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mild bibasilar atelectasis. possible mild pulmonary vascular congestion.
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left picc ends in the upper svc. otherwise, unchanged compared to <unk> at <time> a.m.
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<num>. unchanged port-a-cath tip, terminating in the low svc. <num>. incompletely characterized right proximal humerus lesion, corresponding to known langerhans cell histiocytosis.
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atelectasis and small pleural effusions are seen bilaterally.
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no acute cardiopulmonary abnormality.
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no pneumonia.
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no acute cardiopulmonary abnormality.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p19365165/s52403002/af9e6cbb-f28175f3-573e4c87-2337c3c5-77b5fed8.jpg
small bilateral pleural effusions with overlying atelectasis, greater on the left. incompletely evaluated gaseous distention of multiple upper abdominal bowel loops.
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stable mediastinal contour which is not widened.
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no evidence of acute cardiopulmonary process.
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no evidence of acute cardiopulmonary process.
/mnt/data/chayan/MIMIC-CXR-JPG/2.0.0/files/p13035993/s51744321/94635fc5-9895b266-92a66fe7-b126d655-8dd49332.jpg
no acute intrathoracic process.
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minor bibasilar atelectasis and possible small pleural effusions.
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mild pulmonary edema with no evidence of pneumonia.
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right peribronchial opacity may be attributed to a central airway infection such as a bronchopnuemonia. however, if symptoms persist, repeat radiographs should be performed with shallow oblique views to reassess the finding. this recommendation was communicated with the ed <unk> nurse team via email at <unk> on <unk>
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no acute cardiopulmonary process.
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trace right pleural effusion. subtle opacity at the left lower lung may represent overlap of structures or focal pneumonia.
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as above.
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interval worsening.
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appropriate positioning of right picc, ending in the low svc. no acute cardiac or pulmonary process.
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no acute intrathoracic process.
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small right-sided pleural effusion.
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no acute cardiopulmonary process identified.
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no acute cardiopulmonary process.
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<num>. right perihilar opacity, concerning for pneumonia. <num>. additional ground-glass opacities throughout the lungs bilaterally, which may reflect pulmonary edema, or additional foci of infection. <num>. bilateral pleural effusions, right greater than left. s
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<num>. substantially improved mild interstitial pulmonary edema. slightly decreased mild-to-moderate cardiomegaly. <num>. possible small right pleural effusion, not significantly changed.
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<num>. interval removal of the right chest tube without evidence of increasing pneumothorax. <num>. all other lines and tubes unchanged in positioning.
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no acute cardiopulmonary process.
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no acute intrathoracic process.
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<num>. increased opacification posterior to the heart could represent loculated effusion in the major fissure. however, if further imaging is required, ct of the chest is recommended.
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patchy bibasilar airspace opacities, more pronounced on the left, concerning for multifocal pneumonia.
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area of opacification in the right lower lobe is concerning for pneumonia. these findings do not confirm nor exclude the presence of a pulmonary embolism, and if pulmonary embolism is suspected, cta of the chest is appropriate.
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cardiomegaly with moderate edema and small bilateral pleural effusions.
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mild decrease in pulmonary vascular congestion.
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increased opacity projecting over the heart on lateral view, potentially prominent fat pad at the right cardiophrenic angle. given that this finding is more conspicuous compared to most recent exam and is new from more remote prior, chest ct is suggested to more for fully evaluate.
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no acute intrathoracic process.
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worsening cardiomegaly, with vascular congestion small left pleural effusion. no overt pulmonary edema. no pneumonia.
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normal radiographs of the chest.
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no focal consolidation. pulmonary nodules and bronchial wall thickening are better demonstrated on the same-day chest ct.
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subtle left lower lung opacity, which could represent atelectasis or pneumonia.
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<num>. increased size of large left pleural effusions from <unk> is expected status post left pneumonectomy. <num>. decreased size of small right pleural effusion with resolution of right lower lobe opacity from <unk>. <num>. resolved subcutaneous emphysema.
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effusion, edema and bibasilar opacities have significantly improved as the lung volume are more expanded lungs. mild interstitial edema remains.
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<num>. no acute intrathoracic process. <num>. ett tip is too high - should be advanced <num>-<num> cm to avoid inadvertant extubation. <num>. note that chest radiograph is sub optimal for evaluation of chest wall trauma / rib fractures.
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post treatment changes in the right lung with associated right middle lobe atelectasis as seen on prior. more conspicuous left basilar opacity potentially due to atelectasis however new region of consolidation is possible. two-view chest x-ray may help further characterize.
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<num>. no pneumoperitoneum. <num>. no acute cardiopulmonary process.
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interval resolution of loculated pneumothorax. slight interval increase in moderate subcutaneous emphysema. the patient was assessed asymptomatic and saturating well on room air. the patient was notified of the subcutaneous air and given instructions for symptoms to look out for and when to return to the hospital.
