gem_id stringlengths 20 25 | id stringlengths 24 24 | title stringlengths 3 59 | context stringlengths 151 3.71k | question stringlengths 1 270 | target stringlengths 1 270 | references list | answers dict |
|---|---|---|---|---|---|---|---|
gem-squad_v2-train-9400 | 5a6b9825a9e0c9001a4e9f93 | Myocardial_infarction | Tobacco smoking (including secondhand smoke) and short-term exposure to air pollution such as carbon monoxide, nitrogen dioxide, and sulfur dioxide (but not ozone) have been associated with MI. Other factors that increase the risk of MI and are associated with worse outcomes after an MI include lack of physical activity and psychosocial factors including low socioeconomic status, social isolation, and negative emotions. Shift work is also associated with a higher risk of MI. Acute and prolonged intake of high quantities of alcoholic drinks (3-4 or more) increase the risk of a heart attack. | How many cigarettes per day increase the risk of a heart attack? | How many cigarettes per day increase the risk of a heart attack? | [
"How many cigarettes per day increase the risk of a heart attack?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9401 | 5a6b9825a9e0c9001a4e9f94 | Myocardial_infarction | Tobacco smoking (including secondhand smoke) and short-term exposure to air pollution such as carbon monoxide, nitrogen dioxide, and sulfur dioxide (but not ozone) have been associated with MI. Other factors that increase the risk of MI and are associated with worse outcomes after an MI include lack of physical activity and psychosocial factors including low socioeconomic status, social isolation, and negative emotions. Shift work is also associated with a higher risk of MI. Acute and prolonged intake of high quantities of alcoholic drinks (3-4 or more) increase the risk of a heart attack. | Increased physical activity is bad for what? | Increased physical activity is bad for what? | [
"Increased physical activity is bad for what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9402 | 5a6b9825a9e0c9001a4e9f95 | Myocardial_infarction | Tobacco smoking (including secondhand smoke) and short-term exposure to air pollution such as carbon monoxide, nitrogen dioxide, and sulfur dioxide (but not ozone) have been associated with MI. Other factors that increase the risk of MI and are associated with worse outcomes after an MI include lack of physical activity and psychosocial factors including low socioeconomic status, social isolation, and negative emotions. Shift work is also associated with a higher risk of MI. Acute and prolonged intake of high quantities of alcoholic drinks (3-4 or more) increase the risk of a heart attack. | Short term exposure to 3-4 servings of what is dangerous? | Short term exposure to 3-4 servings of what is dangerous? | [
"Short term exposure to 3-4 servings of what is dangerous?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9403 | 5a6b98aca9e0c9001a4e9f9a | Myocardial_infarction | The evidence for saturated fat is unclear. Some state there is evidence of benefit from reducing saturated fat, specifically a benefit from eating polyunsaturated fat instead of saturated fat. While others state there is little evidence that reducing dietary saturated fat or increasing polyunsaturated fat intake affects heart attack risk. Dietary cholesterol does not appear to have a significant effect on blood cholesterol and thus recommendations about its consumption may not be needed. Trans fats do appear to increase risk. | Evidence for what product has proven to be unequivocally dangerous? | Evidence for what product has proven to be unequivocally dangerous? | [
"Evidence for what product has proven to be unequivocally dangerous?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9404 | 5a6b98aca9e0c9001a4e9f9b | Myocardial_infarction | The evidence for saturated fat is unclear. Some state there is evidence of benefit from reducing saturated fat, specifically a benefit from eating polyunsaturated fat instead of saturated fat. While others state there is little evidence that reducing dietary saturated fat or increasing polyunsaturated fat intake affects heart attack risk. Dietary cholesterol does not appear to have a significant effect on blood cholesterol and thus recommendations about its consumption may not be needed. Trans fats do appear to increase risk. | Study show you should replace polyunsaturated fat with what? | Study show you should replace polyunsaturated fat with what? | [
"Study show you should replace polyunsaturated fat with what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9405 | 5a6b98aca9e0c9001a4e9f9c | Myocardial_infarction | The evidence for saturated fat is unclear. Some state there is evidence of benefit from reducing saturated fat, specifically a benefit from eating polyunsaturated fat instead of saturated fat. While others state there is little evidence that reducing dietary saturated fat or increasing polyunsaturated fat intake affects heart attack risk. Dietary cholesterol does not appear to have a significant effect on blood cholesterol and thus recommendations about its consumption may not be needed. Trans fats do appear to increase risk. | Blood cholesterol has a significant impact on what? | Blood cholesterol has a significant impact on what? | [
"Blood cholesterol has a significant impact on what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9406 | 5a6b98aca9e0c9001a4e9f9d | Myocardial_infarction | The evidence for saturated fat is unclear. Some state there is evidence of benefit from reducing saturated fat, specifically a benefit from eating polyunsaturated fat instead of saturated fat. While others state there is little evidence that reducing dietary saturated fat or increasing polyunsaturated fat intake affects heart attack risk. Dietary cholesterol does not appear to have a significant effect on blood cholesterol and thus recommendations about its consumption may not be needed. Trans fats do appear to increase risk. | What type of cholesterol requires careful limitations? | What type of cholesterol requires careful limitations? | [
"What type of cholesterol requires careful limitations?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9407 | 5a6b98aca9e0c9001a4e9f9e | Myocardial_infarction | The evidence for saturated fat is unclear. Some state there is evidence of benefit from reducing saturated fat, specifically a benefit from eating polyunsaturated fat instead of saturated fat. While others state there is little evidence that reducing dietary saturated fat or increasing polyunsaturated fat intake affects heart attack risk. Dietary cholesterol does not appear to have a significant effect on blood cholesterol and thus recommendations about its consumption may not be needed. Trans fats do appear to increase risk. | Trans what decreases risk? | Trans what decreases risk? | [
"Trans what decreases risk?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9408 | 5a6b99eea9e0c9001a4e9fb8 | Myocardial_infarction | Genome-wide association studies have found 27 genetic variants that are associated with an increased risk of myocardial infarction. Strongest association of MI has been found with the 9p21 genomic locus, which contains genes CDKN2A & 2B, although the single nucleotide polymorphisms that are implicated are within a non-coding region. The majority of these variants are in regions that have not been previously implicated in coronary artery disease. The following genes have an association with MI: PCSK9, SORT1, MIA3, WDR12, MRAS, PHACTR1, LPA, TCF21, MTHFDSL, ZC3HC1, CDKN2A, 2B, ABO, PDGF0, APOA5, MNF1ASM283, COL4A1, HHIPC1, SMAD3, ADAMTS7, RAS1, SMG6, SNF8, LDLR, SLC5A3, MRPS6, KCNE2. | How many genetic variants are shown to decrease risk of an MI? | How many genetic variants are shown to decrease risk of an MI? | [
"How many genetic variants are shown to decrease risk of an MI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9409 | 5a6b99eea9e0c9001a4e9fb9 | Myocardial_infarction | Genome-wide association studies have found 27 genetic variants that are associated with an increased risk of myocardial infarction. Strongest association of MI has been found with the 9p21 genomic locus, which contains genes CDKN2A & 2B, although the single nucleotide polymorphisms that are implicated are within a non-coding region. The majority of these variants are in regions that have not been previously implicated in coronary artery disease. The following genes have an association with MI: PCSK9, SORT1, MIA3, WDR12, MRAS, PHACTR1, LPA, TCF21, MTHFDSL, ZC3HC1, CDKN2A, 2B, ABO, PDGF0, APOA5, MNF1ASM283, COL4A1, HHIPC1, SMAD3, ADAMTS7, RAS1, SMG6, SNF8, LDLR, SLC5A3, MRPS6, KCNE2. | PCSK9 is found in what locus? | PCSK9 is found in what locus? | [
"PCSK9 is found in what locus?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9410 | 5a6b99eea9e0c9001a4e9fba | Myocardial_infarction | Genome-wide association studies have found 27 genetic variants that are associated with an increased risk of myocardial infarction. Strongest association of MI has been found with the 9p21 genomic locus, which contains genes CDKN2A & 2B, although the single nucleotide polymorphisms that are implicated are within a non-coding region. The majority of these variants are in regions that have not been previously implicated in coronary artery disease. The following genes have an association with MI: PCSK9, SORT1, MIA3, WDR12, MRAS, PHACTR1, LPA, TCF21, MTHFDSL, ZC3HC1, CDKN2A, 2B, ABO, PDGF0, APOA5, MNF1ASM283, COL4A1, HHIPC1, SMAD3, ADAMTS7, RAS1, SMG6, SNF8, LDLR, SLC5A3, MRPS6, KCNE2. | How many genetic variants are in the 9p21 genomic locus? | How many genetic variants are in the 9p21 genomic locus? | [
"How many genetic variants are in the 9p21 genomic locus?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9411 | 5a6b99eea9e0c9001a4e9fbb | Myocardial_infarction | Genome-wide association studies have found 27 genetic variants that are associated with an increased risk of myocardial infarction. Strongest association of MI has been found with the 9p21 genomic locus, which contains genes CDKN2A & 2B, although the single nucleotide polymorphisms that are implicated are within a non-coding region. The majority of these variants are in regions that have not been previously implicated in coronary artery disease. The following genes have an association with MI: PCSK9, SORT1, MIA3, WDR12, MRAS, PHACTR1, LPA, TCF21, MTHFDSL, ZC3HC1, CDKN2A, 2B, ABO, PDGF0, APOA5, MNF1ASM283, COL4A1, HHIPC1, SMAD3, ADAMTS7, RAS1, SMG6, SNF8, LDLR, SLC5A3, MRPS6, KCNE2. | What was implicated in the coding region of 9p21? | What was implicated in the coding region of 9p21? | [
