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P21802
O76093
0
binding
up-regulates
0.73
Fgfs bind and activate high-affinity receptor tyrosine kinases. The cloning of fgf receptors (fgfrs) has identified four distinct genes
SIGNOR-42368
P51812
P27361
0
phosphorylation
up-regulates
0.73
We have generated two monoclonal antibodies that recognize two phosphorylated sites, p-ser227 and p-thr577, in the n- and c-terminal kinase domains of rsk2, respectively. phosphorylation and activation of rsk2 by uv light involves the erk pathway
SIGNOR-81460
O15146
O75096
0
binding
up-regulates activity
0.73
AGRN is released by the nerve and binds to LRP4, which then binds to MuSK. This interaction leads to MuSK autophosphorylation and activation of its kinase function, leading to anterograde signalling by subsequent phosphorylation of DOK7 (not shown), which binds MuSK as a dimer.
SIGNOR-273850
P45984
P45985
0
phosphorylation
up-regulates
0.729
Mkk4, which activates p38gamma, p38delta, and jnk2 to phosphorylate p53 on ser-33 and cause a transient g(1) arrest. A map kinase kinase kinase (mapkkk), termed ask1, was identified that activated two different subs of map kinase kinases (mapkk), sek1 (or mkk4) and mkk3/mapkk6 (or mkk6), which in turn activated stress-activated protein kinase (sapk, also known as jnk;c-jun amino-terminal kinase) here we report that mkk4 shows a striking preference for the tyrosine residue (tyr-185), and mkk7 a striking preference for the threonine residue (thr-183) in three sapk1/jnk1 isoforms tested (jnk1 alpha 1, jnk2 alpha 2 and jnk3 alpha 1)
SIGNOR-197998
Q9Y6Q6
Q9Y4K3
0
binding
up-regulates activity
0.729
TRAF6 interacts with a novel motif located between residues 340 and 358 of RANK. TRAF6-binding region (340-358), but not the TRAF2 or TRAF5-binding region, is necessary and sufficient for RANK-induced NF-kappaB activation.
SIGNOR-253045
P49815
P31751
0
phosphorylation
down-regulates
0.729
We demonstrate here that tuberin is phosphorylated on s939 and t1462 in response to pi3k activation. Our results are consistent with akt being the pi3k-depen-dent tuberin kinase. The pi3k-akt-mediated phosphorylation of tuberin would inhibit the function of the tuberin-hamartin complex.
SIGNOR-91041
P37231
P17676
0
transcriptional regulation
up-regulates quantity
0.729
 Induction of C/EBP beta DNA-binding activity in NIH-3T3 beta 2 cells exposed to dexamethasone in the presence of insulin and fetal bovine serum activates the expression of an adipocyte-specific nuclear hormone receptor, PPAR gamma, that stimulates the conversion of these fibroblasts into committed preadipocytes
SIGNOR-255730
Q99683
P31749
0
phosphorylation
down-regulates activity
0.729
Akt phosphorylates and negatively regulates apoptosis signal-regulating kinase 1 akt decreased ask1 kinase activity stimulated by both oxidative stress and overexpression in 293 cells by phosphorylating a consensus akt site at serine 83 of ask1.
SIGNOR-252465
O60674
P29350
0
dephosphorylation
down-regulates activity
0.729
Direct association with and dephosphorylation of Jak2 kinase by the SH2-domain-containing protein tyrosine phosphatase SHP-1
SIGNOR-248466
O14965
Q6PGQ7
0
binding
up-regulates
0.729
Both drosophila and human bora can bind to aurora-a and activate the kinase in vitro.
SIGNOR-148661
P45985
Q13233
0
phosphorylation
up-regulates activity
0.729
The gck-ctd-mekk1 interaction is sufficiently stable to support mekk1 s phosphorylation of its substrate, sek1
SIGNOR-236380
P22736
P31749
0
phosphorylation
down-regulates activity
0.729
We show that akt interacts with nur77 and inactivates nur77 by phosphorylation at ser-350
SIGNOR-252466
Q9HB89
Q5H8A3
0
binding
up-regulates
0.729
Here we identify a novel neuropeptide of 36 amino-acid residues in rat brain as an endogenous ligand for the orphan g protein-coupled receptor fm-4/tgr-1, which was identified to date as the neuromedin u (nmu) receptor, and designate this peptide 'neuromedin s (nms)' because it is specifically expressed in the suprachiasmatic nuclei (scn) of the hypothalamus.
