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no acute intrathoracic process.
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no convincing signs of pneumonia. extensive calcified pleural plaque. probable mild improvement in previously noted small bilateral effusions.
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no significant interval change.
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<num>. limited visualization of subglottic and proximal tracheal air columns as described above. <num>. linear left basilar atelectasis.
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no focal consolidation. increased interstitial markings, unchanged, potentially due to chronic interstitial abnormality. atypical infection or edema are possible.
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no signs of pneumonia or other acute intrathoracic process.
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<num>. massive cardiomegaly and mild interstitial pulmonary edema. <num>. cholelithiasis.
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<num>. missing tooth is identified within the stomach. <num>. persistent mild pulmonary edema. <num>. decreased left pleural effusion, now small.
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no pneumothorax.
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no evidence of acute cardiopulmonary process.
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no acute cardiopulmonary process. interval enlargement and of the density at the posterior aspect of the mediastinum on the right. this is not definitively a hiatal hernia. it demonstrates intervcal enlargement since <unk>. consider nonurgent chest ct in to further characterize.
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stable appearance of the chest.
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stable chest findings. no evidence of new acute infiltrates. unchanged minimal scar formations.
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no signs of pneumonia.
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elevated right hemidiaphragm, similar to prior, with small to moderate right pleural effusion. adjacent right lower lobe atelectasis is also similar to prior but superimposed consolidation cannot be excluded.
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no acute cardiopulmonary abnormality.
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mild pulmonary edema. bibasilar atelectasis. although an early pneumonic infiltrate might be difficult to exclude, no definite change compared with <unk> and no frank consolidation is identified.
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no radiographic evidence for acute cardiopulmonary process.
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significant chest trauma in right side. observe that next previous examination of <unk> did not show similar changes. does patient have history of severe thorax trauma during that time? acute parenchymal infiltrates in left upper lobe lingula as well as right lower lobe posterior segment are probably new.. followup che...
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interval increase in bibasilar reticular opacities which may be due to worsening of chronic interstitial lung disease, but superimposed infection may be present.
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mild improvement in left basilar opacity with continued atelectasis at the left base.
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no evidence of acute cardiopulmonary abnormality.
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small to moderate right pleural effusion, grossly stable since the prior chest ct.
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no acute cardiopulmonary process. nodular density projecting over the anterior right sixth rib. this could be within the bone although underlying pulmonary nodule is possible. shallow obliques suggested on a nonurgent basis to further assess.
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in comparison to <unk> exam, moderate right pleural effusion has resolved. stable marked cardiomegaly.
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pulmonary edema with bibasilar opacities likely due to some combination of pleural effusions and atelectasis.
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no evidence of acute disease.
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left basilar atelectasis. no radiographic evidence for pneumonia.
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retrocardiac opacity, which likely represents pneumonia.
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dense left hilus and heterogeneity of the right lung parenchyma. recommend further evaluation with routine oblique views. telephone notification regarding change in wet read and recommendations to dr <unk> by dr <unk> at <time> on <unk>.
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top normal heart size with calcified tortuous aorta, without radiographic evidence for acute change.
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no evidence of acute cardiopulmonary disease. similar chronic opacity at the left lung base with resolution of relative increased on recent chest radiography.
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no acute intrathoracic process.
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<num>. no significant interval change.
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no acute intrathoracic abnormality.
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cardiomegaly without superimposed acute cardiopulmonary process.
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persistent right-sided pleural effusion. small left pleural effusion. increased opacities at the lung bases particularly on the right likely due to superimposed infection given interval change since recent exam. recommend repeat after treatment to document resolution.
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significant interval decrease in cardiomegaly no evidence of hemodynamic instability.
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interstitial pulmonary edema.
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no evidence of pneumonia.
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no evidence of pulmonary edema or pneumonia. probable copd. possible trace bilateral pleural effusions versus pleural thickening.
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clear lungs.
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there is an increasing left pleural effusion with associated consolidation suggestive of partial lower lobe atelectasis. in addition, there has been interval appearance of mild perihilar edema. pneumoperitoneum is still present but has decreased in conspicuity. tiny left apical pneumothorax is stable. no right-sided pn...
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no acute intrathoracic process.
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no focal consolidation.
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no acute cardiopulmonary process.
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<num>. resolution of previous bibasilar atelectasis. <num>. diaphragmatic flattening suggesting hyperinflation.