"What was implicated in the coding region of 9p21?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9412 | 5a6b99eea9e0c9001a4e9fbc | Myocardial_infarction | Genome-wide association studies have found 27 genetic variants that are associated with an increased risk of myocardial infarction. Strongest association of MI has been found with the 9p21 genomic locus, which contains genes CDKN2A & 2B, although the single nucleotide polymorphisms that are implicated are within a non-coding region. The majority of these variants are in regions that have not been previously implicated in coronary artery disease. The following genes have an association with MI: PCSK9, SORT1, MIA3, WDR12, MRAS, PHACTR1, LPA, TCF21, MTHFDSL, ZC3HC1, CDKN2A, 2B, ABO, PDGF0, APOA5, MNF1ASM283, COL4A1, HHIPC1, SMAD3, ADAMTS7, RAS1, SMG6, SNF8, LDLR, SLC5A3, MRPS6, KCNE2. | SORT1 is associated with decreasing risk of what? | SORT1 is associated with decreasing risk of what? | [
"SORT1 is associated with decreasing risk of what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9413 | 5a6b9bf84eec6b001a80a4a2 | Myocardial_infarction | Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated and ST-elevated MIs, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Bloodstream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote the formation of a blood clot that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary arteries, it leads to MI (necrosis of downstream myocardium). It is estimated that one billion cardiac cells are lost in a typical MI. | How many subtypes does ST-elevated MI have? | How many subtypes does ST-elevated MI have? | [
"How many subtypes does ST-elevated MI have?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9414 | 5a6b9bf84eec6b001a80a4a3 | Myocardial_infarction | Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated and ST-elevated MIs, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Bloodstream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote the formation of a blood clot that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary arteries, it leads to MI (necrosis of downstream myocardium). It is estimated that one billion cardiac cells are lost in a typical MI. | What are always a manifestation of artery disease? | What are always a manifestation of artery disease? | [
"What are always a manifestation of artery disease?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9415 | 5a6b9bf84eec6b001a80a4a4 | Myocardial_infarction | Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated and ST-elevated MIs, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Bloodstream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote the formation of a blood clot that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary arteries, it leads to MI (necrosis of downstream myocardium). It is estimated that one billion cardiac cells are lost in a typical MI. | How long does an MI take? | How long does an MI take? | [
"How long does an MI take?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9416 | 5a6b9bf84eec6b001a80a4a5 | Myocardial_infarction | Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated and ST-elevated MIs, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Bloodstream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote the formation of a blood clot that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary arteries, it leads to MI (necrosis of downstream myocardium). It is estimated that one billion cardiac cells are lost in a typical MI. | How many plaques become unstable in a typical MI? | How many plaques become unstable in a typical MI? | [
"How many plaques become unstable in a typical MI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9417 | 5a6b9bf84eec6b001a80a4a6 | Myocardial_infarction | Acute myocardial infarction refers to two subtypes of acute coronary syndrome, namely non-ST-elevated and ST-elevated MIs, which are most frequently (but not always) a manifestation of coronary artery disease. The most common triggering event is the disruption of an atherosclerotic plaque in an epicardial coronary artery, which leads to a clotting cascade, sometimes resulting in total occlusion of the artery. Atherosclerosis is the gradual buildup of cholesterol and fibrous tissue in plaques in the wall of arteries (in this case, the coronary arteries), typically over decades. Bloodstream column irregularities visible on angiography reflect artery lumen narrowing as a result of decades of advancing atherosclerosis. Plaques can become unstable, rupture, and additionally promote the formation of a blood clot that occludes the artery; this can occur in minutes. When a severe enough plaque rupture occurs in the coronary arteries, it leads to MI (necrosis of downstream myocardium). It is estimated that one billion cardiac cells are lost in a typical MI. | How long does an angiography take? | How long does an angiography take? | [
"How long does an angiography take?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9418 | 5a6bb05e4eec6b001a80a4b6 | Myocardial_infarction | If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in their place. Recent studies indicate that another form of cell death, apoptosis, also plays a role in the process of tissue damage following an MI. As a result, the person's heart will be permanently damaged. This myocardial scarring also puts the person at risk for potentially life-threatening abnormal heart rhythms (arrhythmias), and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. | What does necrosis trigger? | What does necrosis trigger? | [
"What does necrosis trigger?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9419 | 5a6bb05e4eec6b001a80a4b7 | Myocardial_infarction | If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in their place. Recent studies indicate that another form of cell death, apoptosis, also plays a role in the process of tissue damage following an MI. As a result, the person's heart will be permanently damaged. This myocardial scarring also puts the person at risk for potentially life-threatening abnormal heart rhythms (arrhythmias), and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. | What is a collagen scar called? | What is a collagen scar called? | [
"What is a collagen scar called?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9420 | 5a6bb05e4eec6b001a80a4b8 | Myocardial_infarction | If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in their place. Recent studies indicate that another form of cell death, apoptosis, also plays a role in the process of tissue damage following an MI. As a result, the person's heart will be permanently damaged. This myocardial scarring also puts the person at risk for potentially life-threatening abnormal heart rhythms (arrhythmias), and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. | What is another name for a ventricular aneurysm? | What is another name for a ventricular aneurysm? | [
"What is another name for a ventricular aneurysm?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9421 | 5a6bb05e4eec6b001a80a4b9 | Myocardial_infarction | If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in their place. Recent studies indicate that another form of cell death, apoptosis, also plays a role in the process of tissue damage following an MI. As a result, the person's heart will be permanently damaged. This myocardial scarring also puts the person at risk for potentially life-threatening abnormal heart rhythms (arrhythmias), and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. | apoptosis is the death of what artery? | apoptosis is the death of what artery? | [
"apoptosis is the death of what artery?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9422 | 5a6bb05e4eec6b001a80a4ba | Myocardial_infarction | If impaired blood flow to the heart lasts long enough, it triggers a process called the ischemic cascade; the heart cells in the territory of the occluded coronary artery die (chiefly through necrosis) and do not grow back. A collagen scar forms in their place. Recent studies indicate that another form of cell death, apoptosis, also plays a role in the process of tissue damage following an MI. As a result, the person's heart will be permanently damaged. This myocardial scarring also puts the person at risk for potentially life-threatening abnormal heart rhythms (arrhythmias), and may result in the formation of a ventricular aneurysm that can rupture with catastrophic consequences. | What is impaired blood flow called? | What is impaired blood flow called? | [
"What is impaired blood flow called?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9423 | 5a6bb13e4eec6b001a80a4c0 | Myocardial_infarction | Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-tach/VT), which can cause sudden cardiac death. However, VT usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. | Normal heart tissue is slower than what? | Normal heart tissue is slower than what? | [
"Normal heart tissue is slower than what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9424 | 5a6bb13e4eec6b001a80a4c1 | Myocardial_infarction | Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-tach/VT), which can cause sudden cardiac death. However, VT usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. | What is the name of the arrhythmia that is slower than normal? | What is the name of the arrhythmia that is slower than normal? | [
"What is the name of the arrhythmia that is slower than normal?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9425 | 5a6bb13e4eec6b001a80a4c2 | Myocardial_infarction | Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-tach/VT), which can cause sudden cardiac death. However, VT usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. | V-tach is the leading cause of what? | V-tach is the leading cause of what? | [
"V-tach is the leading cause of what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9426 | 5a6bb13e4eec6b001a80a4c3 | Myocardial_infarction | Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-tach/VT), which can cause sudden cardiac death. However, VT usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. | Electrical impulses trigger what? | Electrical impulses trigger what? | [
"Electrical impulses trigger what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9427 | 5a6bb13e4eec6b001a80a4c4 | Myocardial_infarction | Injured heart tissue conducts electrical impulses more slowly than normal heart tissue. The difference in conduction velocity between injured and uninjured tissue can trigger re-entry or a feedback loop that is believed to be the cause of many lethal arrhythmias. The most serious of these arrhythmias is ventricular fibrillation (V-Fib/VF), an extremely fast and chaotic heart rhythm that is the leading cause of sudden cardiac death. Another life-threatening arrhythmia is ventricular tachycardia (V-tach/VT), which can cause sudden cardiac death. However, VT usually results in rapid heart rates that prevent the heart from pumping blood effectively. Cardiac output and blood pressure may fall to dangerous levels, which can lead to further coronary ischemia and extension of the infarct. | A rising blood pressure can cause additional what? | A rising blood pressure can cause additional what? | [
