
DDI-DrugBank.d142.s0.p0
The administration of local anesthetic solutions containing epinephrine or norepinephrine to patients receiving monoamine oxidase inhibitors or tricyclic antidepressants may produce severe, prolonged hypertension.
norepinephrine
monoamine oxidase inhibitors
EFFECT
Lidocaine_ddi.xml
DDI-DrugBank.d564.s0
DDI-DrugBank.d564.s0.p7
Although not observed in this study, adverse effects could potentially arise from co-administration of cephalexin and metformin by inhibition of tubular secretion via organic cationic transporter systems.
cephalexin
metformin
MECHANISM
Cephalexin_ddi.xml
DDI-DrugBank.d303.s2
DDI-DrugBank.d303.s2.p0
- Cholestyramine and colestipol resins: Cholestytamine and colestipol resins have the potential of binding thiazide diuretics and reducing diuretic absorption from the gastrointestinal tract
Cholestyramine
resins
NONE
Chlorothiazide_ddi.xml
DDI-DrugBank.d46.s7
DDI-DrugBank.d46.s7.p1
Cytochalasin D at 10 microM preferentially blocked the secretory effect of carbachol and its synergism with cAMP, whereas it had no effect on histamine- or cAMP-stimulated acid secretion within 15 min.
Cytochalasin D
carbachol
EFFECT
11121387.xml
DDI-MedLine.d59.s4
DDI-MedLine.d59.s4.p0
As with other agents with b-blocking properties, if COREG is to be administered orally with calcium channel blockers of the verapamil or diltiazem type, it is recommended that ECG and blood pressure be monitored.
verapamil
diltiazem
NONE
Carvedilol_ddi.xml
DDI-DrugBank.d269.s17
DDI-DrugBank.d269.s17.p9
The benzodiazepines, including alprazolam, produce additive CNS depressant effects when co-administered with other psychotropic medications, anticonvulsants, antihistaminics, ethanol, and other drugs which themselves produce CNS depression.
benzodiazepines
anticonvulsants
EFFECT
Alprazolam_ddi.xml
DDI-DrugBank.d131.s0
DDI-DrugBank.d131.s0.p2
Agents that are CYP inducers that have been found, or are expected, to decrease plasma levels of EQUETROTM are the following: Cisplatin, doxorubicin HCL, felbamate, rifampin, phenobarbital, Phenytoin(2), primidone, methsuximide, and theophylline Thus, if a patient has been titrated to a stable dosage on EQUETROTM, and then begins a course of treatment with one of these CYP3A4 inducers, it is reasonable to expect that a dose increase for EQUETROTM may be necessary.
EQUETROTM
methsuximide
MECHANISM
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s8
DDI-DrugBank.d94.s8.p7
The following are examples of substances that may increase the blood-glucose-lowering effect and susceptibility to hypoglycemia: oral antidiabetes products, ACE inhibitors, disopyramide, fibrates, fluoxetine, MAO inhibitors, propoxyphene, salicylates, somatostatin analog (e.g., octreotide), sulfonamide antibiotics.
ACE inhibitors
salicylates
NONE
Insulin Glargine recombinant_ddi.xml
DDI-DrugBank.d527.s1
DDI-DrugBank.d527.s1.p5
Although bupropion is not metabolized by this isoenzyme, bupropion and hydroxybupropion are inhibitors of the CYP2D6 isoenzyme in vitro.
bupropion
hydroxybupropion
NONE
Bupropion_ddi.xml
DDI-DrugBank.d5.s13
DDI-DrugBank.d5.s13.p1
While all the selective serotonin reuptake inhibitors (SSRIs), e.g., citalopram, escitalopram, fluoxetine, sertraline, and paroxetine, inhibit P450 2D6, they may vary in the extent of inhibition.