SIGNOR-133074
P53675
Q13492
0
binding
up-regulates
0.729
Calm interacts with the clathrin heavy chain through its c-terminal third and with phophoinositides through its ap180 n-terminal homology (anth) domain, promoting assembly of clathrin triskelia into clathrin cagesin vitro
SIGNOR-144733
Q9BQQ3
P53350
0
phosphorylation
down-regulates quantity
0.729
As GRASP65 is a substrate of cdc2 and polo-like kinase, manipulation of GRASP65 level may affect the localization and activity of these kinases in cell cycle progression, as suggested by a previous study ( ).|During mitosis, GRASP65 is phosphorylated by two mitotic kinases, cdc2 and polo-like kinase (plk), which leads to GRASP65 deoligomerization and thus Golgi unstacking ( xref , xref ).
SIGNOR-279554
Q15465
Q96F81
0
binding
up-regulates activity
0.729
We show that the vertebrate homologue, dispatched-a (dispa) interacts with human sonic hedgehog (hshh) via its cholesterol anchor, and that this interaction is necessary for hshh secretion. binding to dispa is necessary but not sufficient for hshh secretion
SIGNOR-191888
Q13094
P42681
0
phosphorylation
up-regulates
0.728
Resting lymphocyte kinase (rlk/txk) targets lymphoid adaptor slp-76 in the cooperative activation of interleukin-2 transcription in t-cells. In this study, we report that rlk phosphorylates slp-76 at its n-terminal yesp/yepp sites. A third tyrosine within the amino-terminal region (y145) appears to be the most important for optimal slp-76 function
SIGNOR-44669
P51812
P28482
0
phosphorylation
up-regulates
0.728
Erk-activates the rsk family of serine/threonine kinases,rsk1, rsk2, and rsk3.
SIGNOR-161518
Q16539
P46734
0
phosphorylation
up-regulates activity
0.728
Two human MAP kinase kinases (MKK3 and MKK4) were cloned that phosphorylate and activate p38 MAP kinase.
SIGNOR-232156
P11142
Q9UNE7
0
polyubiquitination
down-regulates quantity by destabilization
0.728
BAG-1 stimulates CHIP-induced degradation of the glucocorticoid hormone receptor (GR). A model for the cooperation of CHIP and BAG-1 in coupling Hsc/Hsp70 to the ubiquitin/proteasome system. CHIP associates with Hsc/Hsp70 via its TPR chaperone adaptor (TPR) and, at the same time, recruits E2 ubiquitin-conjugating enzymes of the Ubc4/5 family to the chaperone complex. BAG-1 binds to Hsp70 via its BAG domain (BAG) and utilizes its ubiquitin-like domain (ubl) for proteasomal association
SIGNOR-272588
P05198
P19525
0
phosphorylation
down-regulates activity
0.728
Besides PERK, eIF2α can also be phosphorylated by three other kinases: heme-regulated inhibitor kinase (HRI), general control nonderepressible 2 (GCN2), and PKR. PKR is an interferon-stimulated gene (ISG) activated by binding of double-stranded RNA (dsRNA), a common intermediate during the replication of DNA and RNA viruses. Together, these four eIF2α kinases and their convergent downstream signaling pathways are known as the integrated stress response (ISR)
SIGNOR-260168
O75197
Q9H461
0
binding
up-regulates activity
0.728
Ligands such as Wnt1, Wnt3a, and Wnt8 couple the seven-transmembrane domain receptor Frizzled (Fzd) and the single-membrane-spanning low-density receptor-related protein 5/6 (LRP5/6) to activate Wnt–Beta-catenin signaling.
SIGNOR-169635
Q9Y463
P61962
0
binding
up-regulates activity
0.728
Two isoforms of DYRK, DYRK1A and DYRK1B, co-immunoprecipitate with HAN11 when coexpressed in COS cells indicating that the proteins interact in mammalian cells. HAN11 might target DYRKs to cytosolic locations for regulation of specific cellular functions.
SIGNOR-260631
P84022
P12755
0
binding
down-regulates activity
0.728
Smad2/3 interacts with c-ski through its c-terminal mh2 domain in a tgf-beta-dependent mannerc-ski is incorporated in the smad dna binding complex, interferes with the interaction of smad3 with a transcriptional co-activator, p300, and in turn recruits hdac. c-ski is thus a transcriptional co-repressor that links smads to hdac in tgf-beta signaling.
SIGNOR-232123
P17181
P05000
0
binding
up-regulates
0.728
Ifn-alpha, ifn-beta, and ifn-omega, induce somewhat different cellular effects but act through a common receptor complex, ifnar, composed of subunits ifnar-1 and ifnar-2.