"A rising blood pressure can cause additional what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9428 | 5a6bb1d34eec6b001a80a4d4 | Myocardial_infarction | Myocardial infarction in the setting of plaque results from underlying atherosclerosis. Inflammation is known to be an important step in the process of atherosclerotic plaque formation. C-reactive protein (CRP) is a sensitive but nonspecific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes. Moreover, some drugs for MI might also reduce CRP levels. The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in those who already present with other risk factors or known coronary artery disease. Whether CRP plays a direct role in atherosclerosis remains uncertain. | The setting of plaque is called what? | The setting of plaque is called what? | [
"The setting of plaque is called what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9429 | 5a6bb1d34eec6b001a80a4d5 | Myocardial_infarction | Myocardial infarction in the setting of plaque results from underlying atherosclerosis. Inflammation is known to be an important step in the process of atherosclerotic plaque formation. C-reactive protein (CRP) is a sensitive but nonspecific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes. Moreover, some drugs for MI might also reduce CRP levels. The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in those who already present with other risk factors or known coronary artery disease. Whether CRP plays a direct role in atherosclerosis remains uncertain. | CRP is known to play a direct role in what? | CRP is known to play a direct role in what? | [
"CRP is known to play a direct role in what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9430 | 5a6bb1d34eec6b001a80a4d6 | Myocardial_infarction | Myocardial infarction in the setting of plaque results from underlying atherosclerosis. Inflammation is known to be an important step in the process of atherosclerotic plaque formation. C-reactive protein (CRP) is a sensitive but nonspecific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes. Moreover, some drugs for MI might also reduce CRP levels. The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in those who already present with other risk factors or known coronary artery disease. Whether CRP plays a direct role in atherosclerosis remains uncertain. | What specific marker indicates inflammation? | What specific marker indicates inflammation? | [
"What specific marker indicates inflammation?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9431 | 5a6bb1d34eec6b001a80a4d7 | Myocardial_infarction | Myocardial infarction in the setting of plaque results from underlying atherosclerosis. Inflammation is known to be an important step in the process of atherosclerotic plaque formation. C-reactive protein (CRP) is a sensitive but nonspecific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes. Moreover, some drugs for MI might also reduce CRP levels. The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in those who already present with other risk factors or known coronary artery disease. Whether CRP plays a direct role in atherosclerosis remains uncertain. | Drugs for MI tend to raise what? | Drugs for MI tend to raise what? | [
"Drugs for MI tend to raise what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9432 | 5a6bb1d34eec6b001a80a4d8 | Myocardial_infarction | Myocardial infarction in the setting of plaque results from underlying atherosclerosis. Inflammation is known to be an important step in the process of atherosclerotic plaque formation. C-reactive protein (CRP) is a sensitive but nonspecific marker for inflammation. Elevated CRP blood levels, especially measured with high-sensitivity assays, can predict the risk of MI, as well as stroke and development of diabetes. Moreover, some drugs for MI might also reduce CRP levels. The use of high-sensitivity CRP assays as a means of screening the general population is advised against, but it may be used optionally at the physician's discretion in those who already present with other risk factors or known coronary artery disease. Whether CRP plays a direct role in atherosclerosis remains uncertain. | What type of screening is generally recommended? | What type of screening is generally recommended? | [
"What type of screening is generally recommended?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9433 | 5a6bb2904eec6b001a80a4de | Myocardial_infarction | For a person to qualify as having a STEMI, in addition to reported angina, the ECG must show new ST elevation in two or more adjacent ECG leads. This must be greater than 2 mm (0.2 mV) for males and greater than 1.5 mm (0.15 mV) in females if in leads V2 and V3 or greater than 1 mm (0.1 mV) if it is in other ECG leads. A left bundle branch block that is believed to be new used to be considered the same as ST elevation; however, this is no longer the case. In early STEMIs there may just be peaked T waves with ST elevation developing later. | How many anginas must a person report to have a STEMI? | How many anginas must a person report to have a STEMI? | [
"How many anginas must a person report to have a STEMI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9434 | 5a6bb2904eec6b001a80a4df | Myocardial_infarction | For a person to qualify as having a STEMI, in addition to reported angina, the ECG must show new ST elevation in two or more adjacent ECG leads. This must be greater than 2 mm (0.2 mV) for males and greater than 1.5 mm (0.15 mV) in females if in leads V2 and V3 or greater than 1 mm (0.1 mV) if it is in other ECG leads. A left bundle branch block that is believed to be new used to be considered the same as ST elevation; however, this is no longer the case. In early STEMIs there may just be peaked T waves with ST elevation developing later. | ST elevation can develop into what? | ST elevation can develop into what? | [
"ST elevation can develop into what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9435 | 5a6bb2904eec6b001a80a4e0 | Myocardial_infarction | For a person to qualify as having a STEMI, in addition to reported angina, the ECG must show new ST elevation in two or more adjacent ECG leads. This must be greater than 2 mm (0.2 mV) for males and greater than 1.5 mm (0.15 mV) in females if in leads V2 and V3 or greater than 1 mm (0.1 mV) if it is in other ECG leads. A left bundle branch block that is believed to be new used to be considered the same as ST elevation; however, this is no longer the case. In early STEMIs there may just be peaked T waves with ST elevation developing later. | The ECG must show less than how many mV for a male to be considered a STEMI? | The ECG must show less than how many mV for a male to be considered a STEMI? | [
"The ECG must show less than how many mV for a male to be considered a STEMI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9436 | 5a6bb2904eec6b001a80a4e1 | Myocardial_infarction | For a person to qualify as having a STEMI, in addition to reported angina, the ECG must show new ST elevation in two or more adjacent ECG leads. This must be greater than 2 mm (0.2 mV) for males and greater than 1.5 mm (0.15 mV) in females if in leads V2 and V3 or greater than 1 mm (0.1 mV) if it is in other ECG leads. A left bundle branch block that is believed to be new used to be considered the same as ST elevation; however, this is no longer the case. In early STEMIs there may just be peaked T waves with ST elevation developing later. | What has recently been added as a qualification for a STEMI? | What has recently been added as a qualification for a STEMI? | [
"What has recently been added as a qualification for a STEMI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9437 | 5a6bb2904eec6b001a80a4e2 | Myocardial_infarction | For a person to qualify as having a STEMI, in addition to reported angina, the ECG must show new ST elevation in two or more adjacent ECG leads. This must be greater than 2 mm (0.2 mV) for males and greater than 1.5 mm (0.15 mV) in females if in leads V2 and V3 or greater than 1 mm (0.1 mV) if it is in other ECG leads. A left bundle branch block that is believed to be new used to be considered the same as ST elevation; however, this is no longer the case. In early STEMIs there may just be peaked T waves with ST elevation developing later. | Which two named leads only have to be above 1 mm to be counted? | Which two named leads only have to be above 1 mm to be counted? | [
"Which two named leads only have to be above 1 mm to be counted?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9438 | 5a6bb3444eec6b001a80a4e8 | Myocardial_infarction | In stable patients whose symptoms have resolved by the time of evaluation, technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiological or pharmacological stress. Thallium may also be used to determine viability of tissue, distinguishing whether nonfunctional myocardium is actually dead or merely in a state of hibernation or of being stunned. Medical societies and professional guidelines recommend that the physician confirm a person is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis. Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging. Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG, and cardiac biomarkers suggest the likelihood of a problem. | What is used to examine blood flood in unstable patients? | What is used to examine blood flood in unstable patients? | [
"What is used to examine blood flood in unstable patients?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9439 | 5a6bb3444eec6b001a80a4e9 | Myocardial_infarction | In stable patients whose symptoms have resolved by the time of evaluation, technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiological or pharmacological stress. Thallium may also be used to determine viability of tissue, distinguishing whether nonfunctional myocardium is actually dead or merely in a state of hibernation or of being stunned. Medical societies and professional guidelines recommend that the physician confirm a person is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis. Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging. Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG, and cardiac biomarkers suggest the likelihood of a problem. | What is another name for thallium-201? | What is another name for thallium-201? | [
