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/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p10752847/s56568141/5f9f962b-f8aca094-9ed407fc-d7533262-025247ed.jpg
cardiomegaly has worsened. worsened bilateral pulmonary opacities, likely edema.
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no acute cardiopulmonary process.
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<num>. no focal consolidation, cardiomegaly, or pulmonary edema. <num>. streaky left basilar atelectasis.
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patchy bibasilar airspace opacities, possibly atelectasis though infection is not completely excluded. mild pulmonary vascular congestion with small bilateral pleural effusions.
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no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p18408724/s55257086/7d544da9-1eaa47bd-8d419a0f-de11bcd6-1e177b48.jpg
no acute cardiopulmonary process. sclerotic focus projecting over the left humeral head and calcific focus projecting over the left axilla of unclear etiology but may represent nonspecific soft tissue calcification. the sclerotic focus projecting over the left humeral head could potentially alternatively represent a bo...
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intervally increased reticular markings compatible with fibrosis with known history of uip. no focal consolidation concerning for pneumonia.
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as above.
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low lung volumes.
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no acute cardiopulmonary process.
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interval removal of the left picc and placement of a right picc, which terminates within the right atrium. the right picc can be pulled back approximately <num> cm to terminate at the cavoatrial junction.
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lung volumes have improved and there streaky bibasilar opacities which may reflect scarring or subsegmental atelectasis but without significant change since <unk>. there is blunting of both posterior costophrenic angles consistent with tiny bilateral effusions or chronic pleural thickening. the bones are osteopenic and...
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a moderate right and a small left pleural effusion are increased in size in comparison to <unk>.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p17092587/s53527460/a71a3ec0-6833e60a-024039ed-2247b29a-ebf17a6f.jpg
no acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p12671607/s59947472/a95ef342-321863df-a684bf66-ab6a513d-40bb7bb8.jpg
no signs of pneumonia.
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status post pacemaker placement with no evidence of pneumothorax.
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<num>. no focal consolidation concerning for pneumonia. <num>. decreased lung volumes with bibasilar atelectasis greater on the left.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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enlarged cardiac silhouette. no overt pulmonary edema is seen. patchy right base opacity could relate to overlap of structures although consolidation due to pneumonia is not excluded in the appropriate clinical setting.
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mild cardiomegaly and pulmonary vascular congestion.
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very mild peribronchial cuffing. because this finding is minimal in this patient, it may be within normal limits, but mild small airways disease cannot be excluded.
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<num>. unchanged mild-to-moderate interstitial pulmonary edema. <num>. small bilateral pleural effusions, unchanged. <num>. subsegmental bibasilar atelectasis, as before.
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no evidence of acute disease.
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persistent left basilar opacity may reflect either pneumonia or aspiration.
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no evidence for acute cardiopulmonary process.
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no change to large layering left pleural effusion and probable small to moderate right pleural effusion. no pulmonary edema.
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no acute cardiopulmonary process. dilated bowel loops likely related to ileus.
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no acute cardiopulmonary abnormality. mild compression deformity of a midthoracic vertebral body.
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there is coalescence of opacity in the left lower lung, concerning for progressive pneumonia.
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bibasal atelectasis but no evidence of pneumonia.
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surgical clips are noted in the right upper quadrant. no significant interval change.
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mild cardiomegaly. no features of cardiac decompensation. no airspace consolidation to suggest pneumonia. this preliminary report was reviewed with dr. <unk>, <unk> radiologist.
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findings compatible with moderate congestive heart failure. follow is recommended after treatment to evaluate for underlying pneumonia. preliminary finding of "chf and right middle lobe pneumonia" was discussed with dr. <unk> by phone at <time>am <unk>. the change in the final read was discussed with dr. <unk> by phone...
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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minimal left basilar atelectasis. no radiopaque foreign bodies.
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no acute cardiopulmonary process.
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no acute intrathoracic process. no displaced rib fractures identified.
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no acute cardiopulmonary process. radiopaque densities in the region of the mid to distal esophagus and stomach which may correlate with patient's ph probe placement.
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bibasilar subsegmental atelectasis with otherwise clear lungs. new trace bilateral pleural effusions.
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stable chest findings with moderate cardiac enlargement. no evidence of acute pulmonary congestion. no evidence of new acute pulmonary infiltrates.