SIGNOR-105979
P50591
Q9UBN6
0
binding
down-regulates
0.728
One function of trail-r4 may be inhibition of trail cytotoxicy. Dcr2 functions as an inhibitory apo2l receptor.
SIGNOR-53447
P04637
P49841
0
phosphorylation
up-regulates activity
0.728
Glycogen synthase kinase3 beta phosphorylates serine 33 of p53 and activates p53's transcriptional activity.
SIGNOR-251258
P15531
Q01105
0
binding
down-regulates
0.728
Tumor suppressor nm23-h1 is a granzyme a-activated dnase during ctl-mediated apoptosis, and the nucleosome assembly protein set is its inhibitor. / nm23-h1 binds to set and is released from inhibition by gzma cleavage of set.
SIGNOR-99205
P36896
P13385
0
binding
up-regulates activity
0.728
Nodal effects are dependent upon interactions with Cripto, a small cysteine-rich extracellular protein that is attached to the plasma membrane through a glycosyl phosphatidyl inositol linkage. Cripto interacts with Nodal and ALK4, independently, and promotes the formation of a stable high affinity complex with activin type II receptors.
SIGNOR-251938
P27986
P10721
0
binding
up-regulates activity
0.727
Tyrosine residue 719 of the c-kit receptor is essential for binding of the P85 subunit of phosphatidylinositol (PI) 3-kinase and for c-kit-associated PI 3-kinase activity in COS-1 cells
SIGNOR-255948
P11717
P01344
0
binding
up-regulates
0.727
Insulin-like growth factor ii receptor (igf2r) is a multifunctional cell surface receptor implicated in tumour suppression. Its growth inhibitory activity has been associated with an ability to bind igf-ii.
SIGNOR-115250
P49407
P28482
0
phosphorylation
down-regulates
0.727
Erk1 and erk2 phosphorylate beta-arrestin1 at ser-412 in vitro. . in the resting state, cytosolic arrestin1 proteins are constitutively phosphorylated by extracellular signal-regulated kinase (erk) at ser412, located within their distal c terminus. erk-phosphorylated arrestin1 is unable to associate with clathrin cages, whereas this constraint is removed upon its dephosphorylation
SIGNOR-67630
Q14457
P53355
0
phosphorylation
up-regulates
0.727
The activated form of DAPK triggers autophagy in a beclin-1-dependent manner. DAPK phosphorylates beclin 1 on Thr 119 located at a crucial position within its BH3 domain, and thus promotes the dissociation of beclin 1 from Bcl-XL and the induction of autophagy.
SIGNOR-183548
Q02241
Q96GD4
0
phosphorylation
down-regulates quantity
0.727
Furthermore, reduced turnover of regulatory phosphorylation on another Aurora B substrate MKlp1 was observed, suggesting that PP2A-B56\u03b3 and -\u03b5 play a general role opposing Aurora B at the central spindle.|In anaphase, the KIF4A-targeted pool of B56\u03b3 and -\u03b5 is ideally placed to counteract Aurora B phosphorylations on other central spindle proteins such as MKlp1.
SIGNOR-280192
Q9Y6D9
O43683
0
relocalization
up-regulates activity
0.727
Spindle checkpoint protein Bub1 is required for kinetochore localization of Mad1, Mad2, Bub3, and CENP-E, independently of its kinase activity
SIGNOR-252017
P48551
Q00978
0
binding
up-regulates activity
0.727
By binding to IFNalphaR2 within the region where two adjacent proline boxes bear phospho-Ser364 and phospho-Ser384, CBP acetylates IFNalphaR2 on Lys399, which in turn serves as the docking site for interferon regulatory factor 9 (IRF9)RF9 interacts with the acetyl-Lys399 motif by means of its IRF homology2 (IH2) domain, leading to formation of the ISGF3 complex that includes IRF9, STAT1, and STAT2.
SIGNOR-217779
P51685
P22362
0
binding
up-regulates
0.726
Ccl1 activates the mapk pathway in ccr8-transfected cho cells.
SIGNOR-99401
P58753
O60603
0
binding
up-regulates activity
0.726
To initiate the innate immune response, Toll-like receptors (TLRs) associate with cytoplasmic adaptor proteins through TIR (Toll/interleukin-1 receptor) domain interactions. The four principal signaling adaptor proteins include MyD88, MAL, TRIF and TRAM, and the fifth protein SARM, involved in negative regulation of TLR pathways, is usually considered a part of the TIR domain-containing adaptor protein group
SIGNOR-266745
P42574
Q14790
0
cleavage
up-regulates activity
0.726
Triggering of the DISC leads to caspase-8 activation. Active caspase-8 cleaves caspase-3 which, in type I cells, leads to cell death induction.
