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300
However , for each time examined , the expression of NF-kappa B was maximum after the 0.5-Gy exposure .
However , for each time examined , the expression of <protein>NF-kappa B</protein> was maximum after the 0.5-Gy exposure .
301
The expression of the p50 and p65 NF-kappa B subunits was also shown to be regulated differentially after exposures to 1.0 and 2.0 Gy .
The expression of the <protein>p50 and p65 NF-kappa B subunits</protein> was also shown to be regulated differentially after exposures to 1.0 and 2.0 Gy .
302
Alternative splicing of RNA transcripts encoded by the murine p105 NF-kappa B gene generates I kappa B gamma isoforms with different inhibitory activities .
Alternative splicing of <rna>RNA transcripts</rna> encoded by the <dna>murine p105 NF-kappa B gene</dna> generates <protein>I kappa B gamma isoforms</protein> with different inhibitory activities .
303
The gene encoding the 105-kDa protein ( p105 ) precursor of the p50 subunit of transcription factor NF-kappa B also encodes a p70 I kappa B protein , I kappa B gamma , which is identical to the C-terminal 607 amino acids of p105 .
The gene encoding the <protein>105-kDa protein ( p105 ) precursor</protein> of the <protein>p50</protein> subunit of transcription factor <protein>NF-kappa B</protein> also encodes a <protein>p70 I kappa B protein</protein> , <protein>I kappa B gamma</protein> , which is identical to the <protein>C-terminal 607 amino acids</protein> of <protein>p105</protein> .
304
Here we show that alternative RNA splicing generates I kappa B gamma isoforms with properties different from those of p70 .
Here we show that alternative RNA splicing generates <protein>I kappa B gamma isoforms</protein> with properties different from those of <protein>p70</protein> .
305
One 63-kDa isoform , termed I kappa B gamma-1 , which lacks 59 amino acids C-terminal to ankyrin repeat 7 , has a novel 35-amino acid C terminus encoded by an alternative reading frame of the p105 gene .
One <protein>63-kDa isoform</protein> , termed <protein>I kappa B gamma-1</protein> , which lacks 59 amino acids C-terminal to <protein>ankyrin repeat 7</protein> , has a novel <protein>35-amino acid C terminus</protein> encoded by an <dna>alternative reading frame</dna> of the <dna>p105 gene</dna> .
306
A 55-kDa isoform , I kappa B gamma-2 , lacks the 190 C-terminal amino acids of p70 I kappa B gamma .
A <protein>55-kDa isoform</protein> , <protein>I kappa B gamma-2</protein> , lacks the <protein>190 C-terminal amino acids</protein> of <protein>p70</protein> <protein>I kappa B gamma</protein> .
307
In contrast to p70 I kappa B gamma , which is a cytoplasmic protein , I kappa B gamma-1 is found in both the cytoplasm and nucleus , whereas I kappa B gamma-2 is predominantly nuclear .
In contrast to <protein>p70</protein> <protein>I kappa B gamma</protein> , which is a <protein>cytoplasmic protein</protein> , <protein>I kappa B gamma-1</protein> is found in both the cytoplasm and nucleus , whereas <protein>I kappa B gamma-2</protein> is predominantly nuclear .
308
The I kappa B gamma isoforms also display differences in specificity and affinity for Rel/NF-kappa B proteins .
The <protein>I kappa B gamma isoforms</protein> also display differences in specificity and affinity for <protein>Rel/NF-kappa B proteins</protein> .
309
While p70 I kappa B gamma inhibits p50- , p65- , and c-Rel-mediated transactivation and/or DNA binding , both I kappa B gamma-1 and I kappa B gamma-2 are specific for p50 and have different affinities for this subunit .
While <protein>p70</protein> <protein>I kappa B gamma</protein> inhibits p50- , p65- , and c-Rel-mediated transactivation and/or DNA binding , both <protein>I kappa B gamma-1</protein> and <protein>I kappa B gamma-2</protein> are specific for <protein>p50</protein> and have different affinities for this subunit .
310
The absence in I kappa B gamma-1 and I kappa B gamma-2 of a protein kinase A site whose phosphorylation modulates p70 I kappa B gamma inhibitory activity suggests that alternative RNA splicing may be used to generate I kappa B gamma isoforms that respond differently to intracellular signals .
