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Investing With Us
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You are here: Home / Blog / Crowdfunding / Crowdfunding
27th June, 2016 /in Crowdfunding /by Admiral
Is it time to dip your toe into the water?
One of the main innovations in both finance and technology over the past few years has been the advent of crowdfunding. Crowdfunding is a way of raising finance by asking a large number of investors each for a small amount of money. In the past, financing a business, project or venture typically involved asking a few investors for large sums of money. Crowdfunding switches this idea around.
If it appeals to you, you’re debt-free, willing to increase your risk tolerance and put money away for a longer term, then the best way is to start by dipping your toe in the water.
Main ways of crowdfunding
There are three different types of crowdfunding: equity, debt and donation.
1. Equity crowdfunding involves a company raising finance by selling a pre-determined amount of equity in a business to investors for a certain sum of money.
2. Debt crowdfunding is when a company raises money by way of loan to investors who do not receive any equity in the business but do receive a pre-agreed rate of return on the money invested.
3. Charitable crowdfunding involves no equity or debt investment; charities raise money for projects from large groups of investors to support their causes.
Funding gap
Since the financial crisis, it has become very difficult for smaller businesses in particular to raise the money required to expand and grow. As high street banks have withdrawn from corporate lending, a ‘funding gap’ has emerged that has in part been filled by the meteoric rise of crowdfunding. Additionally, with interest rates at rock bottom for the past six years, investors have been seeking unique returns and are more prepared than ever before to invest money into new types of investments in the hope of achieving a return higher than that offered by their bank account.
The Government’s promotion of enterprise in the UK has also meant that over 95% of all equity crowdfunding campaigns attract valuable tax benefits (such as Enterprise Investment Scheme [EIS] or Seed Enterprise Investment Scheme [SEIS] relief which allow for any losses to be claimed back on a tax return) which has further enhanced their appeal. It is against this backdrop that crowdfunding has flourished.
However, crowdfunding is still a very high-risk venture and, as with all forms of investment, comes with no guarantee of success or even return of capital. Among the main reasons to exercise caution is the price the investor pays for the equity. When a large business seeks to raise money, the numbers used to calculate the value of the business will have been audited and verified by an independent valuer.
High growth
Another key risk that investors need to consider is one of dilution. Many of the companies raising money on the equity crowdfunding platforms are high-growth businesses expected to go up in value significantly over a very short period of time. To fund this expansion, it is somewhat inevitable that these companies will need to continually raise further funds to maintain their level of growth.
While not all crowdfunding campaigns are run by start-ups, the average age of a company raising money on crowdfunding platforms is 3.32 years. Crowdfunding is undoubtedly a welcome addition to the options available for companies looking to raise money, and the effect that this has on the level of enterprise in the UK is a very positive development.
Capital loss
For investors, however, it is clear that great levels of caution should be taken when making any form of crowdfunding investment, and a reliance on the tax benefits will not be enough to offset the real risk of capital loss.
Time to talk to Admiral Wealth?
For further information, please contact Admiral Wealth Management on 01472 357035 or email info@admiral-online.co.uk – we look forward to hearing from you.
PAST PERFORMANCE IS NOT A RELIABLE INDICATOR OF FUTURE PERFORMANCE.
Tags: Admiral Wealth Management, charitable crowdfunding, crowdfunding, debt crowdfunding, Enterprise Investment Scheme, equity crowdfunding, funding gap, investment, raising finance, Seed Enterprise Investment Scheme
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We are not dictating cashew prices - Cashew Council Ghana.
30 October, 2020 - 0:00 Updated: 2 November, 2020 - 09:32
The Cashew Council Ghana (CCG) has debunked claims that it is making moves to dictate prices of cashew by implementing a pricing regime.
Barely two weeks since its launch in Techiman, there are claims in the media that the Council is attempting to implement “a pricing system” that will “dictate prices” of cashew. These reports also misrepresented the Council as “a group of cashew growers” on the Board of the Tree Crops Development Authority (TCDA).
According to them, these reports are clear misinformation by people who do not have a clear understanding of the CCG, and by extension the TCDA.
The CCG, they explain, is the umbrella body of the various value chain actors’ associations in Ghana, including the Cashew Producers Association and other farmers’ groups. The four people representing the cashew sector on the TCDA, they explained, were selected from the various actor groups, two of whom are cashew farmers.
On pricing, they explained that what it has done is to, as part of reorganizing the sector, set up a pricing committee, with the vast majority of the members being cashew farmers, to formulate a cashew pricing formular. This, according to them, is an extension of the pricing committee of the TCDA.
“At our last meeting, where the Council was officially launched, there were representatives of all the various value chain actors. In fact, farmers were the majority. At this meeting, three working committees were set up, including the Pricing Committee as an extension of the pricing committee of the TCDA. It was a decision that was welcomed by all, particularly farmers,” Mr. Malvin Nii Smith, President of the Association of Cashew Processors Ghana (ACPG) said in an interview.
“Interestingly, because of the sensitive nature of pricing and because we know farmers are the major stakeholders in this regard, of the 19 members on this committee, the farmers have 10 representatives with buyers/ exporters and processors having six (6) and three(3) respectively,” he added.
A Cashew farmer in the Tain District, Mr. Sah Kofi Eric, who represents farmers in the District on the Pricing Committee described the claims as false and misleading, explaining that cashew farmers across the country have been duly represented on the said committee.
“There are 10 farmers from various cashew producing areas across the country on the pricing committee. Truth is not every farmer can be consulted in this process. And so, the various representatives update their constituents and take their inputs. That is why we encourage farmers all over the country to attend the association’s meetings at the local levels to make meaningful contributions,” he said in an interview.
He urged farmers to disregard such claims about the CCG and the TCDA, adding that cashew prices will remain competitive based on quality.
“The mandate of the working committee is to meet and suggest a pricing model and present it to the TCDA Board for further interrogation. The CCG, on its own, cannot and has no intention of dictating price of cashew,” he emphasized.
President of the Cashew Traders Association of Ghana, Mr. Alex Owuso Adjei, also refuted claims that exporters have been excluded from the CCG.
“It is not true that exporters have been excluded. I am an exporter and I serve on the pricing committee along with three other exporters. Our duty is to suggest modalities for pricing of cashew to the Board after consultations,” he said.
Acting Coordinator of the CCG, Yayra Amedzro, explained steps have been taken to bring clarity on the issues to farmers.
“As we speak farmers are having a national representative meeting in Wenchi and I believe the necessary information will be relayed to them and hopefully there will be some clarity on the issues of the new development,” she said.
The CCG was launched on Thursday 15th October 2020 by stakeholders of the cashew sector in Techiman to serve as a mother body for the various associations within the sector and to complement the TCDA in developing the sector.
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Aging Research Centre
We are dedicated to providing a service that allows researchers in this field to find information that is related to the study of the aging process. We also endeavor to introduce this field to laymen who would like to know more about the research that is being conducted in this field.
Steven A. Garan, Director Research
We are engaged in many areas of research such as systems biology, brain imaging via automated imaging microscopy, the study of neuroendocrine changes in the hypothalamus, whole body scanning systems, the study of an entire organism as it ages and the effects of hormonal changes on the aging process
Shortest path algorithms combined with natural language parsing, map pathways involved in neuroendocrine aging. Experimental Gerontology, Volume 44, Issues 1-2, January-February 2009, Page 129.
A computational systems biology approach to neuroendocrine aging: Initial results. Experimental Gerontology, Volume 42, Issues 1-2 , January-February 2007, Page 142
Novel methods in computer-assisted tissue analysis: Customized regional targeting of both cytoplasmic and nuclear-stained tissue. Experimental Gerontology, Volume 42, Issues 1-2 , January-February 2007, Pages 141-142
Tracking changes in hypothalamic IGF-1 sensitivity with aging and caloric restriction. Experimental Gerontology, Volume 42, Issues 1-2 , January-February 2007, Pages 11-12.
Insulin-like growth factor-1 receptor immunoreactive cells are selectively maintained in the paraventricular hypothalamus of calorically restricted mice. Int J Dev Neurosci. 2007 Feb;25(1):23-8
Age-dependent loss of insulin-like growth factor-1 receptor immunoreactive cells in the supraoptic hypothalamus is reduced in calorically restricted mice. Int J Dev Neurosci. 2006 Nov;24(7):431-6.
A study of insulin-like growth factor-I receptor immunoreactivity in the supraoptic nucleus of young and old female B6D2F1 mice. FASEB Journal, April 2006, 357.5
Caloric restriction reduces cell loss and maintains estrogen receptor-alpha immunoreactivity in the pre-optic hypothalamus of female B6D2F1 mice, Neuro Endocrinol Lett. 2005 Jun;26(3):197-203.
A survey of estrogen receptor-alpha immunoreactivity in the hypothalamus of young, old, and old-calorie restricted female B6D2F1 mice, Experimental Gerontology, Volume 39, Issues 11-12, November-December 2004, Page 1771
Phenomics: a new direction for the study of neuroendocrine aging. Experimental Gerontology, Volume 38 (2003), Issues 1-2, Page 218
A comparison of estrogen receptor-alpha immunoreactivity in the arcuate hypothalamus of young and middle-aged C57BL6 female mice. Experimental Gerontology, Volume 38 (2003), Issues 1-2, Page 220
Creating Three-Dimensional Neuronal Maps of the Mouse Hypothalamus Using an Automated Imaging Microscope System. Experimental Gerontology, Volume 35 numbers 9-10, December 2000, page 1421
Automated Imaging Microscope System, Linux Journal 2000, Issue 70, Februrary, Page 32-35
Automated Imaging
Microscope System
(AIMS)
Regional targeting of
cytoplasmic and
nuclear-stained tissue Ontological body system maps
Mapping body systems
- Items of major interest
- Meetings & Lectures
Paola S. Timiras Memorial Award for Aging Research and Lecture
Event: Paola S. Timiras Memorial Award for Aging Research and Lecture
Award Recipient: Dr. Elizabeth H. Blackburn, Ph.D
Date: Wednesday, February 13th 2013, 4:00pm - 5:30pm
Genetic Plant Biology Building (GPB, Auditorium 100)
Web: http://crea.berkeley.edu
The Center for Research and Education in Aging (CREA) will hold it's first Paola S. Timiras Memorial Award for Aging Research. The event will be held on Wednesday, February 13th, 2013 from 4:00pm to 5:30pm. The event will start with a reception, afterwards the awards ceremony will be held, where Dr. Blackburn will accept the first Paola S. Timiras Memorial Award for Aging Research. After the awards ceremony, Dr. Blackburn will give a lecture related to aging research. Dr. Blackburn won the 2009 Nobel Prize in Physiology/Medicine and was on the faculty of the University of California, Berkeley, in the Department of Molecular Biology from 1981 to 1990. In 1990 she moved to the University of California, San Francisco. After Dr. Blackburn's lecture, the audience will be able to ask questions from Dr. Blackburn.
66th Annual Scientific Meeting of the
Gerontological Society of America.
Date: 20th November - 24th November 2013
Location: New Orleans Marriott, New Orleans, Louisiana
http://www.geron.org/
Eleventh International Symposium on
Neurobiology and Neuroendocrinology of Aging
Location: Bregenz Austria
Date: TBD July 2014
The objective of these Symposia is to bring together scientists who have made recent major advances in the study of aging ranging from neuroendocrinology, neurobiology, genetics, and molecular mechanisms to practical issues of treatment and care of the elderly and patients with age-related CNS diseases. Our goals are to bring together speakers who normally would not meet at a meeting devoted to Alzheimer's disease, molecular biology of aging, clinical geriatrics, or other commonly covered topics.
For more information, please write, send a facsimile or email:
Dr. Andrzej Bartke
Director of Research,
Geriatrics Initiative,
Southern Illinois University School of Medicine,
Springfield, IL 62794-9636, USA
E-Mail: abartke@siumed.edu
Dr. Richard E Falvo,
Department of Cell and Molecular Physiology,
Medical Biomolecular Research Building,
103 Mason Farm Road,
School of Medicine,
University of North Carolina at Chapel Hill,
Chapel Hill, NC 27599-7545, USA
E-Mail: rfalvo@med.unc.edu
WWW Site: http://www.neurobiology-and-neuroendocrinology-of-aging.org/
- Recent Books
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Scientific American publication on life span and aging - Featured on pagesalon.com
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Age-dependent loss of insulin-like growth factor-1 receptor immunoreactive cells in the supraoptic hypothalamus is reduced in calorically restricted mice.
Both life-long caloric restriction (CR) and the suppression of insulin-like growth factor-1 (IGF-1) signaling reliably extend the mammalian lifespan. The neuroendocrine system, regulated by the hypothalamus, remains the most convincing site of action for both these modes of life extension. Yet, determining whether CR actions are mediated by the modulation of neuroendocrine IGF-1 signaling remains unclear. Of the hypothalamic nuclei that express the IGF-1 receptor (IGF-1R), the cells of the supraoptic nucleus (SON) display some of the most robust IGF-1R expression. Taking IGF-1R immunoreactivity as an index of sensitivity to IGF-1, we counted IGF-1R immunoreactive and non-immunoreactive cells in the SON of young-ad-libitum fed (young-Al, 6 weeks), old-ad-libitum fed (Old-Al, 22 months), and old-calorie-restricted (Old-CR, 22 months) female B6D2F1 mice. An automated imaging microscopy system (AIMS) was used to generate cell counts for each section of supraoptic hypothalamus. Results show that while the total number of cells in the SON of ad-libitum fed mice does not change significantly with aging, a significant reduction in IGF-1R immunoreactive cells does occur in ad-libitum fed mice with aging. In contrast to this, calorie restricted mice show both a decline in the total number of cells and IGF-1R immunoreactive cells in the SON with age, but with the decrease in the latter being notably attenuated when compared to the degree of loss seen in ad-libitum fed mice. Thus, while CR induces greater loss in the total number of cells in the SON with age, it reduces the degree of age-dependent loss seen in IGF-1R expressing cells. As a result, when compared to Old-AL mice, the SON of Old-CR mice displays a greater proportion of IGF-1R cells and thus possibly enhanced IGF-1 sensitivity with aging.
International Journal of Developmental Neuroscience 2006 Nov;24(7):431-6.
Caloric restriction reduces cell loss and maintains estrogen receptor-alpha immunoreactivity in the pre-optic hypothalamus of female B6D2F1 mice.
Life-long calorie restriction (CR) remains the most robust and reliable means of extending life span in mammals. Among the several theories to explain CR actions, one variant of the neuroendocrine theories of aging postulates that changing hypothalamic sensitivity to endocrine feedback is the clock that times phenotypic change over the life span. If the feedback sensitivity hypothesis is correct, CR animals should display a significantly different pattern of hormone-sensitive cell density and distribution in the hypothalamus. Of the many endocrine signal receptors that may be involved in maintaining hypothalamic feedback sensitivity, our study has selected to begin mapping those for estrogen (E). Altered hypothalamic sensitivity to E is known to schedule reproductive maturation and influence reproductive senescence. Taking estrogen receptor-alpha (ERalpha) immunoreactivity as an index of sensitivity to E, we counted ERalpha immunoreactive and non-immunoreactive cells in the pre-optic hypothalamus of young (6 weeks), ad-libitum (Old-AL) fed old (22 months), and calorie restricted (Old-CR) old (22 months) female B6D2F1 mice. An automated imaging microscopy system (AIMS) was used to generate cell counts for each sampled section of pre-optic hypothalamus. Results show a 38% reduction in ERalpha immunoreactive cells and an 18% reduction in total cell numbers in AL-old mice in comparison to young mice. However, CR mice only show a 19% reduction in ERalpha immunoreactive cells and a 13% reduction in total cell numbers in comparison to young mice. This indicates CR prevents age-related cell loss and maintains estrogen sensitivity in the pre-optic hypothalamus of old female B6D2F1 mice.