fluoxetine
sertraline
NONE
Doxepin_ddi.xml
DDI-DrugBank.d223.s8
DDI-DrugBank.d223.s8.p18
Isoflurane or enflurane administered with nitrous oxide/oxygen to achieve 1.25 MAC [Minimum Alveolar Concentration] may prolong the clinically effective duration of action of initial and maintenance doses of NIMBEX and decrease the required infusion rate of NIMBEX.
enflurane
NIMBEX
EFFECT
Cisatracurium Besylate_ddi.xml
DDI-DrugBank.d60.s6
DDI-DrugBank.d60.s6.p8
Although the mechanism of interaction between fenoprofen and aspirin is not totally known, enzyme induction and displacement of fenoprofen from plasma albumin binding sites are possibilities.
fenoprofen
aspirin
MECHANISM
Fenoprofen_ddi.xml
DDI-DrugBank.d154.s1
DDI-DrugBank.d154.s1.p0
Like ibogaine (40 mg/kg), 18-MC (40 mg/kg) decreases the intravenous self-administration of morphine and cocaine and the oral self-administration of ethanol and nicotine in rats;
cocaine
nicotine
NONE
11085336.xml
DDI-MedLine.d110.s2
DDI-MedLine.d110.s2.p13
The concomitant use of transdermal fentanyl with ritonavir or other potent 3A4 inhibitors such as ketoconazole, itraconazole, troleandomycin, clarithromycin, nelfinavir, and nefazadone may result in an increase in fentanyl plasma concentrations.
fentanyl
clarithromycin
MECHANISM
Fentanyl_ddi.xml
DDI-DrugBank.d170.s2
DDI-DrugBank.d170.s2.p4
Although specific drug interaction studies have not been conducted with ALPHAGAN P, the possibility of an additive or potentiating effect with CNS depressants (alcohol, barbiturates, opiates, sedatives, or anesthetics) should be considered.
CNS depressants
opiates
NONE
Brimonidine_ddi.xml
DDI-DrugBank.d138.s0
DDI-DrugBank.d138.s0.p8
Rifampicin: In a study in healthy volunteers, a six-day course of rifampicin at 600 mg/day followed by a single 5 mg dose of isradipine resulted in a reduction in isradipine levels to below detectable limits.
rifampicin
isradipine
MECHANISM
Isradipine_ddi.xml
DDI-DrugBank.d81.s9
DDI-DrugBank.d81.s9.p3
Other drugs which may enhance the neuromuscular blocking action of nondepolarizing agents such as NIMBEX include certain antibiotics (e. g., aminoglycosides, tetracyclines, bacitracin, polymyxins, lincomycin, clindamycin, colistin, and sodium colistemethate), magnesium salts, lithium, local anesthetics, procainamide, and quinidine.
NIMBEX
tetracyclines
EFFECT
Cisatracurium Besylate_ddi.xml
DDI-DrugBank.d60.s12
DDI-DrugBank.d60.s12.p17
Cytotoxic Agents: Enhanced bone marrow suppression by cyclophosphamide and other cytotoxic agents has been reported among patients with neoplastic disease, except leukemia, in the presence of allopurinol.
cyclophosphamide
allopurinol
EFFECT
Allopurinol_ddi.xml
DDI-DrugBank.d413.s18
DDI-DrugBank.d413.s18.p4
Amiodarone or Verapamil: The risk of myopathy/rhabdomyolysis is increased when either amiodarone or verapamil is used concomitantly with a closely related member of the HMG-CoA reductase inhibitor class (see WARNINGS, Myopathy/Rhabdomyolysis).
amiodarone
HMG-CoA reductase inhibitor class
EFFECT
Lovastatin_ddi.xml
DDI-DrugBank.d567.s12
DDI-DrugBank.d567.s12.p8
CANCIDAS reduced the blood AUC0-12 of tacrolimus by approximately 20%, peak blood concentration (Cmax) by 16%, and 12-hour blood concentration (C12hr) by 26% in healthy subjects when tacrolimus (2 doses of 0.1 mg/kg 12 hours apart) was administered on the 10th day of CANCIDAS 70 mg daily, as compared to results from a control period in which tacrolimus was administered alone.