The absence in <protein>I kappa B gamma-1</protein> and <protein>I kappa B gamma-2</protein> of a <dna>protein kinase A site</dna> whose phosphorylation modulates <protein>p70</protein> <protein>I kappa B gamma</protein> inhibitory activity suggests that alternative RNA splicing may be used to generate <protein>I kappa B gamma isoforms</protein> that respond differently to intracellular signals .
311
Structure and expression of the human GATA3 gene .
Structure and expression of the <dna>human GATA3 gene</dna> .
312
GATA3 , a member of the GATA family that is abundantly expressed in the T-lymphocyte lineage , is thought to participate in T-cell receptor gene activation through binding to enhancers .
<protein>GATA3</protein> , a member of the <protein>GATA family</protein> that is abundantly expressed in the <cell_type>T-lymphocyte lineage</cell_type> , is thought to participate in <protein>T-cell receptor</protein> gene activation through binding to <dna>enhancers</dna> .
313
To understand GATA3 gene regulation , we cloned the human gene and the 5 ' end of the mouse GATA3 gene .
To understand <dna>GATA3 gene</dna> regulation , we cloned the <dna>human gene</dna> and the <dna>5 ' end</dna> of the <dna>mouse GATA3 gene</dna> .
314
We show that the human GATA3 gene contains six exons distributed over 17 kb of DNA .
We show that the <dna>human GATA3 gene</dna> contains six <dna>exons</dna> distributed over 17 kb of DNA .
315
The two human GATA3 zinc fingers are encoded by two separate exons highly conserved with those of GATA1 , but no other structural homologies between these two genes can be found .
The two <protein>human GATA3 zinc fingers</protein> are encoded by two separate <dna>exons</dna> highly conserved with those of <protein>GATA1</protein> , but no other structural homologies between these two genes can be found .
316
The human and mouse GATA3 transcription units start at a major initiation site .
The <dna>human and mouse GATA3 transcription units</dna> start at a <dna>major initiation site</dna> .
317
The promoter sequence analysis of these two genes revealed that they are embedded within a CpG island and share structural features often found in the promoters of housekeeping genes .
The promoter sequence analysis of these two genes revealed that they are embedded within a <dna>CpG island</dna> and share structural features often found in the <dna>promoters</dna> of <dna>housekeeping genes</dna> .
318
Finally , we show that a DNA fragment containing the human GATA3 transcription unit , 3 kb upstream from the initiation site and 4 kb downstream from the polyadenylation site , displays T-cell specificity .
Finally , we show that a <dna>DNA fragment</dna> containing the <dna>human GATA3 transcription unit</dna> , <dna>3 kb upstream</dna> from the <dna>initiation site</dna> and <dna>4 kb downstream</dna> from the <dna>polyadenylation site</dna> , displays T-cell specificity .
319
Characterization of the human gene encoding LBR , an integral protein of the nuclear envelope inner membrane .
Characterization of the <dna>human gene</dna> encoding <protein>LBR</protein> , an <protein>integral protein</protein> of the nuclear envelope inner membrane .
320
We have characterized the human gene encoding LBR , an integral protein of the nuclear envelope inner membrane .
We have characterized the <dna>human gene</dna> encoding <protein>LBR</protein> , an <protein>integral protein</protein> of the nuclear envelope inner membrane .
321
Restriction mapping shows that the transcription unit spans approximately 35 kilobases .
Restriction mapping shows that the <dna>transcription unit</dna> spans approximately 35 kilobases .
322
A transcription start site is located approximately 4 kilobases 5 ' to the translation initiation codon , and an RNA splice of 3863 bases occurs in the 5'-untranslated region to generate mature HeLa cell mRNA .
A transcription start site is located approximately 4 kilobases 5 ' to the <dna>translation initiation codon</dna> , and an RNA splice of 3863 bases occurs in the <dna>5'-untranslated region</dna> to generate <rna>mature HeLa cell mRNA</rna> .
323
5 ' to the identified transcription start site are two CCAAT sequences and potential recognition sites for several transcription factors including Sp1 , AP-1 , AP-2 , and NF-kB .
5 ' to the identified <dna>transcription start site</dna> are two <dna>CCAAT sequences</dna> and <dna>potential recognition sites</dna> for several <protein>transcription factors</protein> including <protein>Sp1</protein> , <protein>AP-1</protein> , <protein>AP-2</protein> , and <protein>NF-kB</protein> .
324
There are 13 protein coding exons in the LBR gene .
There are 13 protein coding <dna>exons</dna> in the <dna>LBR gene</dna> .