"What is another name for thallium-201?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9440 | 5a6bb3444eec6b001a80a4ea | Myocardial_infarction | In stable patients whose symptoms have resolved by the time of evaluation, technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiological or pharmacological stress. Thallium may also be used to determine viability of tissue, distinguishing whether nonfunctional myocardium is actually dead or merely in a state of hibernation or of being stunned. Medical societies and professional guidelines recommend that the physician confirm a person is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis. Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging. Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG, and cardiac biomarkers suggest the likelihood of a problem. | What purpose does technetium serve in terms of identifying tissue? | What purpose does technetium serve in terms of identifying tissue? | [
"What purpose does technetium serve in terms of identifying tissue?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9441 | 5a6bb3444eec6b001a80a4eb | Myocardial_infarction | In stable patients whose symptoms have resolved by the time of evaluation, technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiological or pharmacological stress. Thallium may also be used to determine viability of tissue, distinguishing whether nonfunctional myocardium is actually dead or merely in a state of hibernation or of being stunned. Medical societies and professional guidelines recommend that the physician confirm a person is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis. Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging. Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG, and cardiac biomarkers suggest the likelihood of a problem. | What must be determined after taking an imaging test? | What must be determined after taking an imaging test? | [
"What must be determined after taking an imaging test?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9442 | 5a6bb3444eec6b001a80a4ec | Myocardial_infarction | In stable patients whose symptoms have resolved by the time of evaluation, technetium (99mTc) sestamibi (i.e. a "MIBI scan") or thallium-201 chloride can be used in nuclear medicine to visualize areas of reduced blood flow in conjunction with physiological or pharmacological stress. Thallium may also be used to determine viability of tissue, distinguishing whether nonfunctional myocardium is actually dead or merely in a state of hibernation or of being stunned. Medical societies and professional guidelines recommend that the physician confirm a person is at high risk for myocardial infarction before conducting imaging tests to make a diagnosis. Patients who have a normal ECG and who are able to exercise, for example, do not merit routine imaging. Imaging tests such as stress radionuclide myocardial perfusion imaging or stress echocardiography can confirm a diagnosis when a patient's history, physical exam, ECG, and cardiac biomarkers suggest the likelihood of a problem. | A patient does not need routine imaging when they have what kind of biomarker? | A patient does not need routine imaging when they have what kind of biomarker? | [
"A patient does not need routine imaging when they have what kind of biomarker?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9443 | 5a6bb40d4eec6b001a80a4fc | Myocardial_infarction | There is some controversy surrounding the effect of dietary fat on the development of cardiovascular disease. People are often advised to keep a diet where less than 30% of the energy intake derives from fat, a diet that contains less than 7% of the energy intake in the form of saturated fat, and a diet that contains less than 300 mg/day of cholesterol. Replacing saturated with mono- polyunsaturated fat is also recommended, as the consumption of polyunsaturated fat instead of saturated fat may decrease coronary heart disease. Olive oil, rapeseed oil and related products are to be used instead of saturated fat. | How many mg of fat is suggested for a healthy person's diet? | How many mg of fat is suggested for a healthy person's diet? | [
"How many mg of fat is suggested for a healthy person's diet?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9444 | 5a6bb40d4eec6b001a80a4fd | Myocardial_infarction | There is some controversy surrounding the effect of dietary fat on the development of cardiovascular disease. People are often advised to keep a diet where less than 30% of the energy intake derives from fat, a diet that contains less than 7% of the energy intake in the form of saturated fat, and a diet that contains less than 300 mg/day of cholesterol. Replacing saturated with mono- polyunsaturated fat is also recommended, as the consumption of polyunsaturated fat instead of saturated fat may decrease coronary heart disease. Olive oil, rapeseed oil and related products are to be used instead of saturated fat. | What percentage of mono-polyunsaturated fat should make up a person's daily energy requirements? | What percentage of mono-polyunsaturated fat should make up a person's daily energy requirements? | [
"What percentage of mono-polyunsaturated fat should make up a person's daily energy requirements?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9445 | 5a6bb40d4eec6b001a80a4fe | Myocardial_infarction | There is some controversy surrounding the effect of dietary fat on the development of cardiovascular disease. People are often advised to keep a diet where less than 30% of the energy intake derives from fat, a diet that contains less than 7% of the energy intake in the form of saturated fat, and a diet that contains less than 300 mg/day of cholesterol. Replacing saturated with mono- polyunsaturated fat is also recommended, as the consumption of polyunsaturated fat instead of saturated fat may decrease coronary heart disease. Olive oil, rapeseed oil and related products are to be used instead of saturated fat. | What is a good replacement for olive oil? | What is a good replacement for olive oil? | [
"What is a good replacement for olive oil?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9446 | 5a6bb40d4eec6b001a80a4ff | Myocardial_infarction | There is some controversy surrounding the effect of dietary fat on the development of cardiovascular disease. People are often advised to keep a diet where less than 30% of the energy intake derives from fat, a diet that contains less than 7% of the energy intake in the form of saturated fat, and a diet that contains less than 300 mg/day of cholesterol. Replacing saturated with mono- polyunsaturated fat is also recommended, as the consumption of polyunsaturated fat instead of saturated fat may decrease coronary heart disease. Olive oil, rapeseed oil and related products are to be used instead of saturated fat. | Nutritionists recommend an intake of more than 30% of what in one's diet? | Nutritionists recommend an intake of more than 30% of what in one's diet? | [
"Nutritionists recommend an intake of more than 30% of what in one's diet?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9447 | 5a6bb40d4eec6b001a80a500 | Myocardial_infarction | There is some controversy surrounding the effect of dietary fat on the development of cardiovascular disease. People are often advised to keep a diet where less than 30% of the energy intake derives from fat, a diet that contains less than 7% of the energy intake in the form of saturated fat, and a diet that contains less than 300 mg/day of cholesterol. Replacing saturated with mono- polyunsaturated fat is also recommended, as the consumption of polyunsaturated fat instead of saturated fat may decrease coronary heart disease. Olive oil, rapeseed oil and related products are to be used instead of saturated fat. | What controversy have dieticians largely settled? | What controversy have dieticians largely settled? | [
"What controversy have dieticians largely settled?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9448 | 5a6bb4fa4eec6b001a80a506 | Myocardial_infarction | Aspirin has been studied extensively in people considered at increased risk of myocardial infarction. Based on numerous studies in different groups (e.g. people with or without diabetes), there does not appear to be a benefit strong enough to outweigh the risk of excessive bleeding. Nevertheless, many clinical practice guidelines continue to recommend aspirin for primary prevention, and some researchers feel that those with very high cardiovascular risk but low risk of bleeding should continue to receive aspirin. | What drug has been shown to have significant benefits for people at risk of an MI? | What drug has been shown to have significant benefits for people at risk of an MI? | [
"What drug has been shown to have significant benefits for people at risk of an MI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9449 | 5a6bb4fa4eec6b001a80a507 | Myocardial_infarction | Aspirin has been studied extensively in people considered at increased risk of myocardial infarction. Based on numerous studies in different groups (e.g. people with or without diabetes), there does not appear to be a benefit strong enough to outweigh the risk of excessive bleeding. Nevertheless, many clinical practice guidelines continue to recommend aspirin for primary prevention, and some researchers feel that those with very high cardiovascular risk but low risk of bleeding should continue to receive aspirin. | Most studies tend to focus on people with what disease? | Most studies tend to focus on people with what disease? | [
"Most studies tend to focus on people with what disease?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9450 | 5a6bb4fa4eec6b001a80a508 | Myocardial_infarction | Aspirin has been studied extensively in people considered at increased risk of myocardial infarction. Based on numerous studies in different groups (e.g. people with or without diabetes), there does not appear to be a benefit strong enough to outweigh the risk of excessive bleeding. Nevertheless, many clinical practice guidelines continue to recommend aspirin for primary prevention, and some researchers feel that those with very high cardiovascular risk but low risk of bleeding should continue to receive aspirin. | People with diabetes have an increased likelihood of what side effect? | People with diabetes have an increased likelihood of what side effect? | [
"People with diabetes have an increased likelihood of what side effect?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9451 | 5a6bb4fa4eec6b001a80a509 | Myocardial_infarction | Aspirin has been studied extensively in people considered at increased risk of myocardial infarction. Based on numerous studies in different groups (e.g. people with or without diabetes), there does not appear to be a benefit strong enough to outweigh the risk of excessive bleeding. Nevertheless, many clinical practice guidelines continue to recommend aspirin for primary prevention, and some researchers feel that those with very high cardiovascular risk but low risk of bleeding should continue to receive aspirin. | Based on the lack of benefits, what has largely stopped recommending asprin? | Based on the lack of benefits, what has largely stopped recommending asprin? | [