Neuroendocrinology Letters 2005 Jun;26(3):197-203.
Premature aging in mice expressing defective mitochondrial DNA polymerase
Researchers in Sweden have created homozygous knock-in mice that express a proof-reading-deficient version of PolgA, the nucleus-encoded catalytic subunit of mitochondrial DNA (mtDNA) polymerase. The mice encountered a threefold to fivefold increase in the levels of point mutations as well as increased amounts of deleted mtDNA. The deterioration in the experimental mice started at 25 weeks young adulthood in normal mice. They prematurely experienced a range of familiar age-related complaints, including baldness, osteoporosis, anemia, curvature of the spine and reduced fertility. The lifespan of the experimental mice was markedly reduced, with the median age of death at 48 weeks. The oldest of the experimental mice died before 61 weeks. Normal mice live approximately 110 weeks.
Nature 2004 May 27; 429, 357 - Ageing: Mice and mitochondria
Nature 2004 May 27; 429, 417 - 423 - Premature aging in mice expressing defective mitochondrial DNA polymerase
Scientists release draft of the rat genome
An international research team, supported by the National Institutes of Health (NIH), today announced it has completed a high-quality, draft sequence of the genome of the laboratory rat, and has used that data to explore how the rat's genetic blueprint stacks up against those of mice and humans. In the Nature article, the researchers reported that, at approximately 2.75 billion base pairs, the rat genome is smaller than the human genome, which is 2.9 billion base pairs, and slightly larger than mouse genome, which is 2.6 billion base pairs. Rat genome contains about the same number of genes as the human and mouse genomes. Furthermore, almost all human genes known to be associated with diseases have counterparts in the rat genome and appear highly conserved through mammalian evolution, confirming that the rat is an excellent model for many areas of medical research. The rodent lineage, which gave rise to the rat and mouse, and the primate lineage, which gave rise to humans, diverged about 80 million years ago. Humans have 23 pairs of chromosomes, while rats have 21 and mice have 20.
The Journal Nature - Scientists release draft of the rat genome
Nature. 2004 Apr 1;428(6982):493-521. - Genome sequence of the Brown Norway rat yields insights into mammalian evolution.
Dr. Andrzej Bartke is awarded $850,000.00 for aging research from the Ellison Medical Foundation (EMF)
This award to Dr. Bartke is a four-year national grant for the study of the effects of genes and hormones on aging. Total budget for the grant is $850,000. The study will look at the growth and thyroid hormones and their roles in regulating glucose metabolism and determining the rate of aging and length of life in Ames dwarf mice. Dr. Bartke's research is providing new insights into the mechanisms that control the aging process.
October 10, 2003 - SIU Med School Faculty Member Receives National Research Award
Selected papers
Ageing is reversed, and metabolism is reset to young levels in recovering dauer larvae of C. elegans.
Houthoofd K, Braeckman BP, Lenaerts I, Brys K, De Vreese A, Van Eygen S, Vanfleteren JR.
The nematode Caenorhabditis elegans responds to unfavourable environmental conditions by arresting development and entering diapause as a dauer larva. Dauers can survive several times the normal life span and the duration of the dauer state has no effect on postdauer life span. This led to the suggestion that dauers are non-ageing, and that dauers eventually perish as the consequence of depletion of stored nutrients. We have investigated physiological changes associated with long-term diapause survival, and found that dauer larvae slowly develop senescence-like symptoms, including decrease of metabolic capacity, aconitase enzyme activity, and ATP stores, and increase of lipofuscin- and oxidised flavin-specific fluorescence. However, these changes are reversed when the dauers recover. Thus senescence can occur before attainment of reproductive maturity, and furthermore, is reversible. Other life processes, including respiration rate and heat output, remain unaltered over four weeks of diapause at 24 degrees C. Possible determinants of the enhanced life maintenance include increased resistance to oxidative stress provided by enhanced superoxide dismutase and catalase activities, and a shift to a highly reducing redox status.
Pubmed - Exp Gerontol. 2002 Aug-Sep;37(8-9):1015-21
Exp Gerontol. - Exp Gerontol. 2002 Aug-Sep;37(8-9):1015-21
Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome
Eriksson M, Brown WT, Gordon LB, Glynn MW, Singer J, Scott L, Erdos MR, Robbins CM, Moses TY, Berglund P, Dutra A, Pak E, Durkin S, Csoka AB, Boehnke M, Glover TW, Collins FS
Hutchinson-Gilford progeria syndrome (HGPS) is a rare genetic disorder characterized by features reminiscent of marked premature ageing. Here, we present evidence of mutations in lamin A (LMNA) as the cause of this disorder. The HGPS gene was initially localized to chromosome 1q by observing two cases of uniparental isodisomy of 1q-the inheritance of both copies of this material from one parent-and one case with a 6-megabase paternal interstitial deletion. Sequencing of LMNA, located in this interval and previously implicated in several other heritable disorders, revealed that 18 out of 20 classical cases of HGPS harboured an identical de novo (that is, newly arisen and not inherited) single-base substitution, G608G(GGC > GGT), within exon 11. One additional case was identified with a different substitution within the same codon. Both of these mutations result in activation of a cryptic splice site within exon 11, resulting in production of a protein product that deletes 50 amino acids near the carboxy terminus. Immunofluorescence of HGPS fibroblasts with antibodies directed against lamin A revealed that many cells show visible abnormalities of the nuclear membrane. The discovery of the molecular basis of this disease may shed light on the general phenomenon of human ageing.
Nature - 2003 May 15;423(6937):293-8
Pubmed - Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome
Arclab - May 11, 2003 to May 17, 2003
Scientists release draft of the mouse genome
US and British Scientists released a draft copy of the mouse genome online for researchers to use freely. The information that is contained in the mouse genome will help researchers who use the rodent for studying a host of human ailments. In 2001 Celera Gennomics developed their own map of the mouse genome, but they change a fee in order to access their information, where as the US and British Scientists released their data for free. The mouse genome consists of approximately 2,700,000,000 base pairs that code for approximately 30,000 genes.
Mouse Genome Resources - National Institutes of Health (NIH), USA
Mouse Genome Centre - Medical Research Council, UK
The effects of aging on gene expression in the hypothalamus and cortex of mice
Cecilia H. Jiang, Joe Z. Tsien, Peter G. Schultz, and Yinghe Hu
Proc. Natl. Acad. Sci. USA, Vol. 98, Issue 4, 1930-1934
A better understanding of the molecular effects of aging in the brain may help to reveal important aspects of organismal aging, as well as processes that lead to age-related brain dysfunction. In this study, we have examined differences in gene expression in the hypothalamus and cortex of young and aged mice by using high-density oligonucleotide arrays. A number of key genes involved in neuronal structure and signaling are differentially expressed in both the aged hypothalamus and cortex, including synaptotagmin I, cAMP-dependent protein kinase C , apolipoprotein E, protein phosphatase 2A, and prostaglandin D. Misregulation of these proteins may contribute to age-related memory deficits and neurodegenerative diseases. In addition, many proteases that play essential roles in regulating neuropeptide metabolism, amyloid precursor protein processing, and neuronal apoptosis are up-regulated in the aged brain and likely contribute significantly to brain aging. Finally, a subset of these genes whose expression is affected by aging are oppositely affected by exposure of mice to an enriched environment, suggesting that these genes may play important roles in learning and memory.
The effects of aging on gene expression in the hypothalamus and cortex of mice - Proc. Natl. Acad. Sci. USA, Vol. 98, Issue 4, 1930-1934
Extended Life-Span Conferred by Cotransporter Gene Mutations in Drosophila
Blanka Rogina, Robert A. Reenan, Steven P. Nilsen, Stephen L. Helfand
Science 2000 290: 2137-2140
Aging is genetically determined and environmentally modulated. In a study of longevity in the adult fruit fly, Drosophila melanogaster, we found that five independent P-element insertional mutations in a single gene resulted in a near doubling of the average adult life-span without a decline in fertility or physical activity. Sequence analysis revealed that the product of this gene, named Indy (for I'm not dead yet), is most closely related to a mammalian sodium dicarboxylate cotransporter--a membrane protein that transports Krebs cycle intermediates. Indy was most abundantly expressed in the fat body, midgut, and oenocytes: the principal sites of intermediary metabolism in the fly. Excision of the P element resulted in a reversion to normal life-span. These mutations may create a metabolic state that mimics caloric restriction, which has been shown to extend life-span.
Science - 2000 Dec 15;290(5499):2048.
Pubmed - Extended Life-Span Conferred by Cotransporter Gene Mutations in Drosophila
Arclab - Dec 10, 2000 to Dec 16, 2000
The Center for Research and Education in Aging (CREA) will be officially launched November 8th, 2000
On November 8th, 2000 the Center for Research and Education in Aging, which has a mission of "investigating the basic processes that cause aging, with the goal of improving and extending human health span." will be officially launched. For more information about this important and groundbreaking organization or to find out how you can help CREA achieve its mission, please visit CREA's website at:
http://crea.berkeley.edu
Regulation of C. elegans life-span by insulinlike signaling in the nervous system
Wolkow CA, Kimura KD, Lee MS, Ruvkun G
Science 2000 Oct 6;290(5489):147-50
An insulinlike signaling pathway controls Caenorhabditis elegans aging, metabolism, and development. Mutations in the daf-2 insulin receptor-like gene or the downstream age-1 phosphoinositide 3-kinase gene extend adult life-span by two- to threefold. To identify tissues where this pathway regulates aging and metabolism, we restored daf-2 pathway signaling to only neurons, muscle, or intestine. Insulinlike signaling in neurons alone was sufficient to specify wild-type life-span, but muscle or intestinal signaling was not. However, restoring daf-2 pathway signaling to muscle rescued metabolic defects, thus decoupling regulation of life-span and metabolism. These findings point to the nervous system as a central regulator of animal longevity.
Regulation of C. elegans life-span by insulinlike signaling in the nervous system - Science 2000 Oct 6;290(5489):147-50
International Human Genome Sequencing Consortium Announces "Working Draft" of Human Genome
The Human genome consists approximately 3.12-gigabases that code for 80,000 genes. The work, carried out in 16 centres across the world, means that 85% of the human genome has been accurately deciphered. Further work, still to be finally checked, means in total 97% of the human genome has been read. The existence of this genetic map will lay the foundation for a revolution in medical diagnosis and treatment.
Joint Genome Institute
The Insitute for Genomic Research
University of Utah: Human Genetics Department
University of Washington Genome Center
• The Human Genome Project
• The Human Genome Project: From Maps to Medicine
Human Genome Sequencing
The Human Genome Organisation (HUGO)
Genome Sequence Centre (Canada)
Sanger Centre (U.K.)
CDC - Office of Genetics
Department of Energy-Life Sciences Division
Celera Genome Research
• Genomics Education
Cooperative Human Linkage Center
Whitehead Institute for Biomedical Research, MIT
Genethon Genome Research (France)
Centre for Human Genome Research (Denmark)
Los Alamos National Laboratory: Center for Human Genome Studies
Stanford Human Genome Center
Advanced Center for Genome Technology, University of Oklahoma
CNN - 26 June 2000 - Genome announcement a milestone, but only a beginning
BBC - 26 June 2000 - Scientists crack human code
CBC - 26 June 2000 - Mapping the genetic code
TIME - JULY 3, 2000 VOL. 156 NO. 1 - MAPPING THE GENOME
NOTE: Also refer to the article on sequencing of Drosophila melanogaster, dated March 23rd, 2000.
NOTE: Also refer to the article on sequencing of C. elegans, dated December 11th, 1998.
The Drosophila melanogaster genome has been fully sequenced.
REVIEW The 180-megabase genomic sequence of Drosophila melanogaster reveals 13,601 genes. In Drosophila, 60 Mb of the 180-Mb genome consists of centric heterochromatin. The C. elegans was fully sequenced on December 11th, 1998 and it had 97-megabases and 19,000 genes.
Science 2000 Mar 24;vol 1287, issue 5461. page 2181
The Berkeley Drosophila Genome Project (BDGP)
The European Drosophila Genome Project
FlyBase - At Harvard University
NOTE: Also refer to the article on C. elegans dated December 11th, 1998.
First human chromosome is fully sequenced
Scientists with the Human Genome Project have sequenced chromosome 22, the first chromosome to have been fully mapped. The results of this affort are published in the December 2nd issue of the journal Nature. The sequence obtained consists of 12 contiguous segments spanning 33.4 megabases, contains at least 545 genes and 134 pseudogenes, and provides the first view of the complex chromosomal landscapes that will be found in the rest of the genome. More than 30 human disorders are already associated with changes to genes of chromosome 22. These include schizophrenia, disorders of fetal development and the nervous system. The next chromosome to be fully sequenced will most likely be chromosome 7.
NIH Link to Chromosome 22
Human Genome Organisation link to Chromosome 22
The Sanger Centres link to Chromosome 22
The DOE's Joint Genome Institute
Real Audio 80K clip on the sequenceing of chromosome 22
Suppression of p66shc protein, extends the life span of mice 30%
The following describes mutant mice that seem to live almost one-third longer than wild-type animals. These mice, which have a single targeted mutation in the gene encoding the p66shc protein, develop and eat normally, and have a normal body weight. But the absence of p66shc confers a heightened cellular resistance to agents that cause oxidative damage. Although the conclusions must be regarded as provisional until larger groups of animals are studied, the results support the proposal2 that oxidative damage is involved in ageing. They also indicate that modification of the response to oxidative damage can have a considerable effect on lifespan, without apparent negative side effects.
NIH OMIM link - SHC TRANSFORMING PROTEIN
NIH LocusLink - SHC TRANSFORMING PROTEIN
Nature 1999 Nov 18;402(6759):309-13 The p66shc adaptor protein controls oxidative stress response and life span in mammals.
ABC News - Scientists Find Gene Switch Makes Mice Live Longer
Buck Center for Research in Aging celebrates the opening of the Research, Support and Education Building
From September 30th 1999 to October 1st 1999 the Buck Center for Research in Aging in Novato, California, held an inaugural symposiam on aging to celebrate the opening of the center. The Buck Center is located 20 miles north of the Golden Gate Bridge in northern Marin County. The Buck Center receives 5.5 millions annually from the Buck Trust and is governed by its own Board of Directors.
Buck Center for Research in Aging
Geron acquires Roslin Bio-Med and forms research collaboration with the Roslin Institute
MENLO PARK, CA and ROSLIN, MIDLOTHIAN, SCOTLAND - May 4, 1999
Alliance brings together three complementary breakthrough technologies: human pluripotent stem cells, telomerase expression and nuclear transfer.
The combined technologies are expected to enhance and accelerate the development of new transplantation therapies for numerous degenerative diseases such as diabetes, Parkinson’s disease, cancer and heart disease.
Geron and the Roslin Institute are pioneers in these technologies and together are in a strong position to capitalize on their potential.
Dr. Judith Campisi is awarded $964,000.00 for aging research
Berkeley, CA - March 26, 1999 - The Ellison Medical Foundation has awarded Dr. Judith Campisi $964,000.00 to support her basic investigations into the causes of aging. The Foundation was setup by Larry Ellison, CEO of Oracle, one of the largest database software corporations in the United States. Judith Campisi is a researcher in the Department of Cell and Molecular Biology at the Lawrence Berkeley National Laboratory in Berkeley, California.