325
LBR 's nucleoplasmic domain is encoded by exons 1-4 , and its hydrophobic domain , with eight putative transmembrane segments , is encoded by exons 5-13 .
<protein>LBR</protein> 's <protein>nucleoplasmic domain</protein> is encoded by <dna>exons 1-4</dna> , and its <protein>hydrophobic domain</protein> , with eight putative <protein>transmembrane segments</protein> , is encoded by <dna>exons 5-13</dna> .
326
The hydrophobic domain is homologous to three yeast polypeptides , suggesting that this higher eukaryotic gene could have evolved from recombination between a gene that encoded a soluble nuclear protein and a membrane protein gene similar to those in yeast .
The <protein>hydrophobic domain</protein> is homologous to three yeast polypeptides , suggesting that this <dna>higher eukaryotic gene</dna> could have evolved from recombination between a gene that encoded a <protein>soluble nuclear protein</protein> and a <dna>membrane protein gene</dna> similar to those in yeast .
327
These results are the first to demonstrate the structural organization of a vertebrate gene encoding an integral membrane protein of the nuclear envelope that may be a member of a family of polypeptides conserved in evolution .
These results are the first to demonstrate the structural organization of a <dna>vertebrate gene</dna> encoding an <protein>integral membrane protein</protein> of the nuclear envelope that may be a member of a family of polypeptides conserved in evolution .
328
Retinoic acid-induced expression of CD38 antigen in myeloid cells is mediated through retinoic acid receptor-alpha .
Retinoic acid-induced expression of <protein>CD38 antigen</protein> in <cell_type>myeloid cells</cell_type> is mediated through <protein>retinoic acid receptor-alpha</protein> .
329
CD38 is a leukocyte differentiation antigen that has been thought to be a phenotypic marker of different subpopulations of T- and B- lymphocytes .
<protein>CD38</protein> is a <protein>leukocyte differentiation antigen</protein> that has been thought to be a phenotypic marker of different subpopulations of <cell_type>T- and B- lymphocytes</cell_type> .
330
In myeloid cells , CD38 is expressed during early stages of differentiation .
In <cell_type>myeloid cells</cell_type> , <protein>CD38</protein> is expressed during early stages of differentiation .
331
Virtually no information is available on regulation and functions of CD38 .
Virtually no information is available on regulation and functions of <protein>CD38</protein> .
332
Recently we reported that all-trans-retinoic acid ( ATRA ) is a potent and highly specific inducer of CD38 expression in human promyelocytic leukemia cells .
Recently we reported that all-trans-retinoic acid ( ATRA ) is a potent and highly specific inducer of <protein>CD38</protein> expression in <cell_type>human promyelocytic leukemia cells</cell_type> .
333
Here we report that ATRA-induced expression of CD38 antigen in myeloid cells is mediated through retinoic acid-alpha receptor ( RAR alpha ) .
Here we report that ATRA-induced expression of <protein>CD38 antigen</protein> in <cell_type>myeloid cells</cell_type> is mediated through <protein>retinoic acid-alpha receptor</protein> ( <protein>RAR alpha</protein> ) .
334
ATRA failed to induce CD38 expression in a mutant subclone of the HL-60 myeloid leukemia cell line ( designated HL-60R ) that is relatively resistant to ATRA-induced granulocytic differentiation .
ATRA failed to induce <protein>CD38</protein> expression in a <cell_line>mutant subclone</cell_line> of the <cell_line>HL-60 myeloid leukemia cell line</cell_line> ( designated <cell_line>HL-60R</cell_line> ) that is relatively resistant to ATRA-induced granulocytic differentiation .
335
Retroviral vector-mediated transduction of RA receptor ( RAR alpha ) into this HL-60R subclone completely restored the sensitivity of these cells to ATRA in terms of their ability to express CD38 .
Retroviral vector-mediated transduction of <protein>RA receptor</protein> ( <protein>RAR alpha</protein> ) into this <cell_line>HL-60R subclone</cell_line> completely restored the sensitivity of these cells to ATRA in terms of their ability to express <protein>CD38</protein> .
336
In contrast , CD38 expression was not inducible by ATRA in HL-60R cells , transfected with a functional RAR beta , RAR gamma , or RXR alpha receptor .