SIGNOR-171767
O14793
P19883
0
binding
down-regulates activity
0.726
Follistatin (FST) is a member of the tissue growth factor beta family and is a secreted glycoprotein that antagonizes many members of the family, including activin A, growth differentiation factor 11, and myostatin. FST315-deltaHBS-Fc induced improvements in muscle repair after injury/atrophy by modulating the early inflammatory phase allowing for increased macrophage density, and Pax7-positive cells leading to an accelerated restoration of myofibers and muscle function.
SIGNOR-251717
P16220
P51812
0
phosphorylation
up-regulates activity
0.726
MAPK activates CREB kinase, which in turn phosphorylates and activates CREB. Purification, sequencing, and biochemical characterization of CREB kinase revealed that it is identical to a member of the pp90(RSK) family, RSK2. RSK2 was shown to mediate growth factor induction of CREB serine-133 phosphorylation both in vitro and in vivo. These findings identify a cellular function for RSK2 and define a mechanism whereby growth factor signals mediated by RAS and MAPK are transmitted to the nucleus to activate gene expression
SIGNOR-248951
P42702
P13725
0
binding
up-regulates
0.726
Oncostatin m binds the high-affinity leukemia inhibitory factor receptor
SIGNOR-19873
P02647
P00738
0
binding
up-regulates quantity by stabilization
0.726
Haptoglobin binding to apolipoprotein A-I prevents damage from hydroxyl radicals on its stimulatory activity of the enzyme lecithin-cholesterol acyl-transferase. haptoglobin, when circulating at enhanced levels with free Hb during the acute phase of inflammation, might protect ApoA-I structure and function against hydroxyl radicals.
SIGNOR-252106
Q9NZJ5
P11021
0
binding
down-regulates activity
0.726
In the stressed ER, protein chaperone GRP78 binds to unfolded proteins and dissociates from the luminal domain of PERK, leading to oligomerization and activation of PERK by autophosphorylation.
SIGNOR-260164
Q03164
O00255
0
binding
up-regulates activity
0.726
However, menin dramatically increases the amount of MLL bound at the p27Kip1 and p18Ink4c loci, suggesting that it either directly or indirectly promotes MLL recruitment to these targets. Once recruited, MLL could enhance transcription by a number of mechanisms.Overall, the data suggest that transcriptional regulation by menin involves increasing MLL protein association with target loci.
SIGNOR-255890
Q9NWQ8
P07948
0
phosphorylation
up-regulates activity
0.725
Here we show that Lyn interacts with C-terminal Src kinase-binding protein (Cbp), an adaptor protein that recruits negative regulators C-terminal Src kinase (Csk)/Csk-like protein-tyrosine kinase (Ctk). Lyn phosphorylated Cbp on several tyrosine residues, including Tyr314, which recruited Csk/Ctk to suppress Lyn kinase activity.Thus, a single phosphotyrosine residue on Cbp coordinates a two-phase process involving distinct negative regulatory pathways to inactivate, then degrade, Lyn.
SIGNOR-262898
Q9H461
P04628
0
binding
up-regulates
0.725
Wnt signaling is mediated by the frizzled (fz) family of seven-pass transmembrane receptors that bind wnt via the conserved amino-terminal cysteine-rich domain (crd)
SIGNOR-109250
Q14289
Q06124
0
dephosphorylation
down-regulates activity
0.725
We demonstrate that RAFTK is a direct substrate of SHP2 both in vitro and in vivo, and that Tyr(906) in the C-terminal domain of RAFTK mediates its interaction with SHP2. |We demonstrate that RAFTK is a direct substrate of SHP2 both in vitro and in vivo, and that Tyr(906) in the C-terminal domain of RAFTK mediates its interaction with SHP2. Moreover, overexpression of dominant negative SHP2 blocked the protective effect of IL-6 against Dex-induced apoptosis. These findings demonstrate that SHP2 mediates the anti-apoptotic effect of IL-6 and suggest SHP2 as a novel therapeutic target in MM..... 1) RAFTK is a substrate of SHP2 in vitro and 2) dephosphorylation of RAFTK by SHP2 inhibits its kinase activity.