CANCIDAS
tacrolimus
NONE
Caspofungin_ddi.xml
DDI-DrugBank.d350.s5
DDI-DrugBank.d350.s5.p1
Caution is advised when TRISENOX is coadministered with other medications that can prolong the QT interval (e.g. certain antiarrhythmics or thioridazine) or lead to electrolyte abnormalities (such as diuretics or amphotericin B).
TRISENOX
antiarrhythmics
ADVISE
Arsenic trioxide_ddi.xml
DDI-DrugBank.d470.s1
DDI-DrugBank.d470.s1.p0
Valproic Acid: The mean steady-state trough serum valproic acid concentrations prior to and during concomitant gabapentin administration (400 mg TID;
valproic acid
gabapentin
NONE
Gabapentin_ddi.xml
DDI-DrugBank.d438.s11
DDI-DrugBank.d438.s11.p2
Protein Binding In vitro, diclofenac interferes minimally or not at all with the protein binding of salicylic acid (20% decrease in binding), tolbutamide, prednisolone (10% decrease in binding), or warfarin.
diclofenac
warfarin
MECHANISM
Diclofenac_ddi.xml
DDI-DrugBank.d249.s18
DDI-DrugBank.d249.s18.p3
Before taking glimepiride, tell your doctor if you are taking any of the following medicines: - aspirin or another salicylate such as magnesium/choline salicylate (Trilisate), salsalate (Disalcid, others), choline salicylate (Arthropan), magnesium salicylate (Magan), or bismuth subsalicylate (Pepto-Bismol);
glimepiride
aspirin
ADVISE
Glimepiride_ddi.xml
DDI-DrugBank.d521.s1
DDI-DrugBank.d521.s1.p0
Patients receiving catecholamine-depleting drugs, such as reserpine or guanethidine, should be closely monitored, because the added beta-adrenergic blocking action of ZEBETA may produce excessive reduction of sympathetic activity.
guanethidine
ZEBETA
EFFECT
Bisoprolol_ddi.xml
DDI-DrugBank.d476.s1
DDI-DrugBank.d476.s1.p2
The absorption of tetracycline, furosemide, penicillin G, hydrochlorothiazide, and gemfibrozil was significantly decreased when given simultaneously with colestipol hydrochloride;
hydrochlorothiazide
colestipol hydrochloride
MECHANISM
Colestipol_ddi.xml
DDI-DrugBank.d345.s11
DDI-DrugBank.d345.s11.p13
Diuretic agents reduce the renal clearance of lithium and add a high risk of lithium toxicity.
Diuretic agents
lithium
EFFECT
Hydrochlorothiazide_ddi.xml
DDI-DrugBank.d162.s10
DDI-DrugBank.d162.s10.p1
Anticoagulants including coumarin derivatives, indandione derivatives, and platelet aggregation inhibitors such as nonsteroidal anti-inflammatory drugs (NSAIDs), and aspirin may increase the risk of bleeding when administered concomitantly with ardeparin.
NSAIDs
ardeparin
EFFECT
Ardeparin_ddi.xml
DDI-DrugBank.d105.s0
DDI-DrugBank.d105.s0.p13
Elevated serum levels of cyclosporine have been reported with concomitant use of cyclosporine with norfloxacin.
cyclosporine
norfloxacin
MECHANISM
Norfloxacin_ddi.xml
DDI-DrugBank.d217.s3
DDI-DrugBank.d217.s3.p2
Butalbital, acetaminophen and caffeine may enhance the effects of: other narcotic analgesics, alcohol, general anesthetics, tranquilizers such as chlordiazepoxide, sedative-hypnotics, or other CNS depressants, causing increased CNS depression.
caffeine
chlordiazepoxide
EFFECT
Butalbital_ddi.xml
DDI-DrugBank.d559.s1
DDI-DrugBank.d559.s1.p21
In clinical studies, coadministration of WelChol with atorvastatin, lovastatin, or simvastatin did not interfere with the lipid-lowering activity of the HMG-CoA reductase inhibitor.