In contrast , <protein>CD38</protein> expression was not inducible by ATRA in <cell_line>HL-60R cells</cell_line> , transfected with a functional <protein>RAR beta</protein> , <protein>RAR gamma</protein> , or <protein>RXR alpha receptor</protein> .
337
Induction of CD38 in acute promyelocytic and acute myeloblastic leukemia cells was independent of ATRA-induced cytodifferentiation .
Induction of <protein>CD38</protein> in <cell_type>acute promyelocytic and acute myeloblastic leukemia cells</cell_type> was independent of ATRA-induced cytodifferentiation .
338
Following culture with ATRA , increased CD38 protein levels were also observed in normal CD34+ bone marrow cells , but not on normal circulating granulocytes .
Following culture with ATRA , increased <protein>CD38</protein> protein levels were also observed in <cell_type>normal CD34+ bone marrow cells</cell_type> , but not on <cell_type>normal circulating granulocytes</cell_type> .
339
From these results , we conclude that CD38 is ATRA inducible in myeloid leukemia cells and normal CD34+ bone marrow cells .
From these results , we conclude that <protein>CD38</protein> is ATRA inducible in <cell_type>myeloid leukemia cells</cell_type> and <cell_type>normal CD34+ bone marrow cells</cell_type> .
340
This effect is independent of differentiation and is mediated by RAR alpha in HL-60 cells , suggesting a similar role for RAR alpha in CD38 expression in other hematopoietic cells .
This effect is independent of differentiation and is mediated by <protein>RAR alpha</protein> in <cell_type>HL-60 cells</cell_type> , suggesting a similar role for <protein>RAR alpha</protein> in <protein>CD38</protein> expression in other <cell_type>hematopoietic cells</cell_type> .
341
Some antioxidants inhibit , in a co-ordinate fashion , the production of tumor necrosis factor-alpha , IL-beta , and IL-6 by human peripheral blood mononuclear cells .
Some antioxidants inhibit , in a co-ordinate fashion , the production of <protein>tumor necrosis factor-alpha</protein> , <protein>IL-beta</protein> , and <protein>IL-6</protein> by <cell_type>human peripheral blood mononuclear cells</cell_type> .
342
Some antioxidants , including butylated hydroxyanisole ( BHA ) , tetrahydropapaveroline ( THP ) , nordihydroguiauretic acid , and 10 , 11-dihydroxyaporphine ( DHA ) , were found to be potent inhibitors of the production of tumor necrosis factor ( TNF ) -alpha , IL-1 beta , and IL-6 by human peripheral blood mononuclear cells ( PBMC ) stimulated by lipopolysaccharide ( LPS ) ( IC50s in the low micromolar range ) .
Some antioxidants , including butylated hydroxyanisole ( BHA ) , tetrahydropapaveroline ( THP ) , nordihydroguiauretic acid , and 10 , 11-dihydroxyaporphine ( DHA ) , were found to be potent inhibitors of the production of <protein>tumor necrosis factor ( TNF ) -alpha</protein> , <protein>IL-1 beta</protein> , and <protein>IL-6</protein> by <cell_type>human peripheral blood mononuclear cells</cell_type> ( <cell_type>PBMC</cell_type> ) stimulated by lipopolysaccharide ( LPS ) ( IC50s in the low micromolar range ) .
343
Inhibition of cytokine production was gene selective and not due to general effects on protein synthesis .
Inhibition of <protein>cytokine</protein> production was gene selective and not due to general effects on protein synthesis .
344
Inhibition of cytokine production by PBMC was observed also when other inducers were used ( staphylococci , silica , zymosan ) .
Inhibition of <protein>cytokine</protein> production by <cell_type>PBMC</cell_type> was observed also when other inducers were used ( staphylococci , silica , zymosan ) .
345
Much higher concentrations of other antioxidants -- including ascorbic acid , trolox , alpha-tocopherol , butylated hydroxytoluene , and the 5-lipoxygenase inhibitor zileuton -- did not affect the production of these cytokines .
Much higher concentrations of other antioxidants -- including ascorbic acid , trolox , alpha-tocopherol , butylated hydroxytoluene , and the 5-lipoxygenase inhibitor zileuton -- did not affect the production of these <protein>cytokines</protein> .
346
The active compounds did not inhibit IL-1 -induced production of IL-6 in fibroblasts , showing the cell selectivity of the effect .
The active compounds did not inhibit <protein>IL-1</protein> -induced production of <protein>IL-6</protein> in <cell_type>fibroblasts</cell_type> , showing the cell selectivity of the effect .