"Based on the lack of benefits, what has largely stopped recommending asprin?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9452 | 5a6bb4fa4eec6b001a80a50a | Myocardial_infarction | Aspirin has been studied extensively in people considered at increased risk of myocardial infarction. Based on numerous studies in different groups (e.g. people with or without diabetes), there does not appear to be a benefit strong enough to outweigh the risk of excessive bleeding. Nevertheless, many clinical practice guidelines continue to recommend aspirin for primary prevention, and some researchers feel that those with very high cardiovascular risk but low risk of bleeding should continue to receive aspirin. | Aspirin is recommended for people with a high risk both cardiovascular disease and what? | Aspirin is recommended for people with a high risk both cardiovascular disease and what? | [
"Aspirin is recommended for people with a high risk both cardiovascular disease and what?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9453 | 5a6bb5ff4eec6b001a80a510 | Myocardial_infarction | The main treatment for MI with ECG evidence of ST elevation (STEMI) include thrombolysis and percutaneous coronary intervention. Primary percutaneous coronary intervention (PCI) is the treatment of choice for STEMI if it can be performed in a timely manner. If PCI cannot be performed within 90 to 120 minutes then thrombolysis, preferably within 30 minutes of arrival to hospital, is recommended. If a person has had symptoms for 12 to 24 hours evidence for thrombolysis is less and if they have had symptoms for more than 24 hours it is not recommended. | What is the shortened way of referring to thrombolysis? | What is the shortened way of referring to thrombolysis? | [
"What is the shortened way of referring to thrombolysis?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9454 | 5a6bb5ff4eec6b001a80a511 | Myocardial_infarction | The main treatment for MI with ECG evidence of ST elevation (STEMI) include thrombolysis and percutaneous coronary intervention. Primary percutaneous coronary intervention (PCI) is the treatment of choice for STEMI if it can be performed in a timely manner. If PCI cannot be performed within 90 to 120 minutes then thrombolysis, preferably within 30 minutes of arrival to hospital, is recommended. If a person has had symptoms for 12 to 24 hours evidence for thrombolysis is less and if they have had symptoms for more than 24 hours it is not recommended. | PCI and what treatment must be performed within 90 to 120 minutes? | PCI and what treatment must be performed within 90 to 120 minutes? | [
"PCI and what treatment must be performed within 90 to 120 minutes?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9455 | 5a6bb5ff4eec6b001a80a512 | Myocardial_infarction | The main treatment for MI with ECG evidence of ST elevation (STEMI) include thrombolysis and percutaneous coronary intervention. Primary percutaneous coronary intervention (PCI) is the treatment of choice for STEMI if it can be performed in a timely manner. If PCI cannot be performed within 90 to 120 minutes then thrombolysis, preferably within 30 minutes of arrival to hospital, is recommended. If a person has had symptoms for 12 to 24 hours evidence for thrombolysis is less and if they have had symptoms for more than 24 hours it is not recommended. | How long does the PCI procedure take? | How long does the PCI procedure take? | [
"How long does the PCI procedure take?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9456 | 5a6bb5ff4eec6b001a80a513 | Myocardial_infarction | The main treatment for MI with ECG evidence of ST elevation (STEMI) include thrombolysis and percutaneous coronary intervention. Primary percutaneous coronary intervention (PCI) is the treatment of choice for STEMI if it can be performed in a timely manner. If PCI cannot be performed within 90 to 120 minutes then thrombolysis, preferably within 30 minutes of arrival to hospital, is recommended. If a person has had symptoms for 12 to 24 hours evidence for thrombolysis is less and if they have had symptoms for more than 24 hours it is not recommended. | What treatment is recommended after 24 hours? | What treatment is recommended after 24 hours? | [
"What treatment is recommended after 24 hours?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9457 | 5a6bb5ff4eec6b001a80a514 | Myocardial_infarction | The main treatment for MI with ECG evidence of ST elevation (STEMI) include thrombolysis and percutaneous coronary intervention. Primary percutaneous coronary intervention (PCI) is the treatment of choice for STEMI if it can be performed in a timely manner. If PCI cannot be performed within 90 to 120 minutes then thrombolysis, preferably within 30 minutes of arrival to hospital, is recommended. If a person has had symptoms for 12 to 24 hours evidence for thrombolysis is less and if they have had symptoms for more than 24 hours it is not recommended. | When must ECG evidence be taken? | When must ECG evidence be taken? | [
"When must ECG evidence be taken?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9458 | 5a6bb82b4eec6b001a80a524 | Myocardial_infarction | Thrombolysis involves the administration of medication that activates the enzymes that normally destroy blood clots. Thrombolysis agents include streptokinase, reteplase, alteplase, and tenecteplase. If no contraindications are present (such as a high risk of bleeding), thrombolysis can be given in the pre-hospital or in-hospital setting. When given to people suspected of having a STEMI within 6 hours of the onset of symptoms, thrombolytic drugs save the life of 1 in 43 who received them. The risks were major bleeding (1 in 143) and brain bleeding (1 in 250). It is unclear whether pre-hospital thrombolysis reduces death in people with STEMI compared to in-hospital thrombolysis. Pre-hospital thrombolysis reduces time to thrombolytic treatment, based on studies conducted in higher income countries. | What enzymes in the body typically destroy blood clots? | What enzymes in the body typically destroy blood clots? | [
"What enzymes in the body typically destroy blood clots?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9459 | 5a6bb82b4eec6b001a80a525 | Myocardial_infarction | Thrombolysis involves the administration of medication that activates the enzymes that normally destroy blood clots. Thrombolysis agents include streptokinase, reteplase, alteplase, and tenecteplase. If no contraindications are present (such as a high risk of bleeding), thrombolysis can be given in the pre-hospital or in-hospital setting. When given to people suspected of having a STEMI within 6 hours of the onset of symptoms, thrombolytic drugs save the life of 1 in 43 who received them. The risks were major bleeding (1 in 143) and brain bleeding (1 in 250). It is unclear whether pre-hospital thrombolysis reduces death in people with STEMI compared to in-hospital thrombolysis. Pre-hospital thrombolysis reduces time to thrombolytic treatment, based on studies conducted in higher income countries. | What is an example of a contraindication that must be present for thrombolysis? | What is an example of a contraindication that must be present for thrombolysis? | [
"What is an example of a contraindication that must be present for thrombolysis?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9460 | 5a6bb82b4eec6b001a80a526 | Myocardial_infarction | Thrombolysis involves the administration of medication that activates the enzymes that normally destroy blood clots. Thrombolysis agents include streptokinase, reteplase, alteplase, and tenecteplase. If no contraindications are present (such as a high risk of bleeding), thrombolysis can be given in the pre-hospital or in-hospital setting. When given to people suspected of having a STEMI within 6 hours of the onset of symptoms, thrombolytic drugs save the life of 1 in 43 who received them. The risks were major bleeding (1 in 143) and brain bleeding (1 in 250). It is unclear whether pre-hospital thrombolysis reduces death in people with STEMI compared to in-hospital thrombolysis. Pre-hospital thrombolysis reduces time to thrombolytic treatment, based on studies conducted in higher income countries. | Thrombolytic drugs are given to how many people? | Thrombolytic drugs are given to how many people? | [
"Thrombolytic drugs are given to how many people?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9461 | 5a6bb82b4eec6b001a80a527 | Myocardial_infarction | Thrombolysis involves the administration of medication that activates the enzymes that normally destroy blood clots. Thrombolysis agents include streptokinase, reteplase, alteplase, and tenecteplase. If no contraindications are present (such as a high risk of bleeding), thrombolysis can be given in the pre-hospital or in-hospital setting. When given to people suspected of having a STEMI within 6 hours of the onset of symptoms, thrombolytic drugs save the life of 1 in 43 who received them. The risks were major bleeding (1 in 143) and brain bleeding (1 in 250). It is unclear whether pre-hospital thrombolysis reduces death in people with STEMI compared to in-hospital thrombolysis. Pre-hospital thrombolysis reduces time to thrombolytic treatment, based on studies conducted in higher income countries. | If thrombolytic drugs are given after 6 hours of onset, what are the odds of bleeding? | If thrombolytic drugs are given after 6 hours of onset, what are the odds of bleeding? | [
"If thrombolytic drugs are given after 6 hours of onset, what are the odds of bleeding?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9462 | 5a6bb82b4eec6b001a80a528 | Myocardial_infarction | Thrombolysis involves the administration of medication that activates the enzymes that normally destroy blood clots. Thrombolysis agents include streptokinase, reteplase, alteplase, and tenecteplase. If no contraindications are present (such as a high risk of bleeding), thrombolysis can be given in the pre-hospital or in-hospital setting. When given to people suspected of having a STEMI within 6 hours of the onset of symptoms, thrombolytic drugs save the life of 1 in 43 who received them. The risks were major bleeding (1 in 143) and brain bleeding (1 in 250). It is unclear whether pre-hospital thrombolysis reduces death in people with STEMI compared to in-hospital thrombolysis. Pre-hospital thrombolysis reduces time to thrombolytic treatment, based on studies conducted in higher income countries. | How many hours must pass after onset of symptoms before administering thrombolysis? | How many hours must pass after onset of symptoms before administering thrombolysis? | [