WelChol
simvastatin
NONE
Colesevelam_ddi.xml
DDI-DrugBank.d551.s4
DDI-DrugBank.d551.s4.p2
Therefore, esomeprazole may interfere with the absorption of drugs where gastric pH is an important determinant of bioavailability (eg, ketoconazole, iron salts and digoxin).
esomeprazole
ketoconazole
MECHANISM
Esomeprazole_ddi.xml
DDI-DrugBank.d29.s12
DDI-DrugBank.d29.s12.p0
The benzodiazepines, including alprazolam, produce additive CNS depressant effects when co-administered with other psychotropic medications, anticonvulsants, antihistaminics, ethanol, and other drugs which themselves produce CNS depression.
benzodiazepines
ethanol
EFFECT
Alprazolam_ddi.xml
DDI-DrugBank.d131.s0
DDI-DrugBank.d131.s0.p4
Antihistamines may have additive effects with alcohol and other CNS depressants, e.g., hypnotics, sedatives, tranquilizers, antianxiety agents.
CNS depressants
hypnotics
NONE
Cyproheptadine_ddi.xml
DDI-DrugBank.d492.s1
DDI-DrugBank.d492.s1.p11
Agents that have been found, or are expected to have decreased plasma levels in the presence of EQUETROTM due to induction of CYP enzymes are the following: Acetaminophen, alprazolam, amitriptyline, bupropion, buspirone, citalopram, clobazam, clonazepam, clozapine, cyclosporin, delavirdine, desipramine, diazepam, dicumarol, doxycycline, ethosuximide, felbamate, felodipine, glucocorticoids, haloperidol, itraconazole, lamotrigine, levothyroxine, lorazepam, methadone, midazolam, mirtazapine, nortriptyline, olanzapine, oral contraceptives(3), oxcarbazepine, Phenytoin(4), praziquantel, protease inhibitors, quetiapine, risperidone, theophylline, topiramate, tiagabine, tramadol, triazolam, valproate, warfarin(5) , ziprasidone, and zonisamide.
diazepam
risperidone
NONE
Carbamazepine_ddi.xml
DDI-DrugBank.d94.s11
DDI-DrugBank.d94.s11.p529
Other CNS depressant drugs (e.g. barbiturates, tranquilizers, opioids and general anesthetics) have additive or potentiating effects with INAPSINE.
barbiturates
INAPSINE
EFFECT
Droperidol_ddi.xml
DDI-DrugBank.d254.s0
DDI-DrugBank.d254.s0.p8
Although not studied with alosetron, inhibition of N-acetyltransferase may have clinically relevant consequences for drugs such as isoniazid, procainamide, and hydralazine.
alosetron
procainamide
EFFECT
Alosetron_ddi.xml
DDI-DrugBank.d364.s15
DDI-DrugBank.d364.s15.p1
Etonogestrel may interact with the following medications: acetaminophen (Tylenol), antibiotics such as ampicillin and tetracycline, anticonvulsants (Dilantin, Phenobarbital, Tegretol, Trileptal, Topamax, Felbatol), antifungals (Gris-PEG, Nizoral, Sporanox), atorvastatin (Lipitor), clofibrate (Atromid-S), cyclosporine (Neoral, Sandimmune), HIV drugs classified as protease inhibitors (Agenerase, Crixivan, Fortovase, Invirase, Kaletra, Norvir, Viracept), morphine (Astramorph, Kadian, MS Contin), phenylbutazone, prednisolone (Prelone), rifadin (rifampin), St. Johns wort, temazepam, theophylline (Theo-Dur), and vitamin C.