347
Antioxidant-mediated inhibition of cytokine production was correlated with low levels of the corresponding messenger RNAs .
Antioxidant-mediated inhibition of <protein>cytokine</protein> production was correlated with low levels of the corresponding <rna>messenger RNAs</rna> .
348
Nuclear run-on experiments showed that THP inhibited transcription of the IL-1 beta gene .
Nuclear run-on experiments showed that THP inhibited transcription of the <dna>IL-1 beta gene</dna> .
349
THP decreased the concentration of the transcription factors NF-kappa B and AP-1 detected in nuclear extracts of PBMC cultured in the presence or absence of LPS .
THP decreased the concentration of the <protein>transcription factors</protein> <protein>NF-kappa B</protein> and <protein>AP-1</protein> detected in nuclear extracts of <cell_type>PBMC</cell_type> cultured in the presence or absence of LPS .
350
THP and DHA markedly decreased the levels of TNF-alpha and IL-1 beta in the circulation of mice following LPS injection .
THP and DHA markedly decreased the levels of <protein>TNF-alpha</protein> and <protein>IL-1 beta</protein> in the circulation of mice following LPS injection .
351
Thus antioxidants vary widely in potency as inhibitors of the activation of transcription factors and of the transcription of genes for pro-inflammatory cytokines .
Thus antioxidants vary widely in potency as inhibitors of the activation of <protein>transcription factors</protein> and of the transcription of <dna>genes</dna> for <protein>pro-inflammatory cytokines</protein> .
352
Coordinate inhibition of the transcription of genes for inflammatory cytokines could provide a strategy for therapy of diseases with inflammatory pathogenesis and for septic shock .
Coordinate inhibition of the transcription of genes for inflammatory <protein>cytokines</protein> could provide a strategy for therapy of diseases with inflammatory pathogenesis and for septic shock .
353
An interleukin-4-induced transcription factor : IL-4 Stat .
An <protein>interleukin-4-induced transcription factor</protein> : <protein>IL-4 Stat</protein> .
354
Interleukin-4 ( IL-4 ) is an immunomodulatory cytokine secreted by activated T lymphocytes , basophils , and mast cells .
<protein>Interleukin-4</protein> ( <protein>IL-4</protein> ) is an immunomodulatory cytokine secreted by activated <cell_type>T lymphocytes</cell_type> , <cell_type>basophils</cell_type> , and <cell_type>mast cells</cell_type> .
355
It plays an important role in modulating the balance of T helper ( Th ) cell subsets , favoring expansion of the Th2 lineage relative to Th1 .
It plays an important role in modulating the balance of <cell_type>T helper ( Th ) cell subsets</cell_type> , favoring expansion of the <cell_type>Th2 lineage</cell_type> relative to <cell_type>Th1</cell_type> .
356
Imbalance of these T lymphocyte subsets has been implicated in immunological diseases including allergy , inflammation , and autoimmune disease .
Imbalance of these <cell_type>T lymphocyte subsets</cell_type> has been implicated in immunological diseases including allergy , inflammation , and autoimmune disease .
357
IL-4 may mediate its biological effects , at least in part , by activating a tyrosine-phosphorylated DNA binding protein .
<protein>IL-4</protein> may mediate its biological effects , at least in part , by activating a <protein>tyrosine-phosphorylated DNA binding protein</protein> .
358
This protein has now been purified and its encoding gene cloned .
This protein has now been purified and its encoding gene cloned .
359
Examination of the primary amino acid sequence of this protein indicates that it is a member of the signal transducers and activators of transcription ( Stat ) family of DNA binding proteins , hereby designated IL-4 Stat .
Examination of the primary amino acid sequence of this protein indicates that it is a member of the <protein>signal transducers and activators of transcription ( Stat ) family</protein> of <protein>DNA binding proteins</protein> , hereby designated <protein>IL-4 Stat</protein> .
360
Study of the inhibitory activities of phosphotyrosine-containing peptides derived from the intracellular domain of the IL-4 receptor provided evidence for direct coupling of receptor and transcription factor during the IL-4 Stat activation cycle .
Study of the inhibitory activities of phosphotyrosine-containing peptides derived from the <protein>intracellular domain of the IL-4 receptor</protein> provided evidence for direct coupling of <protein>receptor</protein> and <protein>transcription factor</protein> during the <protein>IL-4 Stat</protein> activation cycle .