"How many hours must pass after onset of symptoms before administering thrombolysis?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9463 | 5a6bb9624eec6b001a80a538 | Myocardial_infarction | People with an acute coronary syndrome where no ST elevation is demonstrated (non-ST elevation ACS or NSTEACS) are treated with aspirin. Clopidogrel is added in many cases, particularly if the risk of cardiovascular events is felt to be high and early PCI is being considered. Depending on whether early PCI is planned, a factor Xa inhibitor or a potentiator of antithrombin (fondaparinux or low molecular weight heparin respectively) may be added. In very high-risk scenarios, inhibitors of the platelet glycoprotein αIIbβ3a receptor such as eptifibatide or tirofiban may be used. | When is clopidogrel removed? | When is clopidogrel removed? | [
"When is clopidogrel removed?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9464 | 5a6bb9624eec6b001a80a539 | Myocardial_infarction | People with an acute coronary syndrome where no ST elevation is demonstrated (non-ST elevation ACS or NSTEACS) are treated with aspirin. Clopidogrel is added in many cases, particularly if the risk of cardiovascular events is felt to be high and early PCI is being considered. Depending on whether early PCI is planned, a factor Xa inhibitor or a potentiator of antithrombin (fondaparinux or low molecular weight heparin respectively) may be added. In very high-risk scenarios, inhibitors of the platelet glycoprotein αIIbβ3a receptor such as eptifibatide or tirofiban may be used. | What is NSTEACS an abbreviation for? | What is NSTEACS an abbreviation for? | [
"What is NSTEACS an abbreviation for?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9465 | 5a6bb9624eec6b001a80a53a | Myocardial_infarction | People with an acute coronary syndrome where no ST elevation is demonstrated (non-ST elevation ACS or NSTEACS) are treated with aspirin. Clopidogrel is added in many cases, particularly if the risk of cardiovascular events is felt to be high and early PCI is being considered. Depending on whether early PCI is planned, a factor Xa inhibitor or a potentiator of antithrombin (fondaparinux or low molecular weight heparin respectively) may be added. In very high-risk scenarios, inhibitors of the platelet glycoprotein αIIbβ3a receptor such as eptifibatide or tirofiban may be used. | Clopidogrel is a form of what inhibitor? | Clopidogrel is a form of what inhibitor? | [
"Clopidogrel is a form of what inhibitor?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9466 | 5a6bb9624eec6b001a80a53b | Myocardial_infarction | People with an acute coronary syndrome where no ST elevation is demonstrated (non-ST elevation ACS or NSTEACS) are treated with aspirin. Clopidogrel is added in many cases, particularly if the risk of cardiovascular events is felt to be high and early PCI is being considered. Depending on whether early PCI is planned, a factor Xa inhibitor or a potentiator of antithrombin (fondaparinux or low molecular weight heparin respectively) may be added. In very high-risk scenarios, inhibitors of the platelet glycoprotein αIIbβ3a receptor such as eptifibatide or tirofiban may be used. | What inhibitors are used in low-risk scenarios? | What inhibitors are used in low-risk scenarios? | [
"What inhibitors are used in low-risk scenarios?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9467 | 5a6bb9624eec6b001a80a53c | Myocardial_infarction | People with an acute coronary syndrome where no ST elevation is demonstrated (non-ST elevation ACS or NSTEACS) are treated with aspirin. Clopidogrel is added in many cases, particularly if the risk of cardiovascular events is felt to be high and early PCI is being considered. Depending on whether early PCI is planned, a factor Xa inhibitor or a potentiator of antithrombin (fondaparinux or low molecular weight heparin respectively) may be added. In very high-risk scenarios, inhibitors of the platelet glycoprotein αIIbβ3a receptor such as eptifibatide or tirofiban may be used. | Eptifibatide has what kind of molecular weight? | Eptifibatide has what kind of molecular weight? | [
"Eptifibatide has what kind of molecular weight?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9468 | 5a6bb9f34eec6b001a80a54c | Myocardial_infarction | Cardiac rehabilitation benefits many who have experienced myocardial infarction, even if there has been substantial heart damage and resultant left ventricular failure; ideally other medical conditions that could interfere with participation should be managed optimally. It should start soon after discharge from hospital. The program may include lifestyle advice, exercise, social support, as well as recommendations about driving, flying, sport participation, stress management, and sexual intercourse. | Cardiac rehabilitation is not an option under what circumstances? | Cardiac rehabilitation is not an option under what circumstances? | [
"Cardiac rehabilitation is not an option under what circumstances?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9469 | 5a6bb9f34eec6b001a80a54d | Myocardial_infarction | Cardiac rehabilitation benefits many who have experienced myocardial infarction, even if there has been substantial heart damage and resultant left ventricular failure; ideally other medical conditions that could interfere with participation should be managed optimally. It should start soon after discharge from hospital. The program may include lifestyle advice, exercise, social support, as well as recommendations about driving, flying, sport participation, stress management, and sexual intercourse. | Cardiac rehabilitation often recommends ceasing what activities? | Cardiac rehabilitation often recommends ceasing what activities? | [
"Cardiac rehabilitation often recommends ceasing what activities?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9470 | 5a6bb9f34eec6b001a80a54e | Myocardial_infarction | Cardiac rehabilitation benefits many who have experienced myocardial infarction, even if there has been substantial heart damage and resultant left ventricular failure; ideally other medical conditions that could interfere with participation should be managed optimally. It should start soon after discharge from hospital. The program may include lifestyle advice, exercise, social support, as well as recommendations about driving, flying, sport participation, stress management, and sexual intercourse. | What should start immediately upon registering at the hospital? | What should start immediately upon registering at the hospital? | [
"What should start immediately upon registering at the hospital?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9471 | 5a6bb9f34eec6b001a80a54f | Myocardial_infarction | Cardiac rehabilitation benefits many who have experienced myocardial infarction, even if there has been substantial heart damage and resultant left ventricular failure; ideally other medical conditions that could interfere with participation should be managed optimally. It should start soon after discharge from hospital. The program may include lifestyle advice, exercise, social support, as well as recommendations about driving, flying, sport participation, stress management, and sexual intercourse. | When are other medical conditions address? | When are other medical conditions address? | [
"When are other medical conditions address?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9472 | 5a6bba8a4eec6b001a80a554 | Myocardial_infarction | Some risk factors for death include age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease, and elevation of cardiac markers. Assessment of left ventricular ejection fraction may increase the predictive power. Prognosis is worse if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs. Morbidity and mortality from myocardial infarction has improved over the years due to better treatment. | How many classes of ST-segmentation are there? | How many classes of ST-segmentation are there? | [
"How many classes of ST-segmentation are there?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9473 | 5a6bba8a4eec6b001a80a555 | Myocardial_infarction | Some risk factors for death include age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease, and elevation of cardiac markers. Assessment of left ventricular ejection fraction may increase the predictive power. Prognosis is worse if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs. Morbidity and mortality from myocardial infarction has improved over the years due to better treatment. | Prognosis improves after what complication? | Prognosis improves after what complication? | [
"Prognosis improves after what complication?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9474 | 5a6bba8a4eec6b001a80a556 | Myocardial_infarction | Some risk factors for death include age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease, and elevation of cardiac markers. Assessment of left ventricular ejection fraction may increase the predictive power. Prognosis is worse if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs. Morbidity and mortality from myocardial infarction has improved over the years due to better treatment. | What has decreased over the years? | What has decreased over the years? | [
"What has decreased over the years?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9475 | 5a6bba8a4eec6b001a80a557 | Myocardial_infarction | Some risk factors for death include age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease, and elevation of cardiac markers. Assessment of left ventricular ejection fraction may increase the predictive power. Prognosis is worse if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs. Morbidity and mortality from myocardial infarction has improved over the years due to better treatment. | What are some examples of ST-segment deviation? | What are some examples of ST-segment deviation? | [
"What are some examples of ST-segment deviation?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9476 | 5a6bba8a4eec6b001a80a558 | Myocardial_infarction | Some risk factors for death include age, hemodynamic parameters (such as heart failure, cardiac arrest on admission, systolic blood pressure, or Killip class of two or greater), ST-segment deviation, diabetes, serum creatinine, peripheral vascular disease, and elevation of cardiac markers. Assessment of left ventricular ejection fraction may increase the predictive power. Prognosis is worse if a mechanical complication such as papillary muscle or myocardial free wall rupture occurs. Morbidity and mortality from myocardial infarction has improved over the years due to better treatment. | What are risk factors called? | What are risk factors called? | [
