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stable chest radiograph without evidence for pneumonia.
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no evidence of acute cardiopulmonary process.
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<num>) resolving left lower lobe pneumonia <num>) previously mild pulmonary edema has improved.
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bilateral atelectasis likely due to low lung volumes. follow up pa and lateral radiographs can be obtained when the patient is stable to better evaluate for lung pathology.
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no pneumothorax identified post removal of chest tube. slightly increased amount of subcutaneous emphysema along the left neck and left lateral chest and abdominal wall. small left pleural effusion and bibasilar atelectasis.
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right lower lobe pneumonia.
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mild interval increase in small left pleural effusion with stable small right pleural effusion.
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no acute cardiopulmonary process.
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bilateral pleural effusions, moderate on the right and small on left. stable cardiomegaly. right basal atelectasis, difficult to exclude underlying pneumonia.
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small suspected increased in right-sided pneumothorax allowing for differences in technique.
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no evidence of acute cardiopulmonary process.
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status post endobronchial coil placement with no evidence of pneumothorax.
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central peribronchial wall thickening without definite focal consolidation.
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normal chest radiographs.
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no acute cardiopulmonary abnormalities
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no acute cardiopulmonary process.
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<num>. right picc terminates in the proximal to mid svc without evidence of pneumothorax. mild left base atelectasis.
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findings suggesting mild pulmonary edema.
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severe right pneumonia, moderate left lower lobe pneumonia, and small left pleural effusion, all worsened over <num> hours.
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no acute cardiopulmonary process. this study is not well suited for detailed evaluation of the known left lower lobe pulmonary mass.
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findings perhaps suggestive of mild vascular congestion.
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no acute findings including no displaced rib fracture.
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persistent left lower lobe opacity which could reflect atelectasis though aspiration or infection cannot be excluded.
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mild interstitial pulmonary edema and small, left greater than right, pleural effusions slightly worse since the prior study.
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et tube terminating <num> cm above the carina. right basilar atelectasis. no pleural effusion or pneumothorax.
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new right ij line with tip in the right atrium and should be withdrawn <num> cm.
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no acute cardiopulmonary process.
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hyperinflated lungs suggesting chronic obstructive pulmonary disease. no focal consolidation to suggest pneumonia.
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no acute cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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<num>. no acute cardiopulmonary process. <num>. no displaced rib fractures.
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left lower lobe pneumonia. recommend followup imaging after resolution of symptoms.
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left subclavian picc with tip projecting over the cavoatrial junction. results were discussed with the <unk> over the telephone by dr. <unk> at <time> on <unk> at time of initial review.
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no acute cardiopulmonary process. left shoulder fracture-dislocation.
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no acute cardiopulmonary abnormality.
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no acute cardiothoracic process.
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new bilateral hazy opacities with persistent moderate cardiomegaly. these findings are likely representative of moderate pulmonary edema due to congestive heart failure.
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hyperinflated lungs without radiographic evidence for acute change.
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mild pulmonary vascular congestion with no pulmonary edema. stable mild cardiomegaly.
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improved aeration in the left lower lung with persistent linear opacity at the site of prior pneumonia, which could represent residual atelectasis.
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low lung volumes which lead to bronchovascular crowding. apparent blunting of the bilateral costophrenic angles may be due to overlying soft tissue although <unk> pleural effusions not excluded.
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no focal consolidations concerning for pneumonia identified.
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no significant interval change.
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heterogeneous right lower and middle lobe consolidation of uncertain chronicity and etiology. in a patient without acute pulmonary symptoms or recent aspiration episode, these findings are concerning for malignancy such as lung adenocarcinoma and a chest ct should be obtained for further characterization.
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no acute cardiopulmonary process.
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overall stable mild bilateral pulmonary edema.
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interstitial lung disease with small bilateral effusions and bibasal compressive atelectasis. stable bony deformities.
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right lower lung partially calcified mass lesion does not appear to have progressed since pet-ct from <unk>, based on single frontal chest x-ray. no superimposed acute cardiopulmonary process.
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no acute cardiopulmonary abnormality.
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no substantial interval change from prior with no acute cardiopulmonary process. chronic blunting of the right costophrenic angle and right lateral pleural thickening, likely related to the patient's history of pleurodesis and pleural effusion.
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no acute cardiopulmonary process.
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increased pulmonary edema, especially in the lung bases and new left small pleural effusion. interval increase of heart size might be due increased pericardial effusion. echocardiography is recommended. finidngs were paged at <time> pm to dr <unk>, by dr <unk>
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no acute cardiopulmonary process.