Mouse model demonstrates role of telomeres and telomerase in aging, cancer and lifespan
Menlo Park, CA - March 4, 1999 - Geron Corporation announced that researchers at the Dana-Farber Cancer Institute of Harvard Medical School and colleagues at Johns Hopkins School of Medicine have published in the March 5 issue of Cell that telomerase-negative mice show telomere loss with age and experience a host of age-related changes. This study expands on a growing body of in vivo proof that age-related telomere erosion contributes to pathology, including cancer. Significant new findings in this work include a causal link between telomere loss and two hallmarks of human aging: an impaired ability to recover from stress and an increased rate of cancer formation. Consistent with other data linking telomerase activity with tumor progression, the authors suggest that telomere loss contributes to cancer formation, but lack of telomerase inhibits long-term tumor growth.
Geron announces first in Vivo data indicating telomerase expression in normal human cells extends their replicative lifespan without oncogenic transformation.
Geron Corp and the University of Texas Southwestern Medical Center at Dallas announced today the publication of two papers in the Jan 1, 1999, issue of Nature Genetics.
Human cells cloned.
A group at Kyunghee University Hospital in Seoul, South Korea, combined an egg and a cell from a single donor to produce the first stages of a human embryo. The experiment was terminated by the reseachers after a few cell divisions had taken place.
Genome sequence of the nematode C. elegans: A platform for investigating biology the C. elegans sequencing consortium.
REVIEW The 97-megabase genomic sequence of the nematode Caenorhabditis elegans reveals over 19,000 genes. More than 40 percent of the predicted protein products find significant matches in other organisms. There is a variety of repeated sequences, both local and dispersed. The distinctive distribution of some repeats and highly conserved genes provides evidence for a regional organization of the chromosomes
Science 1998 Dec 11;282(5396):2012-8
The genetics of aging as it relates to the C. elegans
Life extension and stress resistance in Caenorhabditis elegans modulated by the tkr-1 gene.
Molecular genetics of life span in C. elegans: how much does it teach us?
Genetic control of programmed cell death and aging in the nematode Caenorhabditis elegans.
A genetic pathway conferring life extension and resistance to UV stress in Caenorhabditis elegans.
Direct isolation of longevity mutants in the nematode Caenorhabditis elegans.
Defining genes that govern longevity in Caenorhabditis elegans.
Identification genes that are differentially expressed during aging in Caenorhabditis elegans
First Cows are Cloned
Eight calves, four of which died during birth, are cloned from a single caw by a team led by Dr. Yukio Tsunoda of Kinki University in Nara, Japan
Embryonic Stem Cell Lines Derived from Human Blastocysts.
Thomson JA, Itskovitz-Eldor J, Shapiro SS, Waknitz MA, Swiergiel JJ, Marshall VS, Jones JM J. A. Thomson, M. A. Waknitz, J. J. Swiergiel, V. S. Marshall
Wisconsin Regional Primate Research Center, University of Wisconsin, Madison, WI 53715, USA. J. Itskovitz-Eldor, Department of Obstetrics and Gynecology, Rambam Medical Center, Faculty of Medi cine, Technion, Haifa 31096, Israel. S. S. Shapiro and J. M. Jones, Department of Obstetrics and Gynecology, University of Wisconsin, Madison, WI 53715, USA.
Science 1998 Nov 6;282(5391):1145-1147
Human blastocyst-derived, pluripotent cell lines are described that have normal karyotypes, express high levels of telomerase activity, and express cell surface markers that characterize primate embryonic stem cells but do not characterize other early lin eages. After undifferentiated proliferation in vitro for 4 to 5 months, these cells still maintained the developmental potential to form trophoblast and derivatives of all three embryonic germ layers, including gut epithelium (endoderm); cartilage, bone, smooth muscle, and striated muscle (mesoderm); and neural epithelium, embryonic ganglia, and stratified squamous epithelium (ectoderm). These cell lines should be useful in human developmental biology, drug discovery, and transplantation medicine.
Embryonic Stem Cell Lines Derived from Human Blastocysts Science Volume 282, Number 5391 Issue of 6 Nov 1998, pp. 1145 - 1147
New Potential for Human Embryonic Stem Cells Science Volume 282, Number 5391 Issue of 6 Nov 1998, pp. 1061 - 1062
Derivation of pluripotent stem cells from cultured human primordial germ cells
Michael J. Shamblott*, Joyce Axelman*, Shunping Wang*, Elizabeth M. Bugg*, John W. Littlefield, Peter J. Donovan, Paul D. Blumenthal, George R. Huggins, and John D. Gearhart,
Departments of Gynecology and Obstetrics and Physiology, Johns Hopkins University School of Medicine, Baltimore, MD 21287; Kimmel Cancer Institute, Jefferson Medical College, Philadelphia, PA 19107; and Department of Gynecology and Obstetrics, Johns Ho pkins Bayview Hospital, Baltimore, MD 21224
PNAS Vol. 95, Issue 23, 13726-13731, November 10, 1998
Human pluripotent stem cells would be invaluable for in vitro studies of aspects of human embryogenesis. With the goal of establishing pluripotent stem cell lines, gonadal ridges and mesenteries containing primordial germ cells (PGCs, 5-9 weeks postfertil ization) were cultured on mouse STO fibroblast feeder layers in the presence of human recombinant leukemia inhibitory factor, human recombinant basic fibroblast growth factor, and forskolin. Initially, single PGCs in culture were visualized by alkaline ph osphatase activity staining. Over a period of 7-21 days, PGCs gave rise to large multicellular colonies resembling those of mouse pluripotent stem cells termed embryonic stem and embryonic germ (EG) cells. Throughout the culture period most cells within t he colonies continued to be alkaline phosphatase-positive and tested positive against a panel of five immunological markers (SSEA-1, SSEA-3, SSEA-4, TRA-1-60, and TRA-1-81) that have been used routinely to characterize embryonic stem and EG cells. The cul tured cells have been continuously passaged and found to be karyotypically normal and stable. Both XX and XY cell cultures have been obtained. Immunohistochemical analysis of embryoid bodies collected from these cultures revealed a wide variety of differe ntiated cell types, including derivatives of all three embryonic germ layers. Based on their origin and demonstrated properties, these human PGC-derived cultures meet the criteria for pluripotent stem cells and most closely resemble EG cells.
Derivation of pluripotent stem cells from cultured human primordial germ cells Proceedings of the National Academy of Sciences Vol. 95, Issue 23, 13726-13731, November 10, 1998
The 77 year old John Glenn and his shuttle mission.
John Glenn's science experiments that pertain to the study of aging.
50 mice cloned by researchers at the University of Hawaii
Over 50 female mice were cloned by a team led by Ryuzo Yanagimachi, a University of Hawaii scientist. They used a cumulous cell,a type of cell that comes from the ovaries of females but is not an egg cell,then they extracted the nucleus and placed it inside a hollowed-out egg cell using a tiny pipette. Then they implanted the egg cell into the uterus of a female animal. Cloning shows that adult cells can be reprogrammed to start over and develop into a whole creature, the question is will the cloned animals age normally or will they age at a faster rate? Details of the experiment were published in the July 22nd, 1998 issue of Nature.
Gene found which is linked to Alzheimer's disease
A Gene has been found which is linked to Alzheimer's diseas. The new discovery was reported on the 22 July 1998 at a conference in Amsterdam. Geneticist Rudolph E. Tanzi and his research team at Massachusetts General Hospital in Boston identified a gene called A2M which is responsible for producing a protein which strongly interacts with proteins produced by two of the genes-called LRP and APOE- linked to familial Alzheimer's.The study shows that people with the defective form of the gene are three and one half times more likely to develop late-onset Alzheimer's. The disease is believed to be caused by the accumulation of waxy plaques, composed largely of a protein called beta-amyloid, which kills brain cells. The study will be published in the August 1st, 1998 issue of Nature Genetics.
Nature - Genetics
Fruit flies bred with copies of the human SOD1 gene live as much as 40 percent longer.
June 2, (1998).
Gabrielle Boulianne, a neurobiologist at the Hospital for Sick Children and a professor of molecular and medical genetics at the University of Toronto reports in an article in the June issue of the journal Nature Genetics that she and her co-workers bred Drosophila melanogaster with copies of the human SOD1 gene and the result was a fly that could live as much as 40 percent longer.
Extension of Drosophila lifespan by overexpression of human SOD1 in motorneurons
Tony L. Parkes1, Andrew J. Elia2, Dale Dickinson1, Arthur J. Hilliker1, John P. Phillips1 & Gabrielle L. Boulianne2
Reactive oxygen (RO) has been identified as an important effector in ageing and lifespan determination1-3. The specific cell types, however, in which oxidative damage acts to limit lifespan of the whole organism have not been explicitly identified. The association between mutations in the gene encoding the oxygen radical metabolizing enzyme CuZn superoxide dismutase (SOD1) and loss of motorneurons in the brain and spinal cord that occurs in the life-shortening paralytic disease, Familial Amyotrophic Lateral Sclerosis (FALS; ref. 4), suggests that chronic and unrepaired oxidative damage occurring specifically in motor neurons could be a critical causative factor in ageing. To test this hypothesis, we generated transgenic Drosophila which express human SOD1 specifically in adult motorneurons. We show that overexpression of a single gene, SOD1, in a single cell type, the motorneuron, extends normal lifespan by up to 40% and rescues the lifespan of a short-lived Sod null mutant. Elevated resistance to oxidative stress suggests that the lifespan extension observed in these flies is due to enhanced RO metabolism. These results show that SOD activity in motorneurons is an important factor in ageing and lifespan determination in Drosophila.
REFER TO:
Role of oxidative stress in Drosophila aging.- Mutat Res 1992 Sep;275(3-6):267-279
Expression of bovine superoxide dismutase in Drosophila melanogaster augments resistance of oxidative stress.- Mol Cell Biol 1991 Feb;11(2):632-640
Extension of Life-Span by Introduction of Telomerase into Normal Human Cells
Science, Jan 16, (1998).
Andrea G. Bodnar, Michel Ouellette, Maria Frolkis Shawn E. Holt, Choy-Pik Chiu, Gregg B. Morin, Calvin B. Harley, Jerry W. Shay, Serge Lichtsteiner, Woodring E. Wright
Rel 2.9 - 98/01/29
Letter to Nature -- Viable offspring derived from fetal and adult mammalian cells
Nature, Volume 385, 810 - 813, February 27, 1997
See Nature Web Special: Cloned Sheep
Scientists clone first adult mammal
"They found a way of stopping the clock and
programming the genes so they go back to the
beginning," James told Reuters in a telephone
CNN, February 24, 1997
The Telomerase Picture Fills In
SCIENCE - 25 April 1997 - Volume 276 - Number 5312
Positional Cloning of the Werners syndrome Gene - Abstract, Summary
SCIENCE - 12 Apr 1996; 272 (5259):258-262
THE NEW SCIENCE OF AGING
A TV series that was shown on PBS Wednesday, June 2, 1999, 8:00 p.m. ET
For information about the videocassettes from PBS
For more details about this documentary
If you do not have the Real Audio program that you need to listen to the programs below, please click on this message and you may then download the program on to your system.
Sep 16, 2003 - KQED Forum
Aging Research
Cynthia Kenyon
Herbert Boyer Professor of Biochemistry and Biophysics
Director, Hillblom Center for the Biology of Aging
Dale Bredesen,
founding president and CEO,
Buck Institute for Age Research
David Sinclair,
Professor of pathology,
Philip Cohen,
San Francisco bureau chief for New Scientist magazine
May 9, 2003 - NPR Talk of the Nation
Genetics of Aging
Ted Gavin
SpamCon Foundation
Phil Goldman
MailBlocks
David Sinclair
Assistant Professor of Pathology
August 31, 2001 - NPR Talk of the Nation: Science Friday
Longevity and Aging
Author, "Reversing Human Aging"
Clinical Professor of Medicine
Steven Austad
Author, "Why We Age: What Science Is Discovering About the Body's Journey Through Life" (Wiley)
Associate Professor, Zoology
Gerontologist
Moscow, Idaho
Thomas Perls
Author, "Living to 100: Lessons in Living to Your Maximum Potential at Any Age" (Basic Books)
Geriatrician, Gerontology Division and Biometrics Center
Associate Professor, Division on Aging
Michael McKubre
Director, Energy Research Center
SRI Incorporated
June 26, 2000 - NPR Talk of the Nation
The Human Genome
The ten year race to complete a map of the human genetic code has ended in a tie. The publicly funded Human Genome Project and the private corporation Celera Genomics are announcing today that their work on a rough draft of a map of the human genome is complete. The map won't lead to medical miracles overnight, but once the information has been studied it could lead to the development of new drugs and therapies to treat diseases. Join Juan Williams and guests to discuss what a complete map of the human genome will mean for the future of medicine and humanity.
June 9, 2000 - NPR Talk of the Nation
The Human Genome and a genetics update
The public-private race to get the first complete map of the human
genome is nearing the finish line, with both groups expected to
announce a working draft sometime this month. Then the real work
begins: finding the genes along those long stretches of DNA and
determining their function. This hour, we'll get an update on the latest
genetics news.
Bruce Roe
George Lynn Cross Research Professor of Chemistry and Biology
Director, Advanced Center for Genome Technology
Fred Cohen
DoubleTwist
Professor, Medicine and Pharmacology
Maja Bucan
Associate Professor, Genetics
Center for Neurobiology and Behavior
University of Pennsylvania Medical Center
May 14, 1999 - NPR Talk of the Nation: Science Friday
AGING RESEARCH UPDATE
Researchers from Columbia University announced that they have identified an enzyme crucial to extending the lifespan of roundworms. Articles Discussed: "A cytosolic catalase is needed to extend adult lifespan in C. elegans daf-C and clk-1 mutants, "
by James Taub, Joe F. Lau, Charles Ma, Jang Hee Hahn, Rafaz Hoque, Jonathan Rothblatt and Martin Chalfie.
Nature, May 13, 1999.
Martin Chalfie
Professor, Biological Science
Author, Why We Age: What Science is Discovering About the Body's Journey Through Life (John Wiley & Sons)
January 29, 1999 - NPR Talk of the Nation: Science Friday
STEM CELL UPDATE
Brief Text Description:
Human embryonic stem cell research, which holds the promise of curing many diseases, has been hampered by a lack of funds. But last week, the federal government ruled that much of this work is eligible to receive federal funds, a decision that the National Bioethics Advisory Commission is slated to debate.
John Gearhart
Professor of Gynecology and Obstetrics
Eric Meslin
National Bioethics Advisory Commission
Bruce Torbett
Scripps Research Institute
AGING UPDATE
For the first time, scientists have been able to get human cells in the lab to live much longer than normal, perhaps paving the way for new treatments for age-related diseases and cancer. In this hour, we'll get an update on the latest aging research. Plus, a new way of thinking about how the brain stores memories.