361
Such observations indicate that IL-4 Stat has the same functional domain for both receptor coupling and dimerization .
Such observations indicate that <protein>IL-4 Stat</protein> has the same <protein>functional domain</protein> for both receptor coupling and dimerization .
362
Evaluation of the respiratory epithelium of normals and individuals with cystic fibrosis for the presence of adenovirus E1a sequences relevant to the use of E1a- adenovirus vectors for gene therapy for the respiratory manifestations of cystic fibrosis .
Evaluation of the respiratory epithelium of normals and individuals with cystic fibrosis for the presence of <dna>adenovirus E1a sequences</dna> relevant to the use of E1a- adenovirus vectors for gene therapy for the respiratory manifestations of cystic fibrosis .
363
Lung disease associated with disorders such as cystic fibrosis ( CF ) may be amenable to somatic gene therapy in which there is delivery of the normal gene directly to the respiratory epithelium using E1a- adenovirus ( Ad ) type 2- or 5-based vectors .
Lung disease associated with disorders such as cystic fibrosis ( CF ) may be amenable to somatic gene therapy in which there is delivery of the <dna>normal gene</dna> directly to the respiratory epithelium using E1a- adenovirus ( Ad ) type 2- or 5-based vectors .
364
For safety reasons , the Ad vectors are rendered replication deficient by deletion of the E1a region .
For safety reasons , the Ad vectors are rendered replication deficient by deletion of the <dna>E1a region</dna> .
365
Because there is the theoretical possibility of an E1a- replication-deficient vector replicating as a result of recombination or complementation with Ad 2/5 E1a sequences present in the target cell , this study is directed toward evaluating respiratory epithelium of normals and individuals with CF for the presence of E1a sequences .
Because there is the theoretical possibility of an E1a- replication-deficient vector replicating as a result of recombination or complementation with <dna>Ad 2/5 E1a sequences</dna> present in the target cell , this study is directed toward evaluating respiratory epithelium of normals and individuals with CF for the presence of E1a sequences .
366
Using Ad 2/5 E1a-specific primers and the polymerase chain reaction to evaluate DNA recovered from freshly isolated nasal and bronchial epithelium recovered by brushing , E1a sequences were detected in respiratory epithelium of 19 of 91 normals ( 21 % ) .
Using <dna>Ad 2/5 E1a-specific primers</dna> and the polymerase chain reaction to evaluate DNA recovered from freshly isolated nasal and bronchial epithelium recovered by brushing , <dna>E1a sequences</dna> were detected in respiratory epithelium of 19 of 91 normals ( 21 % ) .
367
In the E1a-positive samples , the average of E1a copy number was 55 +/- 18/10 ( 3 ) recovered cells .
In the E1a-positive samples , the average of E1a copy number was 55 +/- 18/10 ( 3 ) recovered cells .
368
In CF individuals , 7 of 52 ( 13 % ) had detectable E1a sequences in the respiratory epithelium , with E1a copy number in the positive samples of 80 +/- 21/10 ( 3 ) recovered cells .
In CF individuals , 7 of 52 ( 13 % ) had detectable <dna>E1a sequences</dna> in the respiratory epithelium , with E1a copy number in the positive samples of 80 +/- 21/10 ( 3 ) recovered cells .
369
These results demonstrate that there are detectable Ad 2/5 E1a sequences in the respiratory epithelium of a small percentage of normals and individuals with CF .
These results demonstrate that there are detectable <dna>Ad 2/5 E1a sequences</dna> in the respiratory epithelium of a small percentage of normals and individuals with CF .
370
Because of the theoretical potential of such sequences supporting replication of E1a- Ad vectors , human gene therapy protocols for CF utilizing such vectors should consider evaluating study individuals for the presence of Ad 2/5 E1a sequences in the respiratory epithelium .
Because of the theoretical potential of such sequences supporting replication of E1a- Ad vectors , human gene therapy protocols for CF utilizing such vectors should consider evaluating study individuals for the presence of <dna>Ad 2/5 E1a sequences</dna> in the respiratory epithelium .
371
Leiomyosarcoma of the vulva : report of a case .
Leiomyosarcoma of the vulva : report of a case .
372
A 52-year-old female presented with a progressively enlarging vulvar mass .
A 52-year-old female presented with a progressively enlarging vulvar mass .
373
Pathological evaluation revealed a high-grade vulvar leiomyosarcoma .