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significant worsening of the left pleural effusion with associated severe left lower lobe atelectasis. vascular congestion and interstitial pulmonary edema not significantly worsened from the previous exam.
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worsening severe bibasilar atelectasis. please take note of the high ett position. probable pleural effusions.
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no acute intrathoracic process.
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low lung volumes with new left lower lobe atelectasis and/or pneumonia with a small left pleural effusion. compared to <unk> there is a resolution of right upper lobe opacities.
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no acute pneumonia.
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unchanged moderate to severe cardiomegaly with moderate pulmonary edema.
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no acute cardiopulmonary process.
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<num>. increased mild pulmonary edema. <num>. right lower lung consolidation could represent atelectasis, but infection cannot be excluded. <num>. increased right pleural effusion.
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hyperinflation without acute cardiopulmonary process.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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<num>. improved pulmonary vascular congestion. no over pulmonary edema. <num>. persistent left lower lobe atelectasis. <num>. small bilateral pleural effusions.
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no acute cardiopulmonary process.
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no acute cardiopulmonary process.
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well inflated clear lungs with mild vascular prominence in both lower lobes.
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fluid overload superimposed on chronic lung disease. an underlying infectious infiltrate particularly in the left lower lobe cannot be excluded.
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no focal consolidation. top normal to mildly enlarged cardiac silhouette, appears slightly larger as compared to the prior study, although this may relate to differences in inspiration/technique.
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<num>. no evidence of pneumonia. <num>. stable enlargement of the cardiomediastinal silhouette status post cabg.
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no acute cardiopulmonary process.
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no significant interval change.
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no pneumonia.
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worsening multifocal pneumonia accompanied by enlarging pleural effusions, left greater than right.
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no acute abnormalities, no free air below the right hemidiaphragm.
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right apical pleural plaque. no evidence of acute cardiopulmonary processes.
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patchy left lower lobe opacity likely atelectasis. innumerable pulmonary metastases, relatively unchanged.
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<num>. mild pulmonary edema. <num>. interval increase in small bilateral pleural effusions. <num>. mild bibasilar atelectasis. superimposed infection cannot be excluded.
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no acute intrathoracic process.
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stable cardiomegaly. no signs of pneumonia.
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right mainstem intubation.
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<num>. no appreciable pneumothorax after left chest tube removal. <num>. persistent low lung volumes with bibasilar atelectasis and mild pulmonary edema.
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no acute cardiopulmonary process. these findings were discussed with <unk> in the office of dr. <unk> by dr. <unk> at <time> on <unk> by telephone at the time of discovery.
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no acute cardiopulmonary abnormality or evidence of pneumonia
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low lung volumes without evidence of pneumonia.
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no acute intrathoracic abnormality.
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increasing left lower lobe opacity, although similar to the earlier of two studies. recurrence of pneumonia versus waxing and waning atelectasis could be considered. findings also suggest very mild fluid overload.
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mild interstitial pulmonary edema.
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<num>. right lower lobe pneumonia with superimposed mild pulmonary edema. <num>. thickening of the soft tissues posterior to the trachea, concerning for esophagitis.
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findings suggest ards.
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patchy lingular and left lower lobe opacities, which could be seen with atelectasis, although a mild component of aspiration is difficult to exclude.
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no acute cardiopulmonary process.
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increased bibasilar opacities most likely due to combination of pleural effusions and atelectasis although underlying consolidation not excluded. pulmonary edema.
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moderate cardiomegaly, not significantly changed compared with prior studies. no acute cardiopulmonary process.
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unremarkable chest radiographic examination. supporting devices in appropriate position.
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minimal bibasilar atelectasis. otherwise, no acute cardiopulmonary process.
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stable minimal blunting of the posterior costophrenic angles may be due to trace pleural effusions. no significant interval change.
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cardiomegaly, probable mild hilar engorgement.
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no acute cardiopulmonary process.
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stable right apical pneumothorax. no significant changes compared to the prior radiograph.
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severe pulmonary edema. small bilateral pleural effusions.
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no acute cardiopulmonary process.
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right lower lobe cavitary lesion is most suspicious for cavitary pneumonia. follow-up radiograph after treatment is recommended to ensure resolution.
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no acute cardiopulmonary process.
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no acute intrathoracic process. trace fluid on the minor fissure.
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no acute intrathoracic abnormality.
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mild congestion without overt edema.
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no acute cardiopulmonary process.