Calvin Harley
Geron Corporation
Author, Reversing Human Aging, (William Morrow)
Howard Hughes Medical Institute
Columbia University, New York, New York
Dusan Bartsch
September 5, 1997 - NPR Talk of the Nation: Science Friday
Gary Ruvkun
Faculty Member, Department of Genetics,
Author, Reversing Human Aging, William Morrow and Company
Founder and Vice President,
Geron Corporation,
April 12, 1996 - NPR Talk of the Nation: Science Friday
THE SCIENCE OF AGING
Gerard Schellenberg
Associate Director of Research
Geriatric Research, Education, and Clinical Center
Veterans Affairs Puget Sound Health Care System
Stanley Rapoport
Chief of Laboratory of Neuroscience
Professor of Clinical Medicine
Reversing Human Aging
September 29, 1995 - NPR Talk of the Nation: Science Friday
BIOLOGY OF AGING
Dr. Leonard Hayflick
Jerry Shay
Professor of Cell Biology and Neuroscience
Dr. Michael Fossel,
Author of forthcoming book, "Reversing Human Aging"
Ronald Crystal
Professor of Medicine and Chief of the Division of Pulmonary
and Critical Care Medicine
The New York Hospital-Cornell Medical Center
Jeffrey Leiden
Professor of Medicine, Professor of Pathology, and Chief of
the Section of Cardiology
Theories of Aging
Papers on different Theories of Aging
Upcoming Meetings and Conferences
Events without dates should be considered as tentative.
Calender of Upcoming Meetings/Conferences
Articles pertaining to the aging process can be found in the journals listed below.
AGING Clinical and Experimental Research
Ageing Research Reviews
American Journal of Respiratory Cell and Molecular Biology
Biochemistry and Molecular Biology International
Biogerontology
Biology of the Cell
Blood - American Society of Hematology
Cell and tissue research
Cell Biochemistry and Function
Cell Biology International
Cellular and Molecular Neurobiology
Cellular immunology
Current Advances in Cell & Developmental Biology
Cytogenetics and Cell Genetics
Development genes and evolution
Development, growth & differentiation
Developmental Brain Research
Developmental genetics
Developmental Review
EMBO - European Molecular Biology Organization
Endocrine reviews
Experimental Cell Research
FASEB - Federation of American Societies for Experimental Biology
Free Radical Biology and Medicine
Gene Structure and Expression
Genes to cells
Growth Hormone & IGF Research
International Journal of Biochemistry and Cell Biology
International Journal of Developmental Neuroscience
Journal of Anti-Aging Medicine
Journal of Endocrinology
Journal of Molecular Biology
Mechanisms of Ageing and Development
Mechanisms of Development
Molecular and Cellular Endocrinology
Molecular Brain Research
Mutation Research
Mutation Research-DNA Repair
Nature - Biotechnology
Nature - Neuroscience
Neurobiology of Aging
Seminars in Cell and Developmental Biology
Trends in Biochemical Sciences
Trends in Biotechnology
Trends in Cell Biology
Trends in Endocrinology and Metabolism
Bio Tool Box
Scientists Involved in Aging Research
Laboratories Involved in Aging Research
Biotech Companies
Companies Involved in Aging Research
Bill Board - Research Opportunities, Calls For Research and News
Under Construction:
If you have any comments or suggestions with regards to this Web site, please E-mail them to
MAIL BOX: sgaran@arclab.org
Help us make this a bigger and better site.
- Steven A. Garan -
- August 21st 1994 -
aging process
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Moving Forward: A Revival & New Works at Bruce Wood Dance Project
Manuel Mendoza October 20, 2015
Dallas/Ft WorthDance5 min read
Bruce Wood Dance Project dancers Joy Bollinger and Harry Feril in Bruce Wood’s Liturgy.
Photo by Brian Guilliaux.
Bruce Wood Dance Project dancers Albert Drake and Eric Coudron in Albert Drake’s Whispers. Photo by Sharon Bradford.
In the midst of its fifth season, Dallas’ Bruce Wood Dance Project is about to premiere two new works and revive a decade-old piece by its namesake choreographer. But neither of the debuts is by Wood, who died in May 2014 at 53.
The company’s board of directors made the difficult decision to go on without their artistic director and chief choreographer. One of their main conundrums was how to stay relevant without new dances from the prolific Wood. He wouldn’t have wanted the troupe to simply become a museum for his past triumphs.
The Dance Project’s answer has been to tap company members mentored by Wood to create new pieces. The first, Whispers, choreographed by Southern Methodist University graduate Albert Drake, premiered at the group’s season-opening shows in June. Now, longtime Wood muse Kimi Nikaidoh, who has replaced him as artistic director, is making a new work for the Dance Project’s Nov. 13-14 run at Dallas City Performance Hall.
“I never dreamed of being a director, and I certainly would prefer if Bruce were here and I was getting to be a dancer working under his direction. I miss him terribly,” Nikaidoh says in an interview at the Bruce Wood Dance Gallery in the Dallas Design District. “Thankfully, though, I’m growing to love the project of figuring out how to keep his work alive, and I’m getting excited about some of the things that turn out to be the answer, like creating and commissioning new work and taking this company as soon as we can on tour.”
Bruce Wood Dance Project Dancers Albert Drake and Emily Perry in Kimi Nikaidoh’s Find Me. Photo by Brian Guilliaux.
Nikaidoh’s Find Me, set to four Van Morrison songs, will close the November shows. “Bruce used to say that he saw a dance performance like a trip to Luby’s,” she recalls. “You have to finish with pie. So that’s what I chose to do for this program – make sure there was some pie at the end.”
The company also has commissioned a piece from emerging choreographer Bryan Arias, the first time it has gone outside for new work. Arias, a member of Crystal Pite’s Canadian troupe Kidd Pivot, has created My Heart Remembers, a series of four overlapping duets about love.
In addition, the Dance Project is on an aggressive campaign to spread the word about Wood’s oeuvre, which is largely unknown outside North Texas. In fact, many Wood works he choreographed for his first group, the Fort Worth-based Bruce Wood Dance Company, have never been seen in Dallas. The November shows will include one of those, 2005’s Liturgy, set to Bach’s Mass in B Minor for chorus and orchestra.
The company also is booking festivals and gearing up to pitch presenters so it can hit the road. Nikaidoh says that between rehearsing and performing, the dancers will work for 40 weeks next year. “We’re moving toward being a viable, full-time professional dance company.”
In December, the Dance Project will revive Mistletoe Magic, a cabaret-style show that premiered in 2013. It is also licensing Wood’s Red to Dallas Black Dance Theatre, to be taught to DBDT by Drake, company member Emily Perry and former Wood dancer Joy Atkins Bollinger. Then next year, it will participate in the AIDS benefit The Gathering at Winspear Opera House, the Dance!Texas festival in Arlington, the Chamberlain Ballet Gala and a Turtle Creek Chorale show honoring Wood and others as local heroes. A company member will make a new piece for the Heroes show.
Bruce Wood Dance Project dancers Kimi Nikaidoh and Harry Feril in Albert Drake’s Whispers. Photo by Sharon Bradford.
To seek national and international bookings, Nikaidoh plans to attend the Association of Performing Arts Presenters conference in New York in January. It usually takes a year or two for such efforts to come to fruition, which means the Bruce Wood Dance Project intends to be around for at least another five years.
“The fact that Bruce came back to the stage after the Bruce Wood Dance Company closed, that people were hungry enough to see his work again and got behind him and supported him has meant that all of us get to continue dancing and seeing his work,” Nikaidoh says. “We also get to continue creating and commissioning the kind of work that prioritizes the same things that he did, that hopefully touches people, that is food for their souls and not just a feast for their eyes.”
—MANUEL MENDOZA
Manuel Mendoza is a Dallas freelance writer specializing in the arts.
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NCAA Draft Prospects NBA Pre-Draft Camp
Portsmouth Invitational Tournament, Day Four (part one)
-Zabian Dowdell did not participate in todays game. He told a teammate that he had back spasms.
-Avis Wyatt was diagnosed with a lateral ankle sprain and will be out of commission for the next two weeks. He went down hard in yesterdays game and continued to play, but was not able to contribute much of anything after that, hence the poor performance. He was in street clothes supporting his team from the bench today.
Norfolk Naval Shipyard 113 - Tidewater Sealants 94
Carl Landry, 6-7, Power Forward, Purdue
25 points, 11 rebounds, 9-13 FG, 7-8 FT
Landry finally gave us a good reason to evaluate him in his third and final game today, salvaging what has otherwise been a fairly mediocre tournament with a solid all-around encore performance.
Landry finally managed to show off the many positive parts to his games to counter the negatives that were on full display in the first two games. Rather than insisting on digging in the trenches of the paint with his back to the basket and getting blocked underneath the hoop like he did numerous times over the past four days, he took more of a face the basket approach, which suits him much better. This is also clearly the role hell be playing in the NBA as well due to his size and skill-set.
Landry looked very good receiving the ball on the perimeter and either driving to the rim for a nice spin-move and up and under finish, or pulling up off the bounce from mid-range off a single dribble. This is exactly his game what his game could be in the NBA, finding spots from mid-range to get his shot off either spotting up or using a shot-fake to set himself up, ala Ryan Gomes. After getting blocked early on twice underneath the rim, showing his clear lack of explosiveness, he changed strategies and went for this approach instead. Rather than forcing the issue in heavy traffic on a drive, he stopped nicely and elevated for a short jumper.
On the glass, Landry also did a good job mixing it up, despite the fact that hes not particularly tall, long or quick off his feet. Defensively, he did a decent job, but its hard to draw too many conclusions due to the weak matchups he went up against.
At the end of the day, Landry probably isnt going to look back on Portsmouth as one of the highlights of his career. He did salvage a really nice showing out of the last day, though. Regardless, Landry will almost certainly get invited to the Orlando pre-draft camp, and hell have a chance to continue his quest of making an NBA team.
Blake Schilb, 67, Small Forward, Loyola-Chicago
25 points, 5 rebounds, 2 assists, 4 turnovers, 9-14 FG, 3-5 3PT, 4-5 FT
After scoring just two points in his first game, Schilb scored 16 in his second, and now put up an impressive 25-point performance in his last outing. Schilb hasnt really quelled any doubts about his athleticism or quickness here, but the small forward/point forward had a very strong game, showing off his versatile skill-set.
Schilb hit three 3-pointers on the game, boasting good shooting form and a decently quick release. He made one buzzer-beater here to close the second half, which was actually a bit of a runner from 30 feet out. Schilb also did some scoring around the hoop, showing off some nice creativity on his lay-ups, hitting a nice baseline reverse, a floater from five feet out that he was also fouled on, a finger-roll in transition, and a dunk over an opposing guard. He also had two nice post-up moves, taking advantage of his smaller opponents, one of which he backed down his man for a lay-up and the other which he posted up from 10 feet away on the baseline, turned, and swished a smooth fadeaway jumper. He didnt show off much in terms of dribble creation, and that doesnt seem like something hed excel at against this level of competition, as his dribble isnt especially low to the ground and he doesnt possess good quickness, leaving him vulnerable to poke-aways, which happened a couple of times today. He averaged four assists in college, playing a bit of a point forward role, but didnt show off much passing game here, as he seems like more of an opportunity passer as opposed to someone who can create via dribble penetration.
Schilb would have a tough time making an NBA roster with his quickness concerns, but he has an interesting skill-set and he does quite a few things well, which should get him invited to the Orlando camp again (he attended last season) and possibly a training camp. If he cant make it in the NBA, he should be a nice complementary player somewhere in Europe.
Sales Systems 81 - Beach Barton Ford 60
David Teague, 66, SG/SF, Purdue
18 points, 2 rebounds, 2 steals, 1 block, 2 turnovers, 7-12 FG, 2-4 3PT, 2-3 FT
David Teague had yet another strong performance in his third game here at Portsmouth, showing off a side of his game we hadnt yet seen before. Hes already shown an excellent shooting stroke and the ability to finish in transition, but today he displayed a bit of a mid-range game, hitting a few tough shots and showing he had the ability to pull up in the lane just as well as he takes the ball to the hoop. Teague doesnt have the greatest ball-handling skills, but he has a decent crossover and he doesnt try to force the ball through holes that arent there, so turnovers werent much of a problem. Teague hit a nice floater in the lane early in the game, where he recognized there were defenders in his way of the hoop would he have driven all the way there. Later in the game he hit a very impressive turnaround jumper in the lane from 10 feet, which he was also fouled on for the and-1. He also hit another floater in the lane from five feet off a crossover to his left hand.
Teague also did some damage from deep in this game, hitting two three-pointers, both of which came in fast-break situations where he got the dump off and put the ball up with an extremely quick release, once with a hand in his face. His stroke is smooth and his form is pretty solid. He also finished in transition a few times, once with a lay-up off the glass and another time with an explosive jam.
Defensively, Teague does a good job getting into his mans face, using his hands to disrupt and poke at the ball. He moves pretty well laterally, but he sometimes has trouble chasing through screens without the ball, but nothing more than the average player. He should have a very good chance of getting into Orlando as well as into a training camp, and will have a decent chance of making a roster as well, either at the beginning of the season or on a 10-day contract later on should he try his luck in the D-League. In terms of a comparison, he resembles Tarence Kinsey in many ways, though is by no means an identical player. He doesnt have quite the length of Kinsey, and he might need to work a bit harder on his mid-range game to get it to Kinseys level, but Teague is a better outside shooter and has a similar overall build. Kinsey played in both pre-draft camps last season, and was signed as an undrafted free agent, which is the most likely path to the NBA for Teague as well.
Brandon Wallace, 6-9, Power Forward, South Carolina
17 points, 5 rebounds, 0 assists, 2 turnvers, 2 blocks, 8-12 FG, 1-2 3P
This was another intriguing showing by South Carolinas raw power forward Brandon Wallace, doing a good job using his athleticism to the fullest on both ends of the floor. He clearly looks better suited for the up and down tempo we find here rather than the stagnant half-court offense he played in at South Carolina, and as a result has improved notably from game to game here.
Wallace ran the floor extremely well today and did a nice job presenting himself around the rim for easy finishes. He is a very frail power forward who cant finish very well if being contested excessively, but his length, terrific athleticism and excellent activity level help him out tremendously in this area. Wallace also stepped outside on one occasion and knocked down a very good looking 3-pointer. Hes got decent touch and form on his jump-shot, and could reasonably be expected to develop this part of his game into a weapon in a few years via the D-League or overseas. Wallace isnt the smartest or most skilled player in the world right now, but he has some nice tools that could intrigue some teams down the road, and therefore it will be interesting to track his progress over the next few seasons wherever he ends up.
Dan Nwaelele, 6-5, Shooting Guard, Air Force
17 points, 5 rebounds, 7-13 FG, 2-4 3P
The two things that stand out the most about Dan Nwaelele are his exceptionally smooth shot and the intelligence with which he plays. Nwaelele was the most efficient shooter in the NCAA this past season and it showed during the PIT. His ability to move quickly and with purpose to spots on the floor and get into his shooting motion immediately after the catch will be a real asset going forward into his pro career. Nwaeleles first step and overall explosiveness were surprising when seen in person. Hes not a super athlete, but hes certainly ahead of the curve in that department, at least compared to the initial impression you get when watching him play in Air Forces system.
Nwaelele was able to use the threat of his shot very effectively to create driving lanes for himself. Unlike some of the other guards at the PIT, he was able to finish above the rim even when the help defense was able to put a body on him. He is extremely compact and has good core strength for powering through initial contact to still get up.
The offensive element that was really missing from Nwaeleles game was a solid mid range game off of the dribble. He showed nice catch-and-shoot ability, but didnt pull up very often when he had the chance. This is understandable because he was able to finish effectively in the paint, but at his size it will be imperative to recognize that floaters, runners, and straight pull-up shots will most likely be the order of the day for him going forward. Still, there was a lot to like in terms of his total game and he reminds one a bit of Kelenna Azubuike in terms of his stature and build, though hes a superior perimeter shooter at this stage in his career.