Etonogestrel
Topamax
INT
Etonogestrel_ddi.xml
DDI-DrugBank.d484.s0
DDI-DrugBank.d484.s0.p10
The hypoglycemic action of sulfonylureas may be potentiated by certain drugs including nonsteroidal anti-inflammatory agents and other drugs that are highly protein bound, salicylates, sulfonamides, chloramphenicol, probenecid, coumarins, monoamine oxidase inhibitors, and beta adrenergic blocking agents.
sulfonylureas
nonsteroidal anti-inflammatory agents
EFFECT
Glibenclamide_ddi.xml
DDI-DrugBank.d178.s0
DDI-DrugBank.d178.s0.p0
Magnesium- and/or aluminum-containing antacids, products containing ferrous sulfate (iron), multivitamin preparations containing zinc or other metal cations, or Videx (didanosine) chewable/buffered tablets or the pediatric powder for oral solution should not be taken within 3 hours before or 2 hours after FACTIVE.
Videx
FACTIVE
ADVISE
Gemifloxacin_ddi.xml
DDI-DrugBank.d347.s7
DDI-DrugBank.d347.s7.p43
In monkeys, (-)-NANM was about 10 times more potent than (+)-NANM in decreasing responding, whereas in pigeons (-)-NANM was about equipotent with (+)-NANM.
(+)-NANM
(-)-NANM
NONE
3968644.xml
DDI-MedLine.d30.s6
DDI-MedLine.d30.s6.p3
Potentiation of the hypotensive effects of nitrates for patients with ischemic heart disease has not been evaluated, and concomitant use of Vardenafil and nitrates is contraindicated.
Vardenafil
nitrates
ADVISE
Vardenafil_ddi.xml
DDI-DrugBank.d198.s24
DDI-DrugBank.d198.s24.p2
Amiodarone is known to raise serum digoxin levels.
Amiodarone
digoxin
MECHANISM
3964797.xml
DDI-MedLine.d61.s1
DDI-MedLine.d61.s1.p0
Sumatriptan - There have been rare postmarketing reports describing patients with weakness, hyperreflexia, and incoordination following the use of a selective serotonin reuptake inhibitor (SSRI) and sumatriptan.
SSRI
sumatriptan
EFFECT
Escitalopram_ddi.xml
DDI-DrugBank.d568.s16
DDI-DrugBank.d568.s16.p5
Antidepressants, tricyclic Amphetamines may enhance the activity of tricyclic antidepressants or sympathomimetic agents;
Antidepressants
tricyclic
NONE
Lisdexamfetamine_ddi.xml
DDI-DrugBank.d158.s3
DDI-DrugBank.d158.s3.p0
This drug may interact with alcohol or other CNS depressants (may potentiate the CNS depressant effects of either these medications or antihistamines), anticholinergics or other medications with anticholinergic activity (anticholinergic effects may be potentiated when these medications are used concurrently with antihistamines), and monoamine oxidase (MAO) inhibitors (concurrent use with antihistamines may prolong and intensify the anticholinergic and CNS depressant effects of antihistamines).
CNS depressants
antihistamines
NONE
Diphenylpyraline_ddi.xml
DDI-DrugBank.d168.s0
DDI-DrugBank.d168.s0.p7
The effect of a pulmonary surfactant extract from bovine lung, Survanta, on the dissolution rate of aerosol particles of budesonide was determined.
pulmonary surfactant
Survanta
NONE
11064383.xml
DDI-MedLine.d18.s1
DDI-MedLine.d18.s1.p0
Injection: Lorazepam injection, like other injectable benzodiazepines, produces depression of the central nervous system when administered with ethyl alcohol, phenothiazines, barbiturates, MAO inhibitors, and other antidepressants.When scopolamine is used concomitantly with injectable lorazepam, an increased incidence of sedation, hallucinations, and irrational behavior has been observed.
benzodiazepines
antidepressants
EFFECT
Lorazepam_ddi.xml
DDI-DrugBank.d18.s1
DDI-DrugBank.d18.s1.p12