SIGNOR-277084
P63000
P31749
0
phosphorylation
down-regulates activity
0.725
Akt protein kinase inhibits Rac1-GTP binding through phosphorylation at serine 71 of Rac1
SIGNOR-252576
Q15797
Q9HAU4
0
polyubiquitination
down-regulates quantity by destabilization
0.725
Here, we report the identification of Smurf2, a new member of the Hect family of E3 ubiquitin ligases. Smurf2 selectively interacts with receptor-regulated Smads and preferentially targets Smad1 for ubiquitination and proteasome-mediated degradation. At higher expression levels, Smurf2 also decreases the protein levels of Smad2, but not Smad3. 
SIGNOR-272936
P42224
P10721
0
phosphorylation
up-regulates activity
0.725
KIT is responsible for the permanent phosphorylation of all three STAT proteins. STAT1, -3, and -5 were phosphorylated on their activation-specific Tyr701, Tyr704, and Tyr694, respectively, following KIT stimulation.
SIGNOR-251365
P11717
O60664
0
relocalization
up-regulates activity
0.725
TIP47 is present in cytosol and on endosomes and is required for MPR transport from endosomes to the trans-Golgi network in vitro and in vivo. TIP47 recognizes a phenylalanine/tryptophan signal in the tail of the cation-dependent MPR that is essential for its proper sorting within the endosomal pathway. These data suggest that TIP47 binds MPR cytoplasmic domains and facilitates their collection into transport vesicles destined for the Golgi.
SIGNOR-253092
P30622
Q7Z460
0
binding
up-regulates activity
0.725
CLIP-associating protein (CLASP) 1 and CLASP2 are mammalian microtubule (MT) plus-end binding proteins, which associate with CLIP-170 and CLIP-115.|We demonstrate that the middle part of CLASPs binds directly to EB1 and to MTs. | Both EB1- and cortex-binding domains of CLASP are required to promote MT stability.
SIGNOR-265091
P63000
Q9NRY4
0
gtpase-activating protein
down-regulates activity
0.724
We therefore developed a screening-compatible live-cell imaging assay, using FRET-based biosensors for the prototype GTPases RHOA, RAC1 and CDC4215,19,20 (Extended Data Fig. 2 and Supplementary Note 1)|We found catalytic activities for 45/75 RhoGEFs and 48/63 RhoGAPs| Our data thus not only reveal extensive promiscuity among regulators, but also that the inactivating RhoGAPs are less selective than the activating RhoGEFs (p-value=0.02)(Supplementary Table 2).
SIGNOR-260493
Q9UGK3
Q13882
0
phosphorylation
up-regulates activity
0.724
Our previous studies revealed that STAP-2 binds to signal transducer and activator of transcription 3 (STAT3) and STAT5, and regulates the signaling pathways downstream of them. In the present study, we identified tyrosine-250 (Tyr250) in STAP-2 as a major site of phosphorylation by Brk, using a series of STAP-2 YF mutants and anti-phospho-STAP-2 Tyr250 antibody. Furthermore, overexpression of the STAP-2 Y250F mutant protein affected Brk-mediated STAT3 activation.
SIGNOR-247067
Q16555
P49841
0
phosphorylation
down-regulates activity
0.724
Here, we showed that glycogen synthase kinase-3beta (gsk-3beta) phosphorylated crmp-2 at thr-514 and inactivated it
SIGNOR-133255
P07550
P32121
0
binding
down-regulates activity
0.724
The protein, termed beta-arrestin, was expressed and partially purified. It inhibited the signaling function of beta ARK-phosphorylated beta-adrenergic receptors by more than 75 percent, but not that of rhodopsin. It is proposed that beta-arrestin in concert with beta ARK effects homologous desensitization of beta-adrenergic receptors
SIGNOR-256501
O95644
P16298
0
relocalization
up-regulates
0.724
The ca2+ dependent phosphatase calcineurin induces cardiac and skeletal muscle hypertrophy by a process that involves nf-at nuclear translocation, and activation of mef2c.
SIGNOR-84047
P05067
P55212
0
cleavage
up-regulates activity
0.724
Inhibition of caspase-6 activity prevents serum deprivation-mediated increase of Ab. Caspase-6 directly cleaves APP at the C terminus and generates a C-terminal fragment of 3 kDa (Capp3) and an Ab-containing 6.5-kDa fragment, Capp6.5, that increases in serum-deprived neurons
SIGNOR-261762
P33991
Q13535
0
phosphorylation
up-regulates
0.724
Together these data strongly support the conclusion that mec1 directly targets the s/tq sites in mcm4 and mcm6, although it is formally possible that mec1 and mrc1 activate a different s/tq-directed kinase to target mcm4 and mcm6.