Darryl Watkins, 6-11, Center, Syracuse
14 points, 10 rebounds, 1 block, 1 steal, 2 assists, 2 turnover, 6-7 FG, 2-2 FT
Watkins last showing of the PIT was a pretty successful one, again showing some nice skills that we didnt get much of a chance to see in his time at Syracuse, but also displaying some concerning weaknesses that go a long ways in explaining why he underachieved in college the way he did.
Watkins did most of his damage offensively by cutting to the basket and finishing around the hoop. On one occasion he stepped back and knocked down a 12 foot jumper from mid-range, confirming for us the notion that he has slightly more range on his shot than what we saw at Syracuse this year.
He got a little too cute at times finishing around the basket, opting to go for a one-handed scoop shot rather than just dunking it aggressively, a tendency that seems to carry over onto the defensive end, where he often doesnt go after it the way youd hope a guy with such limited offensive skills might. In general hes often a step slower on plays that involve quick decision making skills, sometimes leaving something to be desired in terms of his feel for the game. Its not that hes not quick enough to make sharp rotations, but rather a function of his ability to process information rapidly and be assertive in his moves.
With that said, Watkins has plenty of things working in his favor, all of which could very well secure him a spot in the NBA next year. He is big, strong and athletic, with an NBA frame and a good pedigree after spending four years under Jim Boeheim. His offensive skills are a work in a progress, but he has some decent tools to continue to improve. Well surely see how much progress he can make until late May during the pre-draft camp.
Stephane Lasme, 68, PF, Massachusetts
8 points, 9 rebounds, 3 blocks, 3 steals, 3 turnovers, 4-7 FG, 0-2 FT
Lasme had yet another fairly impressive game here, showing how he can use all his physical abilities to rebound and block shots, while also showing a bit of potential in terms of his scoring abilities. Lasme was very active on the defensive end in this game, netting 3 blocks and 3 steals. All three of his blocks came on driving guards in the lane, and he does a good job of fully extending his arm and avoiding body contact as to avoid fouling the opponent. Hes shown good timing on his blocks here, and with his athletic ability, this allows him to block or disrupt many shots in the lane. He also does a good job on the glass, boxing out well and using his strength and athleticism to secure rebounds.
Offensively, he had some nice plays, scoring on a contested lay-up in the lane off a cut on one possession. On another, he posted up his man from the baseline for a powerful drop-step jam. He also scored on a little fade-away shot from three feet out on a post-up, and put back an offensive rebound off a free throw. Lasmes offensive game is very raw at this stage, though he shows little flashes of one-dribble driving ability and post-up ability. He traveled on one baseline face-up drive in this game, which is a bit of a problem when he puts the ball on the floor.
Lasme is a virtual lock to get invited to the Orlando camp, and with his athletic abilities, defense, and rebounding, has an outside chance of making an NBA team in a garbage man-type role and as a good practice body for his energy. Worst case scenario, he should go to the D-League to work on some of his lesser skills, as he almost certainly can work himself into a contract in time, ala Justin Williams.
Ayinde Ubaka, 6-4, Point Guard, Cal
4 points, 6 rebounds, 1 assist, 3 turnover, 1 steal
After two excellent games to start off the tournament, Ubaka came down to earth and put up a real stinker in his teams 20-point blowout. His outside shot, one of the stronger points of his game, wasnt falling today at all, and he had major trouble getting into the paint against the extremely tough defense of Old Dominions Drew Williamson. Ubakas lack of quickness was exposed to a certain extent in this game, and he got too flashy with his ball-handling, over-dribbling and not doing a very good job running his teams offense. Ubaka should be able to redeem himself at the pre-draft camp in a few months, and he will surely draw interest thanks to his excellent size and intriguing offensive tools.
Zabian Dowdell PG/SG
Avis Wyatt PF/C
8.7 PER
Ryan Gomes SF/PF
Tarence Kinsey SG
Brandon Wallace PF
Dan Nwaelele SG/SF
Kelenna Azubuike SG
Almagra
Justin Williams PF/C
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Software Vendor Speeds Development, Taps New Markets with Cloud-Based Solution
“With all supplier information available in a central location, our customers spend less time discovering supplier information. …We have helped our customers reduce 25 percent or more of their time spent on supplier registration and selection processes, diversity compliance, and overall supplier management.”
Rahul Sethi, President, BusinessIntelligenze
BusinessIntelligenze created BEYOND™, a self-service supplier portal to overcome all supplier registration and diversity compliance issues. It is an out-of-box supplier analytics solution for medium to large enterprises. Based on the Windows Azure platform, the product provides a centralized repository of supplier information, which helps the company’s customers manage their supplier database effectively and save up to 25 percent of their time otherwise spent in managing the same process manually. As a result, BusinessIntelligenze has expanded its customer base, while saving significantly in capital expenses with a reliable, scalable, and highly secure cloud platform.
Country or Region: India
Industry: Information Technology
BusinessIntelligenze, a pure-play SaaS provider, delivers a new generation of business intelligence solutions for information workers and enterprises. The company is based in New Delhi, India.
Business Situation
BusinessIntelligenze wanted to offer its customers an out-of-box analytics solution that could manage the complete lifecycle information of suppliers, starting from their registration, on boarding, and diversity compliances.
BusinessIntelligenze used the Windows Azure cloud computing platform to develop, run, and store data for its product called BEYOND™.
Reduces time spent on supplier information management by 25 percent
Tracks supplier non-conformance
Avoids capital expenditures
BusinessIntelligenze is a dynamic SaaS company that provides analytics solutions and strategic information tools to help customers better manage their supplier database. It engages with the client’s procurement team assisting it in achieving its diversity objectives. Founded in 2010, the company has a presence in India and the United States.
In a sustainable procurement model approach, BusinessIntelligenze empowers decision-makers with critical insights at the right time and place. The company realized that its customers needed a tool that could manage complete lifecycle information about their suppliers along with compliances, and meet diversity objectives. Until now supplier information was received on emails, managed on completely decentralized scenario. Rahul Sethi, President, BusinessIntelligenze says, “We work with several large companies. During our engagements, we realized that there was a genuine need for a system that could completely manage supplier registrations, and also enable customers to meet their diversity objectives. There was no product with this focus in the market.”
This niche requirement motivated the company to create a product that would cater to these needs and at the same time contribute to the process of its client’s diversity objectives in the United States.
The product also addresses all seven levels of the Black Economic Empowerment (BEE) program by the Government of South Africa. It enables customers in South Africa to meet their contribution towards areas such as employment equity, skills development, preferential procurement, management control etc. in a meaningful way.
Determined to overcome the challenges of its customers were facing, BusinessIntelligenze created BEYOND™, a supplier management solution to empower customers to achieve operational excellence whereby they can manage their suppliers more effectively.
While deciding on options to host the product, the company decided that cloud computing was a perfect way to meet all the needs. It chose the Windows Azure platform as its cloud computing and storage environment offered an integrated and complete service set. Windows Azure is a cloud services platform that provides on-demand compute, storage, bandwidth, content delivery, middleware, and marketplace capabilities to build, host, and scale web applications through Microsoft data centers.
The architecture of BEYOND™ takes complete advantage of Windows Azure, which offers a development, service hosting, and service management environment. The data center is located in Seattle, with three backup copies of data stored in data centers across the world.
The company uses SQL Azure for its relational database needs. SQL Azure is a cloud-based relational and self-managed database service built on Microsoft SQL Server technologies.
BEYOND™ offers a platform where prospective suppliers can register and manage their corporate profile along with their products and services. This supplier self-service portal provides a centralized repository for all suppliers and takes care of supplier management and tracking.
BusinessIntelligenze takes advantage of the pay-as-you-go- model that Windows Azure offers, ensuring that it only pays for the services that it uses. This gives the company an affordable, right-size infrastructure―an infrastructure that is neither under-utilized nor over-taxed.
“With Windows Azure, we know that we have a scalable, secure, highly reliable, and highly available environment,” says Rahul. “With less administrative overheads, we have more time to focus on our core competencies.”
The company began working on BEYOND™ in April 2009 and launched it in June 2010. Till date, 20 of its customers are using the product and the prediction is that the number will keep on increasing.
Almost immediately after the launch of BEYOND™ on the Windows Azure platform BusinessIntelligenze began experiencing dramatic benefits. It was able to improve supplier management processes, lower costs, and mitigate risk by delivering supplier master data across the entire company from a central location.
BEYOND™ is an easy-to-use, supplier self- service portal that customers can use to improve their supplier management processes. Rahul explains, “With all supplier information available in a central location, our customers now spend less time discovering supplier information. Tasks such as a 360-degree view of supplier information, non-conformances, updated product or service category management, supplier ratings, eradicating duplicate and repetitive processes, and preferred supplier tracking, which earlier used to take hundreds of hours a month by procurement specialists can now be done in a matter of a few minutes by a simple search on BEYOND™. We have helped our customers reduce 25 percent or more of their time spent on supplier registration and selection processes, diversity compliance, and overall supplier management.”
BEYOND™ offers increased compliance with established processes and procedures. Earlier, supplier information such as contacts, financial information, certifications, IT and logistics, past audit findings, and scorecards would be scattered across multiple systems and was frequently inaccurate. Further, it was hard to keep this information updated. A key benefit of the Windows Azure platform is that Microsoft performs a large share of the routine IT infrastructure tasks required in a hosting environment. As a result,BusinessIntelligenze can increase its focus on its core competencies.
“You no longer have to worry about supplier details like company information, primary contact information, secondary contact information, relationship with licensee, and minority status,” says Rahul. “All information is automatically and constantly updated, reducing high touch, high follow-ups, or miscommunication in our supplier selection process.”
With Windows Azure, BusinessIntelligenze was able to expand its customer base while avoiding capital expenditures. A venture that normally would have cost enormously in infrastructure expenses yielded no capital expenditures because the company was able to take advantage of Microsoft data centers. “We significantly lowered our expenses, which gave us more flexibility in our business model,” says Rahul.
Windows Azure Platform
The Windows Azure platform provides an excellent foundation for expanding online product and service offerings. The main components include:
Windows Azure. Windows Azure is the development, service hosting, and service management environment for the Windows Azure platform. Windows Azure provides developers with on-demand compute and storage to host, scale, and manage Web applications on the Internet through Microsoft data centers. Microsoft SQL Azure. Microsoft SQL Azure offers the first cloud-based relational and self-managed database service built on Microsoft SQL Server 2008 technologies.
Windows Azure platform AppFabric. With Windows Azure platform AppFabric, developers can build and manage applications more easily both on-premises and in the cloud.
AppFabric Service Bus connects services and applications across network boundaries to help developers build distributed applications.
AppFabric Access Control provides federated, claims-based access control for REST Web services.
Microsoft Codename “Dallas.” Developers and information workers can use the new service code-named Dallas to easily discover, purchase, and manage premium data subscriptions in the Windows Azure platform.
To learn more about the Windows Azure platform, visit: www.windowsazure.com
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Score statistics
Vytautas barely escape upset in Kedainiai
09.02.2016, BBL Media Service
KEDAINIAI – The league-best record in the Triobet BBL Regular Season does not guarantee automatic success in the Play-Offs, as Vytautas wasted a double-digit lead and almost fell short against Nevezis, winning the first leg of the Eight-Final only 83-78 on the road in Kedainiai on Tuesday evening.
The start of the game seemed to suggest that coach Virginijus Seskus and his team were in store for a relatively easy game. Having built a 27-17 advantage after the first quarter of play, the guests finished the half up by 12 points at 54-52.
Having momentarily increased the lead to 18 points after the interval, Vytautas players let their guard down, allowing Nevezis to mount a big comeback. The hosts trimmed the deficit to 68-61 by the end of the third quarter and continued to chip away in the final period.
An 11-2 run by Kedainiai allowed them to go up 72-70 with seven minutes remaining, but Vytautas quickly regained the upper hand, winning the game with a not-at-all convincing five-point margin.
Laimonas Kisielius was very close to a triple-double with 22 points, nine rebounds and nine assists for Vytautas, while Vytautas Sulskis chipped in with 21 points, five boards and four assists. The players also added four steals each.
Meanwhile, Darius Gvezdauskas dropped 17 points to lead the hosts in scoring, while big man Mindaugas Kupsas finished the game with 16 points. No one else reached double figures for Nevezis.
Finals MVP Manigat: "You're only as good as your fans"
Pieno Zvaigzdes crowned as BBL champions
Tartu cause upset in Siauliai to claim Bronze
Birutis rakes in March MVP honors
Pieno Zvaigzdes take huge step towards gold
Siauliai bring home two-point lead for Bronze
First-leg win brings Jurmala to the Finals
Pieno Zvaigzdes earn spot in the finals
Jurmala excel in first leg of the Semifinals
Pieno Zvaigzdes upset Siauliai crowd
2021 © BBL.NET. All rights reserved
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YungShin Group
Comfort Vision
About Carlsbad Tech®
Based in San Diego County, California, Carlsbad Tech began life in 1990 as YSP Laboratories, Inc., a subsidiary of Taiwan’s leading YungShin Pharmaceutical Co. Two years later, it was renamed Carlsbad Technology, Inc. to reflect its deep sense of identification with the local community.
Beginning as a contract manufacturer of generic pharmaceuticals, Carlsbad Tech has become an industry leader in manufacturing and distribution of generics, supplements, and medical devices. Our facilities were granted cGMP validation in 1998, and the company’s first ANDA was approved in 1998 for Diclofenac Sodium Delayed Release Tablets.
In 2008, the launch of Cefaclor under the Carlsbad label represented a major shift and the first step in building a trusted national generics brand.
An important milestone by Carlsbad Tech was hit in 2016, with the launch of optical brand, Comfort Vision®. The first offering ProOcular® contact lenses in the USA, successfully bringing to market a highly popular product that has sold over 1 billion units in Asia. As growth continues, Carlsbad Tech strives to become a global health bridge, bringing a world of innovative health products to the markets that need them.
“Bridging Asia and the Americas, we provide our partners with vision and expertise as a manufacturer, distributor, and advisor”
CarlsbadTech.com
YungShinGroup.com
Carlsbad Tech® is a pioneer in global health. Based in San Diego, California, we bring innovative healthcare products to your family.
Bridging Asia and the Americas, we provide our partners with vision and expertise as a manufacturer, distributor, and advisor.
Now Hiring: Regulatory Affairs Assistant (Entry Level)
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Now Hiring: Regulatory Affairs Specialist
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Children's story George The Giant by Pat Boulton - Children's Stories Net
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George The Giant
Reader Star Rating:
Pat Boulton
Childrens-Stories.net Terms of use: you may view online and freely print a single paper copy of the entire story page for your own personal domestic private use, individual qualified Teachers may also freely print additional paper copies for teaching purposes within their own educational establishment. Any other use is strictly prohibited without prior written consent by letter from us, please see the contact us button above.
"I AM GOING TO BE THE TALLEST, AND THE STRONGEST GIANT IN THE WORLD.
ALL THE PEOPLE WILL LOVE ME AND RESPECT ME."
His giant mother had named him Gerry but he changed it to George, because he thought it sounded more grown-up.
Although, even though he was not yet fully-grown, he stood as high as the houses in the village.
He would stand and look over the houses at the grown-ups and children who lived there and often he would make them jump when he roared down to speak to them.
"PLEASE DON'T BE FRIGHTENED OF ME. I WILL NOT HARM YOU. GEORGE THE GIANT IS HERE TO PROTECT YOU ALL AND TO SEE NO ONE HARMS YOU."
There was one little boy in the village, who certainly wasn't scared of George the Giant, his name was George as well.