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new right ij line with tip projecting over the mid svc. no pneumothorax or other change.
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probable mild lower lobe atelectasis. please note evaluation is limited due to underpenetration.
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left axillary pacemaker leads terminate in the expected positions. no complications.
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trace left pneumothorax. bilateral thoracostomy tubes are appropriately positioned.
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no evidence of acute cardiopulmonary process.
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suspected background copd. no focal infiltrate to suggest pneumonia or aspiration. no free air seen beneath the diaphragm.
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limited, negative.
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no acute cardiopulmonary process.
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no evidence of significant cardiopulmonary abnormality.
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no acute cardiopulmonary process.
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worsening cardiogenic pulmonary edema. low-lying endotracheal tube, which could be retracted <num> cm to prevent inadvertent intubation of the right main stem bronchus.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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large left pleural effusion with overlying atelectasis, underlying consolidation not excluded. left-sided hemithorax opacity appears slightly increased in extent as compared to the prior study. possible small right pleural effusion. persistent enlargement of the cardiomediastinal silhouette. central pulmonary vascular ...
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no acute cardiopulmonary process. no pneumothorax.
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no acute cardiopulmonary process.
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chronic changes within the lung apices compatible with prior granulomatous disease. no new areas of focal consolidation to suggest pneumonia. no pneumothorax.
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tracheostomy tube projects over the superior mediastinum. no significant change in the overall radiographic appearance since <unk>.
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large right pleural effusion, worsened
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mild pulmonary edema.
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opacity at the left lung base may reflect atelectasis or alternatively scarring. in the appropriate clinical setting, early pneumonia cannot be excluded.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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normal chest.
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left chest tube in place without evidence of residual pneumothorax.
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essentially normal chest radiograph with no evidence of acute pulmonary process.
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mild increase in the right lower lobe infarct likely due to subsequent hemorrhage. mild cardiomegaly.
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right basilar opacity as above which could represent pneumonia in the proper clinical setting.
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moderate bilateral pleural effusions with overlying atelectasis, appear slightly increased as compared to the prior study.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18634175/s51261016/f56690e6-18f434fc-180014e3-ace29a3c-edcf428a.jpg
no acute cardiopulmonary process.
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the right-sided chest tube has been removed no pneumothorax. the lung volumes are very low with increasing basal atelectasis. the right hilar opacity also appears more prominent could be related to postoperative changes and should be followed up on subsequent imaging. mild pulmonary vascular congestion is new.
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low lung volumes with bibasilar atelectasis and minimal blunting of the left posterior costophrenic angle. no chf. doubt frank consolidation.
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<num>. hyperinflated lungs. <num>. no radiographic evidence of interstitial lung disease. <num>. potential pulmonary hypertension, please correlate with echocardiography.
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interval worsening of moderate pulmonary edema. small left pleural effusion.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17710225/s52808186/5830bfed-3f6b263d-f4d7ffea-6a33cac3-054ae7fe.jpg
no evidence of pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12315463/s51269584/1a7a4fe7-9bc909d3-b5215cc3-1c979417-3c1cc579.jpg
no acute cardiopulmonary radiographic abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13174990/s53179858/dc68e357-f45ecfba-d49b84e3-f183aa4a-0ff610b6.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19152674/s53941550/7d377307-8843f5a0-f90747c4-046b1eff-cadde41a.jpg
minimal pulmonary vascular congestion. no focal consolidation to suggest pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18001523/s58950691/6713bb84-86f94bc9-68794345-7b90614e-d82f55af.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12972442/s53627209/8860e1cb-a5fad957-2fae7b9c-b260967e-1aae95d1.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12997545/s56076335/869a2c56-0d0c48df-fa8335a4-7bd86077-dfa6a93d.jpg
no evidence of pneumonia.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p15184801/s54771566/4aea34c3-ec6816c6-b7ec263e-3a137f92-a191eef2.jpg
low lung volumes with bibasilar atelectasis, left greater than right.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19495580/s57218837/7e338b3f-bb49cb70-7991111a-822fe04f-fd6ad011.jpg
no acute intrathoracic process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18662708/s57752199/9a959ad8-975bc591-27e22fb6-3243eab4-665c1127.jpg
persistent moderate cardiomegaly of mild pulmonary vascular congestion. chronically elevated right hemidiaphragm with mild right basilar atelectasis.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10470244/s52010569/0dde0bd3-2c172058-81f0859a-6d1af934-87af4d33.jpg
no acute cardiopulmonary process.