SIGNOR-169412
P15498
P15391
0
binding
up-regulates activity
0.724
CD19 has an extracellular region containing two C2-type Ig-like domains and a cytoplasmic region of ~240 amino acids with 9 conserved tyrosine residues24. Lyn, a Src-family protein tyrosine kinase member, is the dominant kinase that phosphorylates CD19 upon stimulation. Once tyrosyl-phosphorylated, CD19 serves as a membrane-bound adaptor protein for Src homology 2-containing signaling molecules such as Lyn, Vav, and phosphatidylinositol 3-kinase, which further mediate downstream activation cascades.
SIGNOR-242897
P12757
Q6ZNA4
0
polyubiquitination
down-regulates quantity by destabilization
0.723
Upon TGF-β induction, interaction of Arkadia with phosphorylated Smad2 triggers degradation of SnoN, whereas upon arsenic treatment, interaction of Arkadia with poly-SUMO in PML nuclear bodies induces degradation of polysumoylated PML together with RNF4.
SIGNOR-272885
O14733
Q13233
0
phosphorylation
up-regulates
0.723
Here we show that jnkk2, a novel member of the map kinase kinase family, was phosphorylated and activated by mekk1
SIGNOR-51207
P25025
P19876
0
binding
up-regulates activity
0.723
CXCL2/3, also known as macrophage inflammatory protein-2α/2β (MIP-2α/MIP-2β), share the same receptor CXCR2 with CXCL1 and are able to activate neutrophils effectively
SIGNOR-277719
P01130
Q8WY64
0
ubiquitination
down-regulates quantity by destabilization
0.723
The RING E3 ubiquitin ligase inducible degrader of the LDL receptor (IDOL, also known as MYLIP) promotes ubiquitylation and subsequent lysosomal degradation of the LDL receptor (LDLR), thus acting to limit uptake of lipoprotein-derived cholesterol into cells. 
SIGNOR-271485
P42338
P35568
0
binding
up-regulates activity
0.723
To examine contributions of specific YXXM motifs in human insulin receptor substrate-1 (IRS-1) to mediating the metabolic actions of insulin, we studied IRS-1 mutants containing various substitutions of Phe for Tyr. In transfected NIH-3T3(IR) cells, insulin stimulation caused a 5-fold increase in phosphatidylinositol 3-kinase (PI3K) activity coimmunoprecipitated with wild-type IRS-1
SIGNOR-235487
P48736
P27986
0
binding
up-regulates activity
0.723
Signal transduction pathways triggered by Tie2 have been extensively examined. Tyr-1101of Tie2 directly associates in a phosphotyrosine (pTyr)-dependent manner with the p85 regulatory subunit of phosphatidylinositol (PI) 3-kinase, which in turn activate PI 3-kinase, leading to cell motility and survival
SIGNOR-242646
O43663
P53350
0
phosphorylation
down-regulates activity
0.723
Plk1 negatively regulates PRC1 to prevent premature midzone formation before cytokinesis.|Plk1, but not Cdk1, phosphorylates PRC1 on Thr-602 to prevent premature midzone assembly in metaphase.
SIGNOR-279645
O15151
O96017
0
phosphorylation
down-regulates
0.723
Phosphorylation of s342 and s367 in vivo require the chk2 kinase. Chk2 also stimulates mdmx ubiquitination and degradation by mdm2
SIGNOR-140417
Q15303
P35070
0
binding
up-regulates
0.723
For example, betacellulin binds to and activates both erbb1 and erbb4, whereas epiregulin binds to erbb1, erbb3 and erbb4
SIGNOR-195347
P35568
Q05513
0
phosphorylation
down-regulates activity
0.723
Extensive studies have provided evidence that phosphorylation of Ser307 in IRS-1 inhibits IR/IRS-1 complex formation and IRS-1 tyrosine phosphorylation after prolonged insulin-stimulation similar to our results.
SIGNOR-236760
O95271
Q9NTX7
0
ubiquitination
down-regulates quantity
0.723
We show that RNF146, tankyrase, and Axin form a protein complex, and that RNF146 mediates ubiquitylation of all three proteins to target them for proteasomal degradation.