Many times the little boy had tried to speak to George the giant, but he was too high in the air to hear his voice.
One day little George had an idea.
As he was good at climbing trees, he was sure he could climb up the giant so that he could speak to him and tell him that he wanted to be his friend.
Very early the next morning, before anyone was awake, he left his house.
Looking up to the sky he could see that George the giant was still standing near the house on the corner.
He wasn't moving, so little George guessed he was still asleep.
"Yak!" cried little George. "Your feet are all green and slippery and your toenails need cutting, they are as sharp as knives." Little George held his nose. "AND, goodness me, I think your feet need washing."
Little George was not put off.
He started to grip on to one of the Giants toes to help him climb.
The toes were as big as a thick branch on a tree.
He thought he might be able to crawl along his foot to reach his ankle, where he might find something he could grip again.
"Ouch! What was that?'
George felt the giant's foot move, it felt like a boat rocking in rough seas.
He had to grip harder on the toe, so he didn't fall off.
"SOMETHING IS CRAWLING ON MY TOES." Bellowed the Giant.
"I CAN'T SEE MY TOES FROM UP HERE."
The Giant wasn't very happy.
"WHATEVER THAT IS CRAWLING ON MY TOES, GET OFF RIGHT NOW."
George the giant shook his toes once again, and little George wondered how long he could hold on.
'I'll hold on until the giant has stopped bellowing,' George thought to himself. 'When all is still and quiet again, I will make a move for his ankle.'
'I wish there was another way I could make the giant hear what I am saying, but it looks as though I will have to carry on climbing up his green slimy body to reach his ear.' Little George puffed.
At last, after much slipping and sliding, Little George made a grab for the big bone on the giant's ankle.
It was a huge bone, and it was difficult for little George to get a grip.
George the giant started to bellow again:
"GET OFF MY ANKLE YOU HORRIBLE CREEPY CRAWLY. IF YOU DON'T MOVE STRAIGHT AWAY, I WILL BEND DOWN AND GRAB YOU. THEN YOU WILL BE CRUSHED IN MY HAND."
By this time the giant had bellowed and roared so much that he'd woken all the people in the village. They stood in the road near the corner house where the giant was standing. The houses shook each time he roared and some of the windows had broken.
Little George had his back resting on the giants foot, whilst he tried to think of another way of making the giant hear him.
He could see all the people in the road looking up at the giant.
All of a sudden, WHOOSH!
The giants massive hand swept little George up in the air.
He was scared, and he was sure the giant would crush him in his hand.
Once again, the giant bellowed.
"NOW I WILL BE ABLE TO SEE WHAT SORT OF CREEPY CRAWLY, HAS THE NERVE TO CRAWL ALL OVER MY TOES AND FOOT."
Little George, jumped around in the palm of the giants huge hand and shouted as loud as he could.
"Please Mr Giant, I only wanted to talk to you and let you know that I want to be your friend."
However much little George shouted the giant didn't hear him.
Little George was rocking about in the palm of the giants massive hand for ages. Eventually he could see the light showing through the giant's fingers.
Little George could just about see the outline of the giants face. 'My goodness', he thought, 'You are so ugly, and your huge yellow teeth are smelly and like huge yellow icicles, hanging from a hole in a mountain.'
As the giant slowly opened his hand, he stared down at little George.
"YOU'RE NOT A CREEPY CRAWLY. CREEPY CRAWLIES DON'T WEAR CLOTHES. I KNOW WHO YOU ARE;" the giant roared "YOU'RE ONE OF THE CHILDREN FROM THE VILLAGE."
"Yes, that's right." Little George managed to climb along the side of the giants whiskery face, to reach his ear.
"Can you hear what I'm saying now, Mr Giant?"
"YES, I CAN HEAR YOU NOW."
"How do you do," Little George, reached out for the giant's long finger and shook it. "My name is George, and I was climbing up so that I could talk to you and tell you that I wanted to be your friend."
"WELL, WELL, WELL" The giant started to roar with laughter. "MY NAME IS GEORGE TOO, OF COURSE YOU CAN BE MY FRIEND."
The giant promised little George that he would watch over the village, but stand away from the houses so as not to shake them when he spoke. He also promised that when he was really grown up, and became the king of the giants, he would see that no harm came to the people who lived in the village.
"AS FOR YOU, LITTLE GEORGE, ANY TIME YOU WANT TO TALK TO ME, OR NEED MY HELP, JUST TAP THREE TIMES ON MY LITTLE TOE, AND I WILL LIFT YOU UP TO MY EAR."
"Thank you George the giant, you are the kindest giant in the world."
As the giant put little George safely on the ground he ran off to tell his mum and dad about his adventure.
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Southland Tales - Arrow
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From Bedrooms to Billions: The Amiga Years
Director: Anthony Caulfield, Nicola Caulfield
Writers: Anthony Caulfield, Nicola Caulfield
Starring: Shahid Ahmed, Rich Alpin, Brian Bagnall
BBFC Certificate: E
A year or so ago I reviewed a crowd-funded documentary about the birth and growth of the British video games industry, called From Bedrooms to Billions. I was impressed by the film, which was much more than the fluffy nostalgia piece I expected. So when I heard they were releasing a follow up, focussing on the Commodore Amiga, I was eager to get a copy to review. It wasn’t only the quality of the previous film that attracted me to From Bedrooms to Billions: The Amiga Years though. Like a lot of Brits around my age, my introduction to video games didn’t come in the form of the Nintendo or Sega consoles. These tended to come out a lot later in the UK and weren’t the be all and end all that they were in the US. We had an alternative, and that was the Commodore Amiga. I had an Acorn Electron computer first, but the games on this were very basic and I was very young. Our family replacement to this was the Amiga 500 though and it opened the floodgates to video gaming for me. The graphics were great, many of the games fantastic and it was modelled on a PC in design, so was more flexible than a pure games console in terms of offering word processor or paint programs etc. I loved it and the computer/console has long held a special place in my heart.
The documentary opens by describing the early history of home video game consoles, particularly those offered by Atari and Commodore (with some mention of what Apple were doing on the home computing front). Some designers working with these companies at the time grew unhappy with the way things were moving and decided to branch out on their own to form a new company, called Amiga. They had plans for a console/computer that would blow their competitors out of the water in terms of power and capabilities, yet cost a fraction of the price of the expensive PC’s available at the time. They struggled for a time, coming up with brilliant ideas, but not having the backing to pull it off. After a successful demonstration at an important trade show however, Amiga got thrown in the middle of a bidding war between Atari and Commodore. This war was made even more messy by the fact that Atari had been taken over by Jack Tramiel, formerly one of the bosses at Commodore.
After the dust had settled and Commodore became the company to release the first Amiga, the computer was launched. The initial system, the Amiga 1000, came out in 1985 (though not widely released until 1986) and struggled to find a market. 1987’s cheaper model, the Amiga 500, was a huge success though (in Europe at least). The graphics and sound capabilities were groundbreaking, allowing for near arcade-quality games at a fraction of the price. The documentary goes on to praise the importance of some of the machine’s innovations and how they helped shape today’s video games industry.
Like the previous film, The Amiga Years avoids empty praise of the long list of games released by the computer. In fact, it’s almost an hour and a half into the film before we see any extensive footage of the Amiga 500 games roster. Instead, writer/directors Anthony and Nicola Caulfield take their time to tell the full story of how the Amiga came to be. Given the smaller focus, the film seems even more in-depth than its predecessor in fact. As such, I found some of the segments examining the hardware design a little much to get my head around at times, but the key message was always clear – a groundbreaking piece of kit was being put together. There’s plenty of humour present too, to keep things from getting dry, such as how two developers stayed up all night to improve the system’s demo during a trade show, working so hard they fell asleep in the booth, only to be found by their colleagues the next morning.
The film does spend much of the last hour praising the system, which gets a little repetitive perhaps. The later years of Amiga, when a number of models flopped and the computer was eventually discontinued, are surprisingly passed over (although this is looked at in the special features). The makers instead decide to end on a high, which does make the film feel a little more nostalgic than before. All the praise seems justified and at times quite fascinating though. The Caulfields don’t merely list off great games, in fact little time is given to individual titles. Instead they look at what the Amiga inspired, like the success of the demo scene, the revolutions in music production aided by the computer and the improved possibilities in pixel art. So, although it’s all very pro-Amiga, the film still feels like it has worth and substance beyond the kind of lazy rose-tinted ‘documentaries’ you often see on TV when a gap needs filling in the schedule.
Like the previous film, The Amiga Years comes in at a hefty two and a half hours. Also like before though, the film never feels overlong or dull. It’s pretty tight and fairly fast paced, although not quite as before, largely because of the narrower focus the second time around. Once again the makers have got their hands on a wealth of archive material to keep things visually interesting too. We’re not stuck watching talking heads all the time and there’s more to look at than just game footage. We get to see the original plans for the system, old news reports and hundreds of photos taken by and of those involved in creating the Amiga.
It maybe falls slightly short of its predecessor, largely because the praise of the Amiga can feel a bit much towards the end. The content wasn’t quite as revelatory as before either – I didn’t find the story quite as surprising, but I still found it fascinating to watch. I’m not sure it would be as interesting to those unfamiliar with the Amiga and certainly not to non-gamers, but if, like me, you grew up playing the likes of The Secret of Monkey Island, Syndicate, Lemmings and Populous, you owe it to yourself to check out this rich and enjoyable documentary.
From Bedrooms to Billions: The Amiga Years is out in September on DVD and Blu-Ray exclusively at Funstock (both with a digital download included free). You can get Standard Editions in both formats or Special Editions.
There are tonnes of special features included. Here’s the list:
Disc 1 (both versions):
– Directors Commentary
– The US Video Games Crash
– What Commodore Should Have Done
– What Happened to Larry
– Launching the Amiga CD32
– Creating Turrican II Sound Track with Chris Huelsbeck
– Creating Syndicate with Sean Cooper
– Creating Lemmings – With Dave Jones and Mike Dailly
– Creating The Secret of Monkey Island with Ron Gilbert
– The Amiga Christmas Batman Pack!
Disc 2 (Special Editions only):
– Creating ‘Another World’ – Eric Chahi
– Founding Electronic Arts – Trip Hawkins
– Creating ‘Flashback’ – Paul Cuisset
– Atari Vs Activision – Larry Kaplan
– Ron Gilbert – Insights into game design and working at Lucasfilm
– The Business of Amiga – The continuing story of Commodore and the Amiga line.
– Creating the ‘Desert Dream’ demo – Anders Hansen
– Chris Huelsbeck – Creating music on the Amiga
– Dan Malone – Designing ‘Speedball II’
– Stoo Cambridge – Designing ‘Cannon Fodder’
– Demo Scene Sequence Expanded – Various contributors.
– Founding DICE
– ‘First Samurai’ – Design insights with Mev Dinc
– Team 17 – (Alien Breed, Super Frog), Andreas Tadic, Rico Holmes, Martyn Brown & Allister Brimble.
– Magnetic Fields – Shaun Southern & Andrew Morris insights, including Lotus and Super Cars series.
Deleted scenes from feature fiction films are generally of little interest to me. They tend to have been deleted for a reason. With a documentary like this however, they instead feel like a genuine expansion of the film. I can understand why the Caulfields didn’t keep them in and have a documentary that was 6 hours long. That would be madness. Having them available to watch as supplementary features though means you can really dig into the entire story behind the Amiga. As well as adding to the history behind the computer, the added features allow in depth looks into some of the system’s classic games too, giving you your nostalgia fix you may have felt you didn’t get in the documentary itself. The clips featured on the standard edition are only short, so you might want to look to the special edition for a more substantial package, but the standard edition clips are great nonetheless.
Like with the first film, Gracious Films have really outdone themselves with this package. There’s pretty much all you’ll ever need or want to know about the Amiga. The makers must be applauded for putting this much effort in. I wish more distributors would.
Reader Rating: (0 Votes)
AmigaAnthony CaulfieldCommodoreDocumentaryhistoryNicola Caulfieldvideo games
Editor of films and videos as well as of this site. On top of his passion for film, he also has a great love for music and his family.
Troll: The Complete Collection
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David Brook 100 Non-Disney, Non-Pixar, Non-Ghibli Animated Features – Part 4 (2006-2012)
Morgan 100 Non-Disney, Non-Pixar, Non-Ghibli Animated Features – Part 4 (2006-2012)
David Brook The Ninja Trilogy
LES BICHES
Women play mind games
Simple plot but music helps
Make gripping and tense
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Blueprint:Review
New post: Southland Tales – Arrow https://t.co/ZcweD6zIAm 07:12:02 PM January 15, 2021 from dlvr.it ReplyRetweetFavorite
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Blueprint: Review – reviews of the best in independent, classic and world cinema, as well as video games and soundtracks.
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Hunter's Sunset
The Hunter's Sunset drink recipe is made from Jagermeister, Black Beard spiced rum and pineapple juice, and served over ice in a highball glass.
I Dream of Nelson
The I Dream of Nelson drink recipe is a tribute to Lord Admiral Nelson, the naval hero of the United Kingdom, freeing the high seas for British expansion. An orange colored drink made from Admiral Nelson's spiced rum and orange soda, and served over ice in a highball glass.
Love Potion Number 9
When your lover is not in the mood, try casting a spell over them with this love potion. The Love Potion Number 9 drink recipe is an orange colored cocktail made from Cruzan 9 Spiced rum, coconut schnapps, pineapple juice and cranberry juice, and served over ice in a highball glass.
Show your true naughty side this Valentine's Day, surprise your lover once again. The Naughty by Nature cocktail recipe is made from Xante cognac, Captain Morgan's spiced rum, pear puree, apple juice, lemon juice and vanilla syrup, and served shaken in a chilled cocktail glass.
Nelson's Lady
The Nelson's Lady drink recipe is a brown colored cocktail made from Admiral Nelson's cherry spiced rum and cherry cola, and served over ice in a highball glass.
Nelson's Touch
The Nelson's Touch drink recipe is a relaxing orange colored tropical cocktail made from Admiral Nelson's spiced rum, peach brandy, lime juice and pineapple juice, and served over ice in a rocks glass.
Old Sea Hag
T' Old Sea Hag be more than a pirate's old lady, it's also a pirate's favorite drink. Made from Burnett's spiced rum, orange juice and cola, and served over ice in a rocks glass.
Pineapple Smash
The Pineapple Smash is a tasty tropical drink made from spiced rum, light rum, lime juice, pineapple and club soda, and served over ice in any glass.
Pom-Acai Punch
The Pom-Acai Punch is a red colored drink recipe made from VeeV acai spirit, spiced rum, pomegranate juice, simple syrup, bitters, lemon and cherry, and served over ice in a rocks glass.
Ragin Rootbeer
The Ragin Rootbeer is a brown drink made from root beer schnapps, vanilla schnapps, spiced rum and cola, and served over ice in a highball glass.
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Tag Archives: Voice of Nigeria
NBC prepares for Africast 2018
October 23, 2018 NewsAfricast, BT4 Media Group, Channels TV, Maimuna Jimada, NBC, Nigeria’s National Broadcasting Commission, Viewer Communications, Voice of Nigeria, Zoommobile Telecomms, Zuma RadioGary
International trade event Africast is set to hold its 12th biennial conference of Africa broadcasters and exhibitors in Abuja this month.
Maimuna Jimada
The international trade conference and exhibition runs between October 23 and 25 in the Ladi Kwali Hall at the Sheraton Hotel and Towers.
The event is aimed at providing a platform for manufacturers of broadcasting equipment with the opportunity to showcase their products and services. It is also a way for broadcast professionals to be informed about the latest trends in broadcasting in the international market.