"What are risk factors called?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9477 | 5a6bbb724eec6b001a80a55e | Myocardial_infarction | Complications may occur immediately following the heart attack (in the acute phase), or may need time to develop (a chronic problem). Acute complications may include heart failure if the damaged heart is no longer able to pump blood adequately around the body; aneurysm of the left ventricle myocardium; ventricular septal rupture or free wall rupture; mitral regurgitation, in particular if the infarction causes dysfunction of the papillary muscle; Dressler's syndrome; and abnormal heart rhythms, such as ventricular fibrillation, ventricular tachycardia, atrial fibrillation, and heart block. Longer-term complications include heart failure, atrial fibrillation, and an increased risk of a second MI. | What is Dressler's syndrome? | What is Dressler's syndrome? | [
"What is Dressler's syndrome?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9478 | 5a6bbb724eec6b001a80a55f | Myocardial_infarction | Complications may occur immediately following the heart attack (in the acute phase), or may need time to develop (a chronic problem). Acute complications may include heart failure if the damaged heart is no longer able to pump blood adequately around the body; aneurysm of the left ventricle myocardium; ventricular septal rupture or free wall rupture; mitral regurgitation, in particular if the infarction causes dysfunction of the papillary muscle; Dressler's syndrome; and abnormal heart rhythms, such as ventricular fibrillation, ventricular tachycardia, atrial fibrillation, and heart block. Longer-term complications include heart failure, atrial fibrillation, and an increased risk of a second MI. | What does an aneurysm of the left ventricle lead to? | What does an aneurysm of the left ventricle lead to? | [
"What does an aneurysm of the left ventricle lead to?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9479 | 5a6bbb724eec6b001a80a560 | Myocardial_infarction | Complications may occur immediately following the heart attack (in the acute phase), or may need time to develop (a chronic problem). Acute complications may include heart failure if the damaged heart is no longer able to pump blood adequately around the body; aneurysm of the left ventricle myocardium; ventricular septal rupture or free wall rupture; mitral regurgitation, in particular if the infarction causes dysfunction of the papillary muscle; Dressler's syndrome; and abnormal heart rhythms, such as ventricular fibrillation, ventricular tachycardia, atrial fibrillation, and heart block. Longer-term complications include heart failure, atrial fibrillation, and an increased risk of a second MI. | What does mitral regurgitation cause? | What does mitral regurgitation cause? | [
"What does mitral regurgitation cause?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9480 | 5a6bbb724eec6b001a80a561 | Myocardial_infarction | Complications may occur immediately following the heart attack (in the acute phase), or may need time to develop (a chronic problem). Acute complications may include heart failure if the damaged heart is no longer able to pump blood adequately around the body; aneurysm of the left ventricle myocardium; ventricular septal rupture or free wall rupture; mitral regurgitation, in particular if the infarction causes dysfunction of the papillary muscle; Dressler's syndrome; and abnormal heart rhythms, such as ventricular fibrillation, ventricular tachycardia, atrial fibrillation, and heart block. Longer-term complications include heart failure, atrial fibrillation, and an increased risk of a second MI. | Atrial fibrillation can only be what kind of problem? | Atrial fibrillation can only be what kind of problem? | [
"Atrial fibrillation can only be what kind of problem?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9481 | 5a6bbc0f4eec6b001a80a566 | Myocardial_infarction | In contrast, IHD is becoming a more common cause of death in the developing world. For example, in India, IHD had become the leading cause of death by 2004, accounting for 1.46 million deaths (14% of total deaths) and deaths due to IHD were expected to double during 1985–2015. Globally, disability adjusted life years (DALYs) lost to ischemic heart disease are predicted to account for 5.5% of total DALYs in 2030, making it the second-most-important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date. | What percentage of deaths does unipolar depressive disorder cause? | What percentage of deaths does unipolar depressive disorder cause? | [
"What percentage of deaths does unipolar depressive disorder cause?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9482 | 5a6bbc0f4eec6b001a80a567 | Myocardial_infarction | In contrast, IHD is becoming a more common cause of death in the developing world. For example, in India, IHD had become the leading cause of death by 2004, accounting for 1.46 million deaths (14% of total deaths) and deaths due to IHD were expected to double during 1985–2015. Globally, disability adjusted life years (DALYs) lost to ischemic heart disease are predicted to account for 5.5% of total DALYs in 2030, making it the second-most-important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date. | How many people died from IHD from 1985-2015? | How many people died from IHD from 1985-2015? | [
"How many people died from IHD from 1985-2015?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9483 | 5a6bbc0f4eec6b001a80a568 | Myocardial_infarction | In contrast, IHD is becoming a more common cause of death in the developing world. For example, in India, IHD had become the leading cause of death by 2004, accounting for 1.46 million deaths (14% of total deaths) and deaths due to IHD were expected to double during 1985–2015. Globally, disability adjusted life years (DALYs) lost to ischemic heart disease are predicted to account for 5.5% of total DALYs in 2030, making it the second-most-important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date. | What is the worldwide leading cause of death? | What is the worldwide leading cause of death? | [
"What is the worldwide leading cause of death?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9484 | 5a6bbc0f4eec6b001a80a569 | Myocardial_infarction | In contrast, IHD is becoming a more common cause of death in the developing world. For example, in India, IHD had become the leading cause of death by 2004, accounting for 1.46 million deaths (14% of total deaths) and deaths due to IHD were expected to double during 1985–2015. Globally, disability adjusted life years (DALYs) lost to ischemic heart disease are predicted to account for 5.5% of total DALYs in 2030, making it the second-most-important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date. | What percentage of deaths will IHD be responsible for in 2030? | What percentage of deaths will IHD be responsible for in 2030? | [
"What percentage of deaths will IHD be responsible for in 2030?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9485 | 5a6bbc0f4eec6b001a80a56a | Myocardial_infarction | In contrast, IHD is becoming a more common cause of death in the developing world. For example, in India, IHD had become the leading cause of death by 2004, accounting for 1.46 million deaths (14% of total deaths) and deaths due to IHD were expected to double during 1985–2015. Globally, disability adjusted life years (DALYs) lost to ischemic heart disease are predicted to account for 5.5% of total DALYs in 2030, making it the second-most-important cause of disability (after unipolar depressive disorder), as well as the leading cause of death by this date. | When did IHD begin to be a bigger problem in the developing word? | When did IHD begin to be a bigger problem in the developing word? | [
"When did IHD begin to be a bigger problem in the developing word?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9486 | 5a6bbd284eec6b001a80a57a | Myocardial_infarction | At common law, in general, a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered; however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion. In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from an MI may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane. | What is a myocardial infarction is always considered to be? | What is a myocardial infarction is always considered to be? | [
"What is a myocardial infarction is always considered to be?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9487 | 5a6bbd284eec6b001a80a57b | Myocardial_infarction | At common law, in general, a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered; however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion. In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from an MI may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane. | What occupation cannot have a heart attack classified as work-related? | What occupation cannot have a heart attack classified as work-related? | [
"What occupation cannot have a heart attack classified as work-related?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9488 | 5a6bbd284eec6b001a80a57c | Myocardial_infarction | At common law, in general, a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered; however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion. In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from an MI may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane. | What typically covers an MI? | What typically covers an MI? | [
"What typically covers an MI?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9489 | 5a6bbd284eec6b001a80a57d | Myocardial_infarction | At common law, in general, a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered; however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion. In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from an MI may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane. | When is an MI not considered a work-related injury? | When is an MI not considered a work-related injury? | [
"When is an MI not considered a work-related injury?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9490 | 5a6bbd284eec6b001a80a57e | Myocardial_infarction | At common law, in general, a myocardial infarction is a disease, but may sometimes be an injury. This can create coverage issues in administration of no-fault insurance schemes such as workers' compensation. In general, a heart attack is not covered; however, it may be a work-related injury if it results, for example, from unusual emotional stress or unusual exertion. In addition, in some jurisdictions, heart attacks suffered by persons in particular occupations such as police officers may be classified as line-of-duty injuries by statute or policy. In some countries or states, a person having suffered from an MI may be prevented from participating in activity that puts other people's lives at risk, for example driving a car or flying an airplane. | What generally treats MI as an injury? | What generally treats MI as an injury? | [
"What generally treats MI as an injury?"