SIGNOR-260004
P36897
P37173
0
phosphorylation
up-regulates activity
0.722
Recent studies have revealed that upon TGF-beta binding several serine and threonine residues in the GS domain of TGF-beta type I receptor (T beta R-I) are phosphorylated by TGF-beta type II receptor (T beta R-II) and that the phosphorylation of GS domain is essential for TGF-beta signalingThese observations indicate that serine 172 and threonine 176 of T beta R-I are dispensable for extracellular matrix protein production but essential to the growth inhibition by TGF-beta
SIGNOR-246728
P46108
P08069
0
phosphorylation
down-regulates activity
0.722
On activation of the IGF-I receptor, Crk-II binds to phosphorylated tyrosine residues, especially in the juxtamembrane region. As a result of this binding, the IGF-I receptor kinase phosphorylates Tyr-221 of Crk-II, resulting in a change in intramolecular folding and binding of the SH2 domain to the phosphorylated Tyr-221, which causes rapid disassociation of the Crk-II-IGF-I receptor complex.
SIGNOR-251273
Q15796
P28482
0
phosphorylation
up-regulates
0.722
We show that phosphorylation of smad2, a mediator of the activin/transforming growth factor-beta signal, by activated extracellular signal-regulated kinase 1 (erk1) increases the amount of smad2 protein and leads to enhanced transcriptional activity.
SIGNOR-91714
P48736
P01111
0
binding
up-regulates
0.722
Grb2 binds and activates sos, which then activates ras, and this activates p110 independently of p85. It was also described that ras interacts with pi3k in a direct manner.llysine residue 227 is essential for the interaction of ras with pi3k
SIGNOR-175228
O14733
Q9UQF2
0
binding
up-regulates
0.722
Both jip1 and jip2 selectively bind the mapkk isoform mkk7.
SIGNOR-70848
P60880
P46459
0
binding
down-regulates activity
0.722
NSF is an important regulator of SNARE assembly/disassembly. NSF binds to SNAP-25, while in complex with other SNAREs, and hydrolyzes adenosine triphosphate to disassemble the SNARE complex down to monomers
SIGNOR-263974
Q92847
Q9UBU3
0
binding
up-regulates
0.722
In contrast to wild-type mice, acute treatment of ghsr- mice with ghrelin stimulated neither gh release nor food intake, showing that the ghsr is a biologically relevant ghrelin receptor.
SIGNOR-123948
P30305
O14965
0
phosphorylation
up-regulates
0.722
We show that bypass of the g2/m checkpoint by the chk1 kinase inhibitor ucn-01 results in the activation of aurora-a and phosphorylation of cdc25b on s353
SIGNOR-139396
P16473
P01222
0
binding
up-regulates
0.722
Two novel human glycoprotein hormonelike genes, alpha2 (a2) and beta5 (b5), recently have been identified. Using a yeast two-hybrid assay, the two subunits were found as potential heterodimerization partners.
SIGNOR-88653
P42336
P35568
0
binding
up-regulates activity
0.722
Irs proteins are capable of both regulating and activating pi3k, depending on the cell of origin.
SIGNOR-168985
Q04206
Q96EB6
0
deacetylation
down-regulates activity
0.722
SIRT1 physically interacts with the RelA/p65 subunit of NF-kappaB and inhibits transcription by deacetylating RelA/p65 at lysine 310.
SIGNOR-238817
P10276
P28702
0
binding
up-regulates
0.722
Here we report that the transcriptional activity of rar and rxr can be reciprocally modulated by direct interactions between the two proteins
SIGNOR-16674
Q03135
P06241
0
phosphorylation
down-regulates activity
0.721
Caveolin-1 is phosphorylated on tyr(14) in response to both oxidative and hyperosmotic stress. In the present paper, we show that this phosphorylation requires activation of the src family kinase fyn.Therefore,
SIGNOR-118003
Q08050
O96017
0
phosphorylation
up-regulates
0.721
Chk2 mediates stabilization of the foxm1 transcription factor to stimulate expression of dna repair genesthis phosphorylation of foxm1 on serine residue 361 caused increased stability of the foxm1 protein
SIGNOR-150746
Q12955
Q92823
0
relocalization
up-regulates quantity
0.721
Neurofascin, L1, NrCAM, NgCAM, and neuroglian are membrane-spanning cell adhesion molecules with conserved cytoplasmic domains that are believed to play important roles in development of the nervous system. This report presents biochemical evidence that the cytoplasmic domains of these molecules associate directly with ankyrins, a family of spectrin-binding proteins located on the cytoplasmic surface of specialized plasma membrane domains.
SIGNOR-266720
P46937
P07947
0
phosphorylation
up-regulates activity
0.721
Yes directly phosphorylates YAP and TAZ, resulting in their increased nuclear localization and transcriptional activity.Analysis by mass spectrometry identified Tyr391 and Tyr407 as the two phosphorylation sites of YAP, whereas Tyr305 was the sole phosphorylated residue of TAZ (Fig. 5F and fig. S4, A to C).