Launched in 1996 by Nigeria’s National Broadcasting Commission (NBC), Africast is aimed at professionals, academics and policy makers. Focusing on The Dynamics of Broadcasting in the Digital Era, the 2018 event will also discuss how best digital switch-over can be delivered across Africa.
Nigerian companies at the event will include Channels TV, Zoommobile Telecomms, Viewer Communications, Zuma Radio, BT4 Media Group and Voice of Nigeria.
Maimuna Jimada, coordinator of the event and senior officer at NBC, said: “Africast has fast become the biggest and most comprehensive broadcast and media industry event on the continent and offers a unique platform for knowledge sharing and networking for digital broadcasters, broadcast regulators, communicators, broadcast content providers, equipment manufacturers and vendors, policy makers and other stakeholders from all over Nigeria, Africa and the rest of the world.
“At Africast 2018, our guests will experience three days of varied participation of up to 1000-plus delegates from across the globe. It is a platform for world-class exposition, showcasing the very best in creativity, innovation and technological advancements in Africa’s broadcast and media ecosystem.”
There will be keynote presentations, discussions, expert roundtables, networking opportunities, masterclasses, a content market with exclusive screenings of African content and an expo showcasing the latest broadcast and media technologies. Exhibitors from countries including China and Japan will attend.
tagged in: Africast, BT4 Media Group, Channels TV, Maimuna Jimada, NBC, Nigeria’s National Broadcasting Commission, Viewer Communications, Voice of Nigeria, Zoommobile Telecomms, Zuma Radio
TV show Adam & Eve seeks contestants
Casting call for SA’s Ugly Betty
Showmax, Canal+ coproduce crime drama
Yvonne Orji developing Disney+ show
African OTT revenues tipped to top $1.7bn
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123-Reg drowns in ongoing DDoS tsunami
DDoS DDoS Attacks DDoS Defense
Data centre target of attack of 30+ Gbps
Beleaguered web host 123-Reg has suffered a “huge scale” distributed denial of service (DDoS) attack to its data centre – knocking the Brit outfit’s website offline and a number of users’ services.
The attack began this morning and is still ongoing but no performance-related issues have been reported since the traffic was rerouted.
The Register understands that the outfit experienced a DDoS attack of 30-plus Gbps to its data centre, with its protection systems kicking in within seconds of the attack being detected.
Consequently the business redirected traffic through its secondary “DDoS protection platform” in Germany, which doubled its capacity.
No servers were offline, although customers experienced intermittent connection issues such as our website, control panel, email or websites.
A 123-Reg spokeswoman said: “At about 10:10am we received a huge scale DDoS attack to our data centre.
“Our protection systems kicked in immediately and the attack was contained by 10:40am. We apologise for any intermittent connection issues to our services that some of our customers may have experienced during this time.”
Back in November, internet provider Eclipse was hit by a DDoS attack. ®
Source: http://www.theregister.co.uk/2016/08/02/123reg_suffers/
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Conformational dynamics of the FMN-binding reductase domain of monooxygenase P450BM-3
Verma, Rajni, Schwaneberg, Ulrich and Roccatano, Danilo (2013) Conformational dynamics of the FMN-binding reductase domain of monooxygenase P450BM-3. Journal of Chemical Theory and Computation, 9 (1). pp. 96-105. ISSN 1549-9618
n the cytochrome P450BM-3, the flavin mononucleotide (FMN) binding domain is an intermediate electron donor between the flavin adenine dinucleotide (FAD) binding domain and the HEME domain. Experimental evidence has shown that different redox states of FMN cofactor were found to induce conformational changes in the FMN domain. Herein, molecular dynamics (MD) simulation is used to gain insight into the latter phenomenon at the atomistic level. We have studied the effect of FMN cofactor and its redox states (oxidized and reduced) on the structure and dynamics of the FMN domain. The results of our study show significant differences in the atomic fluctuation amplitude of the FMN domain in both holo- and apoprotein. The change in the protonation state of FMN cofactor mostly affects its binding in holo-protein. In particular, the loops involved in the binding of the isoalloxazine ring (Lβ4) and ribityl side chain (Lβ1) adopt different conformations in both reduced and oxidized states. In addition, the reduced FMN cofactor mainly induces a conformational change in Trp574 residue (Lβ4) that is essential for controlling electron transfer (ET) within P450BM-3 domains. The structure of the apoprotein in solution remains mostly unchanged with respect to the crystal structure of the holo-protein. However, FMN binding loops were more flexible in apoprotein that might favor the rebinding of FMN cofactor. In the holo-protein simulation, the largest conformational changes in FMN cofactor are caused by the ribityl side chain. The isoalloxazine ring of FMN cofactor remains almost planar (∼177°) in the oxidized state and bends along the N5–N10 axis at an angle of ∼160° in the reduced state. The collective modes of the isoalloxazine ring were identical in both protonation states of FMN cofactor except the first eigenvector. In the reduced state, the isoalloxazine ring attains the butterfly motion as a dominant collective motion in the first eigenvector due to the bending along the N5–N10 axis.
Molecular dynamics simulation, Protein complex
F Physical Sciences > F165 Biomolecular Chemistry
C Biological Sciences > C790 Molecular Biology, Biophysics and Biochemistry not elsewhere classified
College of Science > School of Mathematics and Physics
http://dx.doi.org/10.1021/ct300723x
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The Traveler (Fourth Realm Trilogy, Book 1)
Of particular note are the title story from the first collection plus “A Harvest of Wolves” (1983), “Anusazi’s Daughter” (1984) and the amusing “Orc’s Drift” (1997). A newspaper account describing a similar phenomenon, though on a larger scale, in Brazil, discounted as mass hysteria, gives him fresh hope, and he theorizes that some new form of life has appeared in the world, invisible and determined to replace us. It could be argued, for example, that DR. The Beast House (1986) is basically a reprise of the first story with a new cast of characters.
Publisher: Random House Audio; Abridged edition (July 10, 2007)
Single And Single
Sesame and lilies: and, The political economy of art
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Ya-Yas in Bloom (The Ya-Ya Series Book 3)
To Demons Bound
Season in Purgatory
Fancy Strut
The Alley of Wishes
Secrets Of A Whitby Girl
Walnut Shell Days
The 158-Pound Marriage
Captain Vorpatril's Alliance (Vorkosigan Saga)
Turn Left at the Daffodils
A Woman Possessed
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A Sudden Light: A Novel
BEAR MY KISS: Turi's Tale (Edanholme Book 4)
Sun in Splendour
Legacy - A Sweeping Epic of America's Earliest Days!
SANDRA BELLONI
The Dominant Strain (1903)
Twelve Days of Christmas: A Christmas Novel
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HomeShows NewThe Butterfly Effect
“Can a butterfly flapping it’s wings in the Amazon cause a tornado in Texas?”
First performed at the Edinburgh Fringe Festival in 2006. My most successful show to date. We recorded a BBC Radio Comedy pilot based on this show at the Oxford Road studios in Manchester on 16th April 2007. It starred Jo Enright, James Quinn, myself and Dave Turquoise. It was produced by Paul Hardy and Sally Harrison.
Thrills, Spills and a masturbating monkey!
The theory of the Butterfly Effect states how seemingly trivial events can have huge consequences and explains how one ninny’s inability to text the word spaghetti can lead to a 15 mile tailback on the motorway.
The Butterfly Effect is a small part of Chaos theory, one of the most recent and radical sciences which explains how the universe is as unpredictable as a coach load of drunken granddads.
Essentially a show about chain reactions that moves from stand up comedy to stand up fiction with a selection of interconnecting short stories presented and performed by their author.
Never before has there been a show at the fringe festival about chaos yet this year two shows arrive, both from comedians called Ian (the other show by Ian Stone) and both sat next to each other in the fringe programme. How perfectly chaotic!
Kate Copstick – The Scotsman
“Ian Fox is a mild-mannered, long-haired, really rather charming young man the preparation of whose Edinburgh show has been made extremely difficult by circumstances. And George Clooney. But you wouldn’t know, because his hour is a fascinating, funny and engaging one. And yes, it does explore, at some length, The Butterfly Effect – Lorenz’s idea of cause and effect which eventually led to Chaos Theory.
We get there gently, through chat about fetishes and phobias, Dairylea slices, free porn, a great little piece about McDonald’s and some glorious fantasising about producing a film through the internet. Fox’s observations about how great discoveries have all been made by people doing nothing is smartly funny.
The show does slightly die away over the course of the overlong story that he ends with. However, he does, endearingly, warn us that this part of the show could go wrong, and that if it does there is nothing he can do about it because it is a scripted story. Fox’s show is part of the Free Fringe, which is a very happy hunting ground for comedy this year”. (3/5)
Julia Chamberlain – Chortle
“This is an excellent debut show from Manchester based comic Ian Fox. Tucked away at the Meadows Bar; it would not be out of place at one of the swankier venues. His quick delivery engages your attention from the off and he obviously has a great ear for the rhythm of natural speech. There’s something very satisfying about having ASBO, EBay and ER all in the same sentence.
As with many catchwords, we all think we know what the Butterfly Effect is, but his various analogies for it help fix if firmly in the mind. (It demonstrates a chain reaction which leads from a trivial incident to a massive result, by tiny knock-on effects) and the story of its discovery by Edward Lorenz is fascinating and relevant and,in Fox’s hands, very amusing.
The facts and the science in the first section are interspersed with some excellent lines and ideas, seamlessly blending natural club comedy with higher intent there’s plenty to think about here.
The middle section of the show places psychic prediction under scrutiny. (Yes, it is all bollocks). Illustrated with fascinating examples, one cites an uncanny coincidence in a Daily Telegraph crossword and a sinking ship. He touches on phobias and fetishes and leads us into the final section of the show. He warns us that this is essentially a lengthy piece of storytelling from which he can’t pull out if we’re not going with it. There’s no danger of that as he’s already successfully hooked your attention with his intelligent work and relaxed wit. The pace does slow, but it’s not a problem.
This is good use of an afternoon slot, performed without gimmicky technology, if only because Ian Fox has had his own run in with the butterfly effect. The show succeeds admirably in being smart, funny and entertaining without a whiff of smugness.”
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Roundtable with Mr. Ivan Krastev, Chairman of the Centre for Liberal Strategies
Home > Events > Roundtable with Mr. Ivan Krastev, Chairman of the Centre for Liberal Strategies
Roundtable with
Mr. Ivan Krastev,
Chairman of the Centre for Liberal Strategies
On January 9, 2018, GRF organized a roundtable meeting with Mr. Ivan Krastev, Chairman of the Sofia-based Centre for Liberal Strategies, as part of its Task Force on the Future of EU-Turkey Relations.
Mr. Krastev, who is a founding board member of the European Council on Foreign Relations and a member of the board of trustees of the International Crisis Group, shared his assessments on the multiple crises that threatened the unity of the European Union. In his remarks, Mr. Krastev stressed that the most important challenge was posed by the refugee crisis – which he identified as “Europe’s 9/11” in his latest book, “After Europe” – and discussed its implications for the EU’s future.
Mr. Krastev discussed that Brexit and the election of Donald Trump as US President were elements of shock for Brussels. The former brought disintegration into the EU’s agenda whereas the latter professed that Europe is no longer a priority for Washington.
In his speech, Mr. Krastev suggested that the EU endured three major crises over the past few years: the Ukrainian crisis, the Eurozone crisis and the refugee crisis. While arguing that the interplay of these issues destabilized the EU, he stressed that the refugee crisis was unique in changing European attitudes towards “open borders” and hence, drastically changing EU politics.
Following his speech, Mr. Krastev answered the participants’ questions.
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Pataki, Former Governor of New YorkOnline Meeting with Prof. Serhat Ünal, Hacettepe University Head of Department for Infectious Diseases and Clinical Microbiology & Member of the Coronavirus Scientific BoardOnline Meeting with Rear Admiral (R) Cem Gürdeniz, Founding Director of the Koç University Maritime ForumOnline Meeting with Ambassador Christian Berger Head of the European Union (EU) Turkey DelegationOnline Meeting with Dr. Daniel Gros, Director of the Centre for European Policy Studies (CEPS)Online Meeting with Şant Manukyan, International Capital Markets Manager at İş YatırımOnline Meeting with Howard Chase, Senior Director of Government Affairs for Dow Europe, Middle East, Africa and IndiaOnline Meeting with Dr. Constanze Stelzenmüller, Senior Fellow at Brookings InstituteOnline Meeting with Prof. Paul Farmer, Kolokotrones University Professor and Chair of the Department of Global Health and Social Medicine at Harvard Medical SchoolOnline Meeting with Auguste Tano Kouamé, World Bank Country Director for TurkeyOnline Meeting with Mr. Can Kendi, Country Director of McKinsey & Company Turkey and GRF Corporate MemberOnline Meeting with Dr. Fatih Birol, Executive Director of the International Energy Agency (IEA)Online Meeting with Mr. Gideon Rachman, Chief Foreign Affairs Columnist for Financial TimesOnline Meeting with Mr. George A. 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Grupsex hikayeleri ve daha fazlası burada
Yayım tarihi 5 Ocak 2021 (admin)
Redwood Manor: Autumn Pt. 01
****Foreword****
Greetings! This is my first formal entry into the world of Erotic Fiction, after years of tooling around and not actually writing anything substantial. This story will pass through periods of heavy adult content as well as sections of pure fiction, as any good Erotic literature cannot succeed without both. If you take the time to sink into the world of the characters, and believe the tensions and dynamics they are under, each orgasm will be that much more powerful 😉 This story is seen through a man’s perspective, but (I hope) there is enough content to satisfy both sexes!
Feel free to share my work, but please, I ask that all content following this paragraph be attributed to myself, the author, and that this note appear at the head of any reposting. I am happy to answer any mail, time allowing – just drop me a line at the link in my author profile.
Constructive criticism is also welcomed, directed at the same email listed above. Happy reading, and more importantly, I wish you the best of fantasies 😉
===+===
REDWOOD MANOR
Autumn: Part 1
Clayton pulled the sleeves of his sweatshirt down from his elbows against the bitter wind that blew across the parking lot. Fall was here, and gusting with a vengeance. Sun and blue skies be damned, it had been a cold day, and with the sunset rapidly approaching the air temperature was diminishing. Seasons always seemed to turn so quickly in the Seattle Area, right at the end of another scorching summer. Clayton’s Explorer chirped behind him as he clicked the lock button on his key remote, shuffling through auburn and hazel leaves to the sidewalk. Brushing a bit of dirty blonde hair out of his face, he hiked down the path and across the courtyards of Redwood Manor that led to his girlfriends apartment.
The apartment complex was unlike many of the others east of the city. It was laid out as a series of small buildings, each containing four units or so. Some of the buildings were smaller, housing only two or three, set out at the edges of the unbounded suburban complex. Trees, mostly Douglas Fir and Cedar, dotted the property. Ironically enough, Clayton mused, there was technically not even a real redwood on the the entire property. Hadn’t stopped the developers from taking artistic license with the name.
The benefit of living so far outside of Seattle Proper could only be truly realized in the cost savings of the place itself – 1300 square feet, 2 bedrooms, 2 bath for less than half of what you would pay for a unit that size just 3 miles out of the downtown core. The attractive price, granite counter tops, and new appliances is what had drawn Emily to utterly abandon the city in favor of Suburbia. Clayton smiled to himself. It had been about 2 years, and his girlfriend was still as impulsive as ever. Her decision to move so far out had been made in less than two days, much to the chagrin of her sister. At first, Kendra had been very upset about moving so far out into ‘the boonies’. So far from her friends and her job, but hey, when your only option for a roommate is your sister (who also is footing the majority of the rent), how much can you actually complain?