] | {
"text": [],
"answer_start": []
} |
gem-squad_v2-train-9491 | 5725b5d138643c19005acbb5 | Montevideo | It is classified as a Beta World City, ranking seventh in Latin America and 73rd in the world. Described as a "vibrant, eclectic place with a rich cultural life", and "a thriving tech center and entrepreneurial culture", Montevideo ranks 8th in Latin America on the 2013 MasterCard Global Destination Cities Index. By 2014, is also regarded as the fifth most gay-friendly major city in the world, first in Latin America. It is the hub of commerce and higher education in Uruguay as well as its chief port. The city is also the financial and cultural hub of a larger metropolitan area, with a population of around 2 million. | What is Montevideo classified as? | What is Montevideo classified as? | [
"What is Montevideo classified as?"
] | {
"text": [
"a Beta World City"
],
"answer_start": [
20
]
} |
gem-squad_v2-train-9492 | 5725b5d138643c19005acbb6 | Montevideo | It is classified as a Beta World City, ranking seventh in Latin America and 73rd in the world. Described as a "vibrant, eclectic place with a rich cultural life", and "a thriving tech center and entrepreneurial culture", Montevideo ranks 8th in Latin America on the 2013 MasterCard Global Destination Cities Index. By 2014, is also regarded as the fifth most gay-friendly major city in the world, first in Latin America. It is the hub of commerce and higher education in Uruguay as well as its chief port. The city is also the financial and cultural hub of a larger metropolitan area, with a population of around 2 million. | What rank did Montevideo hold in Latin America during 2013? | What rank did Montevideo hold in Latin America during 2013? | [
"What rank did Montevideo hold in Latin America during 2013?"
] | {
"text": [
"8th"
],
"answer_start": [
238
]
} |
gem-squad_v2-train-9493 | 5725b5d138643c19005acbb7 | Montevideo | It is classified as a Beta World City, ranking seventh in Latin America and 73rd in the world. Described as a "vibrant, eclectic place with a rich cultural life", and "a thriving tech center and entrepreneurial culture", Montevideo ranks 8th in Latin America on the 2013 MasterCard Global Destination Cities Index. By 2014, is also regarded as the fifth most gay-friendly major city in the world, first in Latin America. It is the hub of commerce and higher education in Uruguay as well as its chief port. The city is also the financial and cultural hub of a larger metropolitan area, with a population of around 2 million. | Montevideo is the hub of commerce and higher education in what area? | Montevideo is the hub of commerce and higher education in what area? | [
"Montevideo is the hub of commerce and higher education in what area?"
] | {
"text": [
"Uruguay"
],
"answer_start": [
471
]
} |
gem-squad_v2-train-9494 | 5725b5d138643c19005acbb8 | Montevideo | It is classified as a Beta World City, ranking seventh in Latin America and 73rd in the world. Described as a "vibrant, eclectic place with a rich cultural life", and "a thriving tech center and entrepreneurial culture", Montevideo ranks 8th in Latin America on the 2013 MasterCard Global Destination Cities Index. By 2014, is also regarded as the fifth most gay-friendly major city in the world, first in Latin America. It is the hub of commerce and higher education in Uruguay as well as its chief port. The city is also the financial and cultural hub of a larger metropolitan area, with a population of around 2 million. | Montevideo is the chief port of what area? | Montevideo is the chief port of what area? | [
"Montevideo is the chief port of what area?"
] | {
"text": [
"Uruguay"
],
"answer_start": [
471
]
} |
gem-squad_v2-train-9495 | 5725b5d138643c19005acbb9 | Montevideo | It is classified as a Beta World City, ranking seventh in Latin America and 73rd in the world. Described as a "vibrant, eclectic place with a rich cultural life", and "a thriving tech center and entrepreneurial culture", Montevideo ranks 8th in Latin America on the 2013 MasterCard Global Destination Cities Index. By 2014, is also regarded as the fifth most gay-friendly major city in the world, first in Latin America. It is the hub of commerce and higher education in Uruguay as well as its chief port. The city is also the financial and cultural hub of a larger metropolitan area, with a population of around 2 million. | What is the population of Montevideo? | What is the population of Montevideo? | [
"What is the population of Montevideo?"
] | {
"text": [
"around 2 million"
],
"answer_start": [
606
]
} |
gem-squad_v2-train-9496 | 5725b711271a42140099d081 | Montevideo | A Spanish expedition was sent from Buenos Aires, organized by the Spanish governor of that city, Bruno Mauricio de Zabala. On 22 January 1724, the Spanish forced the Portuguese to abandon the location and started populating the city, initially with six families moving in from Buenos Aires and soon thereafter by families arriving from the Canary Islands who were called by the locals "guanches", "guanchos" or "canarios". There was also one significant early Italian resident by the name of Jorge Burgues. | Who organized the Spanish expedition? | Who organized the Spanish expedition? | [
"Who organized the Spanish expedition?"
] | {
"text": [
"Bruno Mauricio de Zabala"
],
"answer_start": [
97
]
} |
gem-squad_v2-train-9497 | 5725b711271a42140099d082 | Montevideo | A Spanish expedition was sent from Buenos Aires, organized by the Spanish governor of that city, Bruno Mauricio de Zabala. On 22 January 1724, the Spanish forced the Portuguese to abandon the location and started populating the city, initially with six families moving in from Buenos Aires and soon thereafter by families arriving from the Canary Islands who were called by the locals "guanches", "guanchos" or "canarios". There was also one significant early Italian resident by the name of Jorge Burgues. | What date did the Spanish force the Portuguese to abandon the location? | What date did the Spanish force the Portuguese to abandon the location? | [
"What date did the Spanish force the Portuguese to abandon the location?"
] | {
"text": [
"22 January 1724"
],
"answer_start": [
126
]
} |
gem-squad_v2-train-9498 | 5725b711271a42140099d083 | Montevideo | A Spanish expedition was sent from Buenos Aires, organized by the Spanish governor of that city, Bruno Mauricio de Zabala. On 22 January 1724, the Spanish forced the Portuguese to abandon the location and started populating the city, initially with six families moving in from Buenos Aires and soon thereafter by families arriving from the Canary Islands who were called by the locals "guanches", "guanchos" or "canarios". There was also one significant early Italian resident by the name of Jorge Burgues. | Who was one significant early italian resident? | Who was one significant early italian resident? | [
"Who was one significant early italian resident? "
] | {
"text": [
"Jorge Burgues"
],
"answer_start": [
492
]
} |
gem-squad_v2-train-9499 | 5725b82838643c19005acbc5 | Montevideo | A few years after its foundation, Montevideo became the main city of the region north of the Río de la Plata and east of the Uruguay River, competing with Buenos Aires for dominance in maritime commerce. The importance of Montevideo as the main port of the Viceroyalty of the Río de la Plata brought it in confrontations with the city of Buenos Aires in various occasions, including several times when it was taken over to be used as a base to defend the eastern province of the Viceroyalty from Portuguese incursions. | What became the main city of the region north of the Rio de la Plata? | What became the main city of the region north of the Rio de la Plata? | [
"What became the main city of the region north of the Rio de la Plata?"
] | {
"text": [
"Montevideo"
],
"answer_start": [
34
]
} |
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