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there are stable left perihilar and suprahilar hilar opacities with associated volume loss in the left lung consistent with post treatment changes for lung cancer. lungs remain hyperinflated with changes suggestive of emphysema. no developing airspace consolidation is appreciated. there may be a small left effusion ver...
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as above.
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no free air under the diaphragm. unremarkable chest radiograph.
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no acute findings.
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left pleural effusion, cardiomegaly, pulmonary edema. vague asymmetric right midlung opacity may represent focal infection versus scarring.
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well placed left-sided picc line, no evidence of pneumothorax or any other placement-related complication.
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<num>. enlargement of cardiac silhouette, which appears to have increased compared to <unk>. <num>. mild-to-moderate pulmonary edema and a small right pleural effusion.
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no evidence of acute cardiopulmonary process.
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no evidence of acute cardiopulmonary disease.
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hyperinflated lungs. no focal consolidation to suggest pneumonia. no pulmonary edema.
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<num>. no acute cardiac or pulmonary process. <num>. near-complete interval resolution of small loculated left pleural effusion.
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no acute cardiopulmonary abnormality.
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no acute intrathoracic process.
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no acute cardiopulmonary process.
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moderate cardiomegaly. no acute infection.
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blunting of right costophrenic sulcus favors pleural thickening in the setting of mild right sided volume loss. comparison to older radiographs would be helpful to document stability. if unavailable, short-term followup radiographs in <unk> weeks may be helpful to exclude an active process.
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no radiographic evidence of pulmonary vasculitis.
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no acute cardiopulmonary process. elevated right hemidiaphragm, similar in appearance to radiographs from <unk>. no evidence of a displaced rib fracture.
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normal chesst radiograph.
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<num>. appropriate positioning of the pacemaker leads without evidence of pneumothorax. <num>. mild pulmonary edema. <num>. probable small bilateral pleural effusions.
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no evidence of acute disease. similar enlargement of central pulmonary arteries.
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left-sided pacemaker with the tips in the right atrium and right ventricle. no pneumothorax.
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no significant clinical changes.
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left lower lobe pneumonia.
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right-sided port-a-cath terminates in the right atrium. no evidence of pneumothorax.
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<num>. et tube is in standard position <num> cm above the carina. <num>. diffuse nodular opacities are mildly improved from <unk> and again suggest disseminated infection, possibly septic emboli. <num>. a transesophageal tube ends in the proximal stomach. advancement <num> cm is recommended to ensure proper placement.
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interval resolution of the previously identified pulmonary edema.
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no acute intrathoracic process.
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no acute cardiopulmonary abnormality. diffuse distention of colonic loops of bowel in the upper abdomen.
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cortical irregularity involving the right seventh lateral rib may reflect a nondisplaced fracture. no acute cardiopulmonary abnormality.
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mild right middle lobe atelectasis.
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mild interstitial edema and small left effusion without superimposed consolidation.
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platelike atelectasis in the left lung base, no convincing signs of pneumonia.
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no acute cardiopulmonary process.
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limited negative.
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no acute intrathoracic process.
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probable retrocardiac opacity, which could represent atelectasis but would be concerning for pneumonia or aspiration in the correct clinical setting. possible small left pleural effusion.
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<num>. decrease in size of moderate left pleural effusion. <num>. stable small right pleural and loculated effusions. <num>. stable bibasilar atelectasis without new consolidation.
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no substantial change from prior.
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no definite acute cardiopulmonary process.
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resolution of the bilateral opacities with continued severe enlargement of the cardiac silhouette. differential includes cardiomyopathy and pericardial effusion.
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low lung volumes with bibasilar atelectasis and mild pulmonary vascular congestion.
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interval improvement of the bibasilar atelectasis with residual scarring at the bases. no evidence of a new pneumonia. large hiatal hernia.
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<num>. mild pulmonary vascular congestion, without overt pulmonary edema. <num>. tortuous and enlarged thoracic aorta, similar to prior studies on the frontal view.
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<num>. complete opacification of the left hemi thorax due to the presence of a large pleural effusion, pleural thickening, and multiple pleural masses, better depicted on previous ct. <num>. mild right basilar atelectasis. no radiographic evidence of pneumonia.
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interval improvement of the right basilar opacification compared to the prior exam. the et tube terminates <num> cm from the carina.
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no acute cardiopulmonary abnormality.
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findings suggest pneumonia in the left lung, probably in the left lower lobe. follow-up radiographs are suggested to show resolution within <unk> weeks.
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no evidence of acute cardiopulmonary process.
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no acute findings in the chest.
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no acute cardiopulmonary process.
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marked cardiomegaly with mild pulmonary edema.
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no definite acute cardiopulmonary process.
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port-a-cath tip ends at the cavoatrial junction.