Clayton reached into his pocket as he felt his phone buzz. New snap from Emily. Sliding his phone unlocked, Clayton opened up the picture message. A pair of brown eyes ringed by golden blonde hair stared up at him, along with a serious pouty face.
‘Gonna be at the office longer than I thought’, the words on the screen read. As the message timed out, another one appeared in the inbox. Clayton couldn’t help but grin deviously. The pout had been replaced by down-the-blouse view of a pair of wonderfully perky breasts being squeezed together, the top of the photo showing Emily nibbling on her lower lip seductively.
‘Don’t have too much fun without me?’
‘How could I?’ Clayton texted back. ‘Am I even going to be able to get into your apartment? Or am I just supposed to freeze to death out here.’
Buzz buzzzzz.
‘You are such a drama queen. You already know it is unlocked. Gotta go, see you later gator.’ Clayton smirked as he slipped his phone back into his pocket. It was too much fun to tease Emily. She was such a small girl for 24, only standing about 5’5, and no more than 115 pounds. Getting her riled up was all too easy, and when she got truly irritated she would turn into a little ball of fire.
It was hard to believe that her and Kendra were related. While Emily was loud and boisterous, Kendra was quiet and shy. Three years younger, and taller than Emily at 5’8, she weighed only a little more than her older sister. This was due to the fact that she was an insanely picky eater. In the 4 months that Clayton had been visiting Emily and Kendra at their new apartment, Kendra had only ever touched 3 of the meals that Clayton had made, preferring taquitos and ramen noodles. He knew he was no master illegal bahis chef, but still. So picky. Despite all her finicky meal habits, she did have those stark, ice blue eyes, complimenting her fair skin and brunette hair so well…
Clayton shook his head. While he was very much satisfied with his relationship with Emily, he had been abjectly trying to avoid the reality that had been growing in his head for weeks now: Kendra was hot, plain and simple. It had been easy to categorize her as ‘the little sister’, but then her 21st had come and gone. It was OK, she wasn’t even single, but then the boyfriend was no longer in the picture. Over the months, Clayton had walked into the apartment a number of times as she was leaving the shower in a small towel. It was always slightly awkward, mostly a blush from Kendra and a quickly averted gaze on his part, but nevertheless, it had happened. Clayton brushed the train of though from his mind. It was a dangerous one, and he knew it. He pulled out his phone again.
‘What is taking so long at the office today anyways? Has your boss really got you that busy?’ It was hard to peck out the words with his cold thumbs as the sun drew lower in the sky. Emily’s reply was quick.
‘Well, the more you BOTHER ME, the LONGER I’LL BE. I already told Kendra that I would be late tonight. I’m not sure if she knows you are on the way, though. Just knock so you don’t scare her or something.’
‘Shouldn’t you text her and say I am coming by? Wouldn’t that be the better option?’
‘What would be the BEST option, I think, is if you handled it yourself.’
Clayton began typing a reply, but thought better of it. Accounting had left Emily more spent than most these past few weeks, and their relationship was beginning to feel the effects of it. She usually came home too weary for any kind of fooling around, leaving Clayton increasingly sexually frustrated. Emily was always feisty, but when her mood was tainted with stress from work, it really permeated every aspect of her social interaction. She could go from playful to testy extremely fast, and without warning.
Blowing out a puff of steam, Clayton rounded the corner of the building that Emily and Kendra lived in. Downside of super cheap property so far out of the city? No need to build these units in any semblance of a compact arrangement – it was a full 7 minute walk from the parking lot to their unit. The hill they were situated on, out at the north end of the property, made this building one of the three unit configurations: a ground floor, an upper floor, and an adjacent upper floor that rested up the hill. Clayton weaved around the shrubbery and assorted landscaping that littered the edges of the path up the hill, making his way to the northernmost division of the building. Living all the way out on the edge was a hassle for hauling groceries and such, but the lack of other residences in any direction but south meant that the window shades were usually left open.
Which is why, much to Clayton’s confusion, the double french doors to the little patio were completely shaded. That was his way in when Emily wasn’t home, as the front door further down the hill was usually locked. Even more confusing than the shades was the steadily pulsing House Music coming from the living room behind the doors. The immediate neighbors were gone, as evidenced by the darkened windows, and the music was playing quite loud.
Clayton had bought them a decent set of home theater speakers as a housewarming gift (for parties and a quality listening experience, or so it was intended), but they had rarely seen use. Emily was not all that into music in the first place, and Kendra never played anything on them when Emily and Clayton were home. Kendra was very much a fan of electronic music, however, and had eagerly taken any music that Clayton had offered. Clayton nodded his head in time with the beat as he recognized one of his favorite songs bumping on the system.
He walked up to the french doors, and gently tried the knob, but it was locked. Superb. Annoyed at the cold, and deciding to scare Emily’s sister after all, Clayton slipped through the branches of a small tree around to the uphill side of the apartment, by the kitchen window. Clayton cursed under his breath as the kitchen window shade, inexplicably, was also up. ‘They never close this one, what the hell,’ Clayton thought.
The kitchen windows were tall, owing their height to the vaulted ceilings of the room, but the shade did not reach all the way to the ceiling. Determined to scare the crap out of Kendra, Clayton began to climb the hill. It was a little steeper here, where the ground had been scooped away to level the foundation of the uphill apartment, and he clambered up the dew-covered grass. He would be able to see over the top of the shades is he went up the hill a few more feet, and might be able to flail his arms at Kendra if she was in the kitchen. Far enough up illegal bahis siteleri the hill, he turned around, and Clayton’s jaw dropped open.
Clayton could only process bits of information at a time. Kendra was in the kitchen, facing away from him. She was not, however, cooking. She was absolutely naked, glistening with sweat, kneeling on top of the granite island. Her left hand was on top of her head, weaved in her long brunette hair, of which strands were sticking to her face and back with perspiration. Her right hand, nails dug in, was pulling her rounded ass to one side, helping to open herself to a large, veined, green dildo. It was buried in her pussy, and as Kendra ground her lithe body up and down upon it in tempo with the music, Clayton could see it was absolutely shining with her wetness. She was taking it in so deeply a small ring of white girlcum had even formed around the base of the toy, just above the suction cup holding it in place.
The look on her face as she twisted it sideways momentarily, the arch in her eyebrows, and the moans Clayton could see Kendra mouthing silently told him all he needed to know – she was very, very close. And yet, she was moving with deliberately slow, almost torturous motions. Three beats of the music took her up, and on the forth, she plunged back down onto the thick, green cock. She would lose control for a moments at a time, quite literally bouncing up and back down upon it every beat, the veins in her neck showing as she stifled a guttural moan. Her chest was rising and falling rapidly, breath short and gasping.
Clayton’s cock had come alive. Before he even knew what he was doing, the length of it was freed of its strain against his jeans, and he was stroking away with a fever he had not known was inside him. He was as hard as iron, the veins of his manhood stark against the smooth skin of his shaft. His head pulsed, feeling like it would explode, with every heartbeat. A breathless groan slid past his lips as Kendra picked up the pace again. He was close enough to see her pink lips pulling at the thickness of the toy as it slid out of her, close enough to see her ass contract as a wave of pleasure washed over her body. Clayton felt precum leaking out the tip of his throbbing cock, and could feel his own orgasm building rapidly. Faced with such a voyeuristic, erotic sight, his body was in complete sensory overload.
Suddenly, Kendra cried out, loud enough for Clayton to hear through the window and over the music. She dropped down on top of the silicone phallus, put both hands on the counter, and abandoned the tempo of her masturbation entirely. Her hips flew back and forth, grinding on the length of the toy, pressing it ever deeper into her soaking hole. Clayton’s hand flew up and down his shaft, his own aching balls begging for a meaningful release, as he watched her climax begin. It had been so long since he was this aroused, and he knew that he was going to have a very powerful orgasm. About 10 seconds passed, and then all of the sudden, it happened. Kendra’s legs snapped together, her body quivering as her own orgasm peaked. A small, but fresh wave of white girlcum squeezed out of her pussy around the toy as her body quaked with contractions.
Clayton lost it completely. With a grunt, his cock erupted into a fountain of cum. Two, three, four times he roped out across the grass, thick white beads covering a distance of three feet. His balls squeezed, dribbling more out of his spent head, and his vision blurred for a brief second. Completely emptied in a single load, Clayton sat back up on the grass. He had not recalled sitting down after his tremendous orgasm. As he wiped his hand on the back of his jeans, Kendra reached a point of recovery. She popped up off the dildo and slid it out. It was more thick than long, Clayton, couldn’t help but notice. She lifted it to her mouth, and slowly licked the toy clean of all her cum before walking over to the sink to finish cleaning it. That sight alone almost had Clayton hard again, despite having nothing left to cum. She disappeared from the kitchen, and the music turned down, then off. She reappeared wearing only sweats, brushing the hair stuck to her face off and tying it up in a pony tail. She began to wipe down the island, and Clayton realized that it now was completely dark. It was time to go in.
He walked down to the door, being careful not to attract the attention of the now less-occupied Kendra, and rapped twice. He heard nothing for a minute, then the hurried sound of somebody running around the place.
“One minute!!” Kendra’s voice frantically called. A few seconds later, the door clicked open. Kendra stood there, now fully dressed and breathing heavy, a few hairs still clinging to her sweaty forehead. She helplessly looked around, trying to avoid any incriminating questions as the smell of sweat, humidity, and sex all but steamed out the door into the fall night.
“Hey Kendra, canlı bahis siteleri I got here before Emily tonight. She’s working late, or something.” Kendra shifted awkwardly.
“Yeah, she told me that, didn’t say anything about you coming over though.” She paused, looked at the potted plant on the deck, and continued in a tone too forced to be natural. “Been here long?”
Clayton had to suppress a smirk. “Only just walked up, actually.” He felt like pushing his luck a little bit further. “What have you been up to, anyways? You are extremely sweaty looking.” Kendra seized up.
“Uhhhhhhh, I was, umm, doing Yoga,” she stammered, gesturing vaguely at her yoga mat, rolled up neatly in the corner. “Yeah, I just finish- wrapped it up, you know. Well, I’m beat, and it’s pretty late, so I’ll be going to bed now, nice to see you.” With that, she turned and all but fled to her room, almost slamming the door behind her. Clayton looked at the clock on the wall. It was 7:30. He chuckled to himself. She really was caught off guard. He walked into the kitchen, and stared at the granite Kendra had been atop minutes ago. As he looked around, he noticed a tiny speck of something white and viscous still on the counter. A new wave of arousal washed over him as he reached down with his finger – it was still lukewarm. Clayton then did something he had never done before in his life, and put his finger to his mouth.
As Kendra’s cum was pungent, a little sour, and savory to the taste. It was powerful, full of pheromones that sent Clayton’s senses on fire. He was used to the taste of Emily by now, but this was something new…and he loved it. The sensual, almost spicy taste of her cum was enough to make him close his eyes for a minute, and bask in the flavor.
Kendra’s voice broke the silence.
“Uh, Clayton, what are you doing?”
Clayton’s eyes snapped open, and he stepped behind the counter to conceal an erection he did not feel rising. He stared at Kendra stupidly for a minute, finger stuck in his mouth, as she looked at him with a look that conveyed confusion, horror, irritation, incredulity, and something more that Clayton could not place. Her eyes snapped down to his waistline, which was behind the counter now, and back up to his face in an instant. Her cheeks reddened. He blinked twice, then pulled his finger out of his mouth slowly.
“I cut my finger on something sharp, on, uh, the counter here,” Clayton pointed momentarily at the island, then slid his hand quickly into his pocket.
“Uh huh,” Kendra said in a flat, annoyed tone. It was obvious she wasn’t buying it. “When did you get here again?”
“Five minutes ago,” Clayton blurted in a deadpan voice. Kendra glared at him, getting more red in the face, and her eyes darting around the room, an awkward tension forming instantly.
“So, I like doing my yoga uninterrupted and stuff, basically you really surprised me showing up like you did, it makes me a little uncomfortable to think about, so yeah.” At this point, the air could be cut with a knife.
“Just – I don’t know, text me if you are early again. So I have time to clean up and stuff, be presentable. You know, clean up from my yoga. Not like I need time to clean. Clean up from anything else, I mean. Not that anything else was going on. Nothing else, actually.” She stopped fumbling over words, and glanced at the counter where Clayton had wiped up her accidentally left-behind cum, and grew 3 shades deeper. She couldn’t look him in the eye. Clayton felt incredibly awkward. It was apparent Kendra knew he had seen enough of the aftermath of her self-gratification to know that indeed, she wasn’t doing any yoga. Clayton opened her mouth to speak, but Kendra cut him off with an abrupt step, and walked around the counter to reach in a drawer Clayton was standing by. His erection still half present, Clayton circled the counter opposite of her, drawing an even more uncomfortable pause into the room. She reached into the drawer, pulled out a pad of sticky notes, and wrote her cell number on it really fast.
“Just text me next time, so I can be sure and let you in.” She breezed out of the kitchen and back into her room without another word.
A few hours later, Emily walked into the apartment. She closed the door to her room, and flopped down face-first next to Clayton on the bed. She let out a tired sigh.
“Hey baby, I’m sorry I’m so late tonight.” She rolled over onto her back, taking her clothes off as she went. “I know I was teasing you a little bit earlier, but I am way too tired. You’ll have to use your imagination tonight, big boy.” She sat up, pulling on the oversize t-shirt she slept in. “Will you be OK?” Clayton lay still, staring at the ceiling.
“Yeah, I’ll be fine.” He said.
“OK,” Emily yawned, “I’m sure it helps your dreams feel more intense anyways. I bet I know exactly what you’ll be dreaming about.” Emily clicked out the light, and a little sigh told him she was already almost fast asleep
“I’m not so sure about that,” Clayton said quietly. He closed his eyes, and couldn’t help but feel a little bit guilty as fair-skinned, glistening lips started sliding down a thick, green toy again.
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Casket of civil rights icon John Lewis crosses Selma bridge
by Lorene Schwartz
The news coverage of the event help galvanise support for the passage of the Voting Rights Act of 1965.
Though Lewis is possibly best known for his march in Selma, he had already emerged as a leading voice in the fight for equal rights and had been arrested a number of times for the cause by then.
Lewis, 80, died July 17 several months after he was diagnosed with advanced pancreatic cancer.
As the wagon approached the bridge, members of the crowd shouted "Thank you, John Lewis!" and "Good trouble" - the phrase Lewis used to describe his tangles with white authorities during the civil rights movement.
Sunday found him crossing alone - instead of arm-in-arm with civil rights and political leaders - after his coffin was loaded atop a horse-drawn wagon that retraced the route through Selma from Brown Chapel African Methodist Episcopal Church, where the 1965 march began.
The bridge became a landmark in the fight for racial justice when Lewis and other marchers were beaten there 55 years ago on Bloody Sunday, a key event in the fight for voting rights for African Americans.
Lewis suffered a fractured skull and was one of dozens of nonviolent protesters who were hospitalized.
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He co-organised and spoke at the 1963 March on Washington for Jobs and Freedom, the rally at which Dr King delivered his historic I Have a Dream speech. Lewis led hundreds of marchers across the Edmund Pettus Bridge on March 7, 1965 as they faced attacks by state troopers.
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Selma officials, however, oppose the name change, according to the Associated Press.
Calls to rename the bridge for Lewis are increasing.
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