Pathological evaluation revealed a high-grade vulvar leiomyosarcoma .
374
Immunohistochemical and ultrastructural studies were performed to support the diagnosis .
Immunohistochemical and ultrastructural studies were performed to support the diagnosis .
375
In an effort to better understand the biology of this tumor additional immunohistochemical studies for the protein product of p53 tumor suppressor gene and estrogen receptor expression by tumor cells , as well as the type of immune cells infiltrating the tumor were performed .
In an effort to better understand the biology of this tumor additional immunohistochemical studies for the <protein>protein product</protein> of <dna>p53 tumor suppressor gene</dna> and <protein>estrogen receptor</protein> expression by <cell_type>tumor cells</cell_type> , as well as the type of <cell_type>immune cells</cell_type> infiltrating the tumor were performed .
376
Tumor cells showed an overexpression of p53 protein and were estrogen receptor -positive .
<cell_type>Tumor cells</cell_type> showed an overexpression of <protein>p53 protein</protein> and were <protein>estrogen receptor</protein> -positive .
377
Macrophages and T and B lymphocytes infiltrated the tumor in moderate numbers with occasional lymphoid aggregate formation .
<cell_type>Macrophages</cell_type> and <cell_type>T and B lymphocytes</cell_type> infiltrated the tumor in moderate numbers with occasional lymphoid aggregate formation .
378
This study is the first attempt to better understand the biology of these tumors .
This study is the first attempt to better understand the biology of these tumors .
379
Stimulation of HIV replication in mononuclear phagocytes by leukemia inhibitory factor .
Stimulation of HIV replication in <cell_type>mononuclear phagocytes</cell_type> by <protein>leukemia inhibitory factor</protein> .
380
This study examined the effects of leukemia inhibitory factor ( LIF ) on human immunodeficiency virus ( HIV ) replication in mononuclear phagocytes ( MNP ) .
This study examined the effects of <protein>leukemia inhibitory factor</protein> ( <protein>LIF</protein> ) on human immunodeficiency virus ( HIV ) replication in <cell_type>mononuclear phagocytes</cell_type> ( <cell_type>MNP</cell_type> ) .
381
LIF induced a dose-dependent increase in p24 antigen production in the chronically infected promonocytic cell line U1 .
<protein>LIF</protein> induced a dose-dependent increase in <protein>p24 antigen</protein> production in the <cell_line>chronically infected promonocytic cell line U1</cell_line> .
382
The magnitude and time kinetics of the LIF effects were similar to interleukin 1 ( IL-1 ) , IL-6 , and tumor necrosis factor ( TNF ) , other cytokines known to induce HIV replication in this cell line .
The magnitude and time kinetics of the <protein>LIF</protein> effects were similar to <protein>interleukin 1</protein> ( <protein>IL-1</protein> ) , <protein>IL-6</protein> , and <protein>tumor necrosis factor</protein> ( <protein>TNF</protein> ) , other cytokines known to induce HIV replication in this cell line .
383
To characterize mechanisms responsible for these LIF effects , levels of HIV mRNA , activation of the DNA binding protein nuclear factor ( NF ) -kB , signal transduction pathways , and potential interactions with other cytokines were analyzed .
To characterize mechanisms responsible for these <protein>LIF</protein> effects , levels of <rna>HIV mRNA</rna> , activation of the <protein>DNA binding protein nuclear factor</protein> <protein>( NF ) -kB</protein> , signal transduction pathways , and potential interactions with other <protein>cytokines</protein> were analyzed .
384
LIF increased steady-state levels of HIV mRNA at 2.0 , 4.3 , and 9.2 kB .
<protein>LIF</protein> increased steady-state levels of <rna>HIV mRNA</rna> at 2.0 , 4.3 , and 9.2 kB .
385
This was detectable by 24 h and persisted until 72 h .
This was detectable by 24 h and persisted until 72 h .
386
The DNA binding protein NF-kB is a central mediator in cytokine activation of HIV transcription .
The <protein>DNA binding protein NF-kB</protein> is a central mediator in <protein>cytokine</protein> activation of HIV transcription .
387
NF-kB levels were higher in unstimulated U1 cells as compared to the parent cell line U937 .
<protein>NF-kB</protein> levels were higher in <cell_line>unstimulated U1 cells</cell_line> as compared to the <cell_line>parent cell line</cell_line> <cell_line>U937</cell_line> .
388
In both cell lines LIF increased NF-kB activity .