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no acute cardiopulmonary abnormality.
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stable examination of the chest with persistent moderate right-sided pneumothorax.
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faint increase in opacity of the right middle <unk>, <unk> be concerning for an infectious process.
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linear opacities in lower lungs, most compatible with scarring or atelectasis.
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persistent left lung base consolidation, most likely atelectasis, with associated small left pleural effusion. asymmetric opacification of the left hemithorax likely represents mild asymmetric pulmonary edema.
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mildly improved left lower lung capacity. small left pleural effusion.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality. no subdiaphragmatic free air
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no acute cardiopulmonary process.
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small bilateral pleural effusions without an acute cardiopulmonary process.
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no radiographic evidence for acute cardiopulmonary process.
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most likely bilateral atelectasis, but pneumonia in the correct clinical setting can be considered.
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as above.
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pacemaker leads in standard position.
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no significant interval change.
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possible minimal central pulmonary vascular congestion without overt pulmonary edema.
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bibasilar opacities likely reflecting atelectasis in the setting of low lung volumes, however infection could be a possibility in the appropriate clinical setting.
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chronic streaky right greater than left bibasilar atelectasis/scarring. no definite new focal consolidation. small right pleural effusion.
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no evidence of pneumonia. possible <num> mm left lung nodule. moderate cardiomegaly predominantly right ventricular.
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<num>. small bilateral pleural effusions. <num>. enteric tube courses into the region of the proximal stomach with apparent sharp kink. suggest repositioning. the above findings were discussed with dr. <unk> at <time> a.m. on <unk> via telephone, <num> minutes after discovery.
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no signs of pneumonia or overt chf. top normal heart size.
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trace linear atelectasis at the left base. no focal consolidation, pleural or pneumothorax.
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no acute cardiopulmonary abnormality.
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retrocardiac opacity unchanged likely represents atelectasis or scarring, though difficult to exclude a subtle pneumonia. small left pleural effusion.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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mild pulmonary edema substantially improved from <unk>. no evidence of pneumonia.
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<num>. no radiographic evidence for acute cardiopulmonary process. <num>. ct of chest may provide more information regarding pericardium and right paratracheal widening if clinically relevant.
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no acute cardiopulmonary abnormality.
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right infrahilar opacity likely represents atelectasis. no evidence of pulmonary edema or pneumonia.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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no definite acute cardiopulmonary process. right-sided pleural based tumor and basilar scarring, as seen on ct scan from earlier the same day.
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multiple right rib fractures, mildly displaced, acute. no pneumothorax.
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no acute cardiopulmonary process.
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subtle increased right lower lobe opacity could be due to early infection versus atelectasis in the appropriate clinical setting. widened superior mediastinum corresponds to underlying lymphadenopathy as seen on recent prior pet-ct from <unk>.
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no acute process.
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no pneumothorax status post water seal placement of right-sided chest tubes. rest of the findings also remain unchanged.
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no acute cardiopulmonary disease to suggest pneumonia.
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no acute cardiopulmonary process.
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progression of mild pulmonary edema since the prior radiograph.
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interval removal of right chest tube. small right apical pneumothorax without tension.
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normal chest radiograph without evidence of pneumonia
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no acute cardiopulmonary process.
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no radiographic evidence for acute cardiopulmonary process.
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hyperinflation consistent with copd. no significant no consolidation or significant effusion. minimal bibasilar atelectasis and possible tiny bilateral effusions. small amount of free air seen beneath the diaphragm, consistent with recent abdominal surgery.
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low lung volumes with patchy opacities in the lung bases. these may reflect areas of atelectasis though infection or aspiration cannot be completely excluded.
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no evidence of focal consolidation, pleural effusion or pneumothorax. right hilar fullness and multiple nodular pulmonary opacities are consistent with the patient's known sarcoidosis and are better demonstrated on recent chest ct from <unk>
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normal chest radiograph
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little interval change compared to the previous exam. persistent moderate size right pleural effusion with bibasilar atelectasis. post radiation changes in the medial right upper lobe. dilated and tortuous aorta status post descending thoracic aortic stent graft.
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apparent right upper lobe nodule. repeat pa and lateral chest radiograph should be performed without any overlying objects on the patient to ensure that this is truly a nodule. findings discussed with <unk> md at <num> am.
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right lower lobe pneumonia.
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no acute cardiopulmonary process.