SIGNOR-277653
O75369
Q8WUP2
0
binding
up-regulates activity
0.721
Kindlin binds migfilin tandem LIM domains and regulates migfilin focal adhesion localization and recruitment dynamics. Two integrin-binding proteins present in FAs, kindlin-1 and kindlin-2, are important for integrin activation, FA formation, and signaling. By binding filamin, migfilin provides a link between kindlin and the actin cytoskeleton.
SIGNOR-266106
Q99640
P53350
0
phosphorylation
down-regulates activity
0.721
Here, we have shown that Plk1 is responsible for part of the phosphorylation of Myt1 during M phase. The kinase activity of human Myt1 is reported to be decreased during M phase, and the decreased activity correlates with hyperphosphorylated forms of Myt1 (35, 37). We then tested the ability of these mutant forms of Myt1 (GST fusion proteins), to serve as a substrate for Plk1 in vitro. Quantification of the result (Fig. 5C) showed that Ser-426 is the major phosphorylation site by Plk1 in vitro and Thr-495 the second major site.
SIGNOR-263096
P01116
P21359
0
binding
down-regulates activity
0.721
Sprouty-related, EVH1 domain-containing (SPRED) proteins negatively regulate RAS/mitogen-activated protein kinase (MAPK) signaling following growth factor stimulation. This inhibition of RAS is thought to occur primarily through SPRED1 binding and recruitment of neurofibromin, a RasGAP, to the plasma membrane. Here, we report the structure of neurofibromin (GTPase-activating protein [GAP]-related domain) complexed with SPRED1 (EVH1 domain) and KRAS. The structure provides insight into how the membrane targeting of neurofibromin by SPRED1 allows simultaneous interaction with activated KRAS.
SIGNOR-273661
O00571
Q9UHD2
0
phosphorylation
up-regulates activity
0.721
Coexpression of TBK1 strongly increased the activity of DDX3X.|This suggests that DDX3X is rather specifically phosphorylated by TBK1 and IKK-i.
SIGNOR-278997
P13569
Q9HD26
0
binding
down-regulates
0.721
Cal binds to cftr / cal affects insertion of cftr to the plasma membrane as well as its half-life in the plasma membrane.
SIGNOR-111671
Q13485
Q15796
0
binding
up-regulates activity
0.721
the receptor-regulated Smad, such as Smad2, forms a heterocomplex with the co-mediator Smad, Smad4
SIGNOR-235183
P10828
P19793
0
binding
up-regulates
0.721
Like many receptors belonging to the superfamily of steroid/thyroid nuclear receptors, thyroid hormone receptors (trs) bind to specific th-dna responsive elements (tre) upstream of target gene in heterodimeric complex with the 9-cis retinoid acid receptor (rxr
SIGNOR-81449
P40763
P27361
0
phosphorylation
up-regulates
0.72
The activation of stat-3 is regulated by phosphorylation of tyrosine 705 by receptor and nonreceptor protein tyrosine kinases these include epidermal growth factor receptor (egfr) kinase,92 src,5 janus-activated kinases (jak), and extracellular signal-regulated kinase (erk)a constitutively active galpha16 mutant, galpha16ql, stimulated stat3-dependent luciferase activity as well as the phosphorylation of stat3 at both tyr705 and ser727. Galpha16ql-induced stat3 activation was enhanced by overexpression of extracellular signal-regulated kinase 1 (erk1
SIGNOR-118596
P23443
P67775
0
dephosphorylation
down-regulates
0.72
Protein phosphatase 2a inactivates the mitogen-stimulated s6 kinase from swiss mouse 3t3 cells
SIGNOR-23575
P46531
O00587
0
binding
up-regulates
0.72
Manic fringe elongates the o-linked fucose saccharides on full-length notch1 and notch1 egf repeats 1923.
SIGNOR-80555
Q92859
O00634
0
binding
up-regulates activity
0.72
Experiments have demonstrated that Neogenin also mediates Netrin-1 attractive functions. Both DCC and Neogenin are type I transmembrane receptors that belong to the immunoglobulin superfamily proteins.
SIGNOR-268171
Q08J23
Q96GD4
0
phosphorylation
down-regulates
0.72
Aurora-b phosphorylated nsun2 at ser139. Aurora-b-phosphorylation and the phosphorylation-mimic mutation (s139e) suppressed methyltransferase activities of nsun2.
SIGNOR-152001