In both cell lines <protein>LIF</protein> increased <protein>NF-kB</protein> activity .
389
Induction of NF-kB and HIV replication by cytokines are at least in part dependent on reactive oxygen intermediates .
Induction of <protein>NF-kB</protein> and HIV replication by <protein>cytokines</protein> are at least in part dependent on reactive oxygen intermediates .
390
The oxygen radical scavenger N-acetyl-L-cysteine , but not an inhibitor of nitric oxide synthase , inhibited LIF -induced HIV replication .
The oxygen radical scavenger N-acetyl-L-cysteine , but not an inhibitor of <protein>nitric oxide synthase</protein> , inhibited <protein>LIF</protein> -induced HIV replication .
391
LIF induces the production of other cytokines in monocytes but its effects on HIV replication were not inhibited by antibodies to IL-1 , TNF , or IL-6 .
<protein>LIF</protein> induces the production of other <protein>cytokines</protein> in monocytes but its effects on HIV replication were not inhibited by <protein>antibodies</protein> to <protein>IL-1</protein> , <protein>TNF</protein> , or <protein>IL-6</protein> .
392
These results identify LIF as a stimulus of HIV replication .
These results identify <protein>LIF</protein> as a stimulus of HIV replication .
393
( ABSTRACT TRUNCATED AT 250 WORDS )
( ABSTRACT TRUNCATED AT 250 WORDS )
394
Mechanisms involved in the inhibition of growth of a human B lymphoma cell line , B104 , by anti-MHC class II antibodies .
Mechanisms involved in the inhibition of growth of a <cell_line>human B lymphoma cell line</cell_line> , <cell_line>B104</cell_line> , by <protein>anti-MHC class II antibodies</protein> .
395
The mechanisms involved in the inhibition of growth of a human B lymphoma cell line , B104 , by anti-MHC class II antibodies ( Ab ) were compared with those in anti-IgM Ab -induced B104 growth inhibition .
The mechanisms involved in the inhibition of growth of a <cell_line>human B lymphoma cell line</cell_line> , <cell_line>B104</cell_line> , by <protein>anti-MHC class II antibodies</protein> ( <protein>Ab</protein> ) were compared with those in <protein>anti-IgM Ab</protein> -induced B104 growth inhibition .
396
Two anti-MHC class II Ab , L227 and 2.06 , inhibited the growth of B104 cells , although 2.06 , but not L227 , needed to be further cross-linked with a goat anti-mouse IgG Ab ( GAM ) to show the effect .
Two <protein>anti-MHC class II Ab</protein> , <protein>L227</protein> and <protein>2.06</protein> , inhibited the growth of <cell_line>B104 cells</cell_line> , although <protein>2.06</protein> , but not <protein>L227</protein> , needed to be further cross-linked with a <protein>goat anti-mouse IgG Ab</protein> ( <protein>GAM</protein> ) to show the effect .
397
L227 induced an increase in intracellular free Ca2+ concentration ( [ Ca2+ ] i ) from the intracellular pool and little or no protein tyrosine phosphorylation , phosphatidyl inositol turnover , or expression of Egr-1 mRNA , whereas 2.06 plus GAM induced an increase in [ Ca2+ ] i from both the intracellular and , in particular , the extracellular pools .
<protein>L227</protein> induced an increase in intracellular free Ca2+ concentration ( [ Ca2+ ] i ) from the intracellular pool and little or no protein tyrosine phosphorylation , phosphatidyl inositol turnover , or expression of <rna>Egr-1 mRNA</rna> , whereas <protein>2.06</protein> plus <protein>GAM</protein> induced an increase in [ Ca2+ ] i from both the intracellular and , in particular , the extracellular pools .
398
The inhibition of B104 cell growth induced by anti-MHC class II Ab was Ca ( 2+ ) -independent and not inhibited by actinomycin D or cyclosporin A , and cell cycle arrest at the G2/M interphase was not observed .
The inhibition of <cell_line>B104 cell</cell_line> growth induced by <protein>anti-MHC class II Ab</protein> was Ca ( 2+ ) -independent and not inhibited by actinomycin D or cyclosporin A , and cell cycle arrest at the G2/M interphase was not observed .
399
These features are very different from those observed in B104 cell death induced by anti-IgM Ab .
These features are very different from those observed in <cell_line>B104 cell</cell_line> death induced by <protein>anti-IgM